Ventricular septal defects (VSDs) occur when structures fail to grow correctly during fetal development, resulting in holes between the ventricles. There are several types of VSDs depending on location. Small VSDs may be asymptomatic, while larger defects can cause symptoms like breathing issues, failure to thrive, and heart failure. VSDs are diagnosed through physical exam, imaging like echocardiogram and chest x-ray, and evaluated based on location, size of defects, and presence of other issues. Prognosis depends on specific defect characteristics and risk of complications like arrhythmias, pulmonary hypertension, and heart failure.
Ventricular septal defects (VSDs) occur when structures fail to grow correctly during fetal development, resulting in holes between the ventricles. There are several types of VSDs depending on location. Small VSDs may be asymptomatic, while larger defects can cause symptoms like breathing issues, failure to thrive, and heart failure. VSDs are diagnosed through physical exam, imaging like echocardiogram and chest x-ray, and evaluated based on location, size of defects, and presence of other issues. Prognosis depends on specific defect characteristics and risk of complications like arrhythmias, pulmonary hypertension, and heart failure.
Ventricular septal defects (VSDs) occur when structures fail to grow correctly during fetal development, resulting in holes between the ventricles. There are several types of VSDs depending on location. Small VSDs may be asymptomatic, while larger defects can cause symptoms like breathing issues, failure to thrive, and heart failure. VSDs are diagnosed through physical exam, imaging like echocardiogram and chest x-ray, and evaluated based on location, size of defects, and presence of other issues. Prognosis depends on specific defect characteristics and risk of complications like arrhythmias, pulmonary hypertension, and heart failure.
Ventricular septal defects (VSDs) occur when structures fail to grow correctly during fetal development, resulting in holes between the ventricles. There are several types of VSDs depending on location. Small VSDs may be asymptomatic, while larger defects can cause symptoms like breathing issues, failure to thrive, and heart failure. VSDs are diagnosed through physical exam, imaging like echocardiogram and chest x-ray, and evaluated based on location, size of defects, and presence of other issues. Prognosis depends on specific defect characteristics and risk of complications like arrhythmias, pulmonary hypertension, and heart failure.
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The ventricular septum form before
8th` week of fetal life and require
downward growth from endocardial cushion and the bulbar ridge separating PA &Aorta. Failure of these structures to grow correctly will result VSD. RISK FACTORS Genetic Factors , Radiation Exposure, Drugs Like Teratogens, Antenatal Effects, Chromosomal Disorders TYPES OF VSD A. PER MEMBRANOUS VSD : These are the most common type of ventricular septal defects. They involve the membranous part of ventricular septum which lies in the outflow tract of the left ventricle immediately beneath the aortic valve. They usually extend into muscular, inlet or outlet portion of ventricular septum. B. Inlet VSD : These VSD account for 5 to 8 % of all the VSD's. They are located posteriorly and inferior to perimembranous portion of ventricular septum. C. Subarterial VSD or Doubly committed VSD or Subpulmonary VSD : These VSD account for 5 to 7 % of all the VSD’s. They are located just beneath the pulmonary valve. D. Muscular VSD : These VSD account for 5 to 20 % of all the VSD’s and are further sub classified depending on their location in the muscular septum. i) Central : Located in the mid-muscular part of the ventricular septum with multiple apparent channels on right ventricular side and single defect on the left ventricular side. ii) Apical : Located in the apical part of the ventricular septum with multiple apparent channels on right ventricular side and single defect on the left ventricular side. iii) Swiss Cheese : Multiple muscular defects in the ventricular septum. CLINICAL MANIFESTATION Small VSD may be asymptomatic Pan systolic murmur Moderate breathing difficulty -slow to gain weight - Respiratory infection - Sign of LV failure - Mid systolic murmur Large VSD Hepatomegaly &oliguria Systolic murmur with thrill Failure to thrive Biventricular hypertrophy and CCF CLINICAL MANIFESTATIONS: •Feeding difficulty •Poor Weight Gain •Failure to cry •Increased sweating •Palpitation •Dyspnoea •Cyanosis DIAGNOSTIC EVALUATION History of illness Physical Examination Chest x-ray(cardiomegaly, enlarged LA& LV, PA, increased Pulmonary vascular marking, Pulmonary Edema) ECG(Extreme left axis is characteristics, biventricular hypertrophy) ECO(Chamber size and pressures) Two dimensional echo cardiogram with Doppler study Cardiac catheter o2 content, PA pressure, size and no of defects. PROGNOSIS Risk depends on the location of the defect , the number of defects, and the presence of other associated cardiac defects. Single membranous defects are associated with low mortality (<2%) multiple muscular defects can occur a higher risk. COMPLICATIONS Arrhythmias Right ventricular failure Pulmonary complication-pneumonia, enlarged LA, left lobe collapsed Residual VSD Pulmonary hypertension Pulmonary stenosis Failure to thrive