Activin receptor-like kinase (ALK)1 is an antagonistic mediator of lateral TGFbeta/ALK5 signaling (Q24298472)

Transforming growth factor, beta receptor I

GOA release 2020-03-11

1 reference

GOA release 2020-03-11

author

Gudrun Valdimarsdottir

Gudrun Valdimarsdottir

2

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Franck Lebrin

Franck Lebrin

4

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Susumu Itoh

Susumu Itoh

3

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Christine Mummery

Christine Mummery

6

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author name string

Marie José Goumans

1

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Jonas Larsson

5

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Stefan Karlsson

7

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Peter ten Dijke

Identification of human activin and TGF beta type I receptors that form heteromeric kinase complexes with type II receptors

8

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language of work or name

English

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publication date

October 2003

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published in

Molecular Cell

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volume

12

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issue

4

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page(s)

817-28

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cites work

1 reference

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Type I receptors specify growth-inhibitory and transcriptional responses to transforming growth factor beta and activin

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Smad1 recognition and activation by the ALK1 group of transforming growth factor-beta family receptors

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Direct binding of Smad3 and Smad4 to critical TGF beta-inducible elements in the promoter of human plasminogen activator inhibitor-type 1 gene

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Cloning of a type I TGF-beta receptor and its effect on TGF-beta binding to the type II receptor

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Balancing the activation state of the endothelium via two distinct TGF-beta type I receptors

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A simplified system for generating recombinant adenoviruses

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SB-431542 is a potent and specific inhibitor of transforming growth factor-beta superfamily type I activin receptor-like kinase (ALK) receptors ALK4, ALK5, and ALK7

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Localization of Transforming Growth Factor-β Type I and Type II Receptors in Mouse Development

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Elucidation of Smad requirement in transforming growth factor-beta type I receptor-induced responses

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Identification and functional characterization of a Smad binding element (SBE) in the JunB promoter that acts as a transforming growth factor-beta, activin, and bone morphogenetic protein-inducible enhancer

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Identification and functional characterization of distinct critically important bone morphogenetic protein-specific response elements in the Id1 promoter

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Abnormal angiogenesis but intact hematopoietic potential in TGF-beta type I receptor-deficient mice

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Signaling by chimeric erythropoietin-TGF-beta receptors: homodimerization of the cytoplasmic domain of the type I TGF-beta receptor and heterodimerization with the type II receptor are both required for intracellular signal transduction.

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Positive and negative regulation of type II TGF-beta receptor signal transduction by autophosphorylation on multiple serine residues

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Assignment of transforming growth factor beta1 and beta3 and a third new ligand to the type I receptor ALK-1

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TGF-beta signal transduction

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TGF-beta induced transdifferentiation of mammary epithelial cells to mesenchymal cells: involvement of type I receptors

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TGF-beta receptor-mediated signalling through Smad2, Smad3 and Smad4

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Activin receptor-like kinase 1 modulates transforming growth factor-beta 1 signaling in the regulation of angiogenesis

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Transforming growth factor-beta: vasculogenesis, angiogenesis, and vessel wall integrity

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Physiological actions and clinical applications of transforming growth factor-beta (TGF-beta).

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Characterization of type I receptors for transforming growth factor-beta and activin

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Transforming growth factor beta (TGFbeta ) signaling via differential activation of activin receptor-like kinases 2 and 5 during cardiac development. Role in regulating parasympathetic responsiveness

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Complementation between kinase-defective and activation-defective TGF-beta receptors reveals a novel form of receptor cooperativity essential for signaling.

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Mechanism of activation of the TGF-beta receptor

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Identifiers

DOI

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Consolidated OpenCitations Corpus – April 2017

OpenCitations bibliographic resource ID

3066084

OpenCitations bibliographic resource ID

3066084

1 reference