Effects of pharmacological inhibition of NADPH oxidase or iNOS on pro-inflammatory cytokine, palmitic acid or H2O2-induced mouse islet or clonal pancreatic β-cell dysfunction (Q39736215)

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English	Effects of pharmacological inhibition of NADPH oxidase or iNOS on pro-inflammatory cytokine, palmitic acid or H2O2-induced mouse islet or clonal pancreatic β-cell dysfunction	scientific article

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scholarly article

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Europe PubMed Central

PubMed publication ID

14 September 2017

Effects of pharmacological inhibition of NADPH oxidase or iNOS on pro-inflammatory cytokine, palmitic acid or H2O2-induced mouse islet or clonal pancreatic β-cell dysfunction (English)

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Europe PubMed Central

PubMed publication ID

14 September 2017

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Philip Newsholme

4

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Europe PubMed Central

PubMed publication ID

14 September 2017

author name string

Marta Michalska

1

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Europe PubMed Central

PubMed publication ID

14 September 2017

Gabriele Wolf

2

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Europe PubMed Central

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14 September 2017

Reinhard Walther

3

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PubMed publication ID

14 September 2017

publication date

1 December 2010

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Europe PubMed Central

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14 September 2017

Bioscience Reports

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14 September 2017

30

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Europe PubMed Central

PubMed publication ID

14 September 2017

6

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Europe PubMed Central

PubMed publication ID

14 September 2017

445-453

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Europe PubMed Central

PubMed publication ID

14 September 2017

New insights into fatty acid modulation of pancreatic beta-cell function.

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The expanding role of NADPH oxidases in health and disease: no longer just agents of death and destruction

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Insights into the critical role of NADPH oxidase(s) in the normal and dysregulated pancreatic beta cell

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Phosphorylation of p47phox sites by PKC alpha, beta II, delta, and zeta: effect on binding to p22phox and on NADPH oxidase activation

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21 January 2018

Pro-inflammatory cytokines increase glucose, alanine and triacylglycerol utilization but inhibit insulin secretion in a clonal pancreatic beta-cell line

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Life and death decisions of the pancreatic beta-cell: the role of fatty acids

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Diabetes associated cell stress and dysfunction: role of mitochondrial and non-mitochondrial ROS production and activity

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Glucose, palmitate and pro-inflammatory cytokines modulate production and activity of a phagocyte-like NADPH oxidase in rat pancreatic islets and a clonal beta cell line

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A nuclear magnetic resonance-based demonstration of substantial oxidative L-alanine metabolism and L-alanine-enhanced glucose metabolism in a clonal pancreatic beta-cell line: metabolism of L-alanine is important to the regulation of insulin secreti

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Arachidonic acid, palmitic acid and glucose are important for the modulation of clonal pancreatic beta-cell insulin secretion, growth and functional integrity

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Stimulation of insulin secretion by long-chain free fatty acids. A direct pancreatic effect

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Pleiotropic effects of fatty acids on pancreatic beta-cells

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In vivo imaging of reactive oxygen and nitrogen species in inflammation using the luminescent probe L-012.

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Effect of inhibition of inducible nitric oxide synthase and guanylyl cyclase on endotoxin-induced delay in gastric emptying and intestinal transit in mice

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Hydrogen peroxide alters mitochondrial activation and insulin secretion in pancreatic beta cells.

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Involvement of oxidative stress in the pathogenesis of diabetes

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Protection of insulin-producing RINm5F cells against cytokine-mediated toxicity through overexpression of antioxidant enzymes

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Thiols and pancreatic beta-cell function: a review

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Interleukin-1 and nitric oxide increase NADPH oxidase activity in human coronary artery smooth muscle cells

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Induction of inducible nitric oxide synthase increases the production of reactive oxygen species in RAW264.7 macrophages

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Identification of a classic cytokine-induced enhancer upstream in the human iNOS promoter

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iNOS expression requires NADPH oxidase-dependent redox signaling in microvascular endothelial cells

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Reactive oxygen intermediate-dependent NF-kappaB activation by interleukin-1beta requires 5-lipoxygenase or NADPH oxidase activity

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Reactive oxygen species as a signal in glucose-stimulated insulin secretion

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Association of NAD(P)H oxidase with glucose-induced insulin secretion by pancreatic beta-cells.

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A neuronal isoform of nitric oxide synthase expressed in pancreatic beta-cells controls insulin secretion

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NO induces a cGMP-independent release of cytochrome c from mitochondria which precedes caspase 3 activation in insulin producing RINm5F cells.

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21 January 2018

Identifiers

10.1042/BSR20090138

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

14 September 2017

ResearchGate publication ID

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