Stimulation of CD30 in anaplastic large cell lymphoma leads to production of nuclear factor-kappaB p52, which is associated with hyperphosphorylated Bcl-3 (Q24316194)

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English	Stimulation of CD30 in anaplastic large cell lymphoma leads to production of nuclear factor-kappaB p52, which is associated with hyperphosphorylated Bcl-3	scientific article

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Stimulation of CD30 in anaplastic large cell lymphoma leads to production of nuclear factor-kappaB p52, which is associated with hyperphosphorylated Bcl-3 (English)

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TNF receptor superfamily member 8

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GOA release 2020-03-11

BCL3 transcription coactivator

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GOA release 2020-03-11

positive regulation of TRAIL production

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GOA release 2020-03-11

Takashi Uchiyama

4

object named as

Takashi Uchiyama

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author name string

Momoko Nishikori

1

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Hitoshi Ohno

2

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Hironori Haga

3

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language of work or name

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publication date

August 2005

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96

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8

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487-97

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Molecular cloning and expression of a new member of the nerve growth factor receptor family that is characteristic for Hodgkin's disease

1 reference

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20 April 2017

Diverse effects of BCL3 phosphorylation on its modulation of NF-kappaB p52 homodimer binding to DNA

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Nuclear factor kappaB-dependent gene expression profiling of Hodgkin's disease tumor cells, pathogenetic significance, and link to constitutive signal transducer and activator of transcription 5a activity

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The oncoprotein Bcl-3 directly transactivates through kappa B motifs via association with DNA-binding p50B homodimers

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Death receptors: signaling and modulation

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FADD, a novel death domain-containing protein, interacts with the death domain of Fas and initiates apoptosis

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TNF-dependent recruitment of the protein kinase RIP to the TNF receptor-1 signaling complex

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T cell receptor-dependent cell death of T cell hybridomas mediated by the CD30 cytoplasmic domain in association with tumor necrosis factor receptor-associated factors

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NF-kappa B-inducible BCL-3 expression is an autoregulatory loop controlling nuclear p50/NF-kappa B1 residence

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NF-kappaB p105 is a target of IkappaB kinases and controls signal induction of Bcl-3-p50 complexes

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NF-kappaB-inducing kinase regulates the processing of NF-kappaB2 p100

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CD40 regulates the processing of NF-kappaB2 p100 to p52

1 reference

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The candidate oncoprotein Bcl-3 is an antagonist of p50/NF-kappa B-mediated inhibition

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The Bcl-3 oncoprotein acts as a bridging factor between NF-kappaB/Rel and nuclear co-regulators

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Critical roles for the Bcl-3 oncoprotein in T cell-mediated immunity, splenic microarchitecture, and germinal center reactions

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B cell lymphoma-associated chromosomal translocation involves candidate oncogene lyt-10, homologous to NF-kappa B p50

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Translocations involving anaplastic lymphoma kinase (ALK).

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Hodgkin and Reed-Sternberg cells in Hodgkin's disease represent the outgrowth of a dominant tumor clone derived from (crippled) germinal center B cells.

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High-level expression of BCL3 differentiates t(2;5)(p23;q35)-positive anaplastic large cell lymphoma from Hodgkin disease

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Dual action of CD30 antigen: anti-CD30 antibody induced apoptosis and interleukin-8 secretion in Ki-1 lymphoma cells

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Continuous hematopoietic cell lines as model systems for leukemia-lymphoma research

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The candidate proto-oncogene bcl-3 is related to genes implicated in cell lineage determination and cell cycle control

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Uracil DNA glycosylase activity is dispensable for immunoglobulin class switch

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BCL-3 overexpression in anaplastic lymphoma kinase-positive anaplastic large cell lymphoma.

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Epstein-Barr virus-encoded latent infection membrane protein 1 regulates the processing of p100 NF-kappaB2 to p52 via an IKKgamma/NEMO-independent signalling pathway

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Lymphotoxin and lipopolysaccharide induce NF-kappaB-p52 generation by a co-translational mechanism

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Latent membrane protein 1 of Epstein-Barr virus stimulates processing of NF-kappa B2 p100 to p52.

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Retroviral oncoprotein Tax induces processing of NF-kappaB2/p100 in T cells: evidence for the involvement of IKKalpha

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ALK expression defines a distinct group of T/null lymphomas ("ALK lymphomas") with a wide morphological spectrum.

1 reference

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The TNF receptor superfamily of cellular and viral proteins: activation, costimulation, and death

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The signaling adaptors and pathways activated by TNF superfamily

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NF-kappaB/Rel-mediated regulation of apoptosis in hematologic malignancies and normal hematopoietic progenitors.

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The NPM-ALK oncoprotein abrogates CD30 signaling and constitutive NF-kappaB activation in anaplastic large cell lymphoma

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GSK3-mediated BCL-3 phosphorylation modulates its degradation and its oncogenicity

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NF-kappaB signaling pathways in mammalian and insect innate immunity

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Constitutive expression of Bc1-3 in thymocytes increases the DNA binding of NF-kappaB1 (p50) homodimers in vivo

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Nuclear factor (NF)-kappa B2 (p100/p52) is required for normal splenic microarchitecture and B cell-mediated immune responses

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Mice deficient in nuclear factor (NF)-kappa B/p52 present with defects in humoral responses, germinal center reactions, and splenic microarchitecture

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Impaired Negative Selection of T Cells in Hodgkin's Disease Antigen CD30–Deficient Mice

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7 January 2021

based on heuristic

inferred from DOI database lookup

Molecular analysis of a chromosomal translocation, t(9;14)(p13;q32), in a diffuse large-cell lymphoma cell line expressing the Ki-1 antigen

1 reference

https://api.crossref.org/works/10.1111%2FJ.1349-7006.2005.00078.X

7 January 2021

based on heuristic

inferred from DOI database lookup

The candidate proto-oncogene bcl-3 encodes a transcriptional coactivator that activates through NF-kappa B p50 homodimers

1 reference

https://api.crossref.org/works/10.1111%2FJ.1349-7006.2005.00078.X

7 January 2021

based on heuristic

inferred from DOI database lookup

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10.1111/J.1349-7006.2005.00078.X

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