CRISPR–Cas9 genome editing in human cells occurs via the Fanconi anemia pathway (Q58054240)

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14 April 2020

title

CRISPR-Cas9 genome editing in human cells occurs via the Fanconi anemia pathway (English)

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14 April 2020

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Christopher D Richardson

1

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14 April 2020

Jacob E Corn

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author name string

Elena Zelin

4

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14 April 2020

Katelynn R Kazane

2

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14 April 2020

Sharon J Feng

3

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Nicholas L Bray

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14 April 2020

Axel J Schäfer

6

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Stephen N Floor

7

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language of work or name

English

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publication date

27 July 2018

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14 April 2020

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volume

50

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issue

8

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page(s)

1132-1139

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Expanding the Biologist's Toolkit with CRISPR-Cas9

14 April 2020

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Characterization of the interplay between DNA repair and CRISPR/Cas9-induced DNA lesions at an endogenous locus

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Human Fanconi anemia monoubiquitination pathway promotes homologous DNA repair

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7 January 2021

Homology-directed repair of DNA nicks via pathways distinct from canonical double-strand break repair

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The control of DNA repair by the cell cycle

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The Fanconi anemia DNA repair pathway: structural and functional insights into a complex disorder

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How the fanconi anemia pathway guards the genome

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Maintenance of genome stability by Fanconi anemia proteins

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Inactivation of murine Usp1 results in genomic instability and a Fanconi anemia phenotype.

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7 January 2021

Molecular pathogenesis and clinical management of Fanconi anemia

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7 January 2021

Preventing nonhomologous end joining suppresses DNA repair defects of Fanconi anemia.

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7 January 2021

Impaired TIP60-mediated H4K16 acetylation accounts for the aberrant chromatin accumulation of 53BP1 and RAP80 in Fanconi anemia pathway-deficient cells

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DNA damage response factors from diverse pathways, including DNA crosslink repair, mediate alternative end joining

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DNA Repair Profiling Reveals Nonrandom Outcomes at Cas9-Mediated Breaks

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The Fanconi anemia pathway promotes replication-dependent DNA interstrand cross-link repair

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7 January 2021

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7 January 2021

10.1038/S41588-018-0174-0

Identifiers

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14 April 2020

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