Differential regulation of neutrophil-activating chemokines by IL-6 and its soluble receptor isoforms (Q24337486)

extrinsic apoptotic signaling pathway

GOA release 2020-03-11

1 reference

GOA release 2020-03-11

8

Naoki Yamamoto

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Thomas S. Wilkinson

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author name string

Rachel M McLoughlin

1

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Suzanne M Hurst

2

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Mari A Nowell

3

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Dean A Harris

4

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Sankichi Horiuchi

5

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Llinos W Morgan

6

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Thomas S Wilkinson

7

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Nicholas Topley

9

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Simon A Jones

Chemotactic agents induce IL-6Ralpha shedding from polymorphonuclear cells: involvement of a metalloproteinase of the TNF-alpha-converting enzyme (TACE) type

10

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language of work or name

English

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publication date

1 May 2004

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published in

Journal of Immunology

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volume

172

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issue

9

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page(s)

5676-83

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cites work

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The soluble interleukin-6 receptor is generated by shedding

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Soluble interleukin-6 receptors released from T cell or granulocyte/macrophage cell lines and human peripheral blood mononuclear cells are generated through an alternative splicing mechanism

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Interferon-alpha and interferon-gamma down-regulate the production of interleukin-8 and ENA-78 in human monocytes

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Granulocyte chemotactic protein 2 acts via both IL-8 receptors, CXCR1 and CXCR2

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Soluble interleukin-6 receptor alpha inhibits the cytokine-Induced fractalkine/CX3CL1 expression in human vascular endothelial cells in culture

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Protein kinase C activity is rate limiting for shedding of the interleukin-6 receptor.

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Isolation of an mRNA encoding a soluble form of the human interleukin-6 receptor

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The role of soluble receptors in cytokine biology: the agonistic properties of the sIL-6R/IL-6 complex

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Chemokines: a new classification system and their role in immunity

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Role of IL-6 and its soluble receptor in induction of chemokines and leukocyte recruitment.

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IL-6 and Its Soluble Receptor Orchestrate a Temporal Switch in the Pattern of Leukocyte Recruitment Seen during Acute Inflammation

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Blood monocytes consist of two principal subsets with distinct migratory properties

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IL-6: a regulator of the transition from neutrophil to monocyte recruitment during inflammation

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Chemokines and leukocyte traffic

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Impaired immune and acute-phase responses in interleukin-6-deficient mice.

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High-level production of alternatively spliced soluble interleukin-6 receptor in serum of patients with adult T-cell leukaemia/HTLV-I-associated myelopathy

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Soluble gp130 is the natural inhibitor of soluble interleukin-6 receptor transsignaling responses

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Different functions for the interleukin 8 receptors (IL-8R) of human neutrophil leukocytes: NADPH oxidase and phospholipase D are activated through IL-8R1 but not IL-8R2.

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The CXC Chemokines Growth-regulated Oncogene (GRO) α, GROβ, GROγ, Neutrophil-activating Peptide-2, and Epithelial Cell-derived Neutrophil-activating Peptide-78 Are Potent Agonists for the Type B, but Not the Type A, Human Interleukin-8 Receptor

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Interleukin-8 (IL-8), melanoma growth-stimulatory activity, and neutrophil-activating peptide selectively mediate priming of the neutrophil NADPH oxidase through the type A or type B IL-8 receptor

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Chemokine antagonists that discriminate between interleukin-8 receptors. Selective blockers of CXCR2.

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STAT3 acts as a co-activator of glucocorticoid receptor signaling

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Differential cross-regulation of the human chemokine receptors CXCR1 and CXCR2. Evidence for time-dependent signal generation.

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Chemokine-cytokine cross-talk. The ELR+ CXC chemokine LIX (CXCL5) amplifies a proinflammatory cytokine response via a phosphatidylinositol 3-kinase-NF-kappa B pathway

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Soluble cytokine receptors are present in normal human urine

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C-reactive protein: a physiological activator of interleukin 6 receptor shedding.

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Purification and characterization of the soluble interleukin-6 receptor from human plasma and identification of an isoform generated through alternative splicing

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Retrograde inflammatory signaling from neutrophils to endothelial cells by soluble interleukin-6 receptor alpha

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IL-6 is an antiinflammatory cytokine required for controlling local or systemic acute inflammatory responses

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The detection of a novel neutrophil-activating peptide (ENA-78) using a sensitive ELISA

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Secretion of oncostatin M by infiltrating neutrophils: regulation of IL-6 and chemokine expression in human mesothelial cells

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CCR3 Expression Induced by IL-2 and IL-4 Functioning as a Death Receptor for B Cells

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Soluble IL-6 Receptor Governs IL-6 Activity in Experimental Arthritis: Blockade of Arthritis Severity by Soluble Glycoprotein 130

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