Shock Practice Questions
Shock Practice Questions
Shock Practice Questions
1. A nurse educator is teaching a group of nurses about assessing critically ill clients for multiple
organ dysfunction syndrome (MODS). The nurse educator evaluates understanding by asking the nurses
to identify which client would be at highest risk for MODS. It would be the client who is experiencing
septic shock and is
a) An older adult man with end-stage renal disease and an infected dialysis access site
b) An 8-year-old boy who underwent an appendectomy and then incurred an iatrogenic infection
c) A young female adolescent who developed shock from tampon use during menses
2. A patient is admitted to the emergency department after sustaining abdominal injuries and a
broken femur from a motor vehicle accident. The patient is pale, diaphoretic, and is not talking
coherently. Vital signs upon admission are temperature 98 F (36 C), heart rate 130 beats/minute,
respiratory rate 34 breaths/minute, blood pressure 50/40 mmHg. The healthcare provider suspects
which type of shock?
a.) Hypovolemic
b.) Cardiogenic
c.) Neurogenic
d.) Distributive
3. When caring for a patient in acute septic shock, what should the nurse anticipate?
4. A massive gastrointestinal bleed has resulted in hypovolemic shock in an older patient. What is a
priority nursing diagnosis?
A. Acute pain
B. Impaired tissue integrity
6. A patient's localized infection has progressed to the point where septic shock is now suspected.
What medication is an appropriate treatment modality for this patient?
A. Insulin infusion
B. IV administration of epinephrine
8. The nurse is caring for a 72-year-old man in cardiogenic shock after an acute myocardial
infarction. Which clinical manifestations would be of most concern to the nurse?
C. PaO2 of 38 mm Hg, serum lactate level of 46.5 mcg/dL, and bleeding from puncture sites
D. Agitation, respiratory rate of 32 breaths/minute, and serum creatinine level of 2.6 mg/dL
9. A patient has a spinal cord injury at T4. Vital signs include falling blood pressure and
bradycardia. The nurse recognizes that the patient is experiencing
a. a relative hypervolemia
b. an absolute hypovolemia
10. A patient who has pericarditis related to radiation therapy, becomes dyspneic, and has a rapid,
weak pulse. Heart sounds are muffled, and a 12 mmHg drop in blood pressure is noted on inspiration.
The healthcare provider's interventions are aimed at preventing which type of shock?
a.) Distributive
b.) Neurogenic
c.) Obstructive
d.) Cardiogenic
11. The healthcare provider is caring for a patient who has septic shock. Which of these should the
healthcare provider administer to the patient first?
13. Which of the following assessment findings is an early indication of hypovolemic shock?
c.) Tachycardia
d.) Hypertension
14. When compensatory mechanisms for hypovolemic shock are activated, the nurse would expect
which two patient findings to normalize?
15. Which type of fluid is most appropriate for volume replacement for a patient with non-
hemorrhagic hypovolemic shock?
16. A 78 year old man has confusion and temperature of 104. He is a diabetic with purulent drainage
from his right heel. After an infusion of 3 L of normal saline solution, his assessment findings are BP
84/40, HR 110, RR 42 and shallow, CO 8L/min, and PAWP 4 mm Hg. This patient's symptoms are most
likely indicative o
a. sepsis
b. septic shock
17. ppropriate treatment modalities for the management of cardiogenic shock include (Select all that
apply)
18. Appropriate treatment modalities for the management of cardiogenic shock include (select all that
apply):
myocardium.
19. A massive gastrointestinal bleed has resulted in hypovolemic shock in an older patient. What is a
priority nursing diagnosis?
a-Acute pain
20. What laboratory finding fits with a medical diagnosis of cardiogenic shock?
21. What will the nurse identify as symptoms of hypovolemic shock in a patient?
Note: Credit will be given only if all correct choices and no incorrect choices are selected.
b. Restlessness
22. Which laboratory finding should cause the nurse to suspect that a patient is developing hypovolemic
shock?
23. The nurse recognizes that which patient would be most likely to develop hypovolemic shock? A
patient with:
c. Ascites
24. Which finding indicates that a patient is experiencing increased peripheral resistance and
vasoconstriction?
25. Which solution would be the most appropriate initial volume replacement for a patient with severe
GI bleeding?
26. Which life-threatening complications would the nurse anticipate developing in the patient being
treated for hypovolemic shock?
Note: Credit will be given only if all correct choices and no incorrect choices are selected.
b. Renal insufficiency
c. Cerebral ischemia
e. Pulmonary edema
27. The nurse, caring for a patient in hypovolemic shock, will not utilize a hypotonic solution for fluid
resuscitation because hypotonic solutions:
a. Move quickly into the interstitial spaces and can cause third spacing
b. Stay longer to expand the intravascular space but deplete intracellular fluid levels
c. Do not stay in the intravascular space long enough to expand the circulating blood volume
d. Need a smaller bore needle to run at a slower rate to keep the intravascular space low
28. The nurse should warm intravenous fluids when a rapid infuser is being utilized to prevent which
complication?
a. Hemorrhagic shock
b. Hypothermia
c. Sepsis
d. Cardiogenic shock
29. A patient is treated in the emergency department (ED) for shock of unknown etiology. The first
action by the nurse should be to
30. A diabetic patient who has had vomiting and diarrhea for the past 3 days is admitted to the hospital
with a blood glucose of 748 mg/ml (41.5 mmol/L) and a urinary output of 120 ml in the first hour. The
vital signs are blood pressure (BP) 72/62; pulse 128, irregular and thready; respirations 38; and
temperature 97° F (36.1° C). The patient is disoriented and lethargic with cold, clammy skin and cyanosis
in the hands and feet. The nurse recognizes that the patient is experiencing the
31. A patient with hypovolemic shock has a urinary output of 15 ml/hr. The nurse understands that the
compensatory physiologic mechanism that leads to altered urinary output is
a. activation of the sympathetic nervous system (SNS), causing vasodilation of the renal arteries.
c. release of aldosterone and antidiuretic hormone (ADH), which cause sodium and water retention.
d. movement of interstitial fluid to the intravascular space, increasing renal blood flow.
32. While caring for a seriously ill patient, the nurse determines that the patient may be in the
compensatory stage of shock on finding
33. When assessing the hemodynamic information for a newly admitted patient in shock of unknown
etiology, the nurse will anticipate administration of large volumes of crystalloids when the
a. cardiac output is increased and the central venous pressure (CVP) is low.
b. pulmonary artery wedge pressure (PAWP) is increased, and the urine output is low.
34. . A patient who has been involved in a motor-vehicle crash is admitted to the ED with cool, clammy
skin, tachycardia, and hypotension. All of these orders are written. Which one will the nurse act on first?
35. A patient with massive trauma and possible spinal cord injury is admitted to the ED. The nurse
suspects that the patient may be experiencing neurogenic shock in addition to hypovolemic shock,
based on the finding of
b. shortness of breath.
d. BP of 82/40 mm Hg.
36. The nurse caring for a patient in shock notifies the health care provider of the patient's deteriorating
status when the patient's ABG results include
1. Answer A: An older adult man with end-stage renal disease and an infected dialysis access site
Explanation:
MODS may develop when a client experiences septic shock. Those at increased risk for MODS are
• older clients,
2. Answer A. Hypovolaemic
Septic shock is characterized by a decreased circulating blood volume. Volume expansion with the
administration of IV fluids is the cornerstone of therapy. The administration of diuretics is inappropriate.
VADs are useful for cardiogenic shock not septic shock. Diphenhydramine (Benadryl) may be used for
anaphylactic shock but would not be helpful with septic shock.
The many deleterious effects of shock are all related to inadequate perfusion and oxygenation of every
body system. This nursing diagnosis supersedes the other diagnoses.
The renal hypoperfusion that accompanies cardiogenic shock results in increased BUN and creatinine
levels. Impaired perfusion of the liver results in increased liver enzymes, while white blood cell levels do
not typically increase in cardiogenic shock. Red blood cell indices are typically normal because of relative
hypovolemia.
Patients in septic shock require large amounts of crystalloid fluid replacement. Nitrates and β-adrenergic
blockers are most often used in the treatment of patients in cardiogenic shock. Epinephrine is indicated
in anaphylactic shock, and insulin infusion is not normally necessary in the treatment of septic shock
(but can be).
8. C. PaO2 of 38 mm Hg, serum lactate level of 46.5 mcg/dL, and bleeding from puncture sites
Severe hypoxemia, lactic acidosis, and bleeding are clinical manifestations of the irreversible state of
shock. Recovery from this stage is not likely because of multiple organ system failure. Restlessness,
tachycardia, and hypoactive bowel sounds are clinical manifestations that occur during the
compensatory stage of shock. Decreased mean arterial pressure, jaundice, cold/ clammy skin, agitation,
tachypnea, and increased serum creatinine are clinical manifestations of the progressive stage of shock.
9. D
Rationale:
Obstructive shock can be caused by anything that impedes the heart's ability to contract and pump
blood around the body, as with cardiac tamponade.
Rationale:
Circulation and perfusion are addressed first so IV fluids will be started immediately. After blood cultures
are obtained, broad-spectrum antibiotics should be administered without delay. Vasopressors are
administered if the patient is not responding to the fluid challenge. Corticosteroids may be considered
to address the inflammatory-induced vasodilation and capillary leakage.
Rationale:
Vascular access should be established quickly in order to replace lost volume before shock progresses.
Rationale:
Tachycardia is an early symptom as the body compensates for a declining blood pressure the heart rate
increases to circulate the blood faster to prevent tissue hypoxia.
[1/26, 12:45] Dr Augustine Ndaimani: But here we are saying, in shock. Pulse rate is already high, and BP
low. Compensation releases catecholamines (adrenaline etc). this increases heart rate (further) and may
improve BP (increased vasoconstriction increases BP)
18. A. Dobutamine to increase myocardial contractility and Circulatory assist devices such as an
intraaortic balloon pump.
Rationale: Dobutamine (Dobutrex) is used in patients in cardiogenic shock with severe systolic
dysfunction. Dobutamine increases myocardial contractility, decreases ventricular filling pressures,
decreases systemic vascular resistance and pulmonary artery wedge pressure, and increases cardiac
output, stroke volume, and central venous pressure. Dobutamine may increase or decrease the heart
rate. The workload of the heart in cardiogenic shock may be reduced with the use of circulatory assist
devices such as an intraaortic balloon pump or ventricular assist device.
[1/26, 12:54] Dr Augustine Ndaimani: Answer should be A, C. The SPECIFIC treatment for cardiogenic
shock. D is tru but not specific to any one shock type
Rationale: The many deleterious effects of shock are all related to inadequate perfusion and
oxygenation of every body system. This nursing diagnosis supersedes the other diagnoses.
20. Answer D. Increased blood urea nitrogen (BUN) and serum creatinine levels
Rationale: The renal hypoperfusion that accompanies cardiogenic shock results in increased BUN and
creatinine levels. Impaired perfusion of the liver results in increased liver enzymes, while white blood
cell levels do not typically increase in cardiogenic shock. Red blood cell indices are typically normal
because of relative hypovolemia.
Rationale a: Fever will increase oxygen demands but is unrelated to hypovolemic shock unless prolonged
fever has caused severe dehydration, reducing the circulating blood volume. Hypovolemic shock reduces
temperatures by peripheral shunting of blood away from the extremities and reducing the core
metabolic rate.
Rationale b: Due to decreased blood flow to the brain and peripheral areas when blood is shunted to
maintain the vital organs, cerebral hypoxia occurs, leading to a change in mental status.
Rationale d: Due to decreased blood flow to the brain and peripheral areas when blood is shunted to
maintain the vital organs, capillary refill time will be reduced.
Rationale e: Bradycardia is not present. The compensatory response is to increase the heart rate to
circulate the blood faster to make up for the fluids that are not present in hypovolemic shock.
Rationale a: The sodium level in hypovolemic shock is elevated above the normal values of 135 to 145
mEq/L, not reduced.
Rationale b: Metabolic acidosis is present due to an accumulation of carbonic acid, leaving a bicarbonate
deficit from decreased tissue perfusion.
Rationale c: Serum lactate is greater than 4 mmol/L as a result of tissue ischemia, hypoxia, and
breakdown from decreased blood flow with hypovolemic shock.
Rationale d: SvO2 (mixed venous oxygen saturation) would be less than 60% due to decreased
circulating blood volume or decrease in cells to carry the oxygen. Therefore, O2 is carried less efficiently
and decreased, not increased.
Rationale b: Severe constipation does not affect the circulating blood volume.
Rationale c: Third spacing shifts move the fluids from the intravascular space into the interstitial space,
causing a drop in the circulating blood volume. Therefore, third spacing is a risk factor for the
development of hypovolemic shock.
Rationale d: Overhydration does not lead to hypovolemic shock. It leads to fluid overload, which might
cause cardiogenic shock, congestive heart failure, and pulmonary edema.
Rationale a: An increased blood supply would increase color and bounding pulses as seen with
vasodilation (blood engorgement) and is not present with increased peripheral resistance and
vasoconstriction.
Rationale b: Increased peripheral resistance causes the blood supply to decrease and results in
decreased blood to the tissues, which causes pallor and decreased skin temperatures. The pulses would
decrease in intensity with a decreased blood supply.
Rationale c: Venous engorgement would not result from vasoconstriction of the arteries. Strong pulses
would not be present with vasoconstriction from increased peripheral resistance.
Rationale d: Capillary refill times are delayed or slowed due to decreased blood flow through the vessels
caused by the vasoconstriction from increased peripheral resistance.
[1/26, 13:00] Dr Augustine Ndaimani: Its should be given i shock and many other conditions. its like
morphine which is for pain but can be used to improve preload or afterload
[1/26, 13:01] Dr Augustine Ndaimani: or having a drink during supper. The drink may be needed but its
not supper
Rationale B: The patient requires immediate infusion of an adequate amount of fluid. Fluid resuscitation
begins with 500 to 1,000 mL of an isotonic solution.
Rationale C: This is a hypotonic solution and would not help with fluid resuscitation.
Rationale D: This is a hypotonic solution and would not help with fluid resuscitation.
Rationale c: Early identification and correction of the fluid volume deficit in hypovolemic shock is
necessary to prevent cerebral ischemia.
Rationale d: Although physiologic stress can increase the risk for the development of stress ulcers, it is
not considered one of the common or life-threatening complications of hypovolemic shock.
Rationale B: Hypotonic solutions do not stay in the intravascular space long enough to expand the
circulating blood volume.
Rationale C: Hypotonic solutions do not stay in the intravascular space long enough to expand the
circulating blood volume.
Rationale D: The bore size of the needle does not affect the displacement or shifting of fluids.
Rationale A: Hemorrhagic shock is caused by a loss of cells or blood volume and is not a result of infusing
fluids too quickly.
Rationale B: Hypothermia can result when providing room temperature fluids at a faster pace than the
body can warm them.
Rationale C: Bacterial contamination can be avoided by sterile technique, and sepsis is not caused by the
rate or temperature of the fluid being administered.
Rationale D: Cardiogenic shock results from poor ventricular functioning, not from the temperature of
the intravenous fluids being administered too rapidly.
29. Correct Answer: B
Rationale: The initial actions of the nurse are focused on the ABCs, and assessing the airway and
ventilation is necessary. The other assessments should be accomplished as rapidly as possible after the
oxygen saturation is determined and addressed.
Rationale: The patient's history of hyperglycemia (and the associated polyuria), vomiting, and diarrhea is
consistent with hypovolemia, and the symptoms are most consistent with the progressive stage of
shock. The patient's temperature of 97° F is inconsistent with septic shock. The history is inconsistent
with a diagnosis of cardiogenic shock, and the patient's neurologic status is not consistent with
refractory shock.
Rationale: The release of aldosterone and ADH lead to the decrease in urine output by increasing the
reabsorption of sodium and water in the renal tubules. SNS stimulation leads to renal artery
vasoconstriction. -Receptor stimulation does increase cardiac output, but this would improve urine
output. During shock, fluid leaks from the intravascular space into the interstitial space.
Rationale: Restlessness and apprehension are typical during the compensatory stage of shock. Cold,
mottled extremities, cool and clammy skin, and a systolic BP less than 90 are associated with the
progressive and refractory stages.
Rationale: A high cardiac output and low CVP suggest septic shock, and massive fluid replacement is
indicated. Increased PAWP indicates that the patient has excessive fluid volume (and suggests
cardiogenic shock), and diuresis is indicated. Bradycardia and a low systemic vascular resistance (SVR)
suggest neurogenic shock, and fluids should be infused cautiously.
Rationale: The normal sympathetic response to shock/hypotension is an increase in heart rate. The
presence of bradycardia suggests unopposed parasympathetic function, as occurs in neurogenic shock.
The other symptoms are consistent with hypovolemic shock.
Rationale: The patient's low pH in spite of a respiratory alkalosis indicates that the patient has severe
metabolic acidosis and is experiencing the progressive stage of shock; rapid changes in therapy are
needed. The values in the answer beginning "pH 7.48" suggest a mild respiratory alkalosis (consistent
with compensated shock). The values in the answer beginning "pH 7.41" suggest compensated
respiratory acidosis. The values in the answer beginning "pH 7.38" are normal.
Pulmonary artery wedge pressure. Normally 4-12mmHG. an estimate of left ventricular end-diastolic
pressure.
Homework
Can you draw a detailed nursing care plan, using all the provided nursing diagnoses. A nursing care plan
has the following columns: Nursing diagnoses, Client-centred goal, Nursing Interventions, Evaluation
criteria.
In wards we do evaluation. In theory we use evaluation criteria since we will not be dealing with actual
patients.