JAK2/STAT3, not ERK1/2, mediates interleukin-6-induced activation of inducible nitric-oxide synthase and decrease in contractility of adult ventricular myocytes (Q28567596)

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English	JAK2/STAT3, not ERK1/2, mediates interleukin-6-induced activation of inducible nitric-oxide synthase and decrease in contractility of adult ventricular myocytes	scientific journal article

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scholarly article

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PubMed publication ID

31 January 2017

JAK2/STAT3, not ERK1/2, mediates interleukin-6-induced activation of inducible nitric-oxide synthase and decrease in contractility of adult ventricular myocytes (English)

1 reference

PubMed publication ID

31 January 2017

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author name string

XinWen Yu

1

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PubMed publication ID

31 January 2017

Richard H. Kennedy

2

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PubMed publication ID

31 January 2017

Shi J. Liu

3

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PubMed publication ID

31 January 2017

language of work or name

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publication date

2 May 2003

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PubMed publication ID

31 January 2017

Journal of Biological Chemistry

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PubMed publication ID

31 January 2017

278

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PubMed publication ID

31 January 2017

18

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PubMed publication ID

31 January 2017

16304–16309

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PubMed publication ID

31 January 2017

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Interleukin-6-induced tethering of STAT3 to the LAP/C/EBPbeta promoter suggests a new mechanism of transcriptional regulation by STAT3.

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21 January 2018

Introduction: STATs as essential intracellular mediators of cytokine responses

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STAT3 is required for the gp130-mediated full activation of the c-myc gene

1 reference

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1 reference

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Promoter of the mouse gene encoding calcium-independent nitric oxide synthase confers inducibility by interferon gamma and bacterial lipopolysaccharide

1 reference

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1 reference

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