Drosophila pink1 is required for mitochondrial function and interacts genetically with parkin (Q29547423)

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=16672981

PTEN-induced putative kinase 1 Dmel_CG4523

23 April 2017

main subject

Drosophila

0 references

1 reference

GOA release 2020-03-11

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Clark IE

1

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Dodson MW

2

1 reference

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Jiang C

3

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Cao JH

4

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Huh JR

5

1 reference

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Seol JH

6

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Yoo SJ

7

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Hay BA

8

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Guo M

9

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language of work or name

1 reference

23 April 2017

publication date

29 June 2006

1 reference

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23 April 2017

1 reference

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volume

441

1 reference

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issue

7097

1 reference

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page(s)

1162-6

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Molecular pathophysiology of Parkinson's disease

23 April 2017

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7 January 2021

Hereditary early-onset Parkinson's disease caused by mutations in PINK1

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Mutations in the parkin gene cause autosomal recessive juvenile parkinsonism

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Early-onset parkinsonism associated with PINK1 mutations: frequency, genotypes, and phenotypes

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PINK1, Parkin, and DJ-1 mutations in Italian patients with early-onset parkinsonism

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Mutational analysis of the PINK1 gene in early-onset parkinsonism in Europe and North Africa

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Mitochondrial pathology and apoptotic muscle degeneration in Drosophila parkin mutants

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Drosophila parkin mutants have decreased mass and cell size and increased sensitivity to oxygen radical stress

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7 January 2021

Mitochondrial import and enzymatic activity of PINK1 mutants associated to recessive parkinsonism

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7 January 2021

Drosophila DJ-1 mutants are selectively sensitive to environmental toxins associated with Parkinson's disease

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7 January 2021

Roles of Drosophila DJ-1 in survival of dopaminergic neurons and oxidative stress

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7 January 2021

Inactivation of Drosophila DJ-1 leads to impairments of oxidative stress response and phosphatidylinositol 3-kinase/Akt signaling

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7 January 2021

Drosophila DJ-1 mutants show oxidative stress-sensitive locomotive dysfunction.

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Mutations in the DJ-1 gene associated with autosomal recessive early-onset parkinsonism

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7 January 2021

Reactive oxygen and nitrogen species: weapons of neuronal destruction in models of Parkinson's disease

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7 January 2021

Multiple apoptotic caspase cascades are required in nonapoptotic roles for Drosophila spermatid individualization

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7 January 2021

Increased glutathione S-transferase activity rescues dopaminergic neuron loss in a Drosophila model of Parkinson's disease

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7 January 2021

Genetic mouse models of parkinsonism: strengths and limitations

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7 January 2021

Mitochondrial dysfunction and oxidative damage in parkin-deficient mice

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7 January 2021

Staging of brain pathology related to sporadic Parkinson's disease

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7 January 2021

Respiratory chain abnormalities in skeletal muscle from patients with Parkinson's disease

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7 January 2021

Detection of respiratory chain defects in cultivated skin fibroblasts and skeletal muscle of patients with Parkinson's disease

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7 January 2021

Genetic dissection of behavior / Seymour Benzer. - (1973)

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7 January 2021

The eukaryotic protein kinase superfamily: kinase (catalytic) domain structure and classification 1

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https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=16672981

Identifiers

2 references

23 April 2017

Consolidated OpenCitations Corpus – April 2017

Dimensions Publication ID

1033391441

0 references

1 reference

Consolidated OpenCitations Corpus – April 2017

https://eutils.ncbi.nlm.nih.gov/entrez/eutils/esummary.fcgi?db=pubmed&retmode=json&id=16672981

PubMed publication ID

2 references

23 April 2017

Consolidated OpenCitations Corpus – April 2017