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    Shock Said 20-21

    Uploaded by

    2859bathina
    Buthaina Moza Husain Amal Said

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    © All Rights Reserved
    Download as PDF, TXT or read online from Scribd

    Shock Said 20-21

    Uploaded by

    2859bathina
    8 views10 pages
    Buthaina Moza Husain Amal Said

    Original Description:

    Shk

    Original Title

    Shock Said 20-21.pptx

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    © © All Rights Reserved

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    Buthaina Moza Husain Amal Said

    Copyright:

    © All Rights Reserved
    Download as PDF, TXT or read online from Scribd
    Download as pdf or txt
    8 views10 pages

    Shock Said 20-21

    Uploaded by

    2859bathina
    Buthaina Moza Husain Amal Said

    Copyright:

    © All Rights Reserved
    Download as PDF, TXT or read online from Scribd
    Download as pdf or txt
    of 10

    Said AL Harthy

    Reflect on a
    patient with
    shock you have What signs and
    treated and symptoms were
    relate the stages occurring.
    of shock to this
    patient.
    Activity
    What actions
    In which stage of were taken to
    shock was the stop the next
    patient stage of shock
    from occurring.
    Learning outcomes:

    By the end of this session the students will be able to:


    ∙ Define shock
    ∙ Differentiate between the stages of shock
    ∙ Discuss the pathophysiology, the causes and signs and
    symptoms of hypovolemic, cardiogenic, neurogenic and
    anaphylactic shock
    ∙ Describe the treatment options available for each type of
    shock
    ∙ Explain nursing interventions necessary for each type of
    shock
    Shock is an acute state in which
    tissue perfusion is inadequate to
    maintain the supply of oxygen and
    nutrients necessary for normal cell
    function. (Alexander et al 1994)

    Shock
    Shock is a condition where the
    cardiovascular system fails to perfuse
    the body tissues adequately, bringing
    about a widespread disruption of
    cellular metabolism, resulting in
    functional disturbances at
    organ/tissue level.
    ► Deficiency of blood supply
    ► Oxygen deprivation
    ► Buildup of waste product
    ► Organ failure

    ► Shock caused by any factor which affects


    ► Blood volume
    ► Blood pressure
    ► Cardiac function
    Initial stage

    ► Cells deprivation of oxygen


    ► The mitochondria can no longer
    continue to produce adenosine
    triphosphate (ATP)
    ► Cell membranes become damaged.
    ► Production of lactic and pyruvic acids
    causes
    ► Systemic metabolic acidosis.
    Compensatory stage
    (Non-progressive)
    Physiological Adaptation
    ► Reduced cardiac output • Baroreceptors
    • Sympathetic
    ► Release of adrenaline and noradrenaline nervous
    ► Vasoconstriction system
    ► Increase in BP and HR
    ► Hyperventilation
    ► General blood flow is reduced
    ► Oliguria might develop.
    De-compensated (Progressive
    stage)
    Failure of compensatory mechanisms
    ► Anaerobic metabolism continues
    ► Increase the body’s metabolic acidosis
    ► Hypotension/Hypoxic injury
    ► Persistent hypotension
    ► Increase capillaries permeability

    ► Brain = confusion, altered mental status,


    ► Heart = MI
    ► Renal = Acute tubular necrosis, renal failure U/O less than 20 ml
    ► Gastro = absence peristalsis, GI ulcers and GI bleeding
    ► Liver = Hepatic failure
    ► Resp. = ARDS
    ► Blood Vessels = DIC
    Refractory stage (Irreversible)

    ► Organs failure (MODS)


    ► Death will occur within a few hours.
    Activity:

    1 2 3 4 5 6 7

    Topic Stages Hypovolemic Cardiogenic Septic Anaphylactic Neurogenic Obstru


    of Obstructive ctive
    shock Shock

    Said Said Buthaina Moza Husain Amal Said

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