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    Duodenal Stenosis Kriti Jangir Group 4

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    jangirkriti770
    Duodenal stenosis and atresia occur during embryonic development when the duodenum fails to recanalize properly between weeks 8-10 of gestation. There are three types of duodenal atresia based on the severity of obstruction. Prenatal ultrasound may detect dilated stomach and duodenum while newborns present with bilious vomiting. Diagnosis is confirmed by x-ray showing the classic "double bubble" sign of gas in stomach and proximal duodenum. Treatment involves gastric decompression, resuscitation, and duodenoduodenostomy or duodenotomy surgery.

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    Duodenal Stenosis Kriti Jangir Group 4

    Uploaded by

    jangirkriti770
    18 views8 pages
    Duodenal stenosis and atresia occur during embryonic development when the duodenum fails to recanalize properly between weeks 8-10 of gestation. There are three types of duodenal atresia based on the severity of obstruction. Prenatal ultrasound may detect dilated stomach and duodenum while newborns present with bilious vomiting. Diagnosis is confirmed by x-ray showing the classic "double bubble" sign of gas in stomach and proximal duodenum. Treatment involves gastric decompression, resuscitation, and duodenoduodenostomy or duodenotomy surgery.

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    I want Shilpi mam notes because I want to increase my anatomy concepts clear

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    Duodenal stenosis and atresia occur during embryonic development when the duodenum fails to recanalize properly between weeks 8-10 of gestation. There are three types of duodenal atresia based on the severity of obstruction. Prenatal ultrasound may detect dilated stomach and duodenum while newborns present with bilious vomiting. Diagnosis is confirmed by x-ray showing the classic "double bubble" sign of gas in stomach and proximal duodenum. Treatment involves gastric decompression, resuscitation, and duodenoduodenostomy or duodenotomy surgery.

    Copyright:

    © All Rights Reserved
    Download as PPTX, PDF, TXT or read online from Scribd
    Download as pptx, pdf, or txt
    18 views8 pages

    Duodenal Stenosis Kriti Jangir Group 4

    Uploaded by

    jangirkriti770
    Duodenal stenosis and atresia occur during embryonic development when the duodenum fails to recanalize properly between weeks 8-10 of gestation. There are three types of duodenal atresia based on the severity of obstruction. Prenatal ultrasound may detect dilated stomach and duodenum while newborns present with bilious vomiting. Diagnosis is confirmed by x-ray showing the classic "double bubble" sign of gas in stomach and proximal duodenum. Treatment involves gastric decompression, resuscitation, and duodenoduodenostomy or duodenotomy surgery.

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    © All Rights Reserved
    Download as PPTX, PDF, TXT or read online from Scribd
    Download as pptx, pdf, or txt
    of 8

    DUODENAL

    STENOSIS AND
    ATRESIA

    NAME :- KRITI JANGIR


    GROUP :- 4
    SUBJECT :- INFANT SURGERY
     Embryology
     The hepatobiliary system and pancreas form during the third
    week of gestation, as the second portion of the duodenum
    gives rise to biliary and pancreatic buds at the junction of the
    foregut to the midgut.
     The duodenum also undergoes a solid phase during this time;
    between the eighth and tenth weeks of gestation, the duodenal
    lumen is reestablished by the gathering of vacuoles, and
    recanalization occurs.
     Insults during this crucial period of development are believed
    to result in failure of recanalization and consequent atresias,
    stenoses, and webs.
     In addition, duodenal atresias have been associated with a
    closely surrounding piece of pancreatic tissue.
     Whether this tissue is an annular pancreas or merely a failure
    of duodenal development is debatable .
     Classification
     Esophageal atresia and
     the VATER (vertebral defects, anal atresia,
    tracheoesophageal fistula with esophageal atresia, and
    renal and radial anomalies) syndrome have also been
    associated with duodenal atresia .
     Duodenal atresia is classified into three types: type I
    (duodenal diaphragm) is linked to the presence of a
    mucosal diaphragmatic membrane with an intact muscle
    wall; type II (complete duodenal atresia) characterized
    by a short fibrous cord connecting the two ends or
    pouches of the duodenum; and type III (also complete
    duodenal atresia) which corresponds to a complete
    separation of the two ends of the duodenum, sometimes
    together with annular pancreas.
     Diagnosis
     Prenatal ultrasonography :- such as a dilated stomach and
    proximal duodenum.
     Polyhydramnios is suggestive of GI tract obstruction
    because the fetus is unable to swallow the amniotic fluid.
     Common associated anomalies and chromosomal defects
    may be assessed by screening maternal serum and
    amniotic fluid.
     In the newborn, clear or bilious emesis is evident within
    hours of birth, with or without abdominal distension. An
    output of more than 20 mL of gastric contents is indicative
     Plain radiography is helpful and may reveal the classic
    double bubble sign, representing air in the stomach and
    proximal duodenum, which is associated with complete or
    near complete duodenal obstruction.
     Gas within the intestine distal to the duodenum is
    indicative of an incomplete obstruction.
     Upright and contrast radiography using air or contrast may
    confirm the diagnosis.
     Treatment
     Gastric decompression is essential to prevent
    aspiration, and thermoregulation should be
    monitored at all times.
     When fluid resuscitation and a full assessment
    have been accomplished, the neonate may proceed
    to surgery

     Duodenoduodenostomy and duodenotomy


     OUTCOME
     An anastomotic leak, injury to the bile duct, and
    sepsis are early complications.
     Late complications include peptic ulceration
    secondary to alkaline reflux, blind-loop syndrome
    due to duodenal stasis, and recurrent obstruction
     Recent long-term data demonstrated that a 9%
    combined early and late mortality rate over an
    average 6-year follow-up period was due almost
    exclusively to associated congenital anomalies .
    REFERENCE
    https://www.orpha.net/consor/cgi-bin/OC_Exp.php?Lng=GB&Expert=1203#:~:te
    xt=Duodenal%20atresia%20is%20classified%20into,and%20type%20III%20(also%20c
    omplete

    https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4420424/

    https://emedicine.medscape.com/article/932917-overview

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