Herpes simplex virus type 1 encephalitis is associated with elevated levels of F2-isoprostanes and F4-neuroprostanes (Q24276264)

8

Terence S Dermody

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Thomas J Montine

7

Thomas J Montine

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Tibor Valyi-Nagy

9

Tibor Valyi-Nagy

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Kathleen S. Montine

4

Kathleen S Montine

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author name string

Yueli Zhang

2

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Sandra J Olson

3

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L Jackson Roberts II

5

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Jason D Morrow

6

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L Jackson Roberts

The isoprostanes: Unique bioactive products of lipid peroxidation

5

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language of work or name

English

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publication date

January 2002

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August 2002

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published in

Journal of NeuroVirology

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volume

8

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issue

4

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page(s)

295-305

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cites work

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Suppression of herpes simplex virus type 1 (HSV-1)-induced pneumonia in mice by inhibition of inducible nitric oxide synthase (iNOS, NOS2)

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Human herpes viruses latent infection in the nervous system

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Interferon-γ Protects against Herpes Simplex Virus Type 1-Mediated Neuronal Death

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Human gamma interferon and tumor necrosis factor exert a synergistic blockade on the replication of herpes simplex virus

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Nitric oxide production induced by herpes simplex virus type 1 does not alter the course of the infection in human monocytic cells.

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Herpes simplex virus type 1 in brain and risk of Alzheimer's disease

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Herpes simplex encephalitis

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Herpes simplex type 1 DNA in human brain tissue

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Gamma interferon expression during acute and latent nervous system infection by herpes simplex virus type 1

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Enhanced hippocampal F2-isoprostane formation following kainate-induced seizures

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Persistent elevated expression of cytokine transcripts in ganglia latently infected with herpes simplex virus in the absence of ganglionic replication or reactivation

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Herpes simplex virus DNA sequences in the CNS of latently infected mice.

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Age distribution of latent herpes simplex virus 1 and varicella-zoster virus genome in human nervous tissue

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In Vivo Protective Effect of Tumour Necrosis Factor Against Experimental Infection with Herpes Simplex Virus Type 1

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In vivo expression of inducible nitric oxide synthase in experimentally induced neurologic diseases

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A herpes simplex virus DNA polymerase mutation that specifically attenuates neurovirulence in mice

1 reference

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Herpes simplex virus type 1 mutant strain in1814 establishes a unique, slowly progressing infection in SCID mice

1 reference

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Induction of cellular transcription factors in trigeminal ganglia of mice by corneal scarification, herpes simplex virus type 1 infection, and explantation of trigeminal ganglia

1 reference

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Autocrine interferon-beta stimulation augments nitric oxide production by mouse macrophage J774A.1 cells infected with herpes simplex virus type 1.

1 reference

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Role of nitric oxide in pathogenesis of herpes simplex virus encephalitis in rats

1 reference

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Formation of Isoprostane-like Compounds (Neuroprostanes) in Vivo from Docosahexaenoic Acid

1 reference

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Herpes simplex virus type 1 and Alzheimer's disease

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Highly variable distribution of HSV-1-specific DNA in human geniculate, vestibular and spiral ganglia

1 reference

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Herpes simplex virus type 1 gene expression and reactivation of latent infection in the central nervous system

1 reference

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Herpes simplex virus type 1 latency in the murine nervous system is associated with oxidative damage to neurons

1 reference

https://pubmed.ncbi.nlm.nih.gov/12161814

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Oxidants and antioxidants in viral diseases: disease mechanisms and metabolic regulation.

1 reference

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Tumor Necrosis Factor-α Increases the Steady-state Reduction of Cytochromebof the Mitochondrial Respiratory Chain in Metabolically Inhibited L929 Cells

1 reference

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Effects of herpes simplex virus type 1 infection on the plasma membrane and related functions of HeLa S3 cells

1 reference

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Imbalance in corneal redox state during herpes simplex virus 1-induced keratitis in rabbits. Effectiveness of exogenous glutathione supply.

1 reference

https://pubmed.ncbi.nlm.nih.gov/12161814

12 December 2020