CSOM
CSOM
CSOM
DEFINITION
Presence of non-purulent fluid within the middle ear cleft
SYNONYMS
Secretory otitis media Middle ear effusion Sero-mucinous otitis media Catarrhal otitis media Glue ear Serous otitis media Non-suppurative otitis media
PREVALENCE
Between 20% and 50% of children do have OME at some time between 3 and 10 years of age Two peaks at 2 and 5 years of age
RISK FACTORS
Race Age Gender Season Nasopharyngeal anatomical abnormalities Cleft palate Smoking ? Allergy
HISTOPATHOLOGY
Changes in the mucosa
Vasodilatation & mononuclear cell infiltration Metaplasia of the epithelium to ciliated columnar Mucus secreting gland formation
ETIOPATHOLOGY
Eustachian tube dysfunction
Chronic inflammation
ETIOLOGY
Eustachian tube dysfunction
Poor muscular function Adenoids Barotrauma Others
Infections
Unresolved AOM Adenoiditis and other URTIs
SYMPTOMS
Hearing impairment Otalgia Fluid sensation
Diagnosis
DIAGNOSIS
DIAGNOSIS
Otoscopy Tuning fork tests
DIAGNOSIS
Otoscopy Tuning fork tests PTA
DIAGNOSIS
Otoscopy Tuning fork tests PTA Tympanometry
DIAGNOSIS
Otoscopy Tuning fork tests PTA Tympanometry Myringotomy
TREATMENT
Treatment of the cause if feasible Observation Medical treatment
Antibiotics Decongestants, ?Auto-inflation ?Steroids
Surgical
Myringotomy Ventilation tubes (grommets)
Iatrogenic Cholesteatoma
SEQUELAE
Spontaneous resolution
50% resolve within 3 months. Only 5% persists for more than 12 months
Conclusion
OME is very common in children Etiology is associated with ET dysfunction and or chronic infection In adults: Nasopharyngeal pathology should be considered Most cases resolve spontaneously Conservative treatment is of doubtful value VT insertion restore hearing in the selected cases
Clinical Features
History of CSOM or OME Deafness is usually the only symptoms TM shows various structural changes
Treatment
Observation Surgical treatment Hearing aid
ETIOLOGY
Environmental Genetic Previous OM Upper respiratory tract infections Eustachian tube dysfunction
CLINICO-PATHOLOGICAL TYPES
Tubo-tympanic
Attico-antral
PATHOLOGY
Signs of suppurative infection
Discharge & perforation Chronic inflammatory reaction in the mucosa and the bone (ostietis)
CHOLESTEATOMA
DEFINITION
The presence of a desquamating stratified squamous epithelium in the middle ear
PATHOGENESIS OF CHOLESTEATOMA
Implantation (congenital or acquired) Metaplasia Epithelial migration
CLASSIFICATION OF CHOLESTEATOMA
Congenital Acquired
Primary Secondary
Effect of Cholesteatoma
Keratin encourages persistence of the infection Matrix causes bone erosion
CLINICO-PATHOLOGICAL TYPES
Tubo-tympanic
Attico-antral (cholesteatoma)
SYMPTOMS OF CSOM
Otorrhea
Intermittent, profuse & odorless in TT type Persistent, scanty & malodorous in AA type
OTOSCOPIC EXAMINATION
Discharge
Present in TT type if active but may be absent Usually is present in AA type
Perforation
Central: in TT type Marginal or attic in AA type with cholesteatoma
PERFORATION IN TT CSOM
PERFORATION IN AA CSOM
OTOSCOPIC EXAMINATION
Discharge
Present in TT type if active but may be absent Usually is present in AA type
Perforation
Central: in TT type Marginal or attic in AA type with cholesteatoma
Bacteriology
B A P S P K e r o b s e t a r o le u e s d a c t e A r io l o n a e g y r o b e s ea s c u p t ia
o m o n a s p h y lo c o c c u t e u s b s i e ll a a n d
a e a r c u t ge ir n o oi d s B s P a e u p r t eo uc so c P e p t o s t r e E s c h e r ic h
INVESTIGATIONS
Audiometry Bacteriology Imaging
Congenital Cholesteatoma
Active TT type
Inactive TT type
Conservative treatment
Active TT type
Inactive TT type
Conservative Treatment Treat any predisposing factor Keep the ear dry TYMPANOPLASTY Ear toilet Antibiotics Removal of polyps and granulations
TYMPANOPLASTY
An operation performed to eradicate disease in the middle ear cavity and to reconstruct the hearing mechanism
MYRINGOPLASTY
An operation performed tympanic membrane to repair the
AIMS OF TYMPANOPLASTY
To close the perforation To prevent re-infection To improve hearing
RADICAL MASTOIDECTOMY
An operation in which the mastoid antrum and air cells, attic and middle ear are converted into common cavity, exteriorized to the external canal. The tympanic membrane, malleus and incus are removed leaving only the stapes in situ.
Conclusion
In TT type the discharge is usually copious, intermittent and odorless. The perforation is central. Treatment is conservative (if there is active infection) followed by tympanoplasty to prevent re-infection and improve hearing. In the AA type the discharge is usually scanty, persistent and of bad odor. The perforation is attic or marginal with cholesteatoma. Treatment is by mastoidectomy to provide safety and dry ear