Basic Ecg Interpretation and Arrhythmia Recognition By: Ma. Victoria E. Martinez Er - RN
Basic Ecg Interpretation and Arrhythmia Recognition By: Ma. Victoria E. Martinez Er - RN
Basic Ecg Interpretation and Arrhythmia Recognition By: Ma. Victoria E. Martinez Er - RN
ARRHYTHMIA RECOGNITION
following:
• conditions in which the heart is enlarged - these conditions
can be caused by various factors, such as congenital (present at
birth) heart defects, valve disorders, high blood pressure,
congestive heart failure, or conduction disturbances.
• ischemia - decreased blood flow to the heart muscle due to
clogged or partially-clogged arteries.
• conduction disorders - a dysfunction in the heart's electrical
conduction system, which can make the heart beat too fast, too
slow, or at an uneven rate.
• electrolyte disturbances - an imbalance in the level of
electrolytes, or chemicals, in the blood, such as potassium,
magnesium, or calcium.
• pericarditis - an inflammation or infection of the sac which
surrounds the heart.
• valve disease - malfunction of one or more of the heart valves
may cause an obstruction of the blood flow within the heart.
• chest trauma - blunt trauma to the chest, such as a motorist
hitting the steering wheel in an automobile accident.
An ECG may also be performed for other reasons,
including, but not limited to, the following:
• The heart's conducting system consists of the sinoatrial node (SA node), atrioventicular
node (AV node), the bundle of His, the bundle branches and the Purkinje fibers.
• The electrical impulse that causes rhythmic contraction of heart muscles arises in the SA node
which is the intrinsic pacemaker of the heart. From the SA node, the impulse spreads over the atrial
muscles causing atrial contraction. The impulse is also conducted to the atrioventicular (AV) node.
• From the AV node the electrical impulse is conducted to ventricular muscles via the bundle of His,
the bundle branches and the Purkinje fibers. The bundle branches and the Purkinje fibers are
collectively called the ventricular conduction system.
EKG Leads
• EKG Standard Leads
• There are three of these leads
which are usually designated as I, II
and III.
• P Wave
• P waves are caused by atrial depolarization. In normal sinus rhythm, the
SA node acts as the pacemaker. The electrical impulse from the SA node
spreads over the right and left atria to cause atrial depolarization. The P
wave contour is usually smooth, entirely positive and of uniform size.
The P wave duration is normally less than 0.12 sec and the amplitude is
normally less than 0.25 mV. A negative P-wave can indicate
depolarization arising from the AV node.
• Note that the P wave corresponds to electrical impulses not mechanical
atria contraction. Atrial contraction begins at about the middle of the P
wave and continues during the PR segment.
– The PR Segment
– PR segment is the portion on the EKG wave from the end of the P
wave to the beginning of the QRS complex, lasting about 0.1
seconds. The PR segment corresponds to the time between the end
of atrial depolarization to the onset of ventricular depolarization.
The PR segment is an isoelectric segment, that is, no wave or
deflection is recorded. During the PR segment, the impulse travels
from the AV node through the conducting tissue (bundle branches,
and Purkinje fibers) towards the ventricles. (Note a wave will be
recorded only after the impulses exit the conducting systems and
activates the ventricular muscle to give the QRS complex). Most of
the delay in the PR segment occurs in the AV node.
– PR interval 0.12 ~ 0.20 sec
• The QRS Complex
– In normal sinus rhythm, each P wave is followed by a QRS complex. The QRS
complex represents the time it takes for depolarization of the ventricles.
Activation of the anterioseptal region of the ventricular myocardium
corresponds to the negative Q wave. The Q wave is not always present.
Activation of the rest of the ventricular muscle from the endocardial surface
corresponds to the rest of the QRS wave. The R wave is the point when half of
the ventricular myocardium has been depolarized. Activation of the
posteriobasal portion of the ventricles give the RS line. The normal QRS
duration range is from 0.04 sec to 0.12 sec measured from the initial deflection of
the QRS from the isoelectric line to the end of the QRS complex.
– Normal ventricular depolarization requires normal function of the right and left
bundle branches. A block in either the right or left bundle branch delays
depolarization of the ventricles, resulting in a prolonged QRS duration.
–
• The EKG on the bottom right shows irregularly spaced RR intervals. If the
distances are irregular, count the number of QRS complexes within 30 large
boxes (which each represent 0.2 seconds) and multiply this number by 10 to
obtain the heart rate in beats/minute.
•
• Method II
• If the peaks are regular, the heart rate can be estimated using the EKG
grid. To do this locate a QRS complex on a bold line. If the next QRS
complex is separated by:
• i. One large box, the heart rate is 300 BPM (300/1)
• ii. Two large boxes, the heart rate is 150 BPM (300/2)
• iii. Three large boxes, the heart rate is 100 BPM (300/3)
• iv. Four large boxes, the heart rate is 75 BPM (300/4)
Arrhythmias
• The term "arrhythmia" refers to any change from
the normal sequence of electrical impulses. The
electrical impulses may happen too fast, too
slowly, or erratically – causing the heart to beat
too fast, too slowly, or erratically.
• When the heart doesn’t beat properly, it can’t
pump blood effectively. When the heart doesn’t
pump blood effectively, the lungs, brain and all
other organs can’t work properly and may shut
down or be damaged.
Types of Arrhythmias
• Ventricular fibrillation (v-fib for short) is the most serious cardiac rhythm disturbance.
The lower chambers quiver and the heart can't pump any blood, causing cardiac arrest.
• How it works
The heart's electrical activity becomes disordered. When this happens, the heart's lower
(pumping) chambers contract in a rapid, unsynchronized way. (The ventricles "flutter"
rather than beat.) The heart pumps little or no blood. Collapse and sudden cardiac
arrest follows -- this is a medical emergency !
Premature Ventricular Contraction
are extra, abnormal heartbeats that begin in one of your heart's
two lower pumping chambers (ventricles). These extra beats
disrupt your regular heart rhythm, sometimes causing you to feel
a flip-flop or skipped beat in your chest. Premature ventricular
contractions are very common — they occur in most people at
some point.
• Premature ventricular contractions are also called:
• Premature ventricular complexes
• PVCs
• Ventricular premature beats
• Ectopic heartbeats
• Extrasystoles
• If you have occasional extra beats, but you are an otherwise
healthy person, there's generally no reason for concern, and no
treatment is needed. If you have frequent symptoms or you have
underlying heart disease, you may need treatment to help you
feel better and treat underlying heart problems.
• Rate = variableP waveusually obscured by the QRS,
PST or T wave of the PVC
QRS = wide > 0.12 seconds; morphology
is bizarre with the ST segment and the T wave
opposite in polarity. May be multifocal and exhibit
different morphologies.
Conduction = the impulse originates below the
branching portion of the Bundle of His; full
compensatory pause is characteristic.
Rhythm = irregular. PVC's may occur in singles,
couplets or triplets; or in bigeminy, trigeminy or
quadrigeminy
PVCs can occur in healthy hearts. For example, an increase in
circulating catecholamines can cause PVCs. They also occur in
diseased hearts and from drug (such as digitalis) toxicities.
Interventions include:
lidocaine,
pronestyl, or
quinidine.
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