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35 views2 pages

Labreportnew

Uploaded by

samud57672
Copyright
© © All Rights Reserved
We take content rights seriously. If you suspect this is your content, claim it here.
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Lab No. : CHP/27-11-2024/SR9964120 Lab Add.

: Newtown,Kolkata-700156

Patient Name : SAMUDRANEEL DUTTA Ref Dr. : Dr.UTTAM BANERJEE

Age : 17 Y 10 M 22 D Collection Date : 27/Nov/2024 11:04AM

Gender :M Report Date : 27/Nov/2024 02:04PM

DEPARTMENT OF BIOCHEMISTRY
Test Name Result Bio Ref. Interval Unit

25-HYDROXY VITAMIN D - TOTAL , . 10.51 ***FOR BIOLOGICAL REFERENCE ng/mL


(Method:CLIA) INTERVAL DETAILS , PLEASE
REFER TO THE BELOW
MENTIONED REMARKS/NOTE
WITH ADDITIONAL CLINICAL
INFORMATION ***

Biological Reference Intervals :


<20 ng/mL (deficiency)*
20-30 ng/mL (insufficient)**
30-100 ng/mL (optimum levels)***
>100 ng/mL (toxicity possible)****

* Could be associated with osteomalacia/ rickets


** May be associated with increased risk of osteoporosis or secondary hyperparathyroidism
*** Optimum levels in the normal population
**** 80 ng/mL is the lowest reported level associated with toxicity in patients without primary hyperparathyroidism who have normal renal function. Most
patients with toxicity have levels >150 ng/mL. Patients with renal failure can have very high 25-OH-VitD levels without any signs of toxicity, as
renal conversion to the active hormone 1,25-OH-VitD is impaired or absent.
These reference ranges represent clinical decision values that apply to males and females of all ages, rather than population-based reference values.
Population reference ranges for 25-OH-VitD vary widely depending on ethnic background, age, geographic location of the studied populations, and the
sampling-season. Population-based ranges correlate poorly with serum 25-OH-VitD concentrations that are associated with biologically and clinically
relevant vitamin D effects and are therefore of limited clinical value.

Clinical References :
1.Holick MF. Vitamin D Deficiency. N Engl J Med. 2007;357:266-81.
2. Jones G, Strugnell SA, DeLuca HF: Current understanding of the molecular actions of vitamin D. Physiol Rev 1998 Oct;78(4):1193-1231.
3. Miller WL, Portale AA: Genetic causes of rickets. Curr Opin Pediatr 1999 Aug;11(4):333-339
4. Vieth R: Vitamin D supplementation, 25-hydroxyvitamin D concentrations, and safety. Am J Clin Nutr 1999 May;69(5):842-856.

LIVER FUNCTION TEST , GEL SERUM


BILIRUBIN (TOTAL) 0.6 0.3-1.2 mg/dL
(Method:Vanadate oxidation)
BILIRUBIN (DIRECT) 0.1 <0.2 mg/dL
(Method:Vanadate oxidation)
BILIRUBIN (INDIRECT) 0.5 0.0 - 0.9 mg/dl
(Method:Calculated)
SGPT/ALT 57 7-55 U/L
(Method:Modified IFCC)
SGOT/AST 31 8-48 U/L
(Method:Modified IFCC)
ALKALINE PHOSPHATASE 101 55-149 U/L
(Method:IFCC standardization )
TOTAL PROTEIN 7.7 5.7-8.2 g/dL g/dL
(Method:BIURET METHOD)
ALBUMIN,BLOOD 4.7 3.2-4.5 g/dL
(Method:BCG Dye Binding)
GLOBULIN 3 1.8-3.2 g/dl
(Method:Calculated)
AG Ratio 1.57 1.0-2.5
(Method:Calculated)

Page 1 of 2
Lab No. : CHP/27-11-2024/SR9964120 Lab Add. : Newtown,Kolkata-700156

Patient Name : SAMUDRANEEL DUTTA Ref Dr. : Dr.UTTAM BANERJEE

Age : 17 Y 10 M 22 D Collection Date : 27/Nov/2024 11:04AM

Gender :M Report Date : 27/Nov/2024 02:04PM

DEPARTMENT OF BIOCHEMISTRY
Test Name Result Bio Ref. Interval Unit

Lab No. : CHP/27-11-2024/SR9964120 Page 2 of 2

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