Eating Disorders

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EATING DISORDERS

A. CRITERIA FOR ANOREXIA NERVOSA


According to the latest Diagnostic and Statistical Manual of
Mental Disorders Fourth Edition (DSM-IV) criteria:
Refusal to maintain body at or above a minimally normal weight
for age and height.
Intense fear of gaining weight
The central concern of weight and shape in the evaluation of the
self, in addition to a reference to the denial of the serious
consequence of weight loss.
Amenorrhea.
SUBTYPES:
The restrictor type
The binge purger type

B. CRITERIA FOR BULIMIA NERVOSA


Diagnostic criteria for Bulimia Nervosa:
Recurrent episodes of binge eating .
Recurrent inappropriate compensatory behavior to prevent
weight gain.
The binge eating and inappropriate compensatory behaviors
both occur, on average, at least twice a week for 3 months.
Self evaluation is unduly influenced by body shape and weight.
The disturbance does not occur exclusively during episodes of
anorexia nervosa.
SUBTYPES:
Purging
Non purging

C. ORAL MANIFESTATIONS

Severe erosion of the enamel on the lingual


surfaces of the maxillary teeth - cardinal
oral manifestation of eating disorders, due
to acids from chronic vomiting.
Parotid enlargement - as a result of
starvation
Mandibular teeth may be affected.
Palatal ulcerations

D. Management

Refer to other practitioners.


Support of the patient physically, by
treatment of tooth desensitization
and esthetics.
Support of the patient
psychologically, by demonstrating a
caring and compassionate attitude.

PLASMA CELL NEOPLASIA


I. DEFINITION

Is a group of related malignant disorders of terminally


differentiated B lymphocytes (plasma cells), of which plasma cell
myeloma or multiple myeloma is the most common (90% of cases).

This condition is characterized by bone marrow multifocal


infiltration by malignant plasma cells. There are typically multiple
destructive lesions or diffuse demineralization of bone. The tumour
cells secrete a homogenous, complete or partial, immunoglobulin
molecule, an M component or para protein, most commonly IgG or
IgA.
II. ETIOLOGY
Genetic predisposition
Ionizing radiation
Chronic antigenic

III. ORAL MANIFESTATION


Maxillary or mandibular lesions
Root resorption and loss of lamina
dura may be found.
Amyloid like deposition in the gingiva
and tongue.

IV. DENTAL MANAGEMENT


Patients are prone to postoperative hemorrhage
due to thrombocytopenia.
Development of dental amyloidosis.
Dental hygiene care should focus on controlling
infections associated with a compromised
immune system.
Patients being treated with bisphophonates must
be monitored closely for the development of
osteonecrosis of the jaw.
Educate patient about the necessity of reporting
unusual sores or painful areas in the mouth.

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