CD95-induced apoptosis of lymphocytes in an immune privileged site induces immunological tolerance (Q44051217)

29 November 2017

title

CD95-induced apoptosis of lymphocytes in an immune privileged site induces immunological tolerance (English)

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29 November 2017

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Griffith TS

1

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29 November 2017

Yu X

2

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29 November 2017

Herndon JM

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29 November 2017

Green DR

4

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29 November 2017

Ferguson TA

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29 November 2017

language of work or name

English

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publication date

1 July 1996

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29 November 2017

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29 November 2017

volume

5

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29 November 2017

issue

1

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29 November 2017

page(s)

7-16

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Die and let live: eliminating dangerous lymphocytes

29 November 2017

cites work

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CD28 costimulation can promote T cell survival by enhancing the expression of Bcl-XL.

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Bcl-XL and Bcl-2 repress a common pathway of cell death

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Identification of programmed cell death in situ via specific labeling of nuclear DNA fragmentation

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7 January 2021

Fas Ligand-Induced Apoptosis as a Mechanism of Immune Privilege

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The immune response and the eye. TCR alpha-chain related molecules regulate the systemic immunity to antigen presented in the eye

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Inhibition of lymphocyte proliferation by resident ocular cells

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7 January 2021

Fas (CD95) participates in peripheral T cell deletion and associated apoptosis in vivo

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Fas and Fas ligand: lpr and gld mutations

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Differential ability of Th1 and Th2 T cells to express Fas ligand and to undergo activation-induced cell death

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Fas involvement in Ca(2+)-independent T cell-mediated cytotoxicity

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7 January 2021

The functional role of CD8+ T helper type 2 cells

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7 January 2021

The fas antigen is involved in peripheral but not thymic deletion of T lymphocytes in T cell receptor transgenic mice

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Induction of anterior chamber-associated immune deviation requires an intact, functional spleen

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A critical role for ACAID in the distinctive pattern of retinitis that follows anterior chamber inoculation of HSV-1.

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Generalized lymphoproliferative disease in mice, caused by a point mutation in the Fas ligand

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Distinct roles for the costimulatory ligands B7-1 and B7-2 in T helper cell differentiation?

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7 January 2021

Lymphoproliferation disorder in mice explained by defects in Fas antigen that mediates apoptosis

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Studies on the induction of anterior chamber-associated immune deviation (ACAID). III. Induction of ACAID depends upon intraocular transforming growth factor-beta

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Fluids from immune privileged sites endow macrophages with the capacity to induce antigen-specific immune deviation via a mechanism involving transforming growth factor-beta

1 reference

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7 January 2021

10.1016/S1074-7613(00)80305-2

Identifiers

DOI

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29 November 2017

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