Gastro
Gastro
Upper GI Hernias
- incomplete formation of the esophageal -Cutaway View of Hiatal Hernia
lumen, resulting in the proximal (upper)
esophagus forming a "blind pouch". - Protrude to the hiatus
Type C- Proximal atresia Distal fistula 85% (most Type 3- Combined type 1 and 2
common) Type 4- complex paraesophageal hiatal hernia
Type D- Double fistula with oesophageal atresia (whole stomach is pushed up into the chest)
1%
Type H- Isolated fistula H-type 4% Assessment
-heartburn (after meal)
Assessment -abdominal pain
-Hydramnios -burping or hiccups
-Prematurity -vomiting or spitting up
-Other possible congenital anomalies: verbal, -coughing or hoarseness
anal, cardiac, tracheoesophageal, renal, and
limb anomalies -fullness of bloating
Complications
Assessment
-Colon Cancer
-Intense crying and pain
-Skin, eye and joint inflammation
-Pulling up the legs
-Medication side effects
-Abdominal distention
-Primary sclerosing cholangitis
-Vomiting(30mins or an hour after eating)
-Blood clots
-Lower Barium Xray (diagnosis)
-Severe dehydration
Therapeutic Management
Diagnosis
-Surgery: Bowel resection with anastomosis.
-a combination of endoscopy (Crohn's Disease)
or colonoscopy (ulcerative colitis) and imaging
studies, such as:
H. Celiac Disease
-Contrast radiography
Assessment
-Magnetic resonance imaging
-An immune-mediated abnormal response to
-Computed tomography gluten
-Stool samples
-when children with the disorder ingest gluten, * Passive artificial immunity: immune globulin
flattening of the fingerlike projections (villi) of
the small intestine occurs, preventing the
absorption of foods Hepatitis B
*Causative agent: Hepatitis B Virus (HBV)
-If left undiagnosed: steatorrhea, failure to * Incubation period: 120 days on average
thrive, and malnutrition.
* Period of communicability: Later part of
incubation period and during the acute stage
Assessment * Mode of transmission: Blood and body fluids
-Poor growth * Immunity: one episode includes immunity for
-Bulky stools the specific type of virus
Assessment
I. Hepatitis A
-Increased Liver Enzymes: ALT and AST, and
* Causative agent: Hepatitis A Virus (HAV) alkaline phosphatase levels
* Incubation period: 25 days on average -Hepatitis A: headache, fever, and anorexia,
jaundice
* Period of communicability: 2 weeks preceding
onset of symptoms - Hepatitis B: generalized aching, right upper
quadrant pain, pruritus (itching) and headache,
* Mode of transmission: Oral-fecal route accompanied by a low-grade fever; dark-colored
* Immunity: One episode includes immunity for urine, jaundice of the sclera, generalized
the specific type of virus jaundiced, white or gray stool.
-Pyloromyotomy (perform before hypoglycemia) -Ensure adequate hydration and proper intake
of essential vitamins and minerals
-Correction of electrolyte imbalance; IV fluid
(isotonic or 5% glucose in saline) -TPN
Therapeutic Management
-Laparoscopy exploration and removal of the
vestigial structure