Characterization of atherosclerosis in LDL receptor knockout mice: macrophage accumulation correlates with rapid and sustained expression of aortic MCP-1/JE (Q73575738)

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scientific article published on 01 April 2000

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English	Characterization of atherosclerosis in LDL receptor knockout mice: macrophage accumulation correlates with rapid and sustained expression of aortic MCP-1/JE	scientific article published on 01 April 2000

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5 November 2019

Characterization of atherosclerosis in LDL receptor knockout mice: macrophage accumulation correlates with rapid and sustained expression of aortic MCP-1/JE (English)

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5 November 2019

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M C Kowala

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5 November 2019

R Recce

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5 November 2019

S Beyer

3

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5 November 2019

C Gu

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5 November 2019

M Valentine

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5 November 2019

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1 April 2000

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5 November 2019

Atherosclerosis

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5 November 2019

149

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5 November 2019

2

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5 November 2019

323-330

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5 November 2019

Dietary-induced atherosclerosis in miniature swine

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Studies of hypercholesterolemia in the nonhuman primate. II. Fatty streak conversion to fibrous plaque.

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The hyperlipidemic hamster as a model of experimental atherosclerosis

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Atherosclerosis susceptibility differences among progenitors of recombinant inbred strains of mice.

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Severe hypercholesterolemia and atherosclerosis in apolipoprotein E-deficient mice created by homologous recombination in ES cells

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Spontaneous hypercholesterolemia and arterial lesions in mice lacking apolipoprotein E

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Hypercholesterolemia in low density lipoprotein receptor knockout mice and its reversal by adenovirus-mediated gene delivery

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Massive xanthomatosis and atherosclerosis in cholesterol-fed low density lipoprotein receptor-negative mice

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Atherosclerosis in mice lacking apo E. Evaluation of lesional development and progression

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ApoE-deficient mice develop lesions of all phases of atherosclerosis throughout the arterial tree

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Enhanced reduction of atherosclerosis in hamsters treated with pravastatin and captopril: ACE in atheromas provides cellular targets for captopril

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Atherosclerosis — An Inflammatory Disease

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Monocyte chemotactic protein-1 (MCP-1), -2, and -3 are chemotactic for human T lymphocytes

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Molecular cloning and functional expression of two monocyte chemoattractant protein 1 receptors reveals alternative splicing of the carboxyl-terminal tails

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Monocyte chemoattractant protein-1 in human atheromatous plaques

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Expression of monocyte chemoattractant protein 1 in macrophage-rich areas of human and rabbit atherosclerotic lesions

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Detection of monocyte chemoattractant protein-1 in human atherosclerotic lesions by an anti-monocyte chemoattractant protein-1 monoclonal antibody

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Monocyte transmigration induced by modification of low density lipoprotein in cocultures of human aortic wall cells is due to induction of monocyte chemotactic protein 1 synthesis and is abolished by high density lipoprotein

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Monocytes may amplify their recruitment into inflammatory lesions by inducing monocyte chemotactic protein

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Induction of monocyte chemoattractant protein-1 synthesis in human monocytes during transendothelial migration in vitro

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7 January 2021

Absence of monocyte chemoattractant protein-1 reduces atherosclerosis in low density lipoprotein receptor-deficient mice

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Decreased lesion formation in CCR2-/- mice reveals a role for chemokines in the initiation of atherosclerosis

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Site of intimal rupture or erosion of thrombosed coronary atherosclerotic plaques is characterized by an inflammatory process irrespective of the dominant plaque morphology

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10.1016/S0021-9150(99)00342-1

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5 November 2019

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:10729382%20AND%20SRC:MED&resulttype=core&format=json

5 November 2019

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