ELECTROCARDIOGRAM

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ELECTROCARDIOGRAM

Dr. NCHINDO PIUS – MARY


INTERNIST MD(Hons) DES.
Objectives

• List 3 criteria for identifying a sinus rhythm

• Identify a normal ECG

• Describe the main criteria for identifying ventricular hypertrophies

• Illustrate the signs of coronary artery disease

• Identify atrial fibrillation


Plan
• Normal ECG

• Atrial and ventricular hypertrophies

• Subepicardial ischemia, myocardial necrosis

• Atrial fibrillation and atrial flutter

• Extrasystoles

• Ventricular tachycardia

• BAV, bundle branch blocks


ECG Basics
Normal impulse conduction
ECG Basics
ECG Basics
ECG Basics
Pacemakers of the heart
• SA Node - Dominant pacemaker with an intrinsic rate of 60 - 100
beats/minute.

• AV Node - Back-up pacemaker with an intrinsic rate of 40 - 60


beats/minute.

• Ventricular cells - Back-up pacemaker with an intrinsic rate of 20 - 45


bpm.
ECG Basics - The ECG paper
ECG Basics - Rhythm analysis

• Step 1: Calculate rate.


• Step 2: Determine regularity.
• Step 3: Assess the P waves.
• Step 4: Determine PR interval.
• Step 5: Determine QRS duration.
ECG Basics - Rhythm analysis
ECG Basics - Rhythm analysis
ECG Basics - Rhythm analysis
ECG Basics - Rhythm analysis
ECG Basics - Rhythm analysis
ECG Basics - Rhythm analysis
ECG Basics - Rhythm analysis
ECG Basics - Rhythm analysis
ECG
• Normal
• Rhythm
• Frequency
• P Wave
• P-R Interval
• QRS Complex
• ST Segment
• T Wave
Normal ECG
• Sinus rhythm: P wave precedes each • QRS complex: duration 70 to 90 msec, axis

QRS, PR interval ≥ 0.12 s, positive P wave • ST segment: end QRS - start T, isoelectric
in DI DIII AVF
• T wave: monophasic, positive and
• Frequency: number of beats per minute asymmetric
(bpm), 60-100 bpm, • QT interval: start QRS – end T, normal time

• P-wave: round, duration ≤ 0.12 s, = 380 to 420 msec,

amplitude ≤ 2.5 mm, axis: 0 to 90° • QTc = QT/√RR < 440 msec

• PR interval: between 120 and 200 msec


Normal ECG values
Left atrial hypertrophy
• Left atrial overload or left atrial hypertrophy
• Occurs in mitral stenosis, hypertension, HF
• It results in an increase in the duration of the P wave in DII ≥ 120 ms
with often a bifid appearance
• In V1 there is a predominance of the terminal negative component of
P
• LAH Moritz criteria: 2 out of 3 criteria are sufficient
Left atrial hypertrophy
Left atrial hypertrophy
• In DII and V1
• Axis < 30°
• Duration > 120 msec
• Bifid in DII
• Negative
Right atrial hypertrophy

• Atrial overload or right atrial hypertrophy (HAD)

• Etiologies – COPD, chronic cor pulmonale, acute cor pulmonale

• Increased P-wave amplitude > 2.5 mm in DII

• Predominance of the positive initial part of the P wave in V1


Right atrial hypertrophy
Right atrial hypertrophy
• In DII and V1
• Right axis deviation,
• Pointed P-wave in DII and
Amplitude> 2.5 mm
Left ventricular hypertrophy
• Sokolow – Lyon criteria
RV5 + SV1 > 35 mm
• Cornell index
RaVL + SV3 > 28mm (♂)
and > 20 mm (♀)
Left ventricular hypertrophy
Right ventricular hypertrophy
• R/S > 1, V1 V2
• S ample V5 V6
• Right axis deviation
Sub endocardial ischemia
Sub epicardial necrosis
• ST-segment elevation = myocardial infarction
Topographic terminologies
• Anterior ischemia or necrosis : V1 to V6

• Antero-septal ischemia or necrosis : V1 to V3

• Apical Ischemia or necrosis: V4 and V5

• Anterolateral ischemia or necrosis: V4 to V6

• inferior ischemia or necrosis: D2, D3, aVF

• Reciprocal changes
Extensive anterior Myocardial Infarction
Anterior myocardial infarction
Inferior myocardial infarction

Sus-décalage ST en DII, DIII, aVF avec miroir en DI, aVL


Possible extension au ventricule droit 36
Posterior myocardial infarction
Myocardial infarction
electrical chronology
Atrial fibrillation
Atrial flutter
Atrial extra systoles
Ventricular extra systoles
bigerminy, trigerminy
Ventricular extra systoles
duplet
Ventricular tachycardia
Atrioventricular blocks
• These are conduction disorders located at the level of the atrioventricular
junction

• either at the level of the atrioventricular node: nodal or "suprahisian"


block,

• or at the level of the trunk of the bundle of his: truncular or "intra-hisian"


AVB, or

• at the level of the dividing branches: infra-hisian AVB

• The AVB can be of very variable degree, ranging from a simple slowing
down of conduction to its complete interruption
First degree AV Block
• First-degree BAV: most
often NODAL
• A-V conduction slowing
is expressed by a
prolongation of the PR
interval beyond 0.20 s
AVB II, Mobitz Type I, with Wenckebach Period
• Gradual lengthening of the PR interval, until a blocked P wave occurs
and the cycle begins again
2nd degree AVB: Mobitz type II
• Unexpected occurrence of a blocked P wave without prior
prolongation of the PR interval.
AVB 3: 3rd degree block or complete AVB
block
• No P waves are conducted to the ventricles Blocking all P waves Total
dissociation between atria and ventricles
AVB 3: 3rd degree block or complete AVB
block
Right bundle branch block
• Interruption of conduction on the right branch of the HIS fibres

• Aspect rSR' or rsr' or rsR' in V1,V2.

• Sometimes qRR' in V1

• Deep and serated S wave in V5,V6; DI,aVL

• QRS Widening: Complet RBB if QRS ≥ 120 ms; Incomplete RBB if QRS
[100-120[
Right bundle branch block
Left bundle branch block

• Interruption of conduction on the left branch of the HIS fibres

• Broad, crocheted R-wave or RR' aspect in V5,V6; DI,aVL

• Disappearance of the septal q wave in V5,V6; DI,aVL

• Large S-wave or QS aspect from V1 to V3 QRS

• Enlargement: complete LBB if QRS ≥ 120 ms; Incomplete LBB if QRS


[100-120[
Left bundle branch block

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