Expression of BCR – ABL in M1 myeloid leukemia cells induces differentiation without arresting proliferation (Q62819858)

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9 December 2019

title

Expression of BCR - ABL in M1 myeloid leukemia cells induces differentiation without arresting proliferation (English)

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9 December 2019

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6

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9 December 2019

Andrew Elefanty

7

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9 December 2019

author name string

Cambier N

1

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9 December 2019

Zhang Y

2

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9 December 2019

Vairo G

3

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9 December 2019

Kosmopoulos K

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Metcalf D

5

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9 December 2019

language of work or name

English

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publication date

1 January 1999

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9 December 2019

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9 December 2019

volume

18

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9 December 2019

issue

2

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9 December 2019

page(s)

343-352

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Differential complementation of Bcr-Abl point mutants with c-Myc

9 December 2019

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Signaling by ABL oncogenes through cyclin D1.

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Ras links growth factor signaling to the cell cycle machinery via regulation of cyclin D1 and the Cdk inhibitor p27KIP1.

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Transforming p21ras mutants and c-Ets-2 activate the cyclin D1 promoter through distinguishable regions

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BCR-ABL activates pathways mediating cytokine independence and protection against apoptosis in murine hematopoietic cells in a dose-dependent manner

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Constitutive expression of a c-myb cDNA blocks Friend murine erythroleukemia cell differentiation

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Tyrosine kinase oncogenes abrogate interleukin-3 dependence of murine myeloid cells through signaling pathways involving c-myc: conditional regulation of c-myc transcription by temperature-sensitive v-abl

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Constitutive c-myc oncogene expression blocks mouse erythroleukaemia cell differentiation but not commitment.

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Structural and signaling requirements for BCR-ABL-mediated transformation and inhibition of apoptosis

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The Bcr-Abl tyrosine kinase activates mitogenic signaling pathways and stimulates G1-to-S phase transition in hematopoietic cells

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Transformation of an interleukin 3-dependent hematopoietic cell line by the chronic myelogenous leukemia-specific P210bcr/abl protein

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Induction of chronic myelogenous leukemia in mice by the P210bcr/abl gene of the Philadelphia chromosome

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Expression of a transfected human c-myconcogene inhibits differentiation of a mouse erythroleukaemia cell line

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Factors involved in leukaemogenesis and haemopoiesis

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Hematologic disease induced in BALB/c mice by a bcr-abl retrovirus is influenced by the infection conditions

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7 January 2021

bcr-abl, the hallmark of chronic myeloid leukaemia in man, induces multiple haemopoietic neoplasms in mice.

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7 January 2021

Divalent cation-independent macrophage adhesion inhibited by monoclonal antibody to murine scavenger receptor

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7 January 2021

Initiation of deregulated growth of multipotent progenitor cells by bcr-abl in vitro

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7 January 2021

The BCR/ABL hybrid gene

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7 January 2021

Interleukin-6- and leukemia inhibitory factor-induced terminal differentiation of myeloid leukemia cells is blocked at an intermediate stage by constitutive c-myc

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7 January 2021

Bcl-2, Bcl-XL and adenovirus protein E1B19kD are functionally equivalent in their ability to inhibit cell death.

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Myc and Ras collaborate in inducing accumulation of active cyclin E/Cdk2 and E2F.

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Differentiation of mouse erythroleukemia cells is blocked by late up-regulation of a c-myb transgene

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Deregulated expression of c-myc by murine erythroleukaemia cells prevents differentiation

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7 January 2021

Wild-type p53 induces apoptosis of myeloid leukaemic cells that is inhibited by interleukin-6

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7 January 2021

Induction of protein-tyrosine-phosphatase activity by interleukin 6 in M1 myeloblastic cells and analysis of possible counteractions by the BCR-ABL oncogene

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7 January 2021

Identifiers

DOI

10.1038/SJ.ONC.1202302

1 reference

https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:9927191%20AND%20SRC:MED&resulttype=core&format=json

Dimensions Publication ID

9 December 2019

1007467809

0 references

1 reference