Endoplasmic reticulum: a primary target in various acute disorders and degenerative diseases of the brain (Q35196245)

7 August 2017

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title

Endoplasmic reticulum: a primary target in various acute disorders and degenerative diseases of the brain (English)

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7 August 2017

main subject

endoplasmic reticulum

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author name string

Wulf Paschen

series ordinal

1

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7 August 2017

language of work or name

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1 October 2003

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7 August 2017

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7 August 2017

volume

34

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7 August 2017

issue

4-5

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7 August 2017

page(s)

365-383

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Cell damage in the brain: a speculative synthesis

7 August 2017

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BH-3-only BIK functions at the endoplasmic reticulum to stimulate cytochrome c release from mitochondria

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Transmission of cell stress from endoplasmic reticulum to mitochondria: enhanced expression of Lon protease

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Manganese Superoxide Dismutase Affects Cytochrome c Release and Caspase-9 Activation After Transient Focal Cerebral Ischemia in Mice

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Calcium signaling in the ER: its role in neuronal plasticity and neurodegenerative disorders

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Dysregulation of cellular calcium homeostasis in Alzheimer's disease: bad genes and bad habits

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Calcium dyshomeostasis and intracellular signalling in Alzheimer's disease

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The endoplasmic reticulum as an integrating signalling organelle: from neuronal signalling to neuronal death

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The role of the ubiquitin-proteasomal pathway in Parkinson's disease and other neurodegenerative disorders

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Presenilin-1 mutations downregulate the signalling pathway of the unfolded-protein response

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Characterization of mouse Ire1 alpha: cloning, mRNA localization in the brain and functional analysis in a neural cell line

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Upregulation of BiP and CHOP by the unfolded-protein response is independent of presenilin expression.

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FAD-linked presenilin-1 mutants impede translation regulation under ER stress.

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Ultrastructural analysis of beta-amyloid-induced apoptosis in cultured hippocampal neurons

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Neurotrophic factors [activity-dependent neurotrophic factor (ADNF) and basic fibroblast growth factor (bFGF)] interrupt excitotoxic neurodegenerative cascades promoted by a PS1 mutation

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Beyond the Qs in the polyglutamine diseases

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Length of huntingtin and its polyglutamine tract influences localization and frequency of intracellular aggregates

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Polyglutamine aggregates stimulate ER stress signals and caspase-12 activation

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7 January 2021

ASK1 is essential for endoplasmic reticulum stress-induced neuronal cell death triggered by expanded polyglutamine repeats

1 reference

https://api.crossref.org/works/10.1016%2FS0143-4160%2803%2900139-8

7 January 2021

Familial Parkinson disease gene product, parkin, is a ubiquitin-protein ligase

1 reference

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7 January 2021

Parkin functions as an E2-dependent ubiquitin- protein ligase and promotes the degradation of the synaptic vesicle-associated protein, CDCrel-1

1 reference

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7 January 2021

Astrocytic but not neuronal increased expression and redistribution of parkin during unfolded protein stress

1 reference

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7 January 2021

Functional and genomic analyses reveal an essential coordination between the unfolded protein response and ER-associated degradation

1 reference

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7 January 2021

The sarco/endoplasmic reticulum calcium-ATPase 2b is an endoplasmic reticulum stress-inducible protein

1 reference

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7 January 2021

Ischemic depolarization monitoring: evaluation of protein synthesis in the hippocampal CA1 after brief unilateral ischemia in a gerbil model

1 reference

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7 January 2021

Cerebral protein synthesis and ischemia

1 reference

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7 January 2021

Persistent inhibition of protein synthesis precedes delayed neuronal death in postischemic gerbil hippocampus

1 reference

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7 January 2021

Evidence supporting a role for programmed cell death in focal cerebral ischemia in rats.

1 reference

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7 January 2021

Global ischemia can cause DNA fragmentation indicative of apoptosis in rat brain

1 reference

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7 January 2021

Internucleosomal DNA cleavage involved in ischemia-induced neuronal death

1 reference

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7 January 2021

Apoptosis after experimental stroke: fact or fashion?

1 reference

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7 January 2021

Neurodegenerative disorders and ischemic brain diseases.

1 reference

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7 January 2021

Programmed cell death in cerebral ischemia

1 reference

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7 January 2021

Perk is essential for translational regulation and cell survival during the unfolded protein response

1 reference

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7 January 2021

Diabetes mellitus and exocrine pancreatic dysfunction in perk-/- mice reveals a role for translational control in secretory cell survival

1 reference

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7 January 2021

Endoplasmic reticulum stress-mediated apoptosis in pancreatic beta-cells

1 reference

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7 January 2021

Endoplasmic reticulum stress proteins block oxidant-induced Ca2+ increases and cell death.

1 reference

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7 January 2021

The endoplasmic reticulum stress-responsive protein GRP78 protects neurons against excitotoxicity and apoptosis: suppression of oxidative stress and stabilization of calcium homeostasis

1 reference

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7 January 2021

Calcium-dependent regulation of protein synthesis in intact mammalian cells

1 reference

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7 January 2021

Hypothermic treatment restores glucose regulated protein 78 (GRP78) expression in ischemic brain

1 reference

https://api.crossref.org/works/10.1016%2FS0143-4160%2803%2900139-8

7 January 2021

Immunohistochemical detection of Bax and Bcl-2 proteins in gerbil hippocampus following transient forebrain ischemia

1 reference

https://api.crossref.org/works/10.1016%2FS0143-4160%2803%2900139-8

7 January 2021

Apoptosis repressor genes Bcl-2 and Bcl-x-long are expressed in the rat brain following global ischemia

1 reference

https://api.crossref.org/works/10.1016%2FS0143-4160%2803%2900139-8

7 January 2021

10.1016/S0143-4160(03)00139-8

Identifiers

DOI

1 reference

release_ar6dfynjwzgexm5657n54y3stq

7 August 2017

Fatcat ID

1 reference

Fatcat

https://api.fatcat.wiki/v0/release/ar6dfynjwzgexm5657n54y3stq

24 November 2022

mapped directly with Wikidata item

1 reference