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BURN Note

This document discusses burns, including: 1. Burns are injuries to the skin or body caused by heat, flames, hot objects, chemicals, or electricity. Burn depth is categorized as first, second, or third degree depending on the severity of tissue damage. 2. Treatment depends on the depth, size, and location of the burn. Options range from applying a cold pack for minor burns to emergency care for severe burns. 3. Nursing care for burn patients involves monitoring vital signs, fluid replacement, pain management, infection prevention, wound care, and exercise/positioning to prevent contractures. Proper treatment can help prevent complications and promote healing.

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Clarence Bravio
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0% found this document useful (0 votes)
12 views3 pages

BURN Note

This document discusses burns, including: 1. Burns are injuries to the skin or body caused by heat, flames, hot objects, chemicals, or electricity. Burn depth is categorized as first, second, or third degree depending on the severity of tissue damage. 2. Treatment depends on the depth, size, and location of the burn. Options range from applying a cold pack for minor burns to emergency care for severe burns. 3. Nursing care for burn patients involves monitoring vital signs, fluid replacement, pain management, infection prevention, wound care, and exercise/positioning to prevent contractures. Proper treatment can help prevent complications and promote healing.

Uploaded by

Clarence Bravio
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© © All Rights Reserved
We take content rights seriously. If you suspect this is your content, claim it here.
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BURN site of thrombosed veins), without blisters (3rd

degree burn).
 Damage to the skin or other body parts caused  Silver-colored, raised or charred area, usually at
by extreme heat, flame, contact with heated the site of electrical contact.
objects, or chemicals.
 Burn depth is generally categorized as first,
second, or third degree.
 The treatment of burns depends on depth, area,
and location of the burn, as well as additional
factors, such as material that may be burned
onto or into the skin.
 Treatment options range from simply applying
a cold pack to emergency treatment to skin.
 It is also an alteration in skin integrity resulting
in tissue loss or injury caused by heat, chemicals,
electricity or radiation.

 Etiology

Types of burn injury

1. Thermal- results from dry heat (flames) or moist heat


(steam or hot liquids); it is the most common type; it
causes cellular destruction that results in vascular, bony,
muscle, or nerve complications; thermal burns can also Burn depth:
lead to inhalation injury if the head and neck area are
affected.  Superficial thickness burn (1st degree)- mild to
severe erythema of skin, blanches with pressure;
2. Chemical burns- are caused by direct contact with heals in 3 to 7 days
either acidic or alkaline agents; they alter tissue  Partial thickness types of burn
perfusion leading to necrosis. Burn (2nd degree)- large blisters; painful,
heals 2-3 weeks
3. Electrical burns- severity depends on type and Full thickness burns (3rd degree)- white,
duration of current and amount of voltage; it follows yellow, deep red to black (eschar)
the path of least resistance (muscle, bone, blood vessels, disruption of blood flow, no pain ;
and nerves); sources of electrical injury include current, scarring; and wound contractures will
alternating current and lightning. develop. Grafting is required; healing
takes weeks to months
4. Radiation burns- are usually associated with sunburn Deep full thickness burn (4th degree) –
or radiation treatment for cancer; are usually involves injury to muscle and bone=
superficial; extensive exposure to radiation may lead to appears black (eschar)- hand and
tissue damage. inelastic healing takes weeks to months;
grafts are required.
Pathophysiology
Diagnostics
-it depends on the cause and classification of the burn;
the injuring denatures cellular proteins; some cells die  Rule of Nines chart determines the percentage
because of traumatic or ischemic necrosis; loss of of body surface area (BSA) covered by the
collagen cross-linking also occurs with denaturation, burn.
creating abnormal osmotic and hydrostatic pressure
gradients that causes intravascular fluid to move into
interstitial spaces; cellular injury triggers the release of
mediators of inflammation, contributing to local and in
the case of major burns, systemic increases in capillary
permeability.

Clinical manifestations
 localized pain and erythema, usually without
blisters in the first 24 hours (first degree burn)
 chills, headache, localized edema, nausea and
vomiting (most severe first-degree burn)
 thin-walled, fluid filled blisters appearing within
minutes of the injury, with mild to moderate
edema and pain (second degree superficial
partial thickness burn)
 white, waxy appearance to damaged area
(second degree partial-thickness burn)
 white, brown or black, leathery tissue and
visible thrombosed vessels due to destruction of
skin elasticity (dorsum of hand, most common
 Irrigate the wound with amounts of water
or normal saline solution or decreased
responsiveness
 Irrigate the wound with amounts of water
or normal saline solution for chemical burns
 Place the patient in semi-Fowler’s position
to maximize chest expansion; keep patient
as quiet and comfortable to minimize
oxygen demand
 Administer rapid fluid replacement therapy
as ordered

 For burn patient in shock:


 Monitor v/s and hemodynamic
parameters
 Assess patient’s intake and output every
hour, insert an indwelling catheter
 ABG level may be normal in the early stages  Assess the patient’s level of pain,
but may reveal hypoxemia and metabolic including nonverbal indicators and
acidosis. administer analgesics such as Morphine
 Carboxyhemoglobin level may reveal the Sulfate IV as ordered
extent of smoke inhalation due to the presence  Keep the patient calm, provide periods
of carbon monoxide of uninterrupted rest between
 Complete blood count may reveal a decrease procedures and use nonpharmacologic
haemoglobin due to hemolysis, increased pain relief measures as appropriate
haematocrit and leukocystosis.  Obtain daily weights and monitor
 Electrolyte levels show hyponatremia and intake, including daily calorie counts;
hyperkalemia, other laboratory tests reveals provide high calorie, high protein diet
elevated BUN, decreased total protein and  Administer histamine 2 receptor
albumin antagonists are ordered to reduce risk of
ulcer formation
 Creatinine kinase (CK) and myoglobin levels
 Assess the patient’s sign and symptoms
may be elevated
of infections; may obtain would culture
 Presence of myoglobin in urine may lead to and administer antimicrobials an
acute tubular necrosis. antipyretics as ordered
 Administer tetanus prophylaxis if
Nursing Diagnosis indicated
 Risk for deficient fluid volume  Perform burn wound care as ordered;
 Risk for infection prepare patient for grafting as
 Impaired physical mobility indicated.
 Imbalanced nutrition: less than body  Assess the neurovascular status of the
requirement injured area, including pulses, reflexes,
 Ineffective breathing pattern paraesthesia, colour and temperature of
 Impaired tissue perfusion the injured area at least 2 to 4 hours or
 Risk for ineffective thermoregulation more frequently as indicated
 Pain  Assist with splinting, positioning,
compression therapy and exercise to the
 Impaired skin integrity
burned area as indicated; maintain the
burned area in a neutral position to
Nursing Management
prevent contractures and minimize
 Assess patient’s ABCs; monitor arterial deformity.
oxygen saturation and serial ABG values  Explain all procedure to the patient
and anticipate the need for ET intubation before performing them
and mechanical ventilation
 Auscultate breath sounds Pharmacotherapy
 Administered supplemental humidified  Antibiotic prophylaxis will eradicate
oxygen as ordered bacterial component
 Perform oropharyngeal or tracheal  Pain therapy
suctioning as indicated by the patient’s  Tetanus prophylaxis
inability to clear his airway  Topical antimicrobial
 Monitor the patient’s cardiac and  Enzymatic debriding agents such as
respiratory status collagenase, fibrinolysis
 Assess LOC for changes such as confusion, indesoxyribonuclease papin or sutilins
restlessness or decreased responsiveness are used with a moisture barrier to
protect surrounding tissue
 Recommended dressings include
polyurethane films (Op-site,
Tegaderm), absorbent hydrocolloid
dressings (Douderm).

Burn Medications
 NItrofurazone (Furacin)- broad spectrum
antibiotic ointment or cream – used when
bacterial resistance to other drugs is a
problem: apply 1/16 inch thick film directly
to burn.
 Mafenide (Sulfamylon)- water soluble
cream bacteriostatic gr+- bacteria: apply
1/16 inch directly to burn- notify physician if
hyperventilation occurs as this drug
precipitate metabolic acidosis
 Silver sulfadiazine (Silvadene) – cream
borad spectrum gr+- ; does not cause
metabolic acidosis- keep burn covered at
all times with Sulfadiazine (1/16 inch thick);
monitor CBC- causes leukopenia
 Silver Nitrate- antiseptic solution against
gr-, dressings are applied to the burn and
then kept moist with Silver Nitrate: used on
extensive burns that may precipitate fluid
and electrolyte imbalance

 Parkland (Baxter) Formula for fluid


replacement
 4 ml Lactated Ringer’s sol x kg body
mass x total percentage of body
surface burned
 1st 8 hours= ½ of total 24-hours fluid
replacement
 Next 8 hrs= ¼ of total
 Last 8 hrs= ¼ of total

Client education
 Environment safety: use low temperature
setting for hot water heater, ensure access to
and adequate number of electrical
cords/outlets, isolate household chemicals,
avoid smoking in bed
 Use of household smoke detectors with
emphasis on maintenance
 Proper storage and use of flammable
substances
 Evacuation plan for family
 Care of burn at home
 Signs symptoms of infection
 How to identify risk of skin changes
 Use of sunscreen to protect healing tissue
and other protective skin care

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