llI'S c;ovt.

Medical College for Women,

Khanpur Kalan
""Da!!tnr•nt of l'hy<,iology
f{t,~piratory Sysh•m
I >atcd: - 24/01/2020
'l'i1nc: J:4S hourB :M ax Marks: 50
I nslruclions:
l. J\JJ qu<·s tion s ,1r<~ con1pulc.;ory
2. l)raw neat and cle;,n dit1gra1n wht•n.~v(•r nc<:cssary
<)I . Wl1t1I i:. J',,0, Bol,r'r; rind J,lnlda1H~':, df,•c PI l<>w 1l11.•y i1n~ b1;!nc:ficil,i" lO
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en. Wf 1:11: r,rc t:111.: effec ts of Ii r,h F'art'inl prc~;s 11n· of l',cisc·!> 011 tltl.! body'? \0
. ,,.. \0
() 1J <)xy li,1< ·1n og lol>111 dissociation t u1vt• 1
i111d th ~l) ,111 1H:,11H:t•
\0
() .r, Lu11g vol111nes ;n 1d cap;!dties with tlwi1 sq 1,nific,uH't•
 • - - ◄ • -.... ..... -• - • __ .. Khanpur
BP~ Govt. Medical College for wornen,
hysiology
At ~Sh.. Department o f P .
temp~ Batch- Cl (01/Q1/ZQ21~
Dra questions. Answer should be brief and to the point.
Wneat&
Q labeled diagram/flow charts wherever necessary· , . •.-.,.
1· ExpJ · ning speea.~
w ain mechanism speech formation by cortical areas concer I ~-;:J!-
,.L , Ord is spoken.
~L . EE.G & sleep.
Waves d uring wakefulness
(}] '
· Short & long term memory.
Q.1. Differenti ate between Dorsal column medicaJ lemniscuses and Anterolc
~.<
---<...>. o·1scuss functions of Basal ganglion.
~:~ List properties of Receptors.
p Write notes on-
a) Astigmatism
b) Place theory of hearing.
 BPS Go, t. Medh:al C cd!~b -~:or , vorn.en , l(hanpur Kala;,, Sonepat
~~~.f:f~ar! 1~1tl_ ·~.LP !~ {.5 ~ o j o ~y
- ·- -...'..:·......·ysheQ[Y
I~(•rve 1\luscil - Te.st
Time: 1:45 min. l\1ax l\,1arks: 50
Instructions: 04/12/2019
1. All q uesti,Jn•~; are corrtp u ::-,or~,
2. Dra"\-V neai· and clean ~Hag~ ;!rd '.•V hl:'.re i; e1 h'2 C ?S•,:uy
Q.1. Giv e a bri.~f account of sUdin.g fil a rnent th~~U{:1 1..;f .(Y'~J bel(-. C':,n ti ~ct1,;n. 10
Q.2.. Compare -& cor trast srn.ooth &: s kel<::t(il !lYLi.~..::le ;~n.:--t.. .~r1-in;.. 10
Ste p s () t 1-reur-0111.u~cu!. 1'
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l\... .1,.ll--·:.. -:.J. . .·•
:..;.l..'1C)D .--:~· i n,g~ -:.Fpctirl.)' r ,e·1.1r01-r, ~('J I,.\. "' c.:-· !1:::tr
-'""" -- _ .. , ... _
Q.3. L_ ' _,. .A. .._ I.J • 'I,,- > 1 • J,,.. - , _ . \ • .J' , :.. . •. -
(6+6="12)
junction .
1'Vr1te·\ -n. 1,..; . -.,~ o·- .
Q •4 • V . 1 ·,I_::, 1:. • ·
6
a. tv·:: .'asthe~_; a G r~·1\'i •:,
6
b. \'\-,}lleria1 · dt.:geri21 :iUOP
6
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 -~-·.:.-- - "I:
}( Ian Sonepat
ap5 Govt. Medical College for Women Khanpttr ad· -1~/02/20
20
II ' Date ·
e· 1.4;hours
. Cardio Vascular System · Max Marks: SO
1itlt ' ctl·OllS··
1nstrU
1 ;\11 questions are compulsory
2.· oraVI neat and dean diagram wherever necessary
Q.l Compare and contrast:-
5
a) Draw cardiac action potential & pacemaker potential curve.
5
b) Starling's law & Starling's hypothesis.
5
\ c) Cardiac output & ejection fraction. 5
d) Laplac(~ law & pouiselle hagens formula.
fl... r. 5
I?-.._
Q.2 a) Name the steps •
dunng cara·iac eye1e.
b) Draw the cardiac cycle showing left ventricle pressure, aortic pressure,
heart second & ECG. 55
Q.3 a) Role of Baroreceptors in short term regulation of blood pressure.
5
b) List the compensatory mechanisms during hemorrhagic shock.
5
c) Circu\atory •
,:hanges during exercise.
th
d) Draw a labelled diagram of normal ECG & give e significance of various
5
. waves, segments and intervals.
f
 TIPS Govt. Medical Cor:cge for Women, Khanpur Kalan
Department of Phyf;io'logy
Part-1
UJ;..1;~1 mi11
M ~
··,tempt alJ que ~ti <Jn ~. Answ,:r '>h<,uld b(: hiicf and to the point.
)J nc~t & la~ ';J ';d ct; ~Jgr,;1rn /f1r>w d1arts wherever nece~sary.
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 BPS GoYt. Medical College for Wo1nen, Khanpur Kalan, Sonepat
~ndocrinology Test Dated: - 20/12/2019
Tin1c: 1:30h.rs ·Max Marks: 50
Instructions:
1. All questions are co1npulsory
2. Ora,,~ neat and clean diagrant wherever necessary
Q.1 List the horn1.onal regulating growth.
&
Discuss the role of Growth hormone & its regulation. 10
Q.7 Discuss the actions of glucocorticoids. 10
Explain why? 5x3=15
Q.3
I. Pigmentation is seen in Addison's disease.
II. Estimation of glycosylated haemoglobin is recommended in diabetes
1nellitus.
Polyphagia is observed in diabetes rr.ellitus eYen though blood
III.
glucose leve1 is high. 5'\.3==15
Q.4 Explain Brjefl y.
I. Regulation of ADI-I secretion.
II. I l ypcrth yroidis1n.
III. 1\ctions of Calcitriol (Vit D1).
 Any ove nnl tln e or rnl fln n
n•~ .

GMC KHANPUR
DEPTT OF PHYSIOLOGY, BPS 5o
TIME = 1hr 45 min/ MARKS
"l e.1 1 HEMATOLOGY TEST /C/ 0)
cct for MC Q a, Q Al: A/B
(wr ite sing le cor rec t/ he~t choice in you r 1111swcr 11h
PA RT A: 10 MARKS
to 1,cro mHrk for tha l MC ()
y o,· enn itin i or cut ting rll', in 1111swl•ri11R wlll lcud
An (I mark]
mnd's discusc?
Al What happens in Von Willcb
------==i DJC T (nc rcascs ~ o t h incrca~ I D]Both decreases
A]BT increases -- (I mark]
phenot ype doe s not hav e:
A2 A person with the Bomb.ty
j B] lILA on leukocytes
jJk!I substance -ffJ Jcadherins
A]glycophorins (I mark]
T cell is ma inly affe cted in IIIV / AIDS ?
A3 Which type of
I C] Memory T cells \ DJ NK cells
,;

A) Cytotoxic T cells I :elper T cells

M'H
(1 mark]
Rh antibody is of wh ich typ e
A4
I BJ IgM I CJ lgD ~ gG
A] IgA 2 marks
A5 Why hyperchromi c RB Cs doe s not exi st? --
rks
in Sickle Cell Anemia 2 ma
y in polypeptide chain of Hb
A6 What is the basic abnormalit 2 marks
in Thala sse mic pat ien ts?
A7 Why HbF is raised

rhart. tahle etc.)

p!a in In De tai l wit h the h('lp. of diagram, flow
PA RT !l: .rn MA RK S (E~
ry.
All questions are compulso
ll01
anemia in detail.
1. Explain megaloblastic
l51
:mmunity (with flow chart).
2. Compler-iem system in
(51
ease of newborn.
3. Explain hemolytic dis
(51
phocytes in immunity.
4. Explain role of'I lym
( S)
iated from Hemophilia?
5. How Purpura is different
(4)
6. Bi concavity of RBCs.
(4)
usion .
7. Autologous blood transf
t2)
nsp lan tat ion rejection.
Wr ite tw o im po rta nt factors to pre ven t organ tra
8.
 DEPTT OF PHYSIOLOGY, BPS GMC KHANPUR
3.11.17 HEMATOLOGYTEST - hr 45 min/ MARKS 50
TIME-1
I' \.RT \: lo 1\1 \.RI\S \" rltr ~ingll' rorrrct / hl'st cholc,• in )Our 11 nswcr ti MCQ llS Q Al: AfB/C/D)
shcc or
An, °' rm ritinR or cutting l'tc In en~" erlng "ill lcud to zero murk for lllllt MCQ
\I 1Wh,lt h,,ppl·ns in \ 'on \\~m d's di~cusc'? - --- --- --- --l
--:, ~t· in~~as~·s ~ BlCl inc11:nscs ICJ both increases ID] both decreases
A.. \\ luc-h ot the foll\,\\ ing cll,tting !'actors is not vitamin K dependent?
>----
·~:__ _ _ _,r=-;:--:;-;----,
A] Fac-t\,r n_ - ~ -- IC] vu ID]IX
,\) ,\ persN\ w1th the Hombny phenotype docs nc,t hnve:
·--- ---. ---- ---- --r: =-: .--~ :--- 1
\ ]gl) c-ophorin:.- \ B] IlLA on lcukc,cytes substance \pr,fl
D]cadherins I
A4 Which 1n,c of r cell is mai~1ly affected in lllV /AIDS?
. A] C\ toto,k r cells ~clp cr T cells \ C) Memory T cells \ D] Suppressor T cells
\5 V
,~:.unc the immunoglobulin which can pass through Placenta
~gG B]IgM I IC]IgA ID] lgE
A6 The test that screens the Intrinsic pathway is: --L----------L-------1
A]APT T B]PT \ C] thrombin time \ D] clot lysis time
Ai Define Hapten .
AS What is the basic abnormality in polypeptide ,:hain of Hb in Sickle Cell
Anemia?
\ A9 What is the etiological cause ofThalassemias"' --- 2 marks

PART B: 40 .M ARKS (Expla in In Detail with the help of diagra

m, flowchart, table etc.)

All questions are compulsory.

I. Write Wintrobe's classification of Anemia. Explain briefly the megalo
blastic anemia . ~
{j) Explain functions of plasma protein. (51
(

/4' Explain hemolytic disease of newborn. (5] 2-, 7

Explain Anti-coagulants. lo
(5)

5. Compare active and passive immunity. (5]

~ - Biconcavity of R.BCs. (3)

_;r. Autologous blood transfusion. l3)

__/. How Purpura can be differentiated from Hemophillia by investi
gations? l 2)
j'. Write two important factors to prevent organ transp§.!}!ation rejection . l2)

'I
 4iiii
10.0
l,l7 .. EMATOLOGY TEST
DEPTT o, PHYSIOloe,, BPS GMC KHANPUf':11 Y-[~
TIME =1hr 4S min
/. / MARKS 50
PA
1
R.T A•• 10,.•11\Rf\s (\\ rite ~inglt> l'Otl'f,•t / ht> 8t ,•hol,·,, in ) our 111~,wr ~h1•1•1 for M( () .~QA I·· A/11/('/l)J
1 11
-Al -.,_ Any O\trwruing or l'Ultlntt tk, In 11n~\\rrlnll
. ,. Ill kiul to 11,., mntk for lh111 M( '(
~
0
Whut h,,pp1.•1 sin\""\\ ilkb111111.1's dlSl'llW''

~L~
--;- \lla~ ll\(')'I,', :-,•s
A~ ----4--
1
--~-r- BI(' ( in1.·1 cuscs I(' . I
j both IJ1l'rc11scs I) I Imt Il tIC·cri.:'ISCS
'.

\\ ht,·h \,f th,, !'t_,\1\ \\ ing dotting lhl.'t11rs is nnt vitn111in Kdq1cnd1.·111'
1
A . -,,~ - I n1 v ] c1v~ [ Ill IX -·--~
.) \~~'1th the Bomba) phenotype docs Ill t huvc: ---~ /

A •t Algl~cophorins
\' 'h• h - -
J n1111 A on kiuk, C'Vft•s T1('I II SllhSIHl11'1' lf)lrnrlh,:rin~
J.
~ '" 1c type of r cell is mainly nffccted in 111 1, / /\ll)S? ·

-;-A.,,.]-C:-y-to-to-xic Teell, J:
OJ IIOlpec r cells ] CJ Mcmo,yr cell, ] Ill"""""'" I ecll,
AS Atrophic gastritis ultimately can result in:
A] Fe deficienc) -
. ---
BlFolatc deficiency
- Dj Thalassc.:mia
A6 \Vh~ hyperch-omic RBC's does not exist?
A7 ----- --
Define Adjuvant.

--4----------~:---:-:-- ----------
A8 What is the et,ological cause of hereditary sp,1erocytosis? _________ ~
A9 What is the etiological cause ofThalassemias? (2 marks):

PART B: 40 MARK-; (Explain In Detail with the ' 1clp of diagram. flowchart, tahl" etc.)
All questions ar,· compulsory.

--
l. Class 1"f) Anem1a. .
.. Explain

2. Fate of Hb. Explain hemolytic disease of newb11rn.

·, pr~-v-ilrnl
brieth. the most con mon ancm1,1 ' in India

[3+51

3. Explain Anti-c0agulants. (6)

4. ..
A) Enlist two types of cond1t1ons where passive immunity is used.

B) Complemen system in immunity (onl y tlo\\ i.: hart ). ~2+4)

5. Biconcavity of RBCs.

~
Autologous blood traosfus;oo. I.' • \I, I 'I
6. . differentiated from HemophilH, h1lfo,esUgaUoosl
7. How Purpura is . ·ectlon (2)
t organ transplantation reJ .
8. Write two importan t factors to preven