Retinoic acid expression associates with enhanced IL-22 production by γδ T cells and innate lymphoid cells and attenuation of intestinal inflammation. (Q41970839)

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scientific article published on 20 May 2013

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English	Retinoic acid expression associates with enhanced IL-22 production by γδ T cells and innate lymphoid cells and attenuation of intestinal inflammation.	scientific article published on 20 May 2013

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1 March 2020

Retinoic acid expression associates with enhanced IL-22 production by γδ T cells and innate lymphoid cells and attenuation of intestinal inflammation (English)

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1 March 2020

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1 March 2020

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1 March 2020

Mathilde Raverdeau

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1 March 2020

Joanna R. Groom

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1 March 2020

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1 March 2020

Adrian P Bracken

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1 March 2020

Kingston H Mills

12

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1 March 2020

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Rowan Higgs

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1 March 2020

Anna Stefanska

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1 March 2020

Lara S Dungan

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1 March 2020

Caroline E Sutton

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1 March 2020

Gundula Streubel

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1 March 2020

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20 May 2013

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1 March 2020

Journal of Experimental Medicine

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1 March 2020

210

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1 March 2020

6

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1 March 2020

1117-1124

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1 March 2020

Innate lymphoid cells--a proposal for uniform nomenclature

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The aryl hydrocarbon receptor regulates gut immunity through modulation of innate lymphoid cells

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Exogenous stimuli maintain intraepithelial lymphocytes via aryl hydrocarbon receptor activation

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A retinoic acid-dependent checkpoint in the development of CD4+ T cell-mediated immunity

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The role of retinoic acid in tolerance and immunity

1 reference

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5 June 2018

IL-23-responsive innate lymphoid cells are increased in inflammatory bowel disease

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Essential role for retinoic acid in the promotion of CD4(+) T cell effector responses via retinoic acid receptor alpha

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Co-adjuvant effects of retinoic acid and IL-15 induce inflammatory immunity to dietary antigens

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γδ T cells protect against lung fibrosis via IL-22

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Skin-draining lymph nodes contain dermis-derived CD103(-) dendritic cells that constitutively produce retinoic acid and induce Foxp3(+) regulatory T cells

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GM-CSF and IL-4 synergistically trigger dendritic cells to acquire retinoic acid-producing capacity

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Innate and adaptive interleukin-22 protects mice from inflammatory bowel disease

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Interleukin-22 mediates early host defense against attaching and effacing bacterial pathogens

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Retinoic acid inhibits Th17 polarization and enhances FoxP3 expression through a Stat-3/Stat-5 independent signaling pathway

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Small intestine lamina propria dendritic cells promote de novo generation of Foxp3 T reg cells via retinoic acid

1 reference

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A functionally specialized population of mucosal CD103+ DCs induces Foxp3+ regulatory T cells via a TGF-beta and retinoic acid-dependent mechanism

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Reciprocal TH17 and regulatory T cell differentiation mediated by retinoic acid

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Generation of gut-homing IgA-secreting B cells by intestinal dendritic cells

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Retinoic acid imprints gut-homing specificity on T cells

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Downregulation of Th17 cells in the small intestine by disruption of gut flora in the absence of retinoic acid

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12 December 2020

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10.1084/JEM.20121588

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1 March 2020

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1 March 2020

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1 March 2020

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