Advanced Tooth Wear/ Non Bacterial Loss of Tooth Substance

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ADVANCED TOOTH WEAR/

NON BACTERIAL LOSS OF TOOTH


SUBSTANCE
ADVANCED TOOTH WEAR/
NON BACTERIAL LOSS OF TOOTH SUBSTANCE/
TOOTH SURFACE LOSS

• Chiefly involves
ATTRITION
ABRASION
EROSION
and also abfraction
Inter related with one aspect predominating
PREVALENCE
• One third of pre-school children (wear)
• One half of teen agers (erosion)
• 7 % of adults exhibit (wear)
• Evidence showing tooth wear due to erosion is
increasing in child and young adult population.
DIFFERENCE BETWEEN
TOOTH WEAR AND DENTAL CARIES
• ATTRITION
Marked in Egyptians( 3000 BC) due to sand content n
their diet.
Mechanical wear tooth caused by movement of one
tooth against other as a result of functional
(which surfaces of teeth would be involved?)
or
• Para functional movements of mandible caused by
Bruxism
stress
marked malalignment or malocclusion
loss of posterior teeth
dust/grit mixed with saliva (occupational / diet)-
combination of attrition and abrasion
CLINICAL
APPEARANCE

• Sharp well-defined
interdigitating tooth
wear lesions on the
incisal edges of
anterior teeth that
meet in occlusion.
CLINICAL APPEARANCE

in posterior wear facets


where complete loss of surface enamel dentine is exposed and
stained heavily
SEVERE CASE
Teeth worn down to gingivae level
• ABRASION
Loss resulting from direct frictional forces b/w
teeth and external objects. Caused by
• Abrasive tooth pastes, powders
• Improper brushing tech. or use of hard tooth
brushes
• Habits as holding pipe, pins, biting thread,
chewing tobacco and hard objects as pen
pencils
CLINICAL
APPEARANCE

Dish or saucer shaped


lesions on facial
surfaces.
Usually involving
cervical margins
areas most commonly
affected labial and
buccal surfaces of
canines and premolars
Worn, shiny
polished
yellow brown
areas at the
cervical
margins
exposed root
surfaces when
gingival recession
has exposed
cementum and
subsequently
dentine
CLINICAL
APPEARANCE
May be seen as
worn notch at incisal
surfaces
related to
para functions,
habits, occupation
mainly affecting
incisal edges of
anterior teeth
Habits as
pipe smoking
pencil chewing
nail biting
Intra oral ornaments
across teeth
Occupation related
seamstresses- pins
carpenters- nails
electricians- wires
beauticians- hair pins
• EROSION
Irreversible loss of dental hard tissues by a
chemical process (acid attack) that does not
involve bacteria.

Described as early as 1892 among Sicilian


lemon pickers
MULTIFACTORIAL
ETIOLOGY SUSCEPTIBLE
TOOTH
Acid resistance

HOST
Saliva
TIME Anatomy
EROSION Reflux
Medical

EXRINSIC
Diet
Lifestyle
Medication
Environment
EXTRINSIC SOURCES
 DIETARY EROSION
Crescent or dished defects commonly on labial
surfaces of usually upper anterior teeth.
Caused by excess of food & drink with low pH
• Citrus fruits
• Pickles, foods /drinks containing vinegar
• Carbonated FIZZY drinks, sparkling water,
energy drinks
• ALCOHOL
• Medication-
CLINICAL
PRESENTATION

wide shallow lesion


affecting the labial
surfaces of anterior
teeth

or

cupped or scooped
lesion on incisal
edges or cusp tips
• ACIDS INVOLVED
chiefly citric acid, found in most fruit juices and
soft drinks.
carbonic , acetic acid, hydrochloric, phosphoric
acids found in many fizzy drinks and colas.
• EROSIVE EFFECT
due to low chemical pH or high titratable acidity
or by chelation
TOTAL TITRATABLE
ACIDITY
measure of amount of
alkali that needs to be
added to an acid to
neutralize it
inside mouth- how long
it takes for the saliva to
buffer it
EXTRINSIC SOURCES – HABITS & LIFESTYLES

Change in attitudes
• 3 meals / day to grazing and snacking
• Glass bottles to plastic bottles and cans
• Drinking to swishing and frothing of drinks
• Bed time milk to fruit-based drinks
• Water to energy drinks (dry mouth combined with
dehydration from vigorous exercise)
• Habitual sucking of fruits
• Use of drugs - ecstasy
EXTRINSIC SOURCES –
ENVIRONMENTAL/ INDUSTRIAL/
OCCUPATIONAL EROSION
• Industrial processes using acids
fertilizers, dynamite, battery, chemicals and paint
factories, crystal glass manufacturers
• Occupational requirement
competitive swimmers
wine tasters
athletes
 INDUSTRIAL EROSION
Pitting defects on labial surfaces of upper anterior teeth
INTRINSIC SOURCES

Commonly affecting palatal surfaces of anterior and


occ. & buccal surfaces of lower post. teeth.

Caused by
 REFLUX (GORD)
 VOMIT ( SPONTANEOUS OR SELF INDUCED)
Obesity
INCREASED pregnancy
GASTRIC
PRESSURE ascites
Posture including
exercise

Heavy spicy, fatty


PRINCIPAL INCRREASED late night meals
GASTRIC
CAUSES OF VOLUME Obstruction
GORD and/or spasm
Hiatus hernia
Drugs;
SPHINCTER
diazepam
INCOMPETENCE Neuromuscular
; cerebral palsy
Oesophagitis;
alcohol
METABOLIC Uraemia
ENDOCRINE Diabetes
HORMONAL Pregnancy-morning sickness

Peptic ulcer, gastritis


GASTROINTESTINAL Obstruction
DISORDERS Nervous system disorders
Cerebral palsy
PRINCIPAL
CAUSES OF Primary – cytotoxic
VOMITING Secondary- gastric irritation by
DRUG INDUCED Alcohol, aspirin, NSAIDS
Long term drug induced xerostomia

Eating disorders-
bulimia/ anorexia
nervosa
PSYCHOSOMATIC
Bizarre habits-
rumination
IDIOPATHIC EROSION ALSO CALLED
ABFRACTION ( STRESS COROSION)

Cervical wedge shaped defect caused by heavy


force in eccentric occlusion resulting in flexural
(elastic bending ) of tooth

ETIOLOGY
Occlusal forces cause the tooth to flex, causing
enamel flecks to break off inducing abrasive
lesions
CLINICAL PICTURE
wedge shaped lesions with
sharp angles found at the
cervical margins
Common in patients
PA • with poor
alignment
Can be associated
with
• Anterior open bite
• Occlusal
restorations that
change cuspal
movement
• Abnormal tongue
movement
DIAGNOSIS

• HISTORY
• CLINICAL PICTURE
ASSESS
~ NATURE OF PROBLEM
~ WEAR IS ONGOING OR NOT
then
~ FIND THE CAUSE OF PROBLEM
MANAGEMENT

• CONTROL OF AETIOLOGICAL FACTORS


• SENSITIVITY, PAIN AND CARIES CONTROL
• PERIOD TO DETERMINE IF WEAR IS PROGRESSIVE
• POSTERIOR SUPPORT ( dentures)
• INCREASE IN OVD ( over dentures, bite planes, dahls
appliance)
• DEFINITIVE REHABILITATION ( definitive dentures,
crowns)
• ATW
• TSL
• NCTSL
• NCTTL
• NBTSL
• GERD
• GORD

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