Decompression Sickness Treatment

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Chapter 16

DECOMPRESSION SICKNESS TREATMENT

FIRST AID
Expert advice.
Expert advice from a diving physician, a diving medical organisation (such as DAN) or a hyperbaric facility, should be sought as soon as possible. Appendices B and D contain a list of sources for such expert advice. In the interim, first aid treatment must be applied The principles of first aid management of decompression sickness (DCS) are: basic first aid, ABC Airway, Breathing, Circulation . (see Chapter 42). 100% oxygen (O2). (see Chapter 40). positioning and rest fluid replacement.

Oxygen therapy
If the diver breathes 100% oxygen (O2), nitrogen (N2) is removed from the lungs. The breathing apparatus must supply to the lungs as close to 100% O2 as possible. This means an anaesthetic type mask or an O2 diving regulator as used in technical and commercial diving. It is not the simple plastic hospital oxygen mask that does not produce an air tight seal. 100% O2 in the lungs results in a high diffusion (pressure gradient) of N2 from the blood to the lungs, causing the increased elimination of N2 from the blood and tissues and also from any bubbles there. Chapter 16 1

Unfortunately 100% O2 can be toxic to the lungs if given for 1824 hours on the surface. This may complicate the hyperbaric O2 which is later given to the diver in a recompression chamber (RCC). Ideally O2 therapy should be given under the supervision of a diving physician however, if expert advice is unavailable, then all suspected case of DCS should be given 100% O2 from the outset, before and during movement of the patient and transport to the recompression chamber. Unconscious divers, if not breathing of their own accord, will require assisted ventilation (intermittent positive pressure respiration or I.P.P.R.). Conscious patients can be treated with continuous flow or demand type masks. In such cases, treating personnel must always check the breathing mask on themselves first in order to ensure the oxygen system is working, and that resistance to breathing is not excessive. Fig. 16.1
Photograph of diver with DCS breathing near 100% O2 from a resuscitation bag. Note reservoir bag (lowermost) and high constant flow (>14 litres per minute) through an oxygen delivery tube.

Position and rest.


An unconscious diver should be placed on his side in the coma position, to protect the airway at all times (see Chapter 42). If there is any likelihood of air embolism, the diver is best placed horizontal - preferably on the left side, but this is not essential. Some clinicians recommend that cases of cerebral DCS should be managed with the patient on the side, without a pillow, to prevent buoyant gas bubbles reaching the brain through the circulation. The Trendelenburg (30 degree head down, feet up) position is no longer recommended. Having the head lowered raises the pressure in the brain and this can aggravate the brain injury. Due to the buoyancy of bubbles, sitting or standing may be dangerous in patients with air embolism or cerebral decompression sickness where the bubbles are still in the blood stream. As a general rule, 100% O2 should be given for at least an hour before allowing the patient to sit or stand. After this, the diver can be allowed to adopt any comfortable position but should be kept relatively still and on 100% O2. A diver with "chokes" will be more comfortable sitting up.

Fluid replacement.
Severe DCS results in loss of blood and fluids into the tissues. It may be valuable to replace this fluid orally or intravenously. Oral fluids may be given if the diver has no abdominal pain, nausea or vomiting. Water or electrolyte fluids (eg."Gastrolyte") may be given with the type and volume recorded. Acidic (orange juice) and glucose dinks should be avoided. If there is no bladder involvement (i.e. the patient can urinate), a litre of clear fluid every 2-4 hours should suffice. This fluid intake should be modified by the patient's thirst. Some authorities have recommended large volumes of oral fluids while the diver is being transported to a RCC in an attempt to replace this deficit. One problem with this (as any party goer will attest) is that the fluid load will promote a vigorous urine flow, so the diver arrives Chapter 16 2

at the recompression facility with a stomach and bladder full of fluid. A patient with spinal DCS may be unable to empty the bladder and will therefore be in considerable pain. The patient will usually be treated on a hyperbaric O2 (HBO) treatment table. There is a very real risk of nausea, vomiting and convulsions as complications of this treatment. A full stomach can then possibly result in regurgitation of the stomach contents and aspiration into the lungs further complicating treatment. If the brain or spinal cord is involved and the patient has difficulty in voiding urine, an indwelling urinary catheter should be inserted whenever possible by a trained physician or nurse. If this is not feasible, care must be taken not to overload the patient with fluids. Anyone who is trained to institute and monitor an intravenous infusion can be expected to be able to assess the state of hydration and determine the desirability and quantity of intravenous fluids, remembering that glucose fluids can aggravate cerebral oedema.

Drugs.
Aspirin as a first aid measure has not been demonstrated to be of value in DCS. It may interfere with blood clotting and cause haemorrhage (bleeding) especially in the stomach. Haemorrhage is already a major pathology in spinal cord and inner ear DCS. The authors have seen one patient with severe DCS bleed to death from an internal haemorrhage just before he was to be given an "experimental last-ditch" anti-clotting agent. We are therefore reluctant to advocate the routine use of aspirin either for pain relief or to inhibit clotting in any DCS case. Joint pains of DCS can be significantly eased without the risk of serious side effects by the administration of paracetamol (acetaminophen) 1000 mg (or two tablets) 4 hourly. NSAID drugs may be requested by a diving physician, but are not routinely needed. Other drugs such as steroids, diuretics and special intravenous fluids such as "Rheomacrodex" have been advocated but have not been proven to be beneficial. Antiepileptics and other drugs such as diazepam ("Valium") may be needed to control fitting (convulsions), and for confusional states.

TRANSPORT OF PATIENT WITH DCS


With mild DCS, or if there has been a delay of 12-24 hours or more, and there is no progressive deterioration, local treatment with rest, monitoring and breathing 100% O2 may be all that is necessary. This decision is best made by a diving physician. With more severely affected divers, or those that are deteriorating or need medical attention, transport should be expedited. The diver should be transported with minimum agitation and as close as possible to sea level or at 1 ATA. Mountainous roads should be avoided whenever an evacuation route by land is planned. 100% O2. should be breathed before and during transport (see Chapter 40).

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Transportation in aircraft presents problems. Apart from movement which aggravates DCS, environmental pressure decreases with altitude, causing DCS bubbles to expand (Boyle's Law) and more gas to pass from the tissues into any bubbles. If the patient is evacuated by air, unpressurised aircraft should endeavor to fly at the lowest safe altitude. Even an altitude of 300 metres (1000 ft.) can make the symptoms of DCS worse. However, maintaining such an altitude can be alarming when flying over 297 metre terrain. It should be remembered that most commercial "pressurised" aircraft normally maintain a cabin pressure of around 2000 metres (6000 ft.), which could seriously aggravate DCS. Whenever possible the cabin altitude should be maintained at 1ATA. This is attainable by many modern commercial jet aircraft, executive aircraft such as the King Air and Lear Jet, and some military transport aircraft (Hercules C130). This requirement is not popular with the commercial airlines since it necessitates the aircraft flying at lower than its most efficient altitude, resulting in excessive fuel consumption. This requirement may also limit the range of certain aircraft. Breathing 100% Oxygen before and during the flight may be of value, especially from closed or semi-closed circuit equipment. There are risks to the aircraft and its inhabitants if open circuit O2 is used as many airlines recirculate the cabin atmosphere and fire/explosion is possible.

Fig. 16.2
A portable patient treatment chamber connected to a second compartment for the exchange and transter of attendents and medics. The larger chamber also allows for transfer of patient to a larger recompression facility.

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DEFINITIVE TREATMENT OF DCS


This is best controlled by diving physicians, hyperbaric facilities or specialised diving medical organisations, such as DAN. Therapeutic recompression.
This is the most effective treatment for DCS. Delay increases the likelihood of a poor final result. The diver is placed in a recompression chamber (RCC) and the pressure is increased according to a specified recompression treatment table. The increase in pressure reduces the bubble size (Boyle's Law) and usually relieves the clinical features. It also increases the surface area to volume ratio of the bubble, which may collapse. The increased pressure in the bubble also enhances the diffusion gradient, encouraging nitrogen to leave the bubble. The therapeutic recompression schedule determines the depth and duration of the treatment profile. The table selected may depend on factors such as the time elapsed since the onset of symptoms, the depth of the original dive and the type and severity of symptoms, as well as the capability of the treatment facility and the various breathing gases available. The treatment may be amended depending on the patient's response. In recent years the Oxygen Treatment Tables are preferred because of their increased effectiveness. The injured diver is usually compressed to an equivalent depth of 18 metres (60 ft.) and is decompressed over 35 hours. He breathes oxygen from a mask while the rest of the chamber is filled with air. If the whole chamber is filled with oxygen, the fire risk increases dramatically. Because the attendant breathes chamber air, great care must be taken to monitor his dive profile to avoid the embarrassing predicament of an attendant emerging from the RCC with DCS. The diver breaths oxygen for the duration of the treatment except for 5 minute periods of air or sometimes heliox (He-O2) breathing every 25 minutes, to minimise oxygen toxicity to the lungs. The regimen is not without hazard as there is a significant risk of cerebral oxygen toxicity causing convulsions in such persons.

Fig. 16.3 Chapter 16 5

Other tables are available which involve compressing the diver to a depth equivalent of 30 50 metres sea-water (100165 ft.) in air, heliox and nitrox mixtures and then decompressing over periods ranging from several hours to several days depending on the severity of the symptoms. Many other investigations and treatment modalities will be employed by experienced physicians in the RCC, including fluid balance, medications, etc, which need not concern the average diver. A further emergency procedure, Underwater Oxygen Treatment, has been devised for use under expert supervision in remote localities. The diver is recompressed in the water to a maximum of 9 metres while breathing 100% O2. Details of this procedure are outlined in Appendix C. Treatment in water with the diver breathing air has been used in many parts of the world and water treatment tables are contained in some Navy diving manuals. While success has often been reported and delay in treatment can be avoided, this form of treatment has serious theoretical and practical difficulties which can result in worsening of the diver's condition. The deep water air requirements (30 metres initially, and decompression for many hours)) renders the patient and accompanying divers prone to cold (hypothermia), narcosis, gas exhaustion, tide and other current changes. Attending divers may well develop DCS from extra exposures. This form of treatment is not generally recommended, unless other options (RCC, underwater oxygen treatment, etc.) are unavailable.

Fig. 16.4
Schematic outline of large static RCC treatment facility showing linking of a small portable chamber to the larger fixed unit. Both chambers are pressurised to identical pressures for a "transfer-under-pressure" (TUP).

Hazards of therapeutic recompression.


While therapeutic recompression generally produces a dramatic relief of symptoms, it has several serious hazards. These include oxygen toxicity in the patient, the risk of fire and the risk of producing DCS in the attendant. It should only be used under the close supervision of a medical officer experienced in its use.

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PREVENTION OF DCS
There are a number of factors which predispose to DCS. These are described in Chapter 13. Obviously these should be avoided wherever possible but some are unavoidable. Apart from falsifying a birth certificate there is little an individual can do about the predisposing factor of age. Because of the unreliability of the currently available dive tables and DCs, and the unpredictability of the development of DCS, it is possible for even the most careful, well trained and conscientious diver to develop this condition. The following suggestions will help reduce the risks. It is important that the diver never exceeds the no-decompression limits and ascent rates. In spite of this, all the dive tables, and especially the dive computers so far devised, have a significant failure rate.

"Fudge factors".
The rate of development of DCS varies from less than 1% to as much as 5% depending on the table used, the depth and the duration of the dive, and if tables are pushed to their limits. The apparent safety of the tables is probably improved by intelligent divers incorporating "fudge factors" of their own. This is especially required for older, fatter and less fit divers. They imply ascending before it is theoretically necessary reducing bottom times and depths.

Accurate depth & time.


It is essential that the diver knows accurately the depths and durations of his diving. A depth gauge which indicates the maximum depth attained is useful, because it is common for divers to descend deeper than they realise. An underwater watch or, better, a bottom timer is valuable, as is a D.C.

No-decompression diving.
Although the tables are not totally reliable, they are less reliable for deep diving (greater than 30 metres). It is advisable to avoid pushing the tables to the limits when a no-decompression schedule is followed and to avoid dives requiring decompression.

Slow ascent rates.


A slow ascent is prudent and the diver should certainly not ascend faster than the rate recommended by the tables. Preferably a slower ascent rate should be employed (810 metres or 2533 ft. per minute is an acceptable safe rate) and the extra time taken is deleted from the bottom time. i.e. ascend earlier than permitted by the tables.

Routine decompression stops.


Most authorities recommend a routine minimum safety ("decompression") stop at 3-5 metres for 3-5 minutes, after a no-decompression dive greater than 15 metres, to allow partial nitrogen elimination and trapping of venous emboli in the lung vessels. Chapter 16 7

Dive planning.
When repeated dives or multi-level dives are planned, the deeper dives should always be performed first. With multi-day diving, a rest day is included after each 3 continuous diving days. With deep diving, gradual build up (acclimatisation) is achieved by progressively deeper exposures.

Post-dive restrictions.
It is advisable to rest for an hour or more after a deep or long dive to ensure elimination of nitrogen from the fast tissues. Surface intervals should be > 2-4 hours. Flying and significant altitude exposures within 1224 hours of diving is not recommended. In addition to the above precautions, the diver is also advised to buy a good quality waterproof rabbit's foot. A healthy scepticism towards reliance on any mechanical equipment, especially if promoted by a glossy brochure or a dive computer salesman, also has good survival value.

Dive computers (See Chapter 14).

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