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Varicose Vein

Varicose veins are dilated, elongated superficial veins caused by venous hypertension. They commonly affect the left lower limb and long saphenous system. The venous system consists of deep, superficial, and perforating veins. Valves and muscle pumps aid venous return. Varicose veins can be primary or secondary and are caused by factors like prolonged standing. Symptoms include leg aches and heaviness. Diagnosis involves history, exam, ultrasound, and venography. Complications include bleeding, thrombophlebitis, and ulcers. Conservative management focuses on compression, elevation, and sclerotherapy using chemicals to obliterate veins. Surgical options are high ligation and stripping or laser ablation. Post-procedure care

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Arslan Khan
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61 views

Varicose Vein

Varicose veins are dilated, elongated superficial veins caused by venous hypertension. They commonly affect the left lower limb and long saphenous system. The venous system consists of deep, superficial, and perforating veins. Valves and muscle pumps aid venous return. Varicose veins can be primary or secondary and are caused by factors like prolonged standing. Symptoms include leg aches and heaviness. Diagnosis involves history, exam, ultrasound, and venography. Complications include bleeding, thrombophlebitis, and ulcers. Conservative management focuses on compression, elevation, and sclerotherapy using chemicals to obliterate veins. Surgical options are high ligation and stripping or laser ablation. Post-procedure care

Uploaded by

Arslan Khan
Copyright
© © All Rights Reserved
Available Formats
Download as PDF, TXT or read online on Scribd
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VARICOSE VEIN

Definition

• Varicose veins are defined as dilated,


elongated, tortuous and palpable
superficial veins as a result of
venous hypertension.
• Varicose presentation
•More common in males in India
•Left lower limb more commonly involved
•Long saphenous system affected in 2/3 rd
of cases
Venous System of lower limb
Consists of:
•Deep system of veins which lies below the deep fascia.
•Superficial system of veins which lies outside
the deep fascia (carry 10% blood)
•Perforating veins which pass through the deep
fascia joining the superficial to the deep system
of veins.
Valves in the veins
• Valves present in superficial veins.
• Prevent flow of blood from proximal to distal and from
deep to superficial
• Absent from above groin level
• Valves can resist pressure up to 300 mm of Hg.
Factors Helping in Venous
return
• Negative pressure in thorax during inspiration to -6 mm.
• Calf muscle pump: Normal venous pressure in relaxed state 20mm
of Hg. Rises to 80-100 mm of Hg during muscle contraction.
• Vis a tergo: arterial pressure transmitted to venous side through
capillary bed
• Competent valves
• Venae commitants: lie by the side of artery, helped by arterial
pulsation to propel blood.
Types
• Primary ( idiopathic)
• More common in women
• Lower extremities
• Strong family history

SECONDARY
PREVIOUS DVT
other identifiable obstruction
Also occur in esophagus, haemorrhoids, arterivenous malformation
Etiology
• •Long hours of standing, • Deep vein thrombosis
which increase the • Oral contraceptives
hydrostatic pressure of • obesity
gravity,
• Family history
• Pregnancy
• Ageing
Pathophysiology
• Etiological factors

enlargement of veins

valves are stretched and become incompetent

Back flow of the venous blood

further increased distention of veins

clinical manifestations
CLINICAL MANIFESTATION
• Cosmetically disfigurement
• Dull aches, muscle cramps, and increased muscle fatigue in the
lower legs.
• Ankle edema and a feeling of heaviness of the legs
• Nocturnal cramps
DIAGNOSIS
• HISTORY COLLECTION
• PHYSICAL EXAMINATION
• DUPLEX ULTRASONOGRAPHY
• VENOGRAPHY
Ascending
descending
Complications
• Bleeding
• Thrombophlebitis
• Venous Hypertension leading to venous ulcer
• Calcification
• Eczematoid dermatitis and pigmentation
Conservative management
• Avoiding prolonged standing
• Crepe bandaging and elastic stockings from toe to thigh,
which causes decreased edema, venous volume and reflux
and increases venous return.
• Limb elevation above the level of heart while lying down
• Sclerotherapy
A chemical is injected into the vein, irritating the venous
endothelium and producing localized phlebitis and fibrosis,
thereby obliterating the lumen of the vein
• Under Ultrasound guidance.
• hypertonic sodium chloride solution
• Sodium morrhuate
• Ethanolamine oleate
• Polidocanol
• Spread of foam monitored under USG guidance as it spreads.
• Apex of saphenous opening compressed by probe to prevent foam
entering deep veins.
• Leg also elevated
• After leg is wrapped with
elastic bandage 24-72 hrs
Surgical management
High end ligation and stripping
• Ligation of entire vein and dissection and removal of its tributaries
• Laser fiber produce endoluminal heat that destroy the
vascular endothelium
Nursing management
• Bed rest is maintained for 24 hours, after which the patient begins
walking every 2 hours for 5 to 10 minutes.
• Elastic compression stockings are used to maintaincompression of
the leg. They are worn continuously for about 1 week after vein
stripping
• The foot of the bed should be elevated, Standing still and sitting are
discouraged
• Usually, the patient may shower after the first 24 hours. The
patient is instructed to dry the incisions well with a clean towel
using a patting technique rather than rubbing
• If the patient underwent sclerotherapy, a burning sensation in
the injected leg may be experienced for 1 or 2 days. The nurse
may encourage the use of a mild analgesi
(eg,acetaminophen,ibuprofen)

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