Glaucoma-1
Glaucoma-1
Glaucoma-1
Kam
-
and4IOP is
the risk
major factor
Common blindness
cause
of reversible
cataract
->
ratio
qup-disc
4
[ID =
<0.5 (normal
- Cuppings
↓
loss axon
of
up size increases, more
whitish,
Angle ↓
↓
all into posterior
Tolle the Chamber
wer
↓
through pupil
↓
Anterior chamber
/
composition:clear, transparent 2 drainage system
He0
->
Trabecular pathway
19001
->
Ho
->
Uneoscleral
- Na! Kt, Don,
->
pathway.
-> Ascorbic acid, lactic acid
Glucose
->
humor.
Hypopyema-pus in
aqueous
↓
corneal user.
Endophthalmites.
vwith Hyperthyroidism.
problem
supply.
as wel
pressure glaucoma (CCA)
normal - this.
thickness more
Ocular HTN- central corneal
([c+)
optic nerve
(neuroretinal aim)
superficial
- had
earachut
ene
.......
-ary
lauer
atrophy.
Idue to retinal
issue
damage)
sharp bendings
vessels.
of
-> of
Nasalization venels.
Bayoneting of vessels.
Bedside test
Peripheralwe
·
disease.
for neurological
·
30 vision
- Humphrey usual
field
Stimulus - static.
Kinetic perimetry.
(159
temporar
neede
Because of eyebrow prominence
50'
-
60'
Because nasal bridge nasal side -
of
lower - to
e
temporal 90%
-
X
X defect
on
goe
tubular
Aston
Paracentral Scotoma:
visual
non seeing area in the field.
↓ Blind
spot scotoma.
-
remained.
Silent-ofeye
->
Bopter.
area is
5060 to 90 joining
it
paracentral scotoma when gothed with blind spot forms
Siedel's scotoma
sinel shaped and called
line
touches the horizontal
Arcuate -
Tubular vision:
(Advanced Series
X-Haunome pigmentosa
>
-
laser extension
ens.
Gonio
-
Goldman applantation
tonometer
b
Sl-Schwalbe's line
AtM= Anterior trabecular
meshwork
Ptm- posterior,
shallow
->
-> shallow
normal.
->
scotoma.
Armate
-
armate fiber
-superior damage
Green- normal