Pages that link to "Q24650383"

The following pages link to Expression of a beta-adrenergic receptor kinase 1 inhibitor prevents the development of myocardial failure in gene-targeted mice (Q24650383):

Displaying 50 items.

• Targeted beta-adrenergic receptor kinase (betaARK1) inhibition by gene transfer in failing human hearts (Q24322049) ‎ (← links)
• Understanding molecular recognition by G protein βγ subunits on the path to pharmacological targeting (Q24635233) ‎ (← links)
• Myocardial adeno-associated virus serotype 6-betaARKct gene therapy improves cardiac function and normalizes the neurohormonal axis in chronic heart failure (Q24648449) ‎ (← links)
• Preservation of myocardial beta-adrenergic receptor signaling delays the development of heart failure after myocardial infarction (Q24656335) ‎ (← links)
• Alterations in cardiac adrenergic signaling and calcium cycling differentially affect the progression of cardiomyopathy (Q24680224) ‎ (← links)
• Signaling effectors underlying pathologic growth and remodeling of the heart (Q26825154) ‎ (← links)
• Targeting G protein coupled receptor-related pathways as emerging molecular therapies (Q26829964) ‎ (← links)
• GRK2 in the heart: a GPCR kinase and beyond (Q26851548) ‎ (← links)
• New advances in beta-blocker therapy in heart failure (Q26865919) ‎ (← links)
• G protein-coupled receptor kinase 2: a link between myocardial contractile function and cardiac metabolism (Q27014769) ‎ (← links)
• Cardiovascular gene therapy for myocardial infarction (Q27014770) ‎ (← links)
• Paroxetine Is a Direct Inhibitor of G Protein-Coupled Receptor Kinase 2 and Increases Myocardial Contractility (Q27671474) ‎ (← links)
• Molecular Mechanism for Inhibition of G Protein-Coupled Receptor Kinase 2 by a Selective RNA Aptamer (Q27681349) ‎ (← links)
• Molecular mechanisms underlying β-adrenergic receptor-mediated cross-talk between sympathetic neurons and immune cells (Q28083146) ‎ (← links)
• Regulation of beta-adrenergic receptor signaling by S-nitrosylation of G-protein-coupled receptor kinase 2 (Q28300709) ‎ (← links)
• Beta-arrestin- and c-Src-dependent degradation of G-protein-coupled receptor kinase 2 (Q28359741) ‎ (← links)
• Chronic phospholamban-sarcoplasmic reticulum calcium ATPase interaction is the critical calcium cycling defect in dilated cardiomyopathy (Q28505358) ‎ (← links)
• Endocytosis machinery is required for beta1-adrenergic receptor-induced hypertrophy in neonatal rat cardiac myocytes (Q28578076) ‎ (← links)
• Seven-transmembrane receptors (Q29619896) ‎ (← links)
• The expanding GRK interactome: Implications in cardiovascular disease and potential for therapeutic development (Q30250314) ‎ (← links)
• The role of G protein coupled receptor kinases in neurocardiovascular pathophysiology (Q30459130) ‎ (← links)
• G protein-coupled receptor kinase 2 ablation in cardiac myocytes before or after myocardial infarction prevents heart failure (Q30487539) ‎ (← links)
• Deletion of the β2-adrenergic receptor prevents the development of cardiomyopathy in mice (Q30548452) ‎ (← links)
• The inotropic peptide βARKct improves βAR responsiveness in normal and failing cardiomyocytes through G(βγ)-mediated L-type calcium current disinhibition. (Q30577637) ‎ (← links)
• The role of HTS in drug discovery at the University of Michigan. (Q31147980) ‎ (← links)
• Vascular beta-adrenergic receptor system is dysfunctional after myocardial infarction (Q31940102) ‎ (← links)
• Evaluation of rat neutrophil beta-adrenergic receptors by microphysiometry (Q32177657) ‎ (← links)
• RyR2R420Q catecholaminergic polymorphic ventricular tachycardia mutation induces bradycardia by disturbing the coupled clock pacemaker mechanism (Q33168540) ‎ (← links)
• Young MLP deficient mice show diastolic dysfunction before the onset of dilated cardiomyopathy (Q33217903) ‎ (← links)
• Ankyrin-B syndrome: enhanced cardiac function balanced by risk of cardiac death and premature senescence (Q33302911) ‎ (← links)
• G protein-coupled Receptor Kinase 2 as a Therapeutic Target for Heart Failure (Q33604042) ‎ (← links)
• G-protein coupled receptor kinases as modulators of G-protein signalling (Q33606979) ‎ (← links)
• Stress pathways and heart failure (Q33734297) ‎ (← links)
• Myocardial G protein-coupled receptor kinases: implications for heart failure therapy (Q33750874) ‎ (← links)
• Evaluating docking methods for prediction of binding affinities of small molecules to the G protein betagamma subunits (Q33757270) ‎ (← links)
• Regulation of cardiac contractility by Rab4-modulated beta2-adrenergic receptor recycling (Q33782643) ‎ (← links)
• Chronic treatment with clenbuterol modulates endothelial progenitor cells and circulating factors in a murine model of cardiomyopathy. (Q33789760) ‎ (← links)
• β-Adrenergic receptor subtype signaling in heart: from bench to bedside (Q33828730) ‎ (← links)
• RGS4 causes increased mortality and reduced cardiac hypertrophy in response to pressure overload (Q33857945) ‎ (← links)
• Transgenic animals in integrative biology: approaches and interpretations of outcome (Q33858644) ‎ (← links)
• Structure-activity relationships of G protein-coupled receptors (Q33861164) ‎ (← links)
• Heterotrimeric G proteins in heart disease (Q33865993) ‎ (← links)
• Mechanisms of regulation and function of G-protein-coupled receptor kinases (Q33866666) ‎ (← links)
• Reversal of impaired myocardial beta-adrenergic receptor signaling by continuous-flow left ventricular assist device support (Q33876021) ‎ (← links)
• Meeting Koch's postulates for calcium signaling in cardiac hypertrophy (Q33919182) ‎ (← links)
• Cellular and functional defects in a mouse model of heart failure (Q33925912) ‎ (← links)
• In vivo ventricular gene delivery of a beta-adrenergic receptor kinase inhibitor to the failing heart reverses cardiac dysfunction (Q33936885) ‎ (← links)
• Functional consequences of altering myocardial adrenergic receptor signaling (Q33938713) ‎ (← links)
• Effects of deletion of muscle LIM protein on myocyte function (Q33947079) ‎ (← links)
• Molecular aspects and gene therapy prospects for diastolic failure (Q34032476) ‎ (← links)