Cellular Respiration
Cellular Respiration
Cellular Respiration
x 45 o ¥s
- 0 s
I . @ I 3
↳ D8 ± E E
= .
i
- GOP
.
⇐ ⇐ ÷ , ±
.
.
N
¥ D W O O Z
3 .
s D w g
pas
S I
Q ¥ §
E .
= -
g e.§
• . .
* .
.
E D
S 3
-
"÷÷¥¥?÷ sin ,
. -
n
←5¥¥ a O
a. E of a EES Tl
u -0
, I T
*¥ p
.
. F
' t
↳
¥÷÷÷*
¥¥¥
§
:* age
? I
c
8
%
§•¥•¥ I s
f
C At
o e -
o
D
3 '
- a o # o
I t
is p
. :
§;
§ .
a o o u
s '
f
I . I got * of I -0
-
O I
.
8 ④ 88 88¥ E
t - f
S go ±§ 58g too it is X
s O
133 03 to 7- g) a Iro
-02 9 o -1
a *I II. + s D
' so 9 or so @
3
39C
s
-05 .
E
,
=
① I @
u @ I ,
1-
↳ no ⇐ 98 -
x
.
5 I. § s 9- § go -0
% .
e
Ex 95 GI O
a
¥S← e
.
51 l e )
e
:
i¥÷ ¥÷¥ ÷ ÷ ⇐ ¥ ⑧¥
4. KE
nd 's •
I
E
%
,
§',
am
Es
ex
4 .
I
Acid Based
Amino
-
( H2O *
) ( polar
Hormones * Hydrophilic "
• .
.
.
sensitive
"
•
•
.
[ LOTS of
• a receptors
'
y
O
°o° o
. cemllembrane Fewer -_ LEI
)
receptor receptors
'
(ex
→ what EXOCRINE .
C
mucus
O
off,woNtaameDmY5Iane Hormones
chemical
°
era
mend:EF¥
- .
are that IN
polar
membranes messengers the
* non -
jmcw ,
travel
bloodstream
in
Hormones
steroid •→
*
3
- thyroxine
Estrogen
'
Testosterone
Progesterone Aldosterone
-
cortisol
'
Glucagon
Glucose VESSEL aldosterone
Idarathyro ;¢ BLOOD °
Epinephrine
"
mo
amid for
'
) µ
(EPO B. P .
Insulin
calcitonin natriuretic
(Thyroid)
Breakdown * * Hypothalamus peptides
Fat kidneys
71310001 Glucose
Inflammation milk production
maturity release
COR-ts.CL# sexual3
,
Reproduction -
-
prolactin
Oxytocin
LH
FSH
+
Repair
estrogen
cell Lengthening
progesterone
* Bone Growth
muscle
Testosterone Growth
Hormone
URINARY SYSTEM
'
kidneys*
:
Excretes nitrogenous wastes & filters blood +
produce urine
creatinine uric acid urea -
, ,
'
' TOO
pathogens
HIGH ?
'
could be UTI
Electrolyte Balance
-
*
* Nat
,
cast , Kt etc ) .
WBCIRBCI Platelets +
protein
,
"
Modifies / Deactivates meds NEVER FILTERED
*
-
sign of
kidney dmglinfxn
Abdominal Aorta Glucose is FILTERED t
REABSORBED
vgnavaas ✓
is too HI
1 V Blood
Renal sugar
Rejoins
Artery
ur
aka diabetes
segmental B
arteries @5)
V .
g.
Arcuate arteries
Aa
AN
'
* constricts
afferent arteriole -
many FIGHT
during
or
FLIGHT .
2 blood
REDUCES
L
*
flow t
in kidneys
filtering
Bed site of filtration
Glomerulus capillary
.
>
Raises B. P
ye¥tere
.
- -
systemically
✓
Peri tubular
Bed
capillary
venules s
CHIEF ) RESPIRATORY
contains HI
Tonsils :
conct .
Of WBC to
lhfxn
fight
sinuses
:
righteous
ON inflamed → sinus pressure
*
✓
nmoid u
sphenoid
sin
"
tonsils :
,
adenoid
sinus L -
enlargeder
can cause
wt .
gain
) BB
passage
food
nasal Palatine Tonsils
UVULA : Blocks can live here
> mouth cavity from
entering -
s.
pyogenes
Tonsils
Eustachian muscular Lingual
Tube pressure
ear
Egvilizesmiddle enter opening tongue
* to can THIS
leads
pathogens
via
ear
-
middle
-
Epiglottiscartilage
Thyroid * elastic choking
cartilage# am
's
p - prevents
Apple •
J*
Esophagustube
* muscular
Tracheal :
cartilages
protects
Hyaline trachea Trachea
holds
-
{ * windpipe
-
Hyaline cartilage
cords
* vocal
elongates during puberty
a voice
LOWER
causing inflammation
of the
Laryngitis
=
Larynx
)
( LOWER RESP .
TRACT
)
Box
Y IE Apple
"
's
" Adam
Thyroid cartilage
Trachea
divided into 2-3 lobes Hyaline cartilage
•
Lungs
.
reinforcing
lobe contains several
Each Bronchi
bronchopulmonary segments by primary
elastic CT
to
( stretchy help
easily) bronchi bronchioles
.
Pleura :
Parietal
•
to chest wall
Attached
cavity when chest moves outward,
layer •
w/ it
fluid
.
-
thin
serous layers •
HOLDS
2
friction , Diaphragm visceral Pleura :
together
-
attached to
PREVENTS size
that t when
are * muscle
. .
INHALED
when relaxed ,
lungs deflate lungs
lungs
-
of RFACTANT #
changing
when go fluid that DEC
soapy Islip pen,
.
*
tension i "
-
inflammation
"
surface the
""
pespifgartoonrchioles
humid alveolus
*p
OR
ALVEOLUS
simple
pleuraterminal squamous
Bronchioles 02
•
#
epithelium
A macrophage
fights infxn
re
's
membrane
,5¥Ee *
cos
ALVEOLUS .
thin
RBC Endothelial
"
cells
"
grape can
-
thicken
* site
of
gas "
disease processes
exchange
has a
-
Each alveolus
next to it
capillary
.
02-1 capillary
Alveolus C-
CO2