Polycystic kidney disease: primary extracellular matrix abnormality or defective cellular differentiation? (Q40876083)

24 September 2017

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title

Polycystic kidney disease: primary extracellular matrix abnormality or defective cellular differentiation? (English)

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polycystic kidney disease

24 September 2017

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Calvet JP

1

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24 September 2017

language of work or name

English

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publication date

1 January 1993

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24 September 2017

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24 September 2017

volume

43

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24 September 2017

issue

1

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24 September 2017

page(s)

101-108

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Acquired cystic disease: replacing one kidney disease with another

24 September 2017

cites work

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Polycystic kidney disease: Clues to pathogenesis

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Renal cystic disease. Insights from recent experimental investigations

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Tubulocystic epithelium

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Comparison of proteoglycans synthesized by porcine normal and polycystic renal tubular epithelial cells in vitro

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The mouse polycystic kidney disease mutation (cpk) is located on proximal chromosome 12

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Glomerulosclerosis and renal cysts in mice transgenic for the early region of SV40

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c-myc as an inducer of polycystic kidney disease in transgenic mice

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Renal cystic disease induced by diphenylthiazole

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Nephron obstruction in nordihydroguaiaretic acid-induced renal cystic disease

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Basement membrane alterations during development and regression of tubular cysts.

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Altered basement membrane protein biosynthesis by primary cultures of cpk/cpk mouse kidney

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A new method for studying human polycystic kidney disease epithelia in culture

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Toxic metabolic defect in polycystic disease of kidney. Evidence from microscope studies

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Ultrastructure and function of cysts from human adult polycystic kidneys

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Potential causes and pathogenesis in autosomal dominant polycystic kidney disease

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Autosomal dominant polycystic kidney disease. Early diagnosis and consideration of pathogenesis

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Reversible changes of tubular cell and basement membrane in drug-induced renal cystic disease.

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Tubular basement membrane change occurs pari passu with the development of cyst formation

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The SGP-2 gene is developmentally regulated in the mouse kidney and abnormally expressed in collecting duct cysts in polycystic kidney disease.

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

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7 January 2021

In vitro modulation of tubular cyst regression in murine polycystic kidney disease

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7 January 2021

Elevated c-myc protooncogene expression in autosomal recessive polycystic kidney disease

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7 January 2021

Localization of overexpressed c-myc mRNA in polycystic kidneys of the cpk mouse

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7 January 2021

Ischemia induces partial loss of surface membrane polarity and accumulation of putative calcium ionophores

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7 January 2021

Ischemia induces surface membrane dysfunction. Mechanism of altered Na+-dependent glucose transport

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7 January 2021

Characterization of ischemia-induced loss of epithelial polarity

1 reference

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7 January 2021

In situ hybridization reveals temporal and spatial changes in cellular expression of mRNA for a laminin receptor, laminin, and basement membrane (type IV) collagen in the developing kidney

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7 January 2021

Identifiers

DOI

10.1038/KI.1993.17

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24 September 2017

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