EMT Notes Final
EMT Notes Final
DIRECTION
STAR OF LIFE (6 SYSTEM FUNCTIONS OF EMS) 1. Follow standing orders and protocols
1. Dedication/ Detection 2. Establish telephone and radio communications
2. Reporting 3. Communicate clearly and completely and follow orders given in
3. Response response
4. Care on scene 4. Consult medical direction for any question about the scope and
5. Care on transit direction of care
6. Transfer to definitive care
ETHICAL RESPONSIBILITIES
COMPONENTS OF EMS 1. Serve the needs of the patient with respect
1. Regulation and Policy 2. Maintain skill mastery
2. Resource Management 3. Keep abreast of changes in EMS that effect patient care
4. Critically review performances
3. Human Resource and Training
5. Report with honesty
4. Transportation 6. Work harmoniously with others
5. Communications
6. Public Information and Education TYPES OF CONSENT
7. Medical Direction 1. Expressed/ Formal/ Informed Consent
8. Trauma Systems 2. Implied Consent
9. Evaluation 3. Consent to treat a minor or a mentally incompetent adult
SMOOTH MUSCLE – under the control of autonomic nervous system 5. SESAMOID BONES – small bones developed in the tendons
1. around arterioles around certain joints
2. Bronchioles
BASIC PARTS
NINE REGIONS OF THE ABDOMEN
1. PELVIC GIRDLE
SOLID ORGANS
1. diaphragm
2. spleen
3. liver
4. pancreas
5. kidneys
HOLLOW ORGANS
1. stomach
2. gallbladder
3. duodenum
4. large intestine
3. SKELETAL SYSTEM JOINTS – junction of 2 or more bones
TYPES:
1. IMMOVABLE (FIBROUS) – bones of the skull and pelvis
2. SLIGHTLY MOVABLE (CARTILAGINOUS) – symphysis pubis
and intervertebral
3. FREELY MOVABLE (SYNOVIAL)
a) Elbow
b) Hip
c) Wrist
d) Knee
1. HINGE JOINT – allows flexion and extension in one direction only
i) Elbow
ii) Knees
RESPIRATORY SYSTEM
FUNCTIONS:
1. Extract O2 from the atmosphere and transfer it to the lungs
2. Excrete H2O vapour and CO2
3. Maintain acid-base balance
4. Ventilate the lungs
INSPIRED AIR
5. AUDITORY OSSICLES 1. 78% Nitrogen
2. 21% O2 (atmospheric concentration)
3. 0.04 % Carbon dioxide
4. 1 % inert gases
5. water vapour - variable
EXPIRED AIR
1. 78% Nitrogen
2. 17% O2
3. 4 % Carbon dioxide
4. 1 % inert gases
5. water vapour - increased amount to saturation
6. FACIAL BONES
EXTERNAL RESPIRATION 3 LAYERS OF THE HEART
-takes place in the lungs (alveolar level)
INTERNAL RESPIRATION
-takes place in the tissues (circulatory/ cellular level)
MECHANISM OF RESPIRATION
1. INSPIRATION (BREATHING IN) – contracted diaphragm and
intercoastal muscles; (-) pressure
2. EXPIRATION (BREATHING OUT) – relaxed diaphragm and
intercoastal muscles; (+) pressure; more gas exchange
CIRCULATORY SYSTEM
BLOOD VESSELS
COMPONENTS OF CIRCULATION
PATHWAY OF ELECTRICAL ACTIVITY
1. BLOOD – Adult: 5liters
-Child: weight x 80cc =volume
2. HEART
3. BLOOD VESSELS
Tx
1. Rest INFECTIOUS DISEASE
2. Oxygenation
3. Recompression in serious cases especially where arterial air
SYPHILIS – acute &chronic disease caused by the spiral shaped bacterium
embolism is present
(Treponema Pallidum)
ARTERIAL AIR EMBOLISM:
3 STAGES:
Rapid onsent of
1. Primary – characterized by painful chancre with indurated borders
1. Altered LOC w/in 10mins
on the penis, vulva or other area with sexual contact
2. Hemiplegia
3. Unequal pupils
2. Secondary – occurs 3-6weeks after the end of the primary stage.
4. Cardiopulmonary failure
Sx: rash(trunks & Flexor surfaces, palms & soles) &
5. Vertigo
Lymphadenopathy
6. Visual disturbances
3. Tertiary – CVS & nervous involvement. Characteristics of this
Treatment and management:
stage occur years after the initial infection. S/S: acute meningitis,
1. CAB’s
dementia, neuropathy to thoracic aneurysm
2. Transport to decompression chamber
3. 100% oxygen, assist ventilation as needed
TUBERCULOSIS (TB) – transmission of the bacteria Myobacterium by
droplet. Communicable period last as long as infective tubercle bacilli are
DECOMPRESSION SICKNESS
being discharged in the sputum that is until about 24-48 hours after antibiotic
1. Gas bubbles trapped deep in the tissues, particularly
treatment
slow circulated tissues
S/S: night sweats, headache, cough & weighted loss
2. Excessive nitrogen dissolve in blood & tissues
oversaturating
RABIES – caused by an RNA containing RHABDOVIRUS & is transmitted
3. Diver does not surface at correct rate to allow nitrogen
by inoculation with infectious saliva from an animal
to escape from blood & tissues
- Incubation period shortest is 12 days & longest is 700 days
TYPES:
- NEGRIBODIES are the characteristics histologic findings
1. DECOMPRESSION SICKNESS – SKIN
- Cutaneous bends
S/S
1. EARLY STAGE – fever, malaise, headache, anorecia, sore throat, DRUG – presumed to have some therapeutic effects when given under
cough & pruritis & paresthesia on the bite site appropriate dosage and circumstances
2. LATE STAGE – restlessness, agitation, altered mental status,
painful bulbar & peripheral muscular spasm, opisthotonous, 3 leading cause of Poisoning
hypersensitive to sensory stimuli, & hydrophobia results from 1. Alcohol intoxication
bulbar spasm that occur until swallowing 2. Methamphetamine
3. Izoniazid
TREATMENT:
1. Safety/BSI APPROACH
2. CAB’s C – Maintain adequate circulation
3. Scrubbing and cleansing of wound to remove rabies A – Maintain adequate airway
4. Transport to medical center for HUMAN IMMUNO GLOBULIN B – Provide adequate breathing
& HYUMAN DIPLOID VACCINE D – Manage drug induced CNS depression
E – Manage electrolyte imbalance
MENINGITIS – inflammation of the meninges which is caused by Neiseria
Meningitidis History taking
1. What was ingested?
S/S: fever, severe headache, stiff neck, changes in state of consciousness, 2. When was the poison ingested?
vomiting, blotchy red or bluish rash 3. How much was ingested?
HEPATITIS – is an infection that cause an inflammation if the liver 4. What else had been taken?
5. Did the patient vomited?
4 Forms of Hepatitis: 6. Why was it taken? (Optional)
1. Hepatitis A
2. Hepatitis B 1. INGESTED POISON– process in which poison enters to the mouth
3. Hepatitis C
4. Hepatitis D Liquefaction necrosis – alkalis
HEPATITIS A – is known as infection hepatitis. Spread by oral fecal route. Coagulation necrosis – acid
Virus is excreted in large amount in feces for 2 weeks before and 1 week after
onset of symptoms Signs / symptoms
1. Papillary changes
HEPATITIS B – is known as serum hepatitis. Spread through contact with 2. Salivation
infectious blood or blood products 3. Sweating
- Incubation period is 45-180 days 4. Respiratory distress
- Complete recovery will take 3-4weeks or may persist a 5. Burn or blister on the lips
lifetime 6. Unusual breath odor
7. Vomiting
HIV-AIDS – caused by HIV. AIDS is the set of conditions that results when 8. Diarrhea
the immune system has been attacked by HIV 9. Nausea
10. Altered mental status
INCUBATION PERIOD: 11. Seizure
1. FIRST – from the time of exposure to the time a person’s blood
test positive for AIDS maybe anywhere from few weeks to a few Management
months. Person who had an accidental exposure to AIDS should be 1. Maintain airway
tested within 2-3weeks after the exposure & then again at 6weeks, 2. NPO
3 months, 6months & year after 3. Collect sample of vomit
2. SECOND – time between documented infection, & the contracted 4. Antihistamine or antiemetic
AIDS from contaminated blood transfusion, incubation period is
approximately 8 years for adults & 2 years for children Note: Do not give high flow O2 for watusi, zinc phosphate and paraquat
Management: Contraindications:
1. Remove patient from environment 1. Methanol
2. Open airway 2. Caustic acids
3. BLS 3. Akalis
4. 02 by NRM 4. Altered Mental Status
Management:
1. Follow protocol PATHOPHYSIOLOGY of ANAPHYLAXIS
2. Maintain open airway
3. Remove jewelry Sting / bite
4. Transport immediately
4. ABSORBED POISON – taken into the body thru unbroken skin Antigen introduced into the body
Management:
1. Move the patient away from the source Antigen – antibody reaction
2. Flood with water
3. Transport
Mast cell release chemical mediator
Watusi Poisoning – yellow phosphorous (Histamine)
Signs / symptoms
1. Direct cardiotoxic effect Chemical mediator exert their efforts on
2. Hypotension END ORGANS
3. Hepatic toxicity
4. Burn
5. Burning pain in the throat (garlic odor) LUNGS
6. Nausea HEART
7. Vomiting BLOOD VESSEL
8. Diarrhea SKIN
9. Abdominal pain HYPOTHERMIA
10. Shock
Contraindications Mild
1. Unconscious or no gag reflex S – Shivering
2. Seizures T – tachypnea
3. Pregnancy A – Ambulatory / alert
4. Acute Myocardial Infarction P – Pain
5. Infants < 6mos P – Peripheral vasoconstriction
6. Volatile hydrocarbon
7. Iodides Moderate
S – slurred speech
Dosage: H –Hallucinations
Adult: 30cc or 1 – 2tbsp I – irrational behavior
Child: 15cc or 3 – 5tsp L – Loss of fine motor skills
D – Dazed
Note: Must be taken with water.
Severe
R – Rigid muscles Management:
A – Assumes fetal position 1. Move patient to a cool dry place
B – bradycardia 2. Give H2O
I – increase lactic acid and CO2 3. ORESOL
D – Dilated pupils 2. HEAT EXHAUSTION
S – Skin is pale / shivering stops Signs / symptoms
1. Headache
2. Extreme weakness
PATHOPHYSIOLOGY 3. Dizziness
EXPOSURE TO COLD 4. Syncope
5. Decrease appetite
6. Rapid pulse
Shivering (400x energy is used) vasoconstriction 7. Dilated pupils
8. Diaphoresis
Release in thyroxine release of nor
epinephrine and Management
epinephrine 1. Move patient to a cool dry place
2. Give H2O
Increase metabolism increase RR, PR, BP 3. ORESOL
4. Elevate leg to 8” – 10”
Decreased in blood glucose 5. Assess VS
3. HEAT STROKE
Shivering stops Types
1. Exertional – prolong exposure to hot places
2. Classic – elderly, alcoholic, obese, CVD, phenothiazine users
Rapid cooling
Signs / symptoms
1. Headache
Decreased in O2 demand 2. Dizziness
3. Irritability
4. Decrease LOC
Cardiac irritability 5. Seizure
6. Bounding pulse
7. No sweat
Hypoxia 8. Dilated pupils
9. Decrease blood pressure
10. Hot red skin
Ventricular fibrillation
Management:
1. Move patient to a cool dry place
DEATH 2. Check CAB
3. Monitor cardiac rhythm
Managemnt: 4. Ice packs
1. Keep it frozen
2. Protect from trauma Preventive measures
3. Do not massage 1. Maintain adequate fluid intake
4. If partly thawed rewarm at 38 – 42 C 2. Allow time to acclimatization
5. transport 3. Limit exposure to hot environment
Methods of Rewarming
1. Active External ACUTE ABDOMEN
a. apply hot pacts to groin, armpits, chest, knees
b. cover with blankets NOTE: Can be referred pain to cardiac emergencies
c. increase body temp gradually (not > 1 C / hr)
d. do not immerse Signs / symptoms
1. Pain
2. Active Internal 2. Tenderness
a. Warmed oxygen 3. Anxiety and Fear
b. Cardiopulmonary bypass 4. Decrease LOC
5. Guarding position
3. Passive 6. Rapid shallow breathing
a. Move patient to warm environment 7. Rapid pulse
8. Nausea Vomiting and Diarrhea
Mechanism of Heat Loss 9. Rigid or tender abdomen
1. Conduction 10. Abdominal Distension
2. Convection
3. Respiration Causes:
4. Radiation 1. Appendicitis – rebound tenderness
5. Evaporation 2. Cholecystitis
3. Instestinal Obstruction
4. Hernia BLEEDING
HEAT EMERGENCIES 5. Pancreatitis
6. UlceR
1. HEAT CRAMPS – sudden painful stiffening of the muscles. 7. Esophageal varices
Sign / symptoms 8. Abdominal Aortic Anuerysm
1. Severe cramping on the abdomen or leg
2. Dizziness Management:
3. Periods of fainting 1. BSI / safety
4. Tachycardia 2. CAB
5. Diaphoresis 3. NPO
4. Watch out for shock
5. Transport comfortably 3. PELVIC INFLAMMATORY DISEASE – caused by bacteria from
vagina (vaginosis)
GYNAECOLOGICAL EMERGENCIES
OBSTETRICAL EMERGENCIES
1. ECTOPIC PREGNANCY – pregnancy outside the uterus. 50% of cases
have history of salphingitis or PID FALSE TRUE
Irregular Regular, predictable
Signs / symptoms From Lower back to
Abdomen
1. Abdominal pain Contractions Abdomen
2. Abnormal vaginal bleeding Disappears in
Continues
3. Amenorrhea ambulation
4. Breast tenderness Duration
5. Low back pain Frequency constant Increases
6. Nausea intensity
Cervical Dilatation none increases
If abnormal pregnancy ruptures and bleeds
1. Fainting Stages of labor
2. Shoulder pain 1st stage - 1st contraction to full dilatation of the cervix
3. Severe sharp, sudden abdominal pain 2nd stage -full dilatation of the cervix to birth of the neonate
4. Shock 3rd stage -birth of the neonate to the expulsion of the placenta
a. Rapid thread pulse
b. Pale, cold clammy skin COMPLICATION OF PREGNANCY
c. Decrease BP 1. (FIRST TRIMESTER)ABORTION– vaginal bleeding less than 28 weeks
of AOG
Management a. Spontaneous
1. Adequate airway i. Threatened – priority 1
2. Assist in ventilation ii. Inevitable – cervix is open
3. NPO iii. Incomplete
4. Anticipate vomiting iv. Missed
5. IV lines large bore b. Criminal
6. Recumbent position c. Therapeutic
7. Assess vital signs
2. (THIRD TRIMESTER) ANTENATAL BLEEDING - vaginal bleeding
2. RUPTURED OVARIAN CYST more than 28 weeks of AOG
Signs/ symptoms a. Abruptio Placenta – premature separation of placenta
1. Sharp piercing pain b. Placenta previa – placenta covers cervical opening.
2. Fever c. Uterine rupture
3. Nausea 3. PREECLAMPSIA/ECLAMPSIA
4. Vomiting Signs / symptoms
5. Weakness
6. Dizziness 1. swelling of the face, hands and feet
7. Fainting 2. weight gain
8. Internal bleeding 3. hypertension
4. headache
5. sensitive to light
6. visual blurring 4. Cutting of the cord
7. pain in upper abdomen
TYPE I – hyperinsulemia
DIABETIC EMERGENCIES -insulin shock
-DKA acetone breathe odor ketone breath end-products of
NORMAL BLOOD GLUCOSE LEVEL: 70-110mg/dl fats
SIGNS AND SYMPTOMS: polyuria
Polydypsia EVALUATION
Polyphagia 1. TYPE I – have you injected yourself with insulin?
TYPE II – have you taken your tablet?
1. HYPERGLYCEMIA 2. Have you taken your medication?
May die because of dehydration 3. After eating, have you vomited?
CAUSES 4. Have you increased your activity other than your routine?
1. Diabetic condition is not been diagnosed and/or treated
2. Has not taken insulin
3. Overeaten, flooding the body with a sudden excess of
carbohydrates PEDIATRIC EMERGENCIES
4. Suffers an infection that disrupts glucose/insulin balance
1. CROUP LARYNGOTRACHEOBRONCHITIS)
SIGNS AND SYMPTOMS Signs / symptoms
1. gradual onset of signs and symptoms over a period of days 1. Seal bark
2. patient complains of dry mouth and intense thirst 2. Whooping sound during inhalation
3. abdominal pain and vomiting common 3. Nasal flaring
4. increase restlessness, confusion followed by stupor and coma 4. Tracheal tagging
5. weak, rapid pulse 5. Retractions
6. signs of air hunger-deep sighing respirations, breath smells of a. Intercostals
acetone-sickly sweet b. Suprasternal
7. warm , red, dry skin 6. Cyanosis
8. normal or slightly low BP
9. sunken eyes Management:
10. kussmauls breathing 1. 02 by mask
2. Do not attempt to examine the throat
EMERGENCY CARE 3. Place in a comfortable position
1. glucogel 4. Transport
2. D50W
3. Packet of sugar 2. EPIGLOTITIS
4. Pulse oximeter: 93% - O2 Signs / symptoms
95% - give sugar/ glucogel 1. Cherry red epiglottis
90% - O2 94% - sugar 2. Odynophagia
5. O2 – 15lpm NRM 3. Dysphagia
6. Responsive:orange juice or glucose drink 4. Drooling
7. Transport immediately 5. High fever
8. ALS assistant
Management:
2. HYPOGLYCEMIA 1. O2
May be delayed for days 2. NPO
CAUSES 3. Do not suction
1. Patient has taken too much insulin 4. Transport in a comfortable position
2. Patient has not eaten enough to provide his normal sugar intake
3. Has over-exercised or overexerted, reducing glucose evel 3. ACUTE ASTHMA ATTACK - characterized by spasm and constriction
4. Vomited a meal of the bronchi, Pulmonary edema and
Hypersecretion of mucus.
SIGNS AND SYMPTOMS
1. Rapid onset of s/sx over a period of minutes Signs / symptoms
2. Copious saliva, drooling
3. Intense hunger 1. Dyspnea
4. Dizziness, headache, sudden fainting, seizures 2. Increase PR
5. Full rapid pulse 3. Shallow RR
6. Normal respiration, no odor 4. Decrease BP
7. Skin cold, pale, clammy profuse perspiration 5. Wheezing, sometimes crakles
8. Normal BP 6. Agitated, anxious and restless
9. Eyes normal
10. Abnormal hostile or aggressive behavior Management
1. O2 by mask
EMERGENCY CARE 2. Aerosolized nebulizer
1. Conscious – administer granular sugar, honey, orange or glucose 3. Semi sitting position
2. Unconscious – glucogel under tongue 4. Transport
3. Recovery position 5. Bronchodilator as per protocol
4. High O2
5. Transport immediately 4. BRONCHIOLITIS – inflammation of bronchioles characterized by low
6. ALS assistant grade fever, runny nose and poor appetite
Emergency care:
NEUROLOGIC EMERGENCY
1. Conscious patient:
a. Ensure open airway
TRANSIENT ISCHEMIC ATTACK - Disorientation of a particular b. Keep patient warm
cerebral artery and vertebral basilar artery and last anywhere from a few c. Give high flow oxygen – check 1st SpO2
seconds to 12 hours. d. Monitor v/s
e. Transport in Semi Fowlers
f. NPO
Signs and symptoms:
g. Sit in front of the patient, maintain eye contact, and
1. Hemiparesis or hemiplegia speak slowly and clearly
2. Unilateral numbness
3. Phasia 2. Unconscious
4. Confusion, coma a. Maintain open airway
5. Convulsion b. Give high flow of oxygen and assist ventilation if
6. Sometimes incontinence necessary – check SpO2
7. Numbness of the face
8. Slurred speech
Classification:
9. Dysphagia
10. Posterior head ache 1. TIA
11. Dizziness or vertigo 2. Reversible ischemic neurologic deficit
3. Stroke in evolution/completed stroke
SEIZURE - Injury, infection, or disease to the normal function of the brain -> Pathomechanism:
the electrical activity becomes irregular -> sudden changes in sensation, Thrombosis, embolism, hemorrhage impaired cerebral blood flow
behavior, or movement called -> seizures cerebral ischemia
Mechanism of spinal injury: Trauma triage criteria indicating nedd for immediate transport:
1. Hyperextension -Excessive posterior movement of head or neck MOI:
a. Face into windshield in MVC 1. Falls >20 ft (3x the victim’s height)
b. Elderly person falling to the floor 2. Pedestrian/ bicyclist vs auto collision
c. Foot ball tackler a. Struck by a vehicle traveling over 5 mph
d. Dive into shallow water b. Thrown or run over by vehicle
3. Motorcyclist impact >20 mph
2. Hyperflexion - Excessive anterior movement of head onto chest 4. Ejection from the vehicle
a. Rider thrown off from the horse or motorcycle 5. Severe vehicle impact
b. Dive into shallow water a. Speed at impact ?40 mph
b. Intrusion of >20 inches into occupants compartment
3. Compression - Weight of head or pelvis driven into stationary c. Vehicle deformity >20 inches
neck or torso 6. Rollover with signs of serious impact
a. Dive into shallow water 7. Death of another occupant in the vehicle
b. Fall of > 10 – 20 feet into head or legs 8. Extrication time >20 mins
Infants and children:
4. Rotation - Excessive rotation of the torso or head and neck, 1. fall >10 ft (3x the victim’s height)
moving one side of the spinal column against the other 2. bicycle/vehicle collision
a. Rollover MVC 3. any vehicle collision where the infant or child was unrestrained
b. Motorcycle accident
Physical findings:
5. Lateral stress - Directly lateral force on spinal column 1. revised trauma score <11
a. T bone MVA 2. GCS <14
b. Fall 3. SBP <90
4. PR <50 or >120
6. Distraction - Excessive stretching of column and cord 5. Penetrating trauma (except distal extremities)
a. Hanging 6. 2 or more paroxysmal long bone fractures
b. Child inappropriately wearing shoulder belt around neck 7. Flail chest
c. Snowmobile or motorcycle under rope or wire
8. Pelvic fractures Clavicle
9. Limb paralysis Shoulder
10. Burns >15% of BSA Hip hits door
11. Burns to face or airway Head or femur driven through acetabulum
Pelvic fractures
C spine injury
Head injury
KINEMATICS
Lateral compression
1. Physics of trauma Ruptured diaphragm, spleen rupture, aortic injury
2. Predictions of injuries based on forces motion involved in injury event
4. Rotational collision (38%)
Off the center impact
Physical principles: Car rotates around impact
Kinetic energy (1/2 mass x velocity) Patient thrown toward impact point
Energy in motion Injuries combination of head on and lateral
Major factor is velocity Point of greatest damage(worst patient)
Speed kills
Newton’s 1st law of motion 5. Roll over collision (6%)
Body in motion stays in motion unless acted on by outside Multiple impacts each time vehicle rolls
motion Injuries unpredictable
Body at rest stays at rest unless acted by an outside force Assume presence of severity of injury
Law of conservation energy Justification for:
Energies are cannot be created or destroyed Transport priority one
Only changed from 1 form to another Trauma team activation
25x the risk when injected
Types of trauma:
1. Penetrating In each collision there are 3 impacts that can occur:
2. Blunt 1. Vehicle
a. Deceleration 2. Occupant
b. Compression 3. Occupant’s organs
Treatment Hallow organs – moves freely and slippery, can absorb impact due to trauma
1. Secure airway
but can rupture (release toxins)
2. High flow 02, assist vent if necessary
3. Rapid transport (if possible)
4. Notify hospital or ALS ASAP Solid organs – has major blood vessel, if ruptured it can lead to hypovolemic
shock (ex. Kidney 25 % of CO)
4. Flail Chest (true emergency) NOTE: Use tape measure in cm to monitor abdominal distension
2 or more ribs in 2 or more places Closed abdominal trauma
Paradoxical movement
Blunt trauma
Most common – sternum
Patient tends to under breath due to pain
S/Sx
Can lacerate blood vessels and puncture lung
1. Large or intense contusion on the abdomen
S/Sx:
2. Coffee ground vomitus
1. SOB
3. Rigid and tender abdomen (abdominal guarding)
2. Paradoxical movement
4. Distended abdomen
3. Bruising and swelling
5. Lies drawn up in an effort to reduce the tension on abdominal
4. Signs of shock
muscles
5. Crepitus (sound of bone grinding)
6. Pain that starts mild then to intolerable pain
7. Indications of developing shock
Treatment:
1. Apply bulky dressing or small pillow position inward – to
immobilizethe injured part and to move together with the chest Open abdominal trauma
2. Have the patient lie on the back Evisceration
3. CAB, high flow of 02 Protruding of abdominal organ through the wound opening
4. Treatment for shock
5. Monitor V/S Emergency care tips for abdominal trauma
1. Stay alert for vomiting and keep airway open
5. Cardiac tamponade 2. Place the patient on his back, legs flexed at the knees to reduce
Most common cause – penetrating injury/ruptured AMI pain by relaxing the abdominal muscle
Blood leaks to pericardial sac 3. Administer high concentration of 02 (Sp02)
Cardiogenic and obstructive shock 4. Monitor V/S
S/Sx: 5. Immediate transport ASAP
1. Distended neck vein 6. Do not touch or try to replace any evisceration or exposed
2. Increase HR organs (cover with occlusive dressing moistened with PNSS)
3. Increase RR 7. For impaled object, leave it to stabilize with bulky dressing and
4. Trachea midline leave the patient’s legs in the position which you found them to
5. lung sound normal avoid muscular movement that may move the impaled object
Preservation of an amputated part NON-URGENT MOVE – there is no danger; can do secondary survey
1. rinse the amputated part free from debris (PNSS)
2. wrap the part loosely in sterile gauze (moistened) PATIENT CARRYING DEVICES
3. seal the amputated part inside a plastic bag and place it in a cool 1. Wheeled stretcher (trolleycot)
container
2. Portable stretcher
4. never warm, place in water, place directly on ice or dry ice to cool
amputated part 3. Carry chair/ stair chair
4. Spine boards
LIFTING AND MOVING 5. Scoop (orthopedic) stretcher
6. Basket stretcher
7. Flexible stretcher
BODY MECHANICS – is the proper use of your body to facilitate lifting and
8. Vacuum mattress
moving
9. Moving patient onto carrying device
10. Patients with suspected spine injury
PRINCIPLES OF BODY MECHANICS
11. Patient with no suspected spine injury
1. keep the weight of the object as close to the body as possible
a. Extremity lift (top and Tail)
2. to move a heavy object use the leg, hip, and gluteal muscle plus
b. Direct ground lift
contracted abdominal muscle
c. Draw sheet method
3. STACK
d. Direct carry
4. Reduce the height or distance through which the object must be
moved
CRITICAL SITUATIONS TO MOVE PATIENT PRIOR TO
ASSESSMENT AND CARE
METHODS TO PREVENT INJURY
1. Fire or threats of fire at the accident scene
1. power lift
2. Explosive and other hazardous materials involved
2. power grip
3. You are unable to protect the accident scene from oncoming traffic,
downed electrical wires, toxic, fumes or other hazards
WHEN REACHING
4. You are unable to reach patient who needs lifesaving care without 1. By agent and source
moving one or more of the victims Chemicals
5. You are unable to properly treat the patient without moving him Electricity
Thermal
Radiation
POSTURE AND FITNESS 2. By depth
1. EXCESSIVE LORDOSIS (SWAYBACK)– abnormal anterior a. Superficial (1st degree)
convexity of the spine involves epidermis and pain at the site ( 3-6 days
2. EXCESSIVE KYPHOSIS (SLOUCH) – abnormal curvature of healing)
the spine with convexity backward b. Partial thickness ( 2nd degree)
Involves epidermis and dermis with deep intense pain,
noticeable reddening, blisters and mottled appearance
BODY ALIGNMENT
(7-21 days healing)
1. Center of gravity c. Full thickness (third degree)
2. Line of gravity Involves all the layers of the skin, patient may feel
severe pain or may not due to damaged nerves,
(requires skin grafting)
INDICATION TO LEAVE THE HELMET IN PLACE d. By severity – consider
1. Good fit, little movement Agent or source
Body regions burned
2. No current or expected airway problems
Circumference burn
3. Removal would be the cause of further injury Depth of the burn
4. Proper immobilization Extent of the burn (BSA estimation)
5. No airway or breathing concerns Rule of nines
Rule of palm
INDICATION FOR REMOVING THE HELMET 3. Age of the patient (<5 or >55)
1. inability to assess or treat airway and breathing problems 4. Other illnesses and injuries
Existing respiratory illness
2. improper fit/ movement within helmet
Patients with heart disease
3. inability to immobilize spine
4. cardiac arrest Infants and children – greater fluid and heat loss, higher the risk of shock,
airway problems and hypothermia
STEPS IN REMOVING THE HELMET
1. stabilize head (EMT – 1) Types of burns
2. fingers should be on patients mandible 1. Dry burn
2. Scald burn (steam, hot liquids)
3. loose strap (EMT – 2)
3. Electrical burn
4. transfer of stabilization to EMT – 2 4. Freeze burn
5. carefully remove the helmet 5. Chemical burn
6. prevent head from falling one helmet is removed 6. Radiation burn
7. begin routine stabilization and immobilization
Classification of burn severity: adults
ADVANTAGE OF VACMAT 1. Minor burns
Full thickness burn of <2%, excluding face, hands, feet,
1. IT IS COMFORTABLE, LESS PRESSURE ON THE BODY
genitalia or respiratory tract
PARTS IN CONTACT WITH THE MATTRESS Partial thickness burns or less than 15%
2. IT IS ADAPTED TO ALL TRAUMA, SPECIALLY SPINE Superficial burns of 50% or less
AND FEMORAL TRAUMA 2. Moderate burns
3. THEVICTIM IS SECURED AS COMPARED TO Full thickness burns of 2% - 10%, excluding face, hands,
BACKBOARDS (LESS OR NO MOVEMENT) feet, genital or respiratory tract
4. PROVIDES THERMAL INSULATION; DECREASED THE Partial thickness burns of 15% - 30%
Superficial burns that involve more than 15%
POTENTIAL FOR HYPOTHERMIA
3. Critical burns
5. FOLLOWS THE CONTOUR OF THE BODY All burns complicated by injuries of the respiratory tract,
other soft tissue injuries and injuries to the bones
BURN EMERGENCIES Partial or thickness burns involving the face, hands, feet,
Burn genitalia or respiratory tract
Is when the body, or a body part, receives more energy that it can Full thickness burns of >10%
absorb without injury Partial thickness burns of >30%
Among the most serious and painful of all injuries Burns complicated by musculoskeletal injuries
Circumferential burns
Function of the skin 4. Moderate burns should be considered critical in person <5 y/o
1. Protection or >55 y/o
2. Water balance
3. Temperature regulation Classification of burn severity: children >5 y/o
4. Excretion 1. Minor burns
5. Shock absorption Partial thickness burns of <10% BSA
2. Moderate burns
Layers of the skin Partial thickness burns of 10% - 20% BSA
Epidermis 3. Critical burns
Dermis Full thickness burns or partial thickness burns of more than
Subcutaneous layer 20% BSA
Communication System in the EMS Ten golden rule in communicating with the patient
1. Make and keep eye contact
EMS communication system 2. Use the patient’s proper name
Is a system that coordinates the many interdependent agencies and 3. Be honest
facilities involved in emergency response and care. 4. Use the language the patient can understand
5. Confidentiality
Basic function of EMS communication system 6. Be aware of your body language
1. Detecting and reporting accidents 7. Speak slowly, clearly and distinctly
2. Assigning personnel to respond 8. If patient hearing is impaired, speak clearly, and face the patient so
3. Maintaining contact between personnel that he/she can read your lips
4. Alerting other personnel as needed 9. Allow time for the patient to answer
5. Relaying patient information and receiving information for 10. Act and speak calm, confident manner while caring
treatment
6. Determining which hospital to transport Emergency medical dispatcher
7. Informing emergency department personnel about numbers, type of A person in the EMS responsible for assigning of emergency
accident, and severity of injury medical resources to a medical emergency
EMOTIONAL STAGES
1. denial
2. anger
3. bargaining
4. depression
5. acceptance
STRESSORS
1. Vomit
2. Unfairness
3. Absence thrown at you, the rescuers, by them, the rescued
4. Interminable waiting
5. Death and dying
6. Administrative insensitivities
7. Long hours and inappropriate pay
8. Demand on you by the ambulance corps