Pages that link to "Q36597197"

The following pages link to Herpes simplex virus infected cell polypeptide 4 preferentially represses Sp1-activated over basal transcription from its own promoter (Q36597197):

Displaying 42 items.

• Bovine Herpes Virus 1 (BHV-1) and Herpes Simplex Virus Type 1 (HSV-1) Promote Survival of Latently Infected Sensory Neurons, in Part by Inhibiting Apoptosis (Q26864175) (← links)
• Towards an understanding of the herpes simplex virus type 1 latency-reactivation cycle (Q33654414) (← links)
• Role of the IE62 consensus binding site in transactivation by the varicella-zoster virus IE62 protein (Q33769312) (← links)
• Persistence and expression of the herpes simplex virus genome in the absence of immediate-early proteins (Q33783411) (← links)
• The polyserine tract of herpes simplex virus ICP4 is required for normal viral gene expression and growth in murine trigeminal ganglia (Q33783767) (← links)
• Perturbation of cell cycle progression and cellular gene expression as a function of herpes simplex virus ICP0. (Q33819612) (← links)
• HSV-1-based vectors for gene therapy of neurological diseases and brain tumors: part I. HSV-1 structure, replication and pathogenesis (Q33995747) (← links)
• Phosphorylation of transcription factor Sp1 during herpes simplex virus type 1 infection (Q34341046) (← links)
• Analysis of herpes simplex virion tegument ICP4 derived from infected cells and ICP4-expressing cells (Q34936278) (← links)
• Herpes simplex virus type 1 and bovine herpesvirus 1 latency (Q35044114) (← links)
• Barrier to auto integration factor becomes dephosphorylated during HSV-1 Infection and Can Act as a host defense by impairing viral DNA replication and gene expression (Q35191248) (← links)
• Intracellular localization of the herpes simplex virus type 1 major transcriptional regulatory protein, ICP4, is affected by ICP27 (Q35828561) (← links)
• Functional interactions between herpes simplex virus immediate-early proteins during infection: gene expression as a consequence of ICP27 and different domains of ICP4 (Q35845959) (← links)
• Herpes simplex virus trans-regulatory protein ICP27 stabilizes and binds to 3' ends of labile mRNA. (Q35851194) (← links)
• The autoregulatory and transactivating functions of the human cytomegalovirus IE86 protein use independent mechanisms for promoter binding (Q35857758) (← links)
• Overexpression of the herpes simplex virus type 1 immediate-early regulatory protein, ICP27, is responsible for the aberrant localization of ICP0 and mutant forms of ICP4 in ICP4 mutant virus-infected cells (Q35866195) (← links)
• Prolonged gene expression and cell survival after infection by a herpes simplex virus mutant defective in the immediate-early genes encoding ICP4, ICP27, and ICP22. (Q35868577) (← links)
• The herpes simplex virus immediate-early protein ICP0 affects transcription from the viral genome and infected-cell survival in the absence of ICP4 and ICP27 (Q35886888) (← links)
• cis elements that contribute to geminivirus transcriptional regulation and the efficiency of DNA replication (Q35892506) (← links)
• Characterization of cis-acting elements required for autorepression of the equine herpesvirus 1 IE gene (Q36073807) (← links)
• Cooperativity among herpes simplex virus type 1 immediate-early regulatory proteins: ICP4 and ICP27 affect the intracellular localization of ICP0 (Q36633412) (← links)
• Relationship between TATA-binding protein and herpes simplex virus type 1 ICP4 DNA-binding sites in complex formation and repression of transcription (Q38292162) (← links)
• Requirements for activation of the herpes simplex virus glycoprotein C promoter in vitro by the viral regulatory protein ICP4. (Q38301836) (← links)
• Temperature-dependent conformational changes in herpes simplex virus ICP4 that affect transcription activation (Q38357725) (← links)
• The herpes viral transcription factor ICP4 forms a novel DNA recognition complex (Q38785758) (← links)
• The N terminus and C terminus of herpes simplex virus 1 ICP4 cooperate to activate viral gene expression (Q39365625) (← links)
• Herpes simplex virus 1 ICP4 forms complexes with TFIID and mediator in virus-infected cells (Q39568162) (← links)
• Identification of a motif in the C terminus of herpes simplex virus regulatory protein ICP4 that contributes to activation of transcription (Q39681678) (← links)
• The initiator element in a herpes simplex virus type 1 late-gene promoter enhances activation by ICP4, resulting in abundant late-gene expression (Q39682517) (← links)
• Repression of activator-mediated transcription by herpes simplex virus ICP4 via a mechanism involving interactions with the basal transcription factors TATA-binding protein and TFIIB. (Q40016568) (← links)
• Induction of Transcription by a Viral Regulatory Protein Depends on the Relative Strengths of Functional TATA Boxes (Q40017145) (← links)
• Interaction of the viral activator protein ICP4 with TFIID through TAF250. (Q40019074) (← links)
• The von Hippel-Lindau tumor suppressor gene product interacts with Sp1 to repress vascular endothelial growth factor promoter activity (Q40023447) (← links)
• Localization of a 34-amino-acid segment implicated in dimerization of the herpes simplex virus type 1 ICP4 polypeptide by a dimerization trap. (Q40041005) (← links)
• Transcription of the Herpes Simplex Virus Genome during Productive and Latent Infection (Q40473966) (← links)
• Differential cellular requirements for activation of herpes simplex virus type 1 early (tk) and late (gC) promoters by ICP4. (Q40551612) (← links)
• Stabilized binding of TBP to the TATA box of herpes simplex virus type 1 early (tk) and late (gC) promoters by TFIIA and ICP4. (Q41817706) (← links)
• DNA-dependent oligomerization of herpes simplex virus type 1 regulatory protein ICP4 (Q41956844) (← links)
• Alphaherpesvirus latency: its role in disease and survival of the virus in nature (Q43586571) (← links)
• Interaction between the varicella zoster virus IE62 major transactivator and cellular transcription factor Sp1. (Q45722653) (← links)
• Herpes Simplex Virus 1 MicroRNA miR-H28 Exported to Uninfected Cells in Exosomes Restricts Cell-to-Cell Virus Spread by Inducing Gamma Interferon mRNA (Q92623263) (← links)
• The HSV-1 ICP4 Transcriptional Auto-Repression Circuit Functions as a Transcriptional "Accelerator" Circuit (Q97525262) (← links)