Loss of PINK1 causes mitochondrial functional defects and increased sensitivity to oxidative stress (Q28592727)

31 January 2017

title

Loss of PINK1 causes mitochondrial functional defects and increased sensitivity to oxidative stress (English)

1 reference

PTEN induced putative kinase 1

31 January 2017

main subject

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GOA release 2020-03-11

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1

Clement A Gautier

1 reference

31 January 2017

2

Tohru Kitada

1 reference

31 January 2017

3

Jie Shen

1 reference

31 January 2017

language of work or name

English

0 references

publication date

12 August 2008

1 reference

Proceedings of the National Academy of Sciences of the United States of America

31 January 2017

published in

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31 January 2017

volume

105

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31 January 2017

issue

32

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31 January 2017

page(s)

11364–11369

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DJ-1 gene deletion reveals that DJ-1 is an atypical peroxiredoxin-like peroxidase

31 January 2017

cites work

1 reference

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PINK1 protects against oxidative stress by phosphorylating mitochondrial chaperone TRAP1

17 March 2017

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Impaired dopamine release and synaptic plasticity in the striatum of PINK1-deficient mice

17 March 2017

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PINK1 protein in normal human brain and Parkinson's disease

17 March 2017

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Mutations in PTEN-induced putative kinase 1 associated with recessive parkinsonism have differential effects on protein stability

17 March 2017

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Hereditary early-onset Parkinson's disease caused by mutations in PINK1

17 March 2017

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Mitochondrial dysfunction and oxidative damage in parkin-deficient mice

17 March 2017

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Mutations in the DJ-1 gene associated with autosomal recessive early-onset parkinsonism

17 March 2017

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Mutations in the parkin gene cause autosomal recessive juvenile parkinsonism

17 March 2017

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Structural and functional diversity of the microbial kinome

17 March 2017

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Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin

22 April 2017

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The PINK1/Parkin pathway regulates mitochondrial morphology

7 April 2017

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Mitochondrial dysfunction in Drosophila PINK1 mutants is complemented by parkin

7 April 2017

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Chronic systemic pesticide exposure reproduces features of Parkinson's disease

7 April 2017

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7 April 2017

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Pink1 regulates mitochondrial dynamics through interaction with the fission/fusion machinery

7 April 2017

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The mitochondrial protease HtrA2 is regulated by Parkinson's disease-associated kinase PINK1.

29 September 2017

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Inflammation as a causative factor in the aetiology of Parkinson's disease

29 September 2017

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Mitochondrial dysfunction, peroxidation damage and changes in glutathione metabolism in PARK6.

29 September 2017

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C-terminal truncation and Parkinson's disease-associated mutations down-regulate the protein serine/threonine kinase activity of PTEN-induced kinase-1.

29 September 2017

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Parkinson's disease brain mitochondrial complex I has oxidatively damaged subunits and is functionally impaired and misassembled.

29 September 2017

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Mitochondria and dopamine: new insights into recessive parkinsonism

29 September 2017

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Mitochondria, oxidative damage, and inflammation in Parkinson's disease

29 September 2017

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Mitochondrial permeability transition and oxidative stress

29 September 2017

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Origin and functional consequences of the complex I defect in Parkinson's disease

29 September 2017

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Parkinson's disease: a case-control study of occupational and environmental risk factors.

29 September 2017

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Mitochondrial complex I and II activities of lymphocytes and platelets in Parkinson's disease

29 September 2017

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Superoxide sensitivity of the Escherichia coli aconitase.

3 June 2018

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Mitochondrial import and enzymatic activity of PINK1 mutants associated to recessive parkinsonism

20 June 2018

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Low platelet mitochondrial complex I and complex II/III activity in early untreated Parkinson's disease

21 January 2018

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Distribution of PINK1 and LRRK2 in rat and mouse brain

21 January 2018

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Inhibition of brain mitochondrial respiration by dopamine and its metabolites: implications for Parkinson's disease and catecholamine-associated diseases

21 January 2018

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Electron transfer complexes I and IV of platelets are abnormal in Parkinson's disease but normal in Parkinson-plus syndromes.

21 January 2018

1 reference

https://pubmed.ncbi.nlm.nih.gov/18687901

12 December 2020

Testing the vicious cycle theory of mitochondrial ROS production: effects of H2O2 and cumene hydroperoxide treatment on heart mitochondria

1 reference

https://pubmed.ncbi.nlm.nih.gov/18687901

12 December 2020

Mono- and double-mutant mouse models of Parkinson's disease display severe mitochondrial damage

1 reference

https://pubmed.ncbi.nlm.nih.gov/18687901

12 December 2020

Mitochondrial complex I deficiency in Parkinson's disease

1 reference

https://pubmed.ncbi.nlm.nih.gov/18687901

12 December 2020

MITOCHONDRIAL FUNCTION IN PARKINSON'S DISEASE

1 reference

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12 December 2020

Identifiers

DOI

10.1073/PNAS.0802076105

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release_lqswu2x2sjc25gu4qsz3utgp4i

0 references

1 reference

Fatcat

https://api.fatcat.wiki/v0/release/lqswu2x2sjc25gu4qsz3utgp4i

24 November 2022

mapped directly with Wikidata item

5546

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5546

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