Electrocardiogram (E.C.G)

Download as pptx, pdf, or txt
Download as pptx, pdf, or txt
You are on page 1of 51

Electrocardiogram

( E.C.G)
Electrocardiogram ( E.C.G)
• A recording of the electrical activity of the
heart .
• The E.C.G records these cardiac electrical
activity by means of metal electrodes.
• Electrical activity is followed by mechanical
event
Metal Electrode placement.
The ECG Paper
• Horizontally
– One small box - 0.04 s
– One large box - 0.20 s
• Vertically
– One large box - 0.5 mV
ECG Graph Paper

• Runs at a paper speed of 25 mm/sec


• Each small block of ECG paper is 1 mm2
• At a paper speed of 25 mm/s, one small block
equals 0.04 s
• Five small blocks make up 1 large block which
translates into 0.20 s (200 msec)
• Hence, there are 5 large blocks per second
• Voltage: 1 mm = 0.1 mV between each individual
block vertically
Normal Impulse Conduction
Sinoatrial node

AV node

Bundle of His

Bundle Branches

Purkinje fibers
Basic ECG waves
P wave - Atrial depolarization
QRS – Ventricular depolarization
T wave - Ventricular repolarization
The 12-Lead ECG

• The 12-Lead ECG sees the heart from 12


different views.
• Therefore, the 12-Lead ECG helps you
see what is happening in different
portions of the heart.
The 12-Leads

The 12-leads include:


–3 Limb leads
(I, II, III)
–3 Augmented leads
(aVR, aVL, aVF)
–6 Precordial leads
(V1- V6)
How waves are formed
• When the wave of depolarisation spreads
towards the positive pole of the lead there is
positive deflection
• When the wave of depolarisation spreads
away from the positive pole of the lead there
is negative deflection
12-Lead ECG Strip
Reading 12-Lead ECGs
The best way to read 12-lead ECGs is to develop a step-by-step
approach
1. Calculate RATE
2. Determine RHYTHM
3. Determine QRS AXIS
4. Calculate DURATION AND INTERVALS
5. Assess for HYPERTROPHY
6. Look for evidence of INFARCTION OR ISCHEMIA
Heart rate
Pacemakers of the Heart
• SA Node - Dominant pacemaker with an intrinsic
rate of 60 - 100 beats/minute.
• AV Node - Back-up pacemaker with an intrinsic
rate of 40 - 60 beats/minute.
• Ventricular cells - Back-up pacemaker with an
intrinsic rate of 20 - 45 bpm.
QUICK WAY

R wave
Heart rate calculation

• Calculate no. of large or small boxes between R-R


interval
Heart rate = no of large boxes / 300
= no of small boxes / 1500.
Heart rate calculation ( cont.)
6 sec (30 large boxes)

– Count the no of R waves in a 6 second( 30 large boxes)


rhythm strip, then multiply by 10.
or
– Count the no of R waves in a 10 second rhythm strip, then
multiply by 6 .
Normal Sinus Rhythm (NSR)

• Etiology: the electrical impulse is formed in


the SA node and conducted normally.
• For rhythm check the rhythms strip ( lead ll )
Determine regularity
R R

• Look at the R-R distances (using a caliper or markings


on a pen or paper).
• Regular (are they equidistant apart)? Occasionally
irregular? Regularly irregular? Irregularly irregular?
Interpretation?
Regular
Assess the P waves

• Are there P waves?


• Do the P waves all look alike?
• Do the P waves occur at a regular rate?
• Is there one P wave before each QRS?
Interpretation?
Normally 1 P wave precedes every QRS
AXIS
Axis refers to the mean QRS axis (or vector) during
ventricular depolarization.
The direction of current flows leftward and downward
because most of the ventricular mass is in the left
ventricle
Position of leads
Limb leads
I = +0o
II = +60o
III = +120o

Augmented
leads
avL = -30o
avF = +90o
avR = -150o
Position of leads wrt Body

LEFT
RIGHT ARM
ARM

RIGHT LEFT LEG


LEG
Axis
• The normal QRS axis lies
between -30o and +100o

QRS Complexes
I II Axis
+ + normal

+ - left axis deviation


• A quick way to determine right axis deviation
- +
the
QRS axis is to look at the - - right superior
QRS complexes in leads I axis deviation
DURATION AND INTERVALS
P wave.
• Depolarization of both atria
• WIDTH = 0.12 seconds ( 3 small boxes)
• Amplitude = 2.5 mm.

• Right atrial enlargement ----- tall p wave


• Left atrial enlargement ------- wide and notched
Right atrial enlargement
– Take a look at this ECG. What do you notice about the P waves?

The P waves are tall, especially in leads II, III and avF.
Left atrial enlargement
– Take a look at this ECG. What do you notice about the P waves?

Notched

Negative deflection

The P waves in lead II are notched and in lead V1


they have a deep and wide negative component.
PR interval
• PR interval: from onset of P wave to onset of QRS

• Normal duration = 0.12-2.0 sec (120-200 ms)


(3-5 small boxes)
• Represents atria to ventricular conduction time
(through His bundle)
• Allows time for ventriculars to fill.
First–degree AV node block
• is when conduction through AV node > 0.2 sec ( 5
small boxes)
– Causes long P-R interval
Second-degree AV node block
Mobitz’s type 1 ( wenckebach) progressive prolongation of pr
interval and then a drop

Mobitz’s type 2 prolonged pr interval but remains constant and


then drop
Third-degree or complete AV node block
• no atrial activity passes to ventricles
– Ventricles driven slowly by bundle of His or
Purkinjes
QRS complex:
• Represents ventricular depolarization
• Normal duration = LESS THAN 100 MSECS
• WIDE --- Ventricular rhythm , bundle
branch block, ventricular
tachycardia ,ventricular fibrillation
• Tall --- ventricular hypertrophy.
Right ventricular hypertrophy
– Take a look at this ECG. What do you notice about the axis and QRS
complexes over the right ventricle (V1, V2)?

There is right axis deviation (negative in I, positive in II) and


there are tall R waves in V1, V2.
Left ventricular hypertrophy
Take a look at this ECG. What do you notice about the axis and QRS
complexes over the left ventricle (V5, V6) and right ventricle (V1, V2)?

There is left axis deviation (positive in I, negative in II) and there are
tall R waves in V5, V6 and deep S waves in V1, V2.
Bundle branch blocks.

Right axis deviation Left axis deviation


Wide QRS complex Wide QRS complex
ST segment and T wave:

• Represents repolarization or recovery of ventricles


• ST segment is isoelectric
• Ischemia • Infarction

• ST depression • ST elevation .

Which Wall = which leads

Anterior wall Lateral wall


v1 v2 v3 v4 l avL V5 V6

Inferior wall
ll lll avf
OTHER MI
• High lateral wall • Posterior wall MI
ONLY IN l and avL Opposite changes seen
• Extensive anterior wall in v1 v2 .
MI Tall R wave ,
V1 – V6 ST depression
• Anterior septal T wave inversion.
V1- V2
• Strict Anterior wall
V3-V4
Different ST Depression
Stages of Infarction

SIGNS OF
INFARCTION

•ST elevation
•T wave
inversion
•Deep and
wide q wave
Anterior wall MI

• ST elevation in v1 v2 v3 v4
• Reciprocal changes ( ST depression in ll lll avf )
Lateral wall MI

• ST elevation in l avL
• Reciprocal changes ( ST depression in ll lll avf v1 v2 v3 )
Inferior wall MI

• ST elevation in ll lll avf


• Reciprocal changes ( ST depression inv1 v2 avL )
QT Interval
• Measured from beginning of QRS to
the end of the T wave
• Normal QT is usually about 0.44 sec
• QT interval varies based on heart
rate
• The faster the heart beats, the faster
the ventricles repolarize so the shorter
the QT interval. Therefore what is a
“normal” QT varies with the heart rate.
For each heart rate you need to
calculate an adjusted QT interval,
called the “corrected QT” (QTc):
• QTc = QT / square root of RR
interval
Arrhythmia Formation
Atrial cells
SA Node
Premature Atrial
Sinus Bradycardia Contractions (PACs)
Sinus Tachycardia Atrial Flutter
Atrial fibrillation

Ventricular cause AV Junctional cause


Premature Ventricular Paroxysmal Supraventricular
Contractions (PVCs) Tachycardia
Ventricular Fibrillation AV Junctional rhythm
Ventricular Tachycardia
Atrial fibrillation

Atrial Flutter
Atrial Premature Contraction

Ventricular Premature Contraction


Junctional Rhythm

Paroxysmal Supraventricular tachycardia

You might also like