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Patient Name : Mrs.D.

RAJAMMA Client Code : AQLAP786


Age/Gender : 63 Y 0 M 0 D /Female Sample Collection : 06/Dec/2024 08:40AM
Visit ID : AQP234839 Registration : 06/Dec/2024 09:39AM
Barcode No : 197631 Reported : 06/Dec/2024 11:21AM
Ref Doctor :SELF
Ref. Lab :HEALTH CARE DIAGNOSTIC CENTRE

DEPARTMENT OF BIOCHEMISTRY
Test Name Result Unit Bio. Ref. Range

Liver Function Test


Total Bilirubin 0.4 mg/dL <1.1
(Method: Diazonium Salt)

Conjugated (D. Bilirubin) 0.1 mg/dL 0.0-0.3


(Method: Diazo Reaction)

Unconjugated ( I.D.Bilirubin) 0.3 mg/dl 0.1-1.0


(Method: Calculated)

Alkaline Phosphatase 133 U/L 35-104


(Method: Kinetic PNPP-AMP)

Alanine Aminotransferase(ALT/SGPT) 18 U/L <34.0


(Method: IFCC without pyridoxal-5- phosphate )

Aspartate Transaminase (AST/SGOT) 13 U/L <31.0


(Method: IFCC without pyridoxal-5- phosphate )

Gamma Glutamyl Transferase(GGT) 20 U/L Upto 60


(Method: g-Glut-3-carboxy-4 nitro)

Total Protein 7.4 g/dL 6.4-8.3


(Method: Biuret)

Albumin 4.3 g/dL 3.4-5.0


(Method: Bromocresol Green (BCG))

Globulin 3.10 g/dl 2.5-3.5


(Method: Calculated)

Albumin/Globulin Ratio 1.39 Ratio 1.0-2.1


(Method: Calculated)

Page 1 of
11

This is an electronically authenticated report. Report printed date: 06-Dec-2024 06:10 PM,
Note: Assay results should be correlated clinically with other clinical findings and the total clinical status of the patient.
Patient Name : Mrs.D.RAJAMMA Client Code : AQLAP786
Age/Gender : 63 Y 0 M 0 D /Female Sample Collection : 06/Dec/2024 08:40AM
Visit ID : AQP234839 Registration : 06/Dec/2024 09:39AM
Barcode No : 197631 Reported : 06/Dec/2024 11:32AM
Ref Doctor :SELF
Ref. Lab :HEALTH CARE DIAGNOSTIC CENTRE

DEPARTMENT OF BIOCHEMISTRY
Test Name Result Unit Bio. Ref. Range

Calcium (SERUM)
Calcium 9.50 mg/dL 8.8-10.2
(Method: Spectrophotometry)

INTERPRETATION:

-Calcium level is increased in patients with hyperparathyroidism, Vitamin D intoxication, metastatic bone tumor, milk-alkali syndrome, multiple myeloma, Paget’s disease.
-Calcium level is decreased in patients with hemodialysis, hypoparathyroidism (primary, secondary), vitamin D deficiency, acute pancreatitis, diabetic Keto-acidosis, sepsis, acute myocardial
infarction (AMI), malabsorption, osteomalacia, renal failure, rickets.

Page 2 of
11

This is an electronically authenticated report. Report printed date: 06-Dec-2024 06:10 PM,
Note: Assay results should be correlated clinically with other clinical findings and the total clinical status of the patient.
Patient Name : Mrs.D.RAJAMMA Client Code : AQLAP786
Age/Gender : 63 Y 0 M 0 D /Female Sample Collection : 06/Dec/2024 08:40AM
Visit ID : AQP234839 Registration : 06/Dec/2024 09:39AM
Barcode No : 197631 Reported : 06/Dec/2024 11:21AM
Ref Doctor :SELF
Ref. Lab :HEALTH CARE DIAGNOSTIC CENTRE

DEPARTMENT OF BIOCHEMISTRY
Test Name Result Unit Bio. Ref. Range

Lipid Profile
Total Cholesterol 455 mg/dl Adults:Desirable:<200
(Method: CHOD-POD)
Borderline:200-239
High risk:>/=240
Cholesterol-HDL 24 mg/dl Major risk factor for Heart
(Method: Enzymatic Colorimetric))
disease<40,br/>Negative risk factor
for heart disease>60
Cholesterol-L D L 413 mg/dl Normal:<100
(Method: Calculated)
Above Optimal:100-129
Borderline High:130-159
High:160-189
Very High:>190
Cholesterol- V L D L 18.4 mg/dl 7-40
(Method: Calculated)

Triglycerides 92 mg/dl Normal:<150


(Method: Glycerol phosphate oxidase/peroxidase)
BorderLine:150-199
High:200-499
Very High:>=500
Total Cholesterol /HDL Ratio 18.96 Desirable: <4
(Method: Calculated)
BorderLine : 4.1-6.0
High Risk : >6.0
LDL / HDL Ratio 17.2 Ratio 0.0-3.5
(Method: Calculated)

Desirable range <100 mg/dL for patients with CHD or diabetes and <70 mg/dL for diabetic patients with known heart disease.
For patients with diabetes plus 1 major Atherosclerotic cardiovascular disease (ASCVD) risk factor, treating to a non-HDL-C goal of <100 mg/dL (LDL-C of <70 mg/dL) is considered a therapeutic option.

Page 3 of
11

This is an electronically authenticated report. Report printed date: 06-Dec-2024 06:10 PM,
Note: Assay results should be correlated clinically with other clinical findings and the total clinical status of the patient.
Patient Name : Mrs.D.RAJAMMA Client Code : AQLAP786
Age/Gender : 63 Y 0 M 0 D /Female Sample Collection : 06/Dec/2024 08:40AM
Visit ID : AQP234839 Registration : 06/Dec/2024 09:39AM
Barcode No : 197630 Reported : 06/Dec/2024 11:45AM
Ref Doctor :SELF
Ref. Lab :HEALTH CARE DIAGNOSTIC CENTRE

DEPARTMENT OF BIOCHEMISTRY
Test Name Result Unit Bio. Ref. Range

Glycosylated Hemoglobin (GHB/HbA1C) (WHOLE BLOOD EDTA)


Glycosylated Hemoglobin (GHB/HbA1C) 5.90 % <6.0:Non Diabetic
(Method: High-performance Liquid Chromatography (HPLC))
6.1-6.5:Prediabetic
6.6-7.0:Good control
7.1-8.0:Poor control
>8.1:Alert
Estimated Average Glucose 122.63 % 70-136
(Method: Calculated)

1. HbA1C has been endorsed by clinical groups and American Diabetes Association guidelines 2017 for diagnosing diabetes using a cut off point of 6.5%
2. Low glycated haemoglobin in a non diabetic individual are often associated with systemic inflammatory diseases, chronic anaemia (especially severe iron deficiency and haemolytic), chronic
renal failure and liver diseases. Clinical correlation suggested.
3. In known diabetic patients, following values can be considered as a tool for monitoring the glycemic control.
Excellent control-6-7 %
Fair to Good control – 7-8 %
Unsatisfactory control – 8 to 10 %
Poor Control – More than 10 %
Note: Source for Reference Range: American Diabetes Association Guidelines
INCREASED IN

1. Chronic renal failure with or without hemodialysis.


2. Iron deficiency anemia. Increased serum triglycerides.
3. Alcohol.
4. Salicylate treatment.

DECREASED IN

1. Shortened RBC life span (hemolytic anemia, blood loss), Pregnancy.


2. Ingestion of large amounts (>1g/day) of vitamin C or E.
3. Hemoglobinopathies (e.g.: spherocytes) produce variable increase or decrease.
4. Results of %HbA1c are not reliable in patients with chronic blood loss and consequent variable erythrocyte life span.

Page 4 of
11

This is an electronically authenticated report. Report printed date: 06-Dec-2024 06:10 PM,
Note: Assay results should be correlated clinically with other clinical findings and the total clinical status of the patient.
Patient Name : Mrs.D.RAJAMMA Client Code : AQLAP786
Age/Gender : 63 Y 0 M 0 D /Female Sample Collection : 06/Dec/2024 08:40AM
Visit ID : AQP234839 Registration : 06/Dec/2024 09:39AM
Barcode No : 197631 Reported : 06/Dec/2024 10:47AM
Ref Doctor :SELF
Ref. Lab :HEALTH CARE DIAGNOSTIC CENTRE

DEPARTMENT OF BIOCHEMISTRY
Test Name Result Unit Bio. Ref. Range

Kidney Function Test


Serum Creatinine 0.60 mg/dL 0.5-1.2
(Method: JAFFE-Kinetic)

Urea 28.0 mg/dL <50.0


(Method: Urease / GLDH)

Blood Urea NItrogen(BUN) 13.1 mg/dL 8.0-23.0


(Method: Calculated)

Bun/Creatinine Ratio 21.8 6-22


(Method: Calculated)

Sodium 139 mmol/L 135-146


(Method: ISE Direct)

Potassium 4.5 mmol/L 3.5-5.0


(Method: ISE Direct)

Chloride 104 mmol/L 95-108


(Method: ISE Direct)

Uric Acid 4.5 mg/dL 3.4-7.0


(Method: Uricase/Peroxidase)

Page 5 of
11

This is an electronically authenticated report. Report printed date: 06-Dec-2024 06:10 PM,
Note: Assay results should be correlated clinically with other clinical findings and the total clinical status of the patient.
Patient Name : Mrs.D.RAJAMMA Client Code : AQLAP786
Age/Gender : 63 Y 0 M 0 D /Female Sample Collection : 06/Dec/2024 08:40AM
Visit ID : AQP234839 Registration : 06/Dec/2024 09:39AM
Barcode No : 197631 Reported : 06/Dec/2024 11:32AM
Ref Doctor :SELF
Ref. Lab :HEALTH CARE DIAGNOSTIC CENTRE

DEPARTMENT OF BIOCHEMISTRY
Test Name Result Unit Bio. Ref. Range

PHOSPHORUS (SERUM)
Phosphrous 4.0 mg/dL 2.5-4.5
(Method: Molybdate-UV/ Endpoint Method)

Page 6 of
11

This is an electronically authenticated report. Report printed date: 06-Dec-2024 06:10 PM,
Note: Assay results should be correlated clinically with other clinical findings and the total clinical status of the patient.
Patient Name : Mrs.D.RAJAMMA Client Code : AQLAP786
Age/Gender : 63 Y 0 M 0 D /Female Sample Collection : 06/Dec/2024 08:40AM
Visit ID : AQP234839 Registration : 06/Dec/2024 09:39AM
Barcode No : 197631 Reported : 06/Dec/2024 11:32AM
Ref Doctor :SELF
Ref. Lab :HEALTH CARE DIAGNOSTIC CENTRE

DEPARTMENT OF BIOCHEMISTRY
Test Name Result Unit Bio. Ref. Range

Iron Profile-1
Iron 38 µg/dL 33-193
(Method: FerroZine-without deproteinization.)

Iron Binding Capacity - Total (TIBC) 363 µg/dL 250-450


(Method: Spectrophotometry)

Transferrin 247.4 ug/dL 176.00 - 280.00


(Method: Immunoturbidimetry)

Transferrin Saturation 10.5 % 20-50


(Method: Calculation)

INTERPRETATION:
SERUM IRON INCREASED IN:
-Hemosiderosis of excessive iron intake (e.g. repeated blood transfusion, iron therapy, iron containing vitamins).
-Decreased formation of RBCs (thalassemia, pyridoxal deficiency anaemia).
-Increased destruction of RBCs (hemolytic anaemia).
-Acute liver damage
-Acute iron toxicity
SERUM IRON DECREASED IN:
-Iron deficiency anaemia
-Normochromic anaemia of infections & chronic diseases
-Nephrosis
-Diurnal variation: Normal in mid morning, low values in mid afternoon, and very low values near midnight.
TIBC/UIBC INCREASED IN:
-Iron deficiency anemia
-Acute & Chronic blood loss
-Acute liver damage
TIBC/UIBC DECREASED IN:
-Hemochromatosis
-Cirrhosis of the liver
-Thalassemia
-Anemia of infective & chronic disease
TRANSFERRIN SATURATION INCREASED IN:
- High Values in iron overload
- Raised transferrin saturation is an early indicator of Iron accumulation in hemochromatosis.
TRANSFERRIN SATURATION DECREASED IN:
- Low Values in iron deficiency

Page 7 of
11

This is an electronically authenticated report. Report printed date: 06-Dec-2024 06:10 PM,
Note: Assay results should be correlated clinically with other clinical findings and the total clinical status of the patient.
Patient Name : Mrs.D.RAJAMMA Client Code : AQLAP786
Age/Gender : 63 Y 0 M 0 D /Female Sample Collection : 06/Dec/2024 08:40AM
Visit ID : AQP234839 Registration : 06/Dec/2024 09:39AM
Barcode No : 197630 Reported : 06/Dec/2024 11:02AM
Ref Doctor :SELF
Ref. Lab :HEALTH CARE DIAGNOSTIC CENTRE

DEPARTMENT OF HAEMATOLOGY
Test Name Result Unit Bio. Ref. Range

Hemogram
Hemoglobin(HB) 12.8 g/dl 12.0-15.0
(Method: Spectrophotometry)

Erythrocyte count (RBC COUNT) 4.7 million/cmm 3.8-4.8~3.8-4.8


(Method: Impedance )

Packed Cell Volume(Hematocrit) 39.4 % 40-50


(Method: Numeric integration)

Platelet Count 3.29 Lakh/cumm 1.50 - 4.10


(Method: Impedance/microscopy)

Red Blood Cell Indices


Mean Cell Volume (MCV) 84 fL 83-101
(Method: Automated/Calculated)

Mean Cell Haemoglobin (MCH) 27.4 pg 27-32


(Method: Automated/Calculated)

Mean Corpuscular Hb Concn. (MCHC) 32.4 g/dl 31.5-34.5


(Method: Automated/Calculated)

Red Cell Distribution Width (RDW)- CV 14.1 % 11.5-14.5


(Method: Automated/Calculated)

Total Count and Differential Count


(Method: Impedance/Microscopy)

Total Leucocytes Count(WBC) 10,200 Cells/Cumm 4000-10000


(Method: Impedance/microscopy )

Neutrophils 51 % 40-80
(Method: Impedance/microscopy )

Lymphocytes 38 % 20-40
(Method: Impedance/microscopy )

Eosinophils 02 % 01-06
(Method: Impedance/microscopy )

Monocytes 09 % 02-10
(Method: Impedance/microscopy )

Basophils 00 % 00-01
(Method: Impedance/microscopy )

Erythrocytes Sedimentation Rate-(ESR)


Erythrocytes Sedimentation Rate (ESR) 14 mm/1st hr 1-14
(Method: Westergren)

Microscopic Examination
RBC Morphology Normocytic Normochromic Cells
WBC Morphology Mild Leucocytosis
Platelet Morphology Adequate
Hemoparasites Not Found
Impression Mild Leucocytosis
Advise Correlate clinically

Page 8 of
11

This is an electronically authenticated report. Report printed date: 06-Dec-2024 06:10 PM,
Note: Assay results should be correlated clinically with other clinical findings and the total clinical status of the patient.
Patient Name : Mrs.D.RAJAMMA Client Code : AQLAP786
Age/Gender : 63 Y 0 M 0 D /Female Sample Collection : 06/Dec/2024 08:40AM
Visit ID : AQP234839 Registration : 06/Dec/2024 09:39AM
Barcode No : 197631 Reported : 06/Dec/2024 11:03AM
Ref Doctor :SELF
Ref. Lab :HEALTH CARE DIAGNOSTIC CENTRE

DEPARTMENT OF HORMONE ASSAYS


Test Name Result Unit Bio. Ref. Range

25-Hydroxy Vitamin D Total (D2 & D3) (SERUM)


25-Hydroxy Vitamin D Total (D2 & D3) 15.90 ng/ml Deficient : <20
(Method: Chemiluminescence)
Insufficient : 20 to <30
Sufficient : 30-100
Upper Safety Limit : >120
INTERPRETATION:

LEVEL REFERENCE RANGE


Deficiency (serious deficient) < 20 ng/ml
Insufficiency (Deficient) 20-30 ng/ml
Sufficient (adequate) 30-100 ng/ml
Upper Safety Limit >100 ng/ml
DECREASED LEVELS:
-Deficiency in children causes Rickets and in adults leads to Osteomalacia. It can also lead to Hypocalcemia and Tetany.
-Inadequate exposure to sunlight.
-Dietary deficiency.
-Vitamin D malabsorption.
-Severe Hepatocellular disease.
-Drugs like Anticonvulsants.
-Nephrotic syndrome.
INCREASED LEVELS:
-Vitamin D intoxication.
COMMENTS:
-Vitamin D (Cholecalciferol) promotes absorption of calcium and phosphorus and mineralization of bones and teeth. Vitamin D status is best determined by
measurement of 25 hydroxy vitamin D, as it is the major circulating form and has longer half life (2-3 weeks) than 1, 25 Dihydronxy vitamin D (5-8 hrs).
-The assay measures D3 (Cholecaciferol) metabolites of vitamin D.
-25 (OH) D is influenced by sunlight, latitude, skin pigmentation, sunscreen use and hepatic function.
-Optimal calcium absorption requires vitamin D 25 (OH) levels exceeding 75 nmol/L.
-It shows seasonal variation, with values being 40-50% lower in winter than in summer.
-Levels vary with age and are increased in pregnancy.
-This is the recommended test for evaluation of vitamin D intoxication.

Page 9 of
11

This is an electronically authenticated report. Report printed date: 06-Dec-2024 06:10 PM,
Note: Assay results should be correlated clinically with other clinical findings and the total clinical status of the patient.
Patient Name : Mrs.D.RAJAMMA Client Code : AQLAP786
Age/Gender : 63 Y 0 M 0 D /Female Sample Collection : 06/Dec/2024 08:40AM
Visit ID : AQP234839 Registration : 06/Dec/2024 09:39AM
Barcode No : 197631 Reported : 06/Dec/2024 10:29AM
Ref Doctor :SELF
Ref. Lab :HEALTH CARE DIAGNOSTIC CENTRE

DEPARTMENT OF HORMONE ASSAYS


Test Name Result Unit Bio. Ref. Range

Vitamin B12 (SERUM)


Vitamin B12 191 pg/mL Deficiency:< 145
(Method: CLIA)
Indeterminate:145 –180
Normal: 180 - 914
Vitamin B12, also known as cyanocobalamin, is a water soluble vitamin that is required for the maturation of erythrocytes and coenzyme form for more than 12 different
enzyme systems. Groupsat risk for vitamin B12 deficiency include those
(1) older than 65 years of age (2) with malabsorption (3) who are vegetarians (4) with autoimmune disorders(5) taking prescribed medication known to interfere with
vitamin absorption or metabolism, including nitrous oxide, phenytoin, dihydrofolate reductase inhibitors, metformin,
and proton pump inhibitors (6) infants with suspected metabolic disorders.
The most common cause of Vitamin B12 deficiency is pernicious anemia. Deficiency of Vitamin B12 is associated with megaloblastic anemia and neuropathy. Excess
Vitamin B12 is excreted in urine. No adverse effects have been associated with excess vitamin B12 intake from food or supplements in healthy people
COMMENTS:
Results may differ between laboratories due to variation in population and test method. Vitamin B12 is implicated in the formation of myelin, and along with Folate is
required for DNA synthesis. The most prominent source of B12 for humans is meat while untreated fresh water can also be a source.
Megaloblastic anaemia has been found to be due to B12 deficiency, a major cause being Pernicious anemia due to poor B12 uptake resulting in below normal serum levels.
Other conditions related to low B12 levels include iron deficiency anemia, pregnancy, vegetarianism, partial gastrectomy, ileal damage, oral contraceptives, parasitic
infestations, pancreatic deficiency, treated epilepsy and advancing age. The correlation of serum B12 levels and Megaloblastic anemia however is not always clear - some
patients with high MCV may have normal B12 levels, while some individuals with B12 deficiency may not have megaloblastic anemia. Disorders renal failure, liver
diseases and myeloproliferative diseases may have elevated vitamin B12 levels.
LIMITATIONS:
For diagnostic purposes, the B12 results should be used in conjunction with other data; e.g.; symptoms results of other testing, clinical impressions, etc.
If the B12 level is inconsistent with clinical evidence, additional testing is suggested to confirm the result.

Page 10 of
11

This is an electronically authenticated report. Report printed date: 06-Dec-2024 06:10 PM,
Note: Assay results should be correlated clinically with other clinical findings and the total clinical status of the patient.
Patient Name : Mrs.D.RAJAMMA Client Code : AQLAP786
Age/Gender : 63 Y 0 M 0 D /Female Sample Collection : 06/Dec/2024 08:40AM
Visit ID : AQP234839 Registration : 06/Dec/2024 09:39AM
Barcode No : 197631 Reported : 06/Dec/2024 10:29AM
Ref Doctor :SELF
Ref. Lab :HEALTH CARE DIAGNOSTIC CENTRE

DEPARTMENT OF HORMONE ASSAYS


Test Name Result Unit Bio. Ref. Range

Thyroid Profile-I (T3, T4, TSH)


Tri-Iodothyronine Total (TT3) 113.4 ng/dL 80.-200
(Method: ECLIA)

Thyroxine Total (TT4) 8.32 µg/dL 5.1-14.1


(Method: ECLIA)

Thyroid Stimulating Hormone (TSH) 5.30 µIU/mL >55years:0.50-8.90


(Method: ECLIA)

INTERPRETATION:
1. Serum T3, T4 and TSH are the measurements form three components of thyroid screening panel and are useful in diagnosing various disorders of thyroid gland
function.
2. Primary hyperthyroidism is accompanied by elevated serum T3 and T4 values along with depressed TSH levels.
3. Primary hypothyroidism is accompanied by depressed serum T3 and T4 values and elevated serum TSH levels.
4. Normal T4 levels accompanied by high T3 levels are seen in patients with T3 thyrotoxicosis. Slightly elevated T3 levels may be found in pregnancy and in estrogen
therapy while depressed levels may be encountered in severe illness, malnutrition, renal failure and during therapy with drugs like propanolol and propylthiouracil.
5. Although elevated TSH levels are nearly always indicative of primary hypothyroidism, rarely they can result from TSH secreting pituitary tumors (secondary
hyperthyroidism).
6. Low levels of Thyroid hormones (T3, T4 & FT3, FT4) are seen in cases of primary, secondary and tertiary hypothyroidism and sometimes in non-thyroidal illness
also.
7. Increased levels are found in Grave’s disease, hyperthyroidism and thyroid hormone resistance.
8. TSH levels are raised in primary hypothyroidism and are low in hyperthyroidism and secondary hypothyroidism.
REFERENCE RANGE:
PREGNANCY TSH in uIU/mL
1 st Trimester 0.60-3.40
2nd Trimester 0.37-3.60
3rd Trimester 0.38-4.04
(Reference range recommended by the American Thyroid Association)
*** End Of Report ***

Page 11 of
11

This is an electronically authenticated report. Report printed date: 06-Dec-2024 06:10 PM,
Note: Assay results should be correlated clinically with other clinical findings and the total clinical status of the patient.

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