Tumor suppressor RBM5 directly interacts with the DExD/H-box protein DHX15 and stimulates its helicase activity (Q24319102)

1

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Wenxing Jin

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Libo Zhang

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Xialu Li

Homozygous deletions at 3p12 in breast and lung cancer

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language of work or name

English

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publication date

5 April 2012

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published in

FEBS Letters

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volume

586

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issue

7

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page(s)

977-83

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cites work

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A comparison of genomic structures and expression patterns of two closely related flanking genes in a critical lung cancer region at 3p21.3

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LUCA-15/RBM5, a putative tumour suppressor, enhances multiple receptor-initiated death signals

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LUCA15, a putative tumour suppressor gene encoding an RNA-binding nuclear protein, is down-regulated in ras-transformed Rat-1 cells

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Candidate tumor suppressor LUCA-15/RBM5/H37 modulates expression of apoptosis and cell cycle genes

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RBM5/Luca-15/H37 regulates Fas alternative splice site pairing after exon definition.

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Up-regulation of the proapoptotic caspase 2 splicing isoform by a candidate tumor suppressor, RBM5

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The human La (SS-B) autoantigen interacts with DDX15/hPrp43, a putative DEAH-box RNA helicase

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The DEAD-box protein family of RNA helicases

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ARF-BP1/Mule is a critical mediator of the ARF tumor suppressor

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Insulin receptor substrate-2 binds to the insulin receptor through its phosphotyrosine-binding domain and through a newly identified domain comprising amino acids 591-786

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Ntr1 activates the Prp43 helicase to trigger release of lariat-intron from the spliceosome

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Proteinases of betaretroviruses bind single-stranded nucleic acids through a novel interaction module, the G-patch

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The ATPase and helicase activities of Prp43p are stimulated by the G-patch protein Pfa1p during yeast ribosome biogenesis

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Interaction between a G-patch protein and a spliceosomal DEXD/H-box ATPase that is critical for splicing

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