Cardiovascular Examination – OSCE Guide

geekymedics.com/cardiovascular-examination-2

Dr Lewis Potter September 29, 2010

Cardiovascular examination frequently appears in OSCEs and you’ll be expected to pick

up the relevant clinical signs using your examination skills. This cardiovascular examination
OSCE guide provides a clear step-by-step approach to examining the cardiovascular
system, with an included video demonstration.

Download the cardiovascular examination PDF OSCE checklist, or use our interactive OSCE
checklist. You may also be interested in our paediatric cardiovascular examination guide.

Introduction
Wash your hands and don PPE if appropriate.

Introduce yourself to the patient including your name and role.

Confirm the patient’s name and date of birth.

Briefly explain what the examination will involve using patient-friendly language.

Gain consent to proceed with the examination.

Adjust the head of the bed to a 45° angle.

Adequately expose the patient’s chest for the examination (offer a blanket to allow
exposure only when required and if appropriate, inform patients they do not need to remove
their bra). Exposure of the patient’s lower legs is also helpful to assess for peripheral
oedema and signs of peripheral vascular disease.

Ask the patient if they have any pain before proceeding with the clinical examination.

You might also be interested in our collection of 900+ OSCE Stations, including a range of
cardiovascular examination stations.

1/33
General inspection

Clinical signs
Inspect the patient from the end of the bed whilst at rest, looking for clinical signs
suggestive of underlying pathology:

Cyanosis: a bluish discolouration of the skin due to poor circulation (e.g. peripheral
vasoconstriction secondary to hypovolaemia) or inadequate oxygenation of the blood
(e.g. right-to-left cardiac shunting).
Shortness of breath: may indicate underlying cardiovascular (e.g. congestive heart
failure, pericarditis) or respiratory disease (e.g. pneumonia, pulmonary embolism).
Pallor: a pale colour of the skin that can suggest underlying anaemia (e.g.
haemorrhage, chronic disease) or poor perfusion (e.g. congestive cardiac failure). It
should be noted that a healthy individual may have a pale complexion that mimics
pallor, however, pathological causes should be ruled out.
Malar flush: plum-red discolouration of the cheeks associated with mitral stenosis.
Oedema: typically presents with swelling of the limbs (e.g. pedal oedema) or abdomen
(i.e. ascites). There are many causes of oedema, but in the context of a cardiovascular
examination OSCE station, congestive heart failure is the most likely culprit.

Objects and equipment

Look for objects or equipment on or around the patient that may provide useful insights into
their medical history and current clinical status:

Medical equipment: note any oxygen delivery devices, ECG leads, medications (e.g.
glyceryl trinitrate spray), catheters (note volume/colour of urine) and intravenous
access.
Mobility aids: items such as wheelchairs and walking aids give an indication of the
patient’s current mobility status.
Pillows: patients with congestive heart failure typically suffer from orthopnoea,
preventing them from being able to lie flat. As a result, they often use multiple pillows to
prop themselves up.
Vital signs: charts on which vital signs are recorded will give an indication of the
patient’s current clinical status and how their physiological parameters have changed
over time.
Fluid balance: fluid balance charts will give an indication of the patient’s current fluid
status which may be relevant if a patient appears fluid overloaded or dehydrated.
Prescriptions: prescribing charts or personal prescriptions can provide useful
information about the patient’s recent medications.

2/33
 General inspection

Hands
The hands can provide lots of clinically relevant information and therefore a focused,
structured assessment is essential.

Inspection

General observations

Inspect the hands for clinical signs relevant to the cardiovascular system:

Colour: pallor suggests poor peripheral perfusion (e.g. congestive heart failure) and
cyanosis may indicate underlying hypoxaemia.
Tar staining: caused by smoking, a significant risk factor for cardiovascular disease
(e.g. coronary artery disease, hypertension).
Xanthomata: raised yellow cholesterol-rich deposits that are often noted on the palm,
tendons of the wrist and elbow. Xanthomata are associated with hyperlipidaemia
(typically familial hypercholesterolaemia), another important risk factor for
cardiovascular disease (e.g. coronary artery disease, hypertension).
Arachnodactyly (‘spider fingers’): fingers and toes are abnormally long and slender,
in comparison to the palm of the hand and arch of the foot. Arachnodactyly is a feature
of Marfan’s syndrome, which is associated with mitral/aortic valve prolapse and aortic
dissection.

Finger clubbing

Finger clubbing involves uniform soft tissue swelling of the terminal phalanx of
a digit with subsequent loss of the normal angle between the nail and the nail bed. Finger
clubbing is associated with several underlying disease processes, but those most likely to
appear in a cardiovascular OSCE station include congenital cyanotic heart disease,
infective endocarditis and atrial myxoma (very rare).

To assess for finger clubbing:

Ask the patient to place the nails of their index fingers back to back.

3/33
 In a healthy individual, you should be able to observe a small diamond-shaped
window (known as Schamroth’s window)
When finger clubbing develops, this window is lost.

Signs in the hands associated with endocarditis

There are several other signs in the hands that are associated with endocarditis including:

Splinter haemorrhages: a longitudinal, red-brown haemorrhage under a nail that

looks like a wood splinter. Causes include local trauma, infective endocarditis, sepsis,
vasculitis and psoriatic nail disease.
Janeway lesions: non-tender, haemorrhagic lesions that occur on the thenar and
hypothenar eminences of the palms (and soles). Janeway lesions are typically
associated with infective endocarditis.
Osler’s nodes: red-purple, slightly raised, tender lumps, often with a pale centre,
typically found on the fingers or toes. They are typically associated with infective
endocarditis.

Palpation

Temperature

Place the dorsal aspect of your hand onto the patient’s to assess temperature:

In healthy individuals, the hands should be symmetrically warm, suggesting adequate

perfusion.
Cool hands may suggest poor peripheral perfusion (e.g. congestive cardiac failure,
acute coronary syndrome).
Cool and sweaty/clammy hands are typically associated with acute coronary
syndrome.

Capillary refill time (CRT)

Measuring capillary refill time (CRT) in the hands is a useful way of assessing peripheral
perfusion:

Apply five seconds of pressure to the distal phalanx of one of a patient’s fingers and
then release.
In healthy individuals, the initial pallor of the area you compressed should return to its
normal colour in less than two seconds.
A CRT that is greater than two seconds suggests poor peripheral perfusion (e.g.
hypovolaemia, congestive heart failure) and the need to assess central capillary refill
time.

4/33
Inspect the hands

Check for finger clubbing

Inspect the palms

Check capillary refill time (CRT)

Pallor due to anaemia 2

5/33
Peripheral cyanosis 3

Tar staining on the hands 4

Xanthomata 5

Arachnodactyly

Finger clubbing 6

6/33
Splinter haemorrhages

Janeway lesions 7

Osler's nodes 8

7/33
8/33
Pulses and blood pressure

Radial pulse
Palpate the patient’s radial pulse, located at the radial side of the wrist, with the tips of your
index and middle fingers aligned longitudinally over the course of the artery.

Once you have located the radial pulse, assess the rate and rhythm.

Heart rate

Assessing heart rate:

You can calculate the heart rate in a number of ways, including measuring for 60
seconds, measuring for 30 seconds and multiplying by 2 or measuring for 15 seconds
and multiplying by 4. The shorter the interval used, the higher the risk of obtaining an
inaccurate result, so wherever possible, you should palpate for a full 60 seconds.

For irregular rhythms, you should measure the pulse for a full 60 seconds to
improve accuracy.

Normal and abnormal heart rates

In healthy adults, the pulse should be between 60-100 bpm.
A pulse <60 bpm is known as bradycardia and has a wide range of aetiologies (e.g.
healthy athletic individuals, atrioventricular block, medications, sick sinus syndrome).
A pulse of >100 bpm is known as tachycardia and has a wide range of aetiologies
(e.g. anxiety, supraventricular tachycardia, hypovolaemia, hyperthyroidism).
An irregular rhythm is most commonly caused by atrial fibrillation, but other causes
include ectopic beats in healthy individuals and atrioventricular blocks.

Radio-radial delay

9/33
Radio-radial delay describes a loss of synchronicity between the radial pulse on each
arm, resulting in the pulses occurring at different times.

To assess for radio-radial delay:

Palpate both radial pulses simultaneously.

In healthy individuals, the pulses should occur at the same time.
If the radial pulses are out of sync, this would be described as radio-radial delay.

Causes of radio-radial delay include:

Subclavian artery stenosis (e.g. compression by a cervical rib)

Aortic dissection
Aortic coarctation

Collapsing pulse

A collapsing pulse is a forceful pulse that rapidly increases and subsequently collapses.
It is also sometimes referred to as a ‘water hammer pulse’.

To assess for a collapsing pulse:

1. Ask the patient if they have any pain in their right shoulder, as you will need to move it
briskly as part of the assessment for a collapsing pulse (if they do, this assessment
should be avoided).
2. Palpate the radial pulse with your right hand wrapped around the patient’s wrist.
3. Palpate the brachial pulse (medial to the biceps brachii tendon) with your left hand,
whilst also supporting the patient’s elbow.
4. Raise the patient’s arm above their head briskly.
5. Palpate for a collapsing pulse: As blood rapidly empties from the arm in diastole, you
should be able to feel a tapping impulse through the muscle bulk of the arm. This is
caused by the sudden retraction of the column of blood within the arm during diastole.

Causes of a collapsing pulse

Normal physiological states (e.g. fever, pregnancy)
Cardiac lesions (e.g. aortic regurgitation, patent ductus arteriosus)
High output states (e.g. anaemia, arteriovenous fistula, thyrotoxicosis)

Brachial pulse

Palpate the brachial pulse

Palpate the brachial pulse in their right arm, assessing volume and character:

1. Support the patient’s right forearm with your left hand.

10/33
2. Position the patient so that their upper arm is abducted, their elbow is partially flexed and
their forearm is externally rotated.

3. With your right hand, palpate medial to the biceps brachii tendon and lateral to the medial
epicondyle of the humerus. Deeper palpation is required (compared to radial pulse palpation)
due to the location of the brachial artery.

Types of pulse character

Normal
Slow-rising (associated with aortic stenosis)
Bounding (associated with aortic regurgitation as well as CO2 retention)
Thready (associated with intravascular hypovolaemia in conditions such as sepsis)

Blood pressure

Measure the blood pressure

Measure the patient’s blood pressure in both arms (see our blood pressure guide for more
details).

A comprehensive blood pressure assessment should also include lying and standing blood
pressure.

In a cardiovascular examination OSCE station, you are unlikely to have to carry out a
thorough blood pressure assessment due to time restraints, however, you should
demonstrate that you have an awareness of what this would involve.

Blood pressure abnormalities

Blood pressure abnormalities may include:

Hypertension: blood pressure of greater than or equal to 140/90 mmHg if under 80

years old or greater than or equal to 150/90 mmHg if you’re over 80 years old.
Hypotension: blood pressure of less than 90/60 mmHg.
Narrow pulse pressure: less than 25 mmHg of difference between the systolic
and diastolic blood pressure. Causes include aortic stenosis, congestive heart
failure and cardiac tamponade.
Wide pulse pressure: more than 100 mmHg of difference between systolic and
diastolic blood pressure. Causes include aortic regurgitation and aortic
dissection.
Difference between arms: more than 20 mmHg difference in blood pressure between
each arm is abnormal and may suggest aortic dissection.

Carotid pulse

11/33
The carotid pulse can be located between the larynx and the anterior border of the
sternocleidomastoid muscle.

Auscultate the carotid artery

Prior to palpating the carotid artery, you need to auscultate the vessel to rule out the
presence of a bruit. The presence of a bruit suggests underlying carotid stenosis, making
palpation of the vessel potentially dangerous due to the risk of dislodging a carotid plaque
and causing an ischaemic stroke.

Place the diaphragm of your stethoscope between the larynx and the anterior border of
the sternocleidomastoid muscle over the carotid pulse and ask the patient to take a deep
breath and then hold it whilst you listen.

Be aware that at this point in the examination, the presence of a ‘carotid bruit’ may, in fact,
be a radiating cardiac murmur (e.g. aortic stenosis).

Palpate the carotid pulse

If no bruits were identified, proceed to carotid pulse palpation:

1. Ensure the patient is positioned safely on the bed, as there is a risk of inducing reflex
bradycardia when palpating the carotid artery (potentially causing a syncopal episode).

2. Gently place your fingers between the larynx and the anterior border of the
sternocleidomastoid muscle to locate the carotid pulse.

3. Assess the character (e.g. slow-rising, thready) and volume of the pulse.

Palpate the radial pulse

Assess for radio-radial delay

12/33
Palpate the brachial pulse

Assess for a collapsing pulse

Auscultate the carotid artery

Palpate the carotid pulse

13/33
Jugular venous pressure (JVP)
Jugular venous pressure (JVP) provides an indirect measure of central venous pressure.
This is possible because the internal jugular vein (IJV) connects to the right atrium without
any intervening valves, resulting in a continuous column of blood. The presence of this
continuous column of blood means that changes in right atrial pressure are reflected in the
IJV (e.g. raised right atrial pressure results in distension of the IJV).

The IJV runs between the medial end of the clavicle and the ear lobe, under the medial
aspect of the sternocleidomastoid, making it difficult to visualise (its double waveform
pulsation is, however, sometimes visible due to transmission through the
sternocleidomastoid muscle).

Because of the inability to easily visualise the IJV, it’s tempting to use the external jugular
vein (EJV) as a proxy for assessment of central venous pressure during clinical assessment.
However, because the EJV typically branches at a right angle from the subclavian vein

14/33
(unlike the IJV which sits in a straight line above the right atrium) it is a less reliable
indicator of central venous pressure.

See our guide to jugular venous pressure (JVP) for more details.

Measure the JVP

1. Position the patient in a semi-recumbent position (at 45°).

2. Ask the patient to turn their head slightly to the left.

3. Inspect for evidence of the IJV, running between the medial end of the clavicle and the ear
lobe, under the medial aspect of the sternocleidomastoid (it may be visible between just
above the clavicle between the sternal and clavicular heads of the sternocleidomastoid. The
IJV has a double waveform pulsation, which helps to differentiate it from the pulsation of the
external carotid artery.

4. Measure the JVP by assessing the vertical distance between the sternal angle and the
top of the pulsation point of the IJV (in healthy individuals, this should be no greater than
3 cm).

Causes of a raised JVP

A raised JVP indicates the presence of venous hypertension. Cardiac causes of a raised
JVP include:

Right-sided heart failure: commonly caused by left-sided heart failure. Pulmonary

hypertension is another cause of right-sided heart failure, often occurring due to
chronic obstructive pulmonary disease or interstitial lung disease.
Tricuspid regurgitation: causes include infective endocarditis and rheumatic heart
disease.
Constrictive pericarditis: often idiopathic, but rheumatoid arthritis and tuberculosis
are also possible underlying causes.

Hepatojugular reflux test

The hepatojugular reflux test involves the application of pressure to the liver whilst
observing for a sustained rise in JVP.

Eliciting hepatojugular reflux

To be able to perform the test, there should be at least a 3cm distance from the upper
margin of the baseline JVP to the angle of the mandible:

Position the patient in a semi-recumbent position (45º).

Apply direct pressure to the liver.

15/33
 Closely observe the IJV for a rise.
In healthy individuals, this rise should last no longer than 1-2 cardiac cycles (it
should then fall).
If the rise in JVP is sustained and equal to or greater than 4cm this is deemed a
positive result.
This assessment can be uncomfortable for the patient and therefore it should only be
performed when felt necessary (an examiner will often prevent you from performing it in
an OSCE but you should mention it).

Conditions associated with hepatojugular reflux

A positive hepatojugular reflux result suggests the right ventricle is unable to

accommodate an increased venous return, but it is not diagnostic of any specific
condition. The following conditions frequently produce a positive hepatojugular reflux test:

Constrictive pericarditis
Right ventricular failure
Left ventricular failure
Restrictive cardiomyopathy

Assess the JVP

Assess for hepatojugular reflux

1. 1
2. 2

Face

Eyes

16/33
Inspect the eyes for signs relevant to the cardiovascular system

Conjunctival pallor: suggestive of underlying anaemia. Ask the patient to gently pull
down their lower eyelid to allow you to inspect the conjunctiva.
Corneal arcus: a hazy white, grey or blue opaque ring located in the peripheral
cornea, typically occurring in patients over the age of 60. In older patients, the condition
is considered benign, however, its presence in patients under the age of 50 suggests
underlying hypercholesterolaemia.
Xanthelasma: yellow, raised cholesterol-rich deposits around the eyes associated with
hypercholesterolaemia.
Kayser-Fleischer rings: dark rings that encircle the iris associated with Wilson’s
disease. The disease involves abnormal copper processing by the liver, resulting in
accumulation and deposition in various tissues (including the heart where it can cause
cardiomyopathy).

Mouth

Inspect the mouth for signs relevant to the cardiovascular system

Central cyanosis: bluish discolouration of the lips and/or the tongue associated with
hypoxaemia (e.g. a right to left cardiac shunt)
Angular stomatitis: a common inflammatory condition affecting the corners of the
mouth. It has a wide range of causes including iron deficiency.
High arched palate: a feature of Marfan syndrome which is associated with
mitral/aortic valve prolapse and aortic dissection.
Dental hygiene: poor dental hygiene is a risk factor for infective endocarditis.

Inspect the eyes

17/33
Look for conjunctival pallor

Inspect the mouth

Conjunctival pallor

Corneal arcus

Xanthelasma 9

18/33
Kayser-Fleischer ring 10

Central cyanosis 11

Central cyanosis 14

Angular stomatitis 12

Angular stomatitis 15

19/33
20/33
Close inspection of the chest

Closely inspect the anterior chest

Look for clinical signs that may provide clues as to the patient’s past medical/surgical
history:

Scars suggestive of previous thoracic surgery: see the thoracic scars section
below.
Pectus excavatum: a caved-in or sunken appearance of the chest.
Pectus carinatum: protrusion of the sternum and ribs.
Visible pulsations: a forceful apex beat may be visible secondary to underlying
ventricular hypertrophy.

Thoracic scars

Median sternotomy scar: located in the midline of the thorax. This surgical approach
is used for cardiac valve replacement and coronary artery bypass grafts (CABG).
Anterolateral thoracotomy scar: located between the lateral border of the sternum
and the mid-axillary line at the 4th or 5th intercostal space. This surgical approach is
used for minimally invasive cardiac valve surgery.
Infraclavicular scar: located in the infraclavicular region (on either side). This surgical
approach is used for pacemaker insertion.
Left mid-axillary scar: this surgical approach is used for the insertion of a
subcutaneous implantable cardioverter-defibrillator (ICD).

21/33
 Inspect for thoracic scars

Inspect for lateral thoracotomy scars

1. 1
2. 2

Palpation
Palpate the chest to assess the location of the apex beat and to identify heaves or thrills.

Apex beat

Palpate the apex beat with your fingers placed horizontally across the chest.
In healthy individuals, it is typically located in the 5th intercostal space in the
midclavicular line. Ask the patient to lift their breast to allow palpation of the
appropriate area if relevant.
Displacement of the apex beat from its usual location can occur due to ventricular
hypertrophy.

Heaves

A parasternal heave is a precordial impulse that can be palpated.

Place the heel of your hand parallel to the left sternal edge (fingers vertical) to
palpate for heaves.
If heaves are present you should feel the heel of your hand being lifted with each
systole.
Parasternal heaves are typically associated with right ventricular hypertrophy.

Thrills

22/33
 A thrill is a palpable vibration caused by turbulent blood flow through a heart valve
(a thrill is a palpable murmur).
You should assess for a thrill across each of the heart valves in turn (see valve
locations below).
To do this place your hand horizontally across the chest wall, with the flats of your
fingers and palm over the valve to be assessed.

Valve locations

Mitral valve: 5th intercostal space in the midclavicular line.

Tricuspid valve: 4th or 5th intercostal space at the lower left sternal edge.
Pulmonary valve: 2nd intercostal space at the left sternal edge.
Aortic valve: 2nd intercostal space at the right sternal edge.

Palpate the apex beat

Assess for a parasternal heave

Assess for thrills

23/33
 Heart valve locations

Auscultation

Auscultate the four heart valves

A systematic routine will ensure you remember all the steps whilst giving you several
chances to listen to each valve area. Your routine should avoid excess repetition whilst
each step should ‘build’ upon the information gathered by the previous steps. Ask the patient
to lift their breast to allow auscultation of the appropriate area if relevant.

1. Palpate the carotid pulse to determine the first heart sound.

24/33
2. Auscultate ‘upwards’ through the valve areas using the diaphragm of the stethoscope
whilst continuing to palpate the carotid pulse:

Mitral valve: 5th intercostal space in the midclavicular line.

Tricuspid valve: 4th or 5th intercostal space at the lower left sternal edge.
Pulmonary valve: 2nd intercostal space at the left sternal edge.
Aortic valve: 2nd intercostal space at the right sternal edge.

3. Repeat auscultation across the four valves with the bell of the stethoscope.

Accentuation manoeuvres

4. Auscultate the carotid arteries using the diaphragm of the stethoscope whilst the patient
holds their breath to listen for radiation of an ejection systolic murmur caused by aortic
stenosis.

5. Sit the patient forwards and auscultate over the aortic area with the diaphragm of the
stethoscope during expiration to listen for an early diastolic murmur caused by aortic
regurgitation.

6. Roll the patient onto their left side and listen over the mitral area with the diaphragm of
the stethoscope during expiration to listen for a pansystolic murmur caused by mitral
regurgitation. Continue to auscultate into the axilla to identify radiation of this murmur.

7. With the patient still on their left side, listen again over the mitral area using the bell of
the stethoscope during expiration for a mid-diastolic murmur caused by mitral stenosis.

Bell vs diaphragm

The bell of the stethoscope is more effective at detecting low-frequency sounds, including
the mid-diastolic murmur of mitral stenosis.

The diaphragm of the stethoscope is more effective at detecting high-frequency sounds,

including the ejection systolic murmur of aortic stenosis, the early diastolic murmur of
aortic regurgitation and the pansystolic murmur of mitral regurgitation.

Palpate the carotid pulse to identify the first heart sound

25/33
Auscultate the mitral valve

Auscultate the tricuspid valve

Auscultate the pulmonary valve

Auscultate over the aortic valve

Repeat auscultation of the four heart valves using the bell of the stethoscope

26/33
Auscultate the carotid arteries for radiation of a murmur

Sit the patient forwards and auscultate over the aortic area with the diaphragm of the
stethoscope during expiration

Auscultate over the mitral area

Auscultate the axilla listening for radiation of a mitral murmur

27/33
28/33
Final steps

Posterior chest wall

Inspection

Inspect the posterior chest wall for any deformities or scars (e.g. posterolateral
thoracotomy scar associated with previous lung surgery).

Auscultation

Auscultate the lung fields posteriorly:

Coarse crackles are suggestive of pulmonary oedema (associated with left

ventricular failure).
Absent air entry and stony dullness on percussion are suggestive of an underlying
pleural effusion (associated with left ventricular failure).

Sacral oedema
Inspect and palpate the sacrum for evidence of pitting oedema.

Legs
Inspect and palpate the patient’s ankles for evidence of pitting pedal oedema (associated
with right ventricular failure).

Inspect the patient’s legs for evidence of saphenous vein harvesting (performed as part of
a coronary artery bypass graft).

Auscultate the posterior lung fields

29/33
Palpate for sacral oedema

Assess for pedal oedema

Pitting pedal oedema 13

30/33
To complete the examination…
Explain to the patient that the examination is now finished.

Thank the patient for their time.

Dispose of PPE appropriately and wash your hands.

Summarise your findings.

Example summary

“Today I examined Mrs Smith, a 64-year-old female. On general inspection, the patient
appeared comfortable at rest and there were no objects or medical equipment around the
bed of relevance.”

“The hands had no peripheral stigmata of cardiovascular disease and were symmetrically
warm, with a normal capillary refill time.”

“The pulse was regular and there was no radio-radial delay. On auscultation of the carotid
arteries, there was no evidence of carotid bruits and on palpation, the carotid pulse had
normal volume and character.”

“On inspection of the face, there were no stigmata of cardiovascular disease noted in the
eyes or mouth and dentition was normal.”

“Assessment of the JVP did not reveal any abnormalities and the hepatojugular reflux test
was negative.”

“Closer inspection of the chest did not reveal any scars or chest wall abnormalities. The
apex beat was palpable in the 5th intercostal space, in the mid-clavicular line. No heaves or
thrills were noted.”

“Auscultation of the praecordium revealed normal heart sounds, with no added sounds.”

“There was no evidence of peripheral oedema and lung fields were clear on auscultation.”

“In summary, these findings are consistent with a normal cardiovascular examination.”

31/33
“For completeness, I would like to perform the following further assessments and
investigations.”

Further assessments and investigations

Suggest further assessments and investigations to the examiner:

Measure blood pressure: to identify hypotension, hypertension or significant

discrepancies between the two arms suggestive of aortic dissection.
Peripheral vascular examination: to identify peripheral vascular disease, which is
common in patients with central cardiovascular pathology.
Record a 12-lead ECG: to look for evidence of arrhythmias or myocardial ischaemia.
Dipstick urine: to identify proteinuria or haematuria which can be associated with
hypertension.
Bedside capillary blood glucose: to look for evidence of underlying diabetes mellitus,
a significant risk factor for cardiovascular disease.
Perform fundoscopy: if there were concerns about malignant hypertension,
fundoscopy would be performed to look for papilloedema.

Reviewer

Dr Matthew Jackson

Interventional Cardiology Consultant

References
1. Heart sounds on auscultation (normal heart sounds, aortic stenosis, aortic
regurgitation). Recorded on a Thinklabs Digital Stethoscope (thinklabs.com).
2. Adapted by Geeky Medics. James Heilman, MD. Peripheral pallor. Licence: CC BY-
SA.
3. Adapted by Geeky Medics. James Heilman, MD. Cyanosis. Licence: CC BY-SA.
4. Adapted by Geeky Medics. James Heilman, MD. Tar staining. Licence: CC BY-SA.
5. Adapted by Geeky Medics. Min.neel. Xanthoma in a child. Licence: CC BY-SA.
6. Adapted by Geeky Medics. Desherinka. Finger clubbing. Licence: CC BY-SA.
7. Adapted by Geeky Medics. Warfieldian. Janeway lesions. Licence: CC BY-SA.
8. Adapted by Geeky Medics. Roberto J. Galindo. Osler’s nodes. Licence CC BY-SA.
9. Adapted by Geeky Medics. Klaus D. Peter, Gummersbach, Germany. Xanthelasma.
Licence: CC BY 3.0 DE.
10. Adapted by Geeky Medics. Herbert L. Fred, MD, Hendrik A. van Dijk. Kayser-Fleischer
ring. Licence: CC BY 3.0

32/33
 11. Adapted by Geeky Medics. Ankit Jain, MBBS, corresponding author Anuradha Patel,
MD, FRCA and Ian C. Hoppe, MD. Central cyanosis. Licence: CC BY-SA.
12. Adapted by Geeky Medics. Matthew Ferguson. Angular stomatitis. Licence: CC BY-SA.
13. Adapted by Geeky Medics. James Heilman, MD. Pedal oedema. Licence: CC BY-SA.
14. Adapted by Geeky Medics. Singhai A et al. Journal of Dr. NTR University of Health
Sciences. Methemoglobinaemia. Licence: CC BY-SA.
15. Adapted by Geeky Medics. Oladokun R et al. Atlas of Paediatric HIV infection.
Licence: CC BY-SA.

Copyright © Geeky Medics

33/33