Abdominal Trauma
Abdominal Trauma
Abdominal Trauma
nursing diagnoses and expected outcomes associated with patients with abdominal trauma.
INTRODUCTION
Epidemiology : Abdominal injuries rank third as a cause of traumatic death preceded by head and chest injuries.
The abdomen is vulnerable to injury since there is minimal bony protection for underlying organs. The retropentoneal location of certain organs and vascular structures (e.g., vena cava, aorta, pancreas, and duodenum) these structures are less frequently injured. The physical examination of the abdomen may not be successful in identifying intra-abdominal pathology; therefore, a description of the mechanism of injury is important.
The most common mechanism of blunt abdominal injury is a motor vehicle crash.Firearms, stabbings, and physical assaults are associated with penetrating abdominal trauma. Injuries to the abdomen can result from acceleration, deceleration, or a combination of both forces. Crushing forces may compress the duodenum or the pancreas against the vertebral column.
During energy transfer, abdominal structures attached by either ligaments or blood vessels may be stressed at their attachment points
Safety restraint devices, particularly three-point safety belts, provide significant protection; however, if they are improperly positioned, they can cause deceleration injuries to the lower abdomen.
Types of Injuries
Blunt or penetrating abdominal injuries are related to the: Type of force applied. Tissue density of structure injured (e.g.. fluid-filled, gasfilled, solid, or encapsulated)
The liver and spleen are the most commonly injured organs from blunt trauma.
Organs of the abdomen are vulnerable to penetrating injury not only through the anterior abdominal ,but through the back, flank area, and lower chest. The liver, small bowel, and stomach are the most commonly injured organs from penetrating trauma.
Because of their anatomical location, fractures of the lower rib cage are often associated with spleen or liver injuries
The patient with abdominal trauma, particularly esophageal and gastric injuries, may have associated chest trauma.
Patients with pelvic fractures frequently have associated intra-abdominal trauma(bladder laceration).
Blood Loss
Injuries to organs or abdominal blood vessels may lead to extensive hemorrhage Some abdominal organs are semi-fixed by ligaments, such as the mesenteric attachments of the intestines. When these organs are stressed at their points of attachment, tears often occur at the point where the vessels enter the organ.
The spleen and the liver have a rich blood supply and store blood. Rapid loss of large blood volumes from their parenchymal or vascular structures can occur.
Blood Loss
Bleeding from organs in the anterior abdomen is usually confined to that cavity. Bleeding from structures in the retro peritoneum lead to hemorrhage in retroperitoneum which is more difficult to evaluate and diagnose .
Pain
Pain, rigidity, guarding, or spasm, of the abdominal musculature are classic signs of intraabdominal pathology. Rebound tenderness and guarding of the abdominal muscles are caused by sudden movement of irritated peritoneal membranes against the abdominal wall.
Pain
Irritation may be because of the presence of free blood or gastric contents in the peritoneal cavity. Manifestations of pancreatic and duodenal injury are related to hemorrhage in the area and the effect of active enzymes on their surrounding tissues. The resultant chemical peritonitis from the enzymes released into the retroperitoneum and the significant tissue swelling may not appear as signs and symptoms for several hours after injury.
Pain
Pain can be referred to other areas of the body. An example is the referred shoulder pain known as Kehrs associated with splenic rupture. The blood that collects under the diaphragm causes irritation of the phrenic nerve which innervates the diaphragm.
Peristalsis
Following abdominal injury, bowel sounds are frequently hypodynamic. Blood in the abdominal cavity, direct bowel injury, or any number of conditions including stress may decrease peristaltic activity.
Hepatic Injuries
Injury to the liver range from controlled subcapsular hematoma and laceration of the parenchyma to severe vascular injury of the hepatic veins. The friability of liver tissue ,the extensive blood supply ,and the blood storage capacity cause hepatic injury to result in profuse hemorrhage. The trend in blunt hepatic trauma is nonoperative management of the hemodynamically stable patient.
Hepatic Injuries
The traditional treatment of liver trauma was exploration and surgical packing but the nontherapeutic laparotomy rate was as high as 67%, largely because most liver injury hemorrhage resolves spontaneously before laparotomy can be performed.
Hepatic Injuries
SIGNS AND SYMPTOMS: Upper right quadrant pain Abdominal wall muscle rigidity, spasm. or involuntary guarding Rebound tenderness Hypoactive or absent bowel sounds Signs of hemorrhage and/or hypovolemic shock
Splenic Injuries
Injury to the spleen is usually associated with blunt trauma, but may also be associated with penetrating trauma. Fractures of the left 10th to 12th ribs are associated with underlying damage to the spleen.
Splenic Injuries
Injury to the spleen usually associated with blunt trauma but may also be associated with penetrating trauma Fractures to 10th to 12th ribs are associated with underlying damage to the spleen Injury to the spleen range from laceration to the capsule or non expanding hematoma to ruptured subcapsular hematoma or parenchymal laceration.
ARTICLES
The American Association for the Surgery of Trauma Organ Injury Severity Scale Liver grading system is as follows:
Grade I - Capsular avulsion; periportal blood tracking; superficial laceration less than 1-cm deep; subcapsular hematoma less than 1-cm thickness Grade II - Laceration 1- to 3-cm deep; subcapsular/central hematoma 1- to 3-cm diameter
Grade III - Laceration greater than 3-cm deep; subcapsular/central hematoma greater than 3-cm diameter
Grade IV - Massive central or subcapsular hematoma greater than 10 cm; lobar tissue maceration or devascularization
ARTICLES
The grade of hepatic injury does not necessarily correlate with the rate of nonoperative treatment success. In grade III and IV liver injuries, a wide range of nonoperative management successes have been reported. Overall, the nonoperative success rate in patients with liver trauma has been reported to be as high as 89-98%. Meredith JW, Young JS, Bowling J, Roboussin D: Nonoperative management of blunt hepatic trauma: the exception or the rule? J Trauma 1994 Apr; 36(4): 529-34; discussion 534-5