Cataract

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CATARACT

DEFINITION
• A cataract is a clouding or opacity that develops in the
crystalline lens of the eye or in its envelope, varying in
degree from slight to capacity and obstructing the passage
of light.
or
• It is a clouding or opaqueness of the crystalline lens which
leads gradual painless blurring and eventual loss of vision

• The term cataract is derived from the Greek word


cataractos, which describes rapidly running water or
falling water.
Epidemiology
1. Cataracts remain the
leading cause of blindness.
2. Age-related cataract
is responsible for 48%
of world blindness,
which represents
about 18 million
people.
3. Cataracts are also an
important cause of low
vision in both developed
and developing countries.
Causes of cataract
• Old age (commonest)>65 Year • Congenital
• Ocular & systemic diseases – Part of a syndrome
– DM – Abnormal galactose
– Uveitis metabolism
–Previous ocular surgery – Hypoglycemia
• Systemic medication • Inherited abnormality
– Steroids – Myotonic
– Phenothiazines dystrophy
• Trauma & intraocular foreign – Marfan’s syndrom
bodies – Rubella
• Ionizing radiation – High myopia
– X-ray
– UV

8
Types of cataracts
The lens is composed of layers, like an onion. The outermost is the
capsule. The layer inside the capsule is the cortex, and the innermost
layer is the nucleus. A cataract may develop in any of these areas.
Cataracts are named for their location in the lens:

A nuclear cataract is located in the center of the lens. The nucleus


tends to darken with age, changing from clear to yellow and
sometimes brown.

A cortical cataract affects the layer of the lens surrounding the


nucleus. The cataract looks like a wedge or a spoke.

A posterior capsular cataract is found in the back outer layer of the


lens. This type often develops more rapidly.
PATHOPHYSIOLOGY
Any physical or chemical cause

Disturbs the intracellular and extracellular equilbrium of water and
electrolytes

Deranges the colloid system in lens fibres

Aberrant fibres are formed from germinal epithelium of lens

Epithelial cell necrosis

Focal
opacification of lens epithelium
(glau
comflecken)


Opa
Opacification of lens takeplace by 3 biochemical changes.

1. Hydration 2.Denaturation of 3.Slow


lens protein
sclerosis
c c
c

Abnormalities of lens proteins & Disorganisation of lens fibres

c
Loss of transparency of lens
c

Cataract
Cataract
Divided to :
• Acquired cataract
Age – related cataract
Metabolic cataract
Radiation or electric cataract
Traumatic cataract
Toxic cataract
Secondary cataract
AGE OF ONSET:

1. CONGENITAL
2. INFANTILE
3. JUVINILE
4. PRE-SENILE
5. SENILE
CONGENITAL CATARACT
INFANTILE AND JUVINILE CATARACT
Age -related cataract
It is the Most commonly occurred.
Classified according to:
Morphological Classification
•Capsular cataract
•Sub capsular cataract
•Cortical cataract
•Supra nuclear cataract
•Nuclear cataract
•Polar cataract
Nuclear cataract
• Most common type
• Age-related
• Occur in the center of the
lens.
• It involves the nucleus of
the crystalline lens. The
nucleus becomes diffusely
cloudy and obstructs the
light rays.
Cortical cataract
• Occur on the outer edge of the lens (cortex).
• Begins as whitish, wedge-shaped opacities.
• The lens fibers of the cortex are mainly affected. There is
hydration due to accumulation of water droplets in between the
fibers and the protein are first denaturated and then are
coagulated forming opacity.
CAPSULAR CATARACT
• It involves the capsule and may be anterior
capsule or posterior capsule.

Subcapsular cataract

•It involves superficial part of the cortex(just below the


capsule) and includes anterior sub capsule or posterior
sub capsule.
MATURITY:
1. Immature catarct
2. Mature cataract
3. Hypermature cataract
MATURE AND IMMATURE CATARACT
Mature Cataract
• Lens is completely opaque.
• Vision reduced to just perception of light
• Iris shadow is not seen
• Lens appears pearly white

Right eye mature cataract, with obvious white


opacity at the centre of pupil
IMMATURE CATARACT
Hypermature Cataract
• Shrunken and wrinkled anterior capsule due to leakage of water
out of the lense.

•This may take any of two forms:


1.Liquefactive/Morgagnian Type
2.Sclerotic Cataract
Liquefactive/Morgagnian Type
• Cortex undergoes auto-lytic liquefaction and turns uniformly
milky white.
• The nucleus loses support and settles to the bottom.
Sclerotic Cataract

• The fluid from the cortex gets absorbed


and the lens becomes shrunken.
• There may be deposition of calcific
material on the lens capsule.
• Iridodonesis: Anterior chamber
deepens and iris becomes tremulous.
• The zonules become weak, increasing
the risk of subluxation / dislocation
of lens.
SUBJECTIVE CLASSIFICATION:
• Grade 0: clear lens
• Grade 1: swollen fibres and sub capsular
opacities
• Grade 2: nuclear cataract and visible lens
fibres
• Grade 3: strong nuclear cataract with
perinuclear area opacity
• Grade 4: total opacity
Subjective classification
Clinical Manifestations
• Gradual painless • Photophobia(light
burning sensitivity)
• Loss of vision due to
lens opacity • Blurred or
• Increased glare in distorted images
bright light • Light scattering
• Decreased color • Leukokoria or
perception
• Decreased visual white pupil
acuity • Reduced light
• Poor vision at transmission
night • Contrast sensitivity is also
lost
Blurred vision due to scattering of light on the
retina
Glared view(trouble driving at night)
Change in colour vision(dimness)
1. H isto ry co llectio n
2. Visual acuity test
3. Dilated eye exam
4. Tonometry
Cataracts are diagnosed through a comprehensive eye examination
This examination may include:

Patient history to determine if vision difficulties are limiting daily activities and
other general health concerns affecting vision.

Visual acuity measurement to determine to what extent a cataract may be limiting


clear distance and near vision.

Refraction to determine the need for changes in an eyeglass or contact lens


prescription.

Evaluation of the lens under high magnification and illumination to determine the
extent and location of any cataracts.

Evaluation of the retina of the eye through a dilated pupil.

Measurement of pressure within the eye.

Supplemental testing for color vision and glare sensitivity.


Treatment
• Glasses: Cataract alters the refractive power of the natural lens
so glasses may allow good vision to be maintained.
• Surgical removal: when visual acuity can't be improved with
glasses.

• Surgical techniques

– Phacoemulsification method.

– Extracapsular cataract extraction.

– Intra capsular cataract extraction.

– Intraocular lens implantation


.

Phacoemulsification in cataract surgery involves


insertion of a tiny, hollowed tip that uses high frequency
(ultrasonic) vibrations to "break up" the eye's cloudy lens
(cataract). The same tip is used to suction out the lens
Intra-capsular Cataract Extraction
Intracapsular Cataract Extraction. From the late
1800s until the 1970s, the technique of choice for
cataract extraction was intracapsular cataract extraction
(ICCE).The entire lens (ie, nucleus, cortex,
and capsule) is removed, and fine sutures close the
incision .

ICCE is infrequently performed today; however, it is


indicated when there is a need to remove the entire lens,
such as with a subluxated cataract (ie, partially or
completely dislocated lens).
Extra-capsular Cataract Extraction
(ECCE)
Extracapsular Surgery. Extracapsular
cataract extraction (ECCE) achieves the
intactness of smaller incisional wounds (less
trauma to the eye) and maintenance of the
posterior capsule of the lens, reducing
postoperative complications, particularly
aphakic retinal detachment and cystoid macular
edema.
Postoperative care after cataract
surgery
• Steroid drops(inflammation)

• Antibiotic drops (infection)

Avoid
• Very strenuous exertion (rise
the pressure in the eyeball)
• Ocular trauma.
Complications of cataract surgery
• Infective endophthalmitis
– Rare but can cause permanent severe reduction of vision.
– Most cases within two weeks of surgery.
– Typically patients present with a short history of a
reduction in their vision and a red painful eye.
– This is an ophthalmic emergency.
– Low grade infection with pathogen such as
Propionibacterium species can lead patients to present
several weeks after initial surgery with a refractory uveitis

• Suprachoroidal haemorrhage.
– Severe intraoperative bleeding can
lead to serious and permanent
reduction in vision.
• Uveitis

• Ocular perforation.

• Postoperative refractive error

• Posterior capsular rupture and

• vitreous loss
Nursing diagnosis
• Disturbed Sensory Perception related to opacity of lens.

• Anxiety related to lack of knowledge about post operative


care.

• Risk for infection related to surgical incision and self care


after surgery.

• Risk for injury related to blurred vision or sensory deficit while


operated eye is patched.

• Acute pain related to trauma to the incision and increased IOP

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