The human immunodeficiency virus-1 Tat protein activates human umbilical vein endothelial cell E-selectin expression via an NF-kappa B-dependent mechanism (Q38292349)

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English	The human immunodeficiency virus-1 Tat protein activates human umbilical vein endothelial cell E-selectin expression via an NF-kappa B-dependent mechanism	scientific article

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scholarly article

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Europe PubMed Central

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1 September 2017

The human immunodeficiency virus-1 Tat protein activates human umbilical vein endothelial cell E-selectin expression via an NF-kappa B-dependent mechanism (English)

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Europe PubMed Central

PubMed publication ID

1 September 2017

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author name string

Adela Cota-Gomez

1

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1 September 2017

Natalia C Flores

2

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1 September 2017

Coral Cruz

3

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1 September 2017

Anna Casullo

4

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1 September 2017

Tak Yee Aw

5

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1 September 2017

Hiroshi Ichikawa

6

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1 September 2017

Jerome Schaack

7

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1 September 2017

Robert Scheinman

8

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1 September 2017

Sonia C Flores

9

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1 September 2017

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publication date

4 February 2002

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1 September 2017

Journal of Biological Chemistry

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1 September 2017

277

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1 September 2017

17

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Europe PubMed Central

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1 September 2017

14390-14399

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Europe PubMed Central

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1 September 2017

Prevalent myocarditis at necropsy in the acquired immunodeficiency syndrome

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Cardiac manifestations of acquired immune deficiency syndrome: A 1991 update

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Transcriptional activation in vitro by the human immunodeficiency virus type 1 Tat protein: evidence for specific interaction with a coactivator(s).

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Multifaceted activities of the HIV-1 transactivator of transcription, Tat.

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The Tat protein of human immunodeficiency virus type 1, a growth factor for AIDS Kaposi sarcoma and cytokine-activated vascular cells, induces adhesion of the same cell types by using integrin receptors recognizing the RGD amino acid sequence

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Identification of an Arg-Gly-Asp (RGD) cell adhesion site in human immunodeficiency virus type 1 transactivation protein, tat

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Tat transactivation: a model for the regulation of eukaryotic transcriptional elongation

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Tat-associated kinase (P-TEFb): a component of transcription preinitiation and elongation complexes

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Trans-activation by human immunodeficiency virus Tat protein requires the C-terminal domain of RNA polymerase II

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Taking a new TAK on tat transactivation

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Requirements for RNA polymerase II carboxyl-terminal domain for activated transcription of human retroviruses human T-cell lymphotropic virus I and HIV-1

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HIV-1 transactivator protein Tat induces proliferation and TGF beta expression in human articular chondrocytes

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HIV-1 tat protein induces the production of interleukin-8 by human brain-derived endothelial cells

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21 January 2018

Transient exposure to HIV-1 Tat protein results in cytokine production in macrophages and astrocytes. A hit and run phenomenon

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Tat protein of human immunodeficiency virus type 1 induces interleukin-10 in human peripheral blood monocytes: implication of protein kinase C-dependent pathway

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Human immunodeficiency virus type 1 Tat protein stimulates inducible nitric oxide synthase expression and nitric oxide production in microglial cultures

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Repression of MHC class I gene promoter activity by two-exon Tat of HIV

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Tat protein of human immunodeficiency virus type 1 represses expression of manganese superoxide dismutase in HeLa cells

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Interferons in the pathogenesis and treatment of human immunodeficiency virus infection

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Differential requirement for p56lck in HIV-tat versus TNF-induced cellular responses: effects on NF-kappa B, activator protein-1, c-Jun N-terminal kinase, and apoptosis

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Extracellular HIV type 1 Tat protein induces CD69 expression through NF-kappaB activation: possible correlation with cell surface Tat-binding proteins.

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Lack of responsiveness of a nuclear factor-kappaB-regulated promoter to transactivation by human immunodeficiency virus 1 Tat in HeLa cells

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Human immunodeficiency virus-1-tat induces matrix metalloproteinase-9 in monocytes through protein tyrosine phosphatase-mediated activation of nuclear transcription factor NF-kappaB.

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Three NF-kappa B binding sites in the human E-selectin gene required for maximal tumor necrosis factor alpha-induced expression

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Trans-activation of human immunodeficiency virus gene expression is mediated by nuclear events

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Tumor necrosis factor alpha-induced E-selectin expression is activated by the nuclear factor-kappaB and c-JUN N-terminal kinase/p38 mitogen-activated protein kinase pathways

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Interleukin-1beta activates a short STAT-3 isoform in clonal insulin-secreting cells

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21 January 2018

Cyclic AMP-Independent ATF Family Members Interact with NF-κB and Function in the Activation of the E-Selectin Promoter in Response to Cytokines

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Nuclear factor kappa B: an oxidative stress-responsive transcription factor of eukaryotic cells (a review)

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Identification of an inducible endothelial-leukocyte adhesion molecule

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Endothelial leukocyte adhesion molecule 1: an inducible receptor for neutrophils related to complement regulatory proteins and lectins

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Tat-mediated delivery of heterologous proteins into cells

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Cellular uptake of the tat protein from human immunodeficiency virus

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The angiogenesis induced by HIV-1 tat protein is mediated by the Flk-1/KDR receptor on vascular endothelial cells.

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Selective CXCR4 antagonism by Tat: implications for in vivo expansion of coreceptor use by HIV-1

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Angiogenic properties of human immunodeficiency virus type 1 Tat protein

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Identifiers

10.1074/JBC.M108591200

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PubMed publication ID

1 reference

Europe PubMed Central

PubMed publication ID

1 September 2017

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