(Q27323951)

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Acidosis activation of the proton-sensing GPR4 receptor stimulates vascular endothelial cell inflammatory responses revealed by transcriptome analysis

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29 February 2020

Acidosis activation of the proton-sensing GPR4 receptor stimulates vascular endothelial cell inflammatory responses revealed by transcriptome analysis (English)

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29 February 2020

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29 February 2020

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29 February 2020

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Lixue Dong

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29 February 2020

Zhigang Li

2

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29 February 2020

Nancy R Leffler

3

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29 February 2020

Adam S Asch

4

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29 February 2020

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16 April 2013

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29 February 2020

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29 February 2020

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29 February 2020

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29 February 2020

e61991

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29 February 2020

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16 April 2013

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0 references

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Analysis of Relative Gene Expression Data Using Real-Time Quantitative PCR and the 2−ΔΔCT Method

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Determinants of blood pH in health and disease

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21 March 2017

Proton-sensing G-protein-coupled receptors

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Inflammatory stress increases receptor for lysophosphatidylcholine in human microvascular endothelial cells

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21 March 2017

Activation of GPR4 by acidosis increases endothelial cell adhesion through the cAMP/Epac pathway

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Lactic acidosis triggers starvation response with paradoxical induction of TXNIP through MondoA

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21 March 2017

Vascular abnormalities in mice deficient for the G protein-coupled receptor GPR4 that functions as a pH sensor

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Crosstalk in NF-κB signaling pathways

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An essential role for NF-kappaB in preventing TNF-alpha-induced cell death

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Functional interaction between responses to lactic acidosis and hypoxia regulates genomic transcriptional outputs.

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28 September 2017

Inhibition of tumor cell migration and metastasis by the proton-sensing GPR4 receptor

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Deletion of the pH sensor GPR4 decreases renal acid excretion

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The genomic analysis of lactic acidosis and acidosis response in human cancers

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Gene expression profile of duodenal epithelial cells in response to chronic metabolic acidosis

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Modulatory effects of hypercapnia on in vitro and in vivo pulmonary endothelial-neutrophil adhesive responses during inflammation

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From endoplasmic-reticulum stress to the inflammatory response

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Genetic endothelial systems biology of sickle stroke risk

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Myocardial reperfusion injury.

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ICAM-1 in acute myocardial infarction: a potential therapeutic target

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Inflammation in stroke and focal cerebral ischemia.

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Anaesthesia for peculiar cells--a century of sickle cell disease

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Anti-adhesion molecule strategies as potential neuroprotective agents in cerebral ischemia: a critical review of the literature

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Multiple control of interleukin-8 gene expression

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Endothelial cell heterogeneity and organ specificity

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Attenuation of extracellular acidic pH-induced cyclooxygenase-2 expression by nitric oxide.

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1 reference

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12 December 2020

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10.1371/JOURNAL.PONE.0061991

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Consolidated OpenCitations Corpus – April 2017

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29 February 2020

2013PLoSO...861991D

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Consolidated OpenCitations Corpus – April 2017

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Consolidated OpenCitations Corpus – April 2017

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Europe PubMed Central

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https://www.ebi.ac.uk/europepmc/webservices/rest/search?query=EXT_ID:23613998%20AND%20SRC:MED&resulttype=core&format=json

29 February 2020

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Consolidated OpenCitations Corpus – April 2017

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Europe PubMed Central

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29 February 2020

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