Remarkable increase in cerebrospinal fluid 3-nitrotyrosine in patients with sporadic amyotrophic lateral sclerosis (Q28369023)

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T Abe

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K Yamazaki

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T Murata

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E Ishizaki

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C Isobe

Mutations in Cu/Zn superoxide dismutase gene are associated with familial amyotrophic lateral sclerosis

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language of work or name

English

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publication date

July 1999

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published in

Annals of Neurology

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volume

46

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issue

1

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page(s)

129-31

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cites work

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Motor neuron degeneration in mice that express a human Cu,Zn superoxide dismutase mutation

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Altered reactivity of superoxide dismutase in familial amyotrophic lateral sclerosis

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A gain-of-function of an amyotrophic lateral sclerosis-associated Cu,Zn-superoxide dismutase mutant: An enhancement of free radical formation due to a decrease in Km for hydrogen peroxide

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Peroxynitrite-mediated tyrosine nitration catalyzed by superoxide dismutase

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Oxidative stress, glutamate, and neurodegenerative disorders

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Increased 3-nitrotyrosine and oxidative damage in mice with a human copper/zinc superoxide dismutase mutation.

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Survival prediction in amyotrophic lateral sclerosis

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Neuronal DNA damage precedes tangle formation and is associated with up-regulation of nitrotyrosine in Alzheimer's disease brain.

21 January 2018

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ALS, SOD and peroxynitrite

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Linkage of a gene causing familial amyotrophic lateral sclerosis to chromosome 21 and evidence of genetic-locus heterogeneity

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Identification of a novel SOD1 mutation in an apparently sporadic amyotrophic lateral sclerosis patient and the detection of Ile113Thr in three others

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Variance of age at onset in a Japanese family with amyotrophic lateral sclerosis associated with a novel Cu/Zn superoxide dismutase mutation

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Increased 3-nitrotyrosine in both sporadic and familial amyotrophic lateral sclerosis

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Upregulation of protein-tyrosine nitration in the anterior horn cells of amyotrophic lateral sclerosis.

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Estimation of hydroxyl free radical levels in vivo based on liquid chromatography with electrochemical detection

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Increase in oxidized NO products and reduction in oxidized glutathione in cerebrospinal fluid from patients with sporadic form of amyotrophic lateral sclerosis

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