Diabetes Mellitus Type2: Case Study
Diabetes Mellitus Type2: Case Study
Diabetes Mellitus Type2: Case Study
Diabetes Mellitus
Type2
Prepared by:
John Carlo E. Garcia, RN, REB
Emerson B. Chavez, RN
Glaiza R. De Guzman, RN
Submitted to:
Ms. Princess Tumambing, MSN, RN
Nurse Supervisor
March 3, 2023
A.1 Introduction
Diabetes Mellitus (DM) is a chronic disease characterized by insufficient insulin production in
the pancreas or when the body cannot efficiently use the insulin it produces. This leads to an
increased concentration of glucose in the bloodstream (hyperglycemia). It is characterized by
disturbances in carbohydrate, protein, and fat metabolism. Sustained hyperglycemia has been
shown to affect almost all tissues in the body. It is associated with significant complications of
multiple organ systems, including the eyes, nerves, kidneys, and blood vessels.
Classification of DM
1. Type 1 DM or Insulin Dependent Diabetes Mellitus- it is due to absolute insulin
deficiency (inability of B cells to secrete sufficient insulin) and cause is unknown but
may be genetically acquired. This condition requires administration of exogenous
insulin for survival. People with this condition are ketosis prone because they utilize
fats and proteins as a source of energy. It is common for people below 30 years of
age. Usually thin at diagnosis and the onset is sudden and dramatic.
3. Gestational DM- due to placental hormones which inhibits the action of insulin.
Glucose intolerance with its onset during pregnancy, usually in the second or third
trimester. Risk factors are obesity, 30 years old and above, family history and
previous large babies.
Risk Factors
1. Non-Modifiable
● Age
● Ethnicity
● Family History
● Genetic predisposition
2. Modifiable
● Physical activity
● Diet
● Excess alcohol intake
● Are 45 years or older
● Have a parent or brother or sister with type 2 diabetes
● Have a family history of diabetes are not physically active because of physical
limitation, sedentary lifestyle or a job that requires sitting for long period of time
● Have a history of gestational diabetes, a type of diabetes that develops pregnancy
or gave birth to a baby weighing or more
Clinical Manifestations
· Classical Symptoms of DM: Polyuria, Polydipsia and Polyphagia.
· Clinical Signs of DM: Hyperglycemia, Glycosuria and Ketonuria.
· Pruritus Vulvae
· Unexplained weight loss
· Weakness and fatigue
· Lowered resistance to infection
· Sudden vision changes
· Skin lesions and wounds that are slow to heal
· Tingling or numbness in hands and feet
· Dry skin
Diagnostic Evaluation:
· Random Blood Sugar
· Fasting Blood Sugar
· Oral Glucose Tolerance Test
· Glycosylated Hemoglobin Test (HBA1c)
· Post-prandial Glucose Test
Diabetic Management
· Nutritional Therapy/ Diet
· Exercise- improves insulin utilization in the body
· Monitoring- to detect episodes of hypoglycemia or hyperglycemia
· Pharmacologic Therapy- Insulin Injection and Oral Antidiabetic Agents
2. Chronic Complications:
a. Macrovascular Complications
o Atherosclerosis and arteriosclerosis
o Hypertension
o Coronary Artery Disease
o Arterial occlusive disease that can lead to diabetic gangrene
b. Microvascular Complications
o Diabetic Retinopathy
o Nephropathy
● Diabetic Neuropathies such as peripheral neuropathy and autonomic
neuropathy.
Prevalence
According to the World Health Organization (WHO) diabetes mellitus is a chronic, metabolic
disease characterized by elevated levels of blood glucose, which leads overtime to damage to the
heart, eyes, kidneys and nerves. Over 90 % of diabetes mellitus cases are Type 2 DM.
According to the latest International Diabetes Federation (IDF), the global prevalence of T2 DM
in adults was 536.6 million people (10.5%) in 2021, and with that there would be 783.2 million
people (12.2%) living with diabetes worldwide by 2045.
Total of 37.3 million people have diabetes (11.3%)of the US population diagnosed 28.7 million
people including 28.5 million adults.
Epidemiology
According to the World Health Organization (WHO) diabetes mellitus is a chronic,
metabolic disease characterized by elevated levels of blood glucose, which leads overtime to
damage to the heart vasculature, eyes, kidneys and nerves.
Endocrine Pancreas
Comprise the Islets of Langerhans: each of which is a cluster of 4 endocrine cell types, whose
products are ultimately secreted into the hepatic portal blood and delivered to the liver.
A key function of pancreatic endocrine cells is to maintain basal blood glucose concentration via
regulated storage and release: glucose is a major source of energy for the body.
In order to maintain homeostatic blood glucose concentrations, the islet cells engage in
communication via neural, hormonal, and cell-to-cell signaling.
Alpha cells
They secrete glucagon, which increases blood glucose concentrations between meals.
Beta cells
They secrete insulin, which lowers blood glucose concentrations after a meal.
Delta cells
Secrete somatostatin, which inhibits insulin and glucagon secretion to moderate their effects on
blood glucose concentrations.
F cells Are rare
Beta cells release insulin in response to multiple stimuli, but glucose is the primary stimulus.
Step 1:
Step 2:
Step 3:
Step 4:
Step 5:
In response to depolarization, calcium channels open, and the intracellular calcium concentration
increases.
Step 6:
This triggers exocytosis of insulin-containing vesicles.
Bear in mind that the beta cell response to fatty acids and proteins is similar, though not
identical.
As discussed in detail elsewhere, insulin is released into the nearby capillaries, drains into the
hepatic portal system, and, eventually, enters systemic circulation to reach its target tissues.
Clinical Correlations:
Destruction of beta cells (often by autoimmune disorders) causes insufficient circulating levels
insulin; thus, carbohydrate, protein, and fat metabolism is inhibited.
Because Type I diabetes is due to insulin insufficiency, insulin replacement therapy is the
primary treatment to normalize blood nutrient concentrations.
Down-regulation of insulin receptors on target tissues, specifically in the liver, skeletal, and
adipose tissues, inhibits metabolism of carbohydrates, protein, and fat.
Despite normal, or even elevated, levels of insulin, the target tissues cannot respond as usual;
thus, treatment includes insulin receptor sensitizers (in addition to insulin therapy).
Your endocrine system is made up of several organs called glands. These glands, located all over
your body, create and secrete (release) hormones.
Hormones are chemicals that coordinate different functions in your body by carrying messages
through your blood to your organs, skin, muscles and other tissues. These signals tell your body
what to do and when to do it.
Your endocrine system continuously monitors the amount of hormones in your blood. Hormones
deliver their messages by locking into the cells they target so they can relay the message.
The pituitary gland senses when your hormone levels rise, and tells other glands to stop
producing and releasing hormones. When hormone levels dip below a certain point, the pituitary
gland can instruct other glands to produce and release more. This process, called homeostasis,
works similarly to the thermostat in your house. Hormones affect nearly every process in your
body, including:
-Metabolism (the way you break down food and get energy from nutrients).
-Sleep.
-Blood pressure.
Sometimes glands produce too much or not enough of a hormone. This imbalance can cause
health problems, such as weight gain, high blood pressure and changes in sleep, mood and
behavior. Many things can affect how your body creates and releases hormones. Illness, stress
and certain medications can cause a hormone imbalance.
The thyroid's main role in the endocrine system is to regulate your metabolism, which is your
body's ability to break down food and convert it to energy. Food essentially fuels the body, and
each of our bodies uses that fuel at different rates.
The endocrine system is made up of organs called glands. Glands produce and release different
hormones that target specific things in the body. You have glands all over your body, including
in your neck, brain and reproductive organs. Some glands are tiny, about the size of a grain of
rice or a pea. The largest gland is the pancreas, which is about 6 inches long.
Hypothalamus: This gland is located in your brain and controls your endocrine system. It uses
information from your nervous system to determine when to tell other glands, including the
pituitary gland, to produce hormones. The hypothalamus controls many processes in your body,
including your mood, hunger and thirst, sleep patterns and sexual function.
Pituitary: This little gland is only about the size of a pea, but it has a big job. It makes hormones
that control several other glands such as the thyroid gland, adrenal glands, ovaries and testicles.
The pituitary gland is in charge of many different functions, including how your body grows. It’s
located at the base of your brain.
Thyroid: Your thyroid is a butterfly-shaped gland in the front of your neck. It’s responsible for
your metabolism (how your body uses energy).
Parathyroid: These four tiny glands are no larger than a grain of rice. They control the level of
calcium in your body. For your heart, kidneys, bones and nervous system to work, you need the
right amount of calcium.
Adrenal: You have two adrenal glands, one on top of each kidney. They control your
metabolism, blood pressure, sexual development and response to stress.
Pineal: This gland manages your sleep cycle by releasing melatonin, a hormone that causes you
to feel sleepy.
Pancreas: Your pancreas is part of your endocrine system, and it plays a significant role in your
digestive system too. It makes a hormone called insulin that controls the level of sugar in your
blood.
Ovaries: In women, the ovaries release sex hormones called estrogen, progesterone and
testosterone. Women have two ovaries in their lower abdomen, one on either side.
Testes: In men, the testes (testicles) make sperm and release the hormone testosterone. This
hormone affects sperm production, muscle strength and sex drive.
Pathogenesis of DM type 2
A.3 Concept Map (Pathophysiology/ Pathogenesis)
3.FAMILY
HISTORY
NONE
moderate drinking
Admission Data
Chief Complaint
The patient sought consultation with the ER with a cough of 3 weeks duration as his main
concern.
Present Illness
3 weeks prior to consultation, the patient experienced cough and fever and was consulted with a
private physician and prescribed with Co-amoxiclav 625mg/tab 3x a day for 7 days. 1 day prior
to consultation, the patient was still with cough and fever, thus decided to consult at ER.
A.6 Genogram
A.7 Gordon’s Functional Health
Health Perception Patient is 54 years old no Patient thinks that he cannot During hospitalization,
history of hospitalization and function well same as before, he thinks he can’t
doesn’t take any medication. but he’ll try to be healthy by function well which
He perceives himself as following the medical advised affects his perception
healthy individual. instructed to him. about health. Perception
of health is natural
progression from relative
nature of health.
(pdhpe.net, 2023).
Elimination Pattern Patient has a normal bowel During hospitalization the Before and during
pattern, he frequently urinates Patient urinates frequently hospitalization the
about 5-6 times a day and and he does not experience Patient still urinates
does not experience pain, his any problems in urinating.. frequently. Frequent
urine is clear and yellow in urination can be sa sign
color. of Diabetes, when
diabetes is not controlled,
excess sugar causes more
fluid to pass from
kidneys into urine.
(Kathleen Smith M.D.
Sept 2022; eveyday
health.com; Frequent
urination symptoms and
treatment).
Activity and Exercise Patient is completely Patient is unable to perform During confinement in
Pattern independent performing ADLs due to hospitalization. hospital, there is
ADLs, he usually lies-down limitation in activities of
after eating. Patient often to daily living and
walk outside the house as disruption in leisure and
form of exercise. recreation pattern. A
problem with the
activity-exercise pattern
may be the primary
reason for the pt. entering
the health care system or
may arise secondary to
problems in another
functional pattern.
(Cox Clinical
Application of Nursing
Diagnosis 5th edition
page 270).
Sleep-Rest Pattern Patient usually sleep 5-6 Patient has no difficulty in Patient is having
hours after work. sleeping and usually sleeps 8- prolonged sleep at night,
10 hours at night. he gains rest/ sleep.
Getting enough sleep is
essential for maintaining
optimal health and well-
being like exercise and a
balanced diet, sleep may
help prevent a range of
health issues.
(medicalnewstoday.com,
2023).
Cognitive and Perceptual Patient has no problem with Nothing has changed in his Patient is coherent and
Pattern his hearing; he has normal cognitive perception. cooperative and alert
cognitive perception. with no problems with
his senses. Pt. manifest
intact sensory
mechanism and
perception.
(Kozier and Erb’s
Fundamentals of Nursing
8th Edition vol 2, page
981).
Self-Perception/ Self Patient views himself as a During the assessment the pt Patients’ self-perception
Concept healthy strong individual. was responsive due to was strongly affected due
hospitalization the pt. can’t do to his condition. The
his daily activity. concept of oneself for
each person is like a
collage. At the center of
the collage are the beliefs
and images that are most
vital for the individual's
identity and self-esteem.
(The Fundamentals of
Nursing, Kozier, page
382).
Role Relationship Pattern Patient lives with his wife Relationship of his family The Patient relationship
and children; he views remains strong; his wife and with his family remains
himself as a responsible his children are the source of strong. Patient
father to his children. his strength after this hospitalization made
hospitalization. their family become
stronger to support, they
used each other as a
source of energy and
hope.
(Cox’s Clinical
Application of Nursing
Diagnosis 5th Edition
page 606).
Coping Stress Patient usually copes stress Patient Family supports him His family supports each
by taking responsibility for for his immediate to recover other in every situation
situation. And share with his from his hospitalization. may occur. Coping
wife to maintain emotionally mechanisms are the
supportive relationship. strategies people often
use in the face of stress
and/or trauma to help
manage painful or
difficult emotions.
Coping mechanisms can
help people adjust to
stressful events while
helping them maintain
their emotional well-
being.
(goodtherapy.org, 2018).
Sexual Reproductive Patient had two children, During hospitalization still no Patient sexual
Pattern according to the Patient no disease/dysfunction in his reproductive pattern is
history of disease/dysfunction reproductive system. not affected. The changes
affecting his reproductive that happen in a person's
system. body or life have an
impact on their sexuality
and reproductive
patterns. "Factors
influence a person’s
sexuality; developmental
level, culture, religion,
values, personal ethics,
disease processes, and
medications." (The
Fundamentals of
Nursing, Kozier, p. 327)
Values-Belief Pattern Ptatient is a Roman Catholic, Patient has a good faith to Patient hospitalization
he rarely goes to church every God. doesn't change his
Sunday, but prays to God values/belief pattern.
often. Spiritual and Religious
beliefs play a significant
approach in major life
events.
(Fundamentals of
Nursing by Kozier page
1048.)
Review of System
URINALYSIS
Date: January 26, 2023
NORMAL FINDINGS RESULT INTERPRETATION
CLINICAL CHEMISTRY
Date: January 31, 2023
RESULT REFERENCE RANGE INTERPRETATION
IMPRESSION:
PTB WITH CAVITATION, LEFT; CANNOT RULE OUT NEW GROWTH
Generic Name: Inhibits protein synthesis of Indication: Side Effects: 1. Monitor daily pattern of
Clindamycin bacterial cell wall by binding to For treatment of respiratory tract Abdominal pain, nausea, bowel activity, stool
bacterial ribosomal receptor infections, skin/ soft tissue vomiting, diarrhea. consistency.
Brand Name: sites. Topically, it decreases infections, sepsis, intra-abdominal Allergic ration, phlebitis
Clinsea fatty acid concentration on skin. infections.
Contraindication: Adverse Reactions: 2. Assess skin for rash
Classification: None known. Antibiotic-associated (dryness, irritation).
Antibiotic, Cautions: Severe hepatic colitis, other
Lincosamide dysfunction; history of GI disease. superinfections (abdominal 3. Instruct pt to notify if they
cramps, severe watery have signs of severe
Dosage: 600mg diarrhea, fever) may occur persistent diarrhea, cramps,
Route: IV during and several weeks
bloody stool.
Frequency: q6h after clindamycin therapy.
Reference:
● Saunders Nursing Drug Handbook, 2016, page 269
DRUG DETAILS MECHANISM OF ACTION INDICATION AND SIDE EFFECTS AND NURSING
CONTRAINDICATION ADVERSE REACTIONS RESPONSIBILITIES
Generic Name: Ciprofloxacin, a Indication: Side Effects: 1. Monitor for signs and
Ciprofloxacin fluoroquinolone anti-infective Lower respiratory tract infections, Nausea, Diarrhea, symptoms of
agent, acts by inhibiting DNA Skin and soft tissue infections, Dyspepsia, vomiting. hypersensitivity reactions
Brand Name: gyrase and topoisomerase IV, Upper respiratory tract infections. constipation, flatulence, and altered mental status.
Acipro both essential in bacterial DNA Crystalluria
replication, transcription, repair Contraindication: 2. Obtain culture and
Classification: and recombination. Hypersensitivity to ciprofloxacin or Adverse Reactions: sensitivity test tests prior to
Fluoroquinolones other quinolones. Irreversible tendinitis, treatment initiation.
tendon rupture, peripheral
Dosage: 400mg neuropathy, CNS effects 3. Monitor daily pattern of
Route: IV (e.g., seizures, increased bowel activity.
Frequency: q12h intracranial pressure).
Nephropathy, 4. Encouraged hydration
Cardiopulmonary arrest,
hypersensitivity reaction 5. Check blood glucose levels,
(rash, pruritus) CBC, renal function, LFT
periodically and during
prolonged therapy.
Reference:
● Ciprofloxacin mims.com
● Saunders Nursing Drug Handbook 2016, page 260
DRUG DETAILS MECHANISM OF ACTION INDICATION AND SIDE EFFECTS AND NURSING
CONTRAINDICATION ADVERSE REACTIONS RESPONSIBILITIES
Generic Name: Humulin R insulin (human Indication: Side Effects: 1. Assess for symptoms of
Insulin (Human recombinant)) is a hormone that Humulin R (insulin (human Hypoglycemia is one of the hypoglycemia.
Recombinant) is produced in the body used to recombinant)) U-100 is indicated as most frequent adverse
Insulin Human (rDna) treat diabetes. an adjunct to diet and exercise to events experienced by 2. Inspect skin areas that will
Brand Name: improve glycemic control in adults insulin users. Symptoms of be used for injection; note
Insurgent R Humulin R: Short Acting and children with type 1 and type 2 mild to moderate any areas that are bruised,
diabetes mellitus. hypoglycemia may occur thickened, or scarred, which
Classification: Onset: 30-60 min suddenly include sweating, could interfere with insulin
Insulin and Analogue Peak: 1-5 hours Contraindication: dizziness and palpitation. absorption and alter
Duration: 6-10 hours Humulin R (insulin (human anticipated response to
Dosage: 6U recombinant)) U-100 is insulin therapy.
Route: SQ contraindicated during episodes of
Frequency: TID hypoglycemia and in patients 3. Give maintenance doses
premeal hypersensitive to Humulin R subcutaneously as needed,
(insulin (human recombinant)) U- rotating injection sites
100 or any of its excipients. regularly to decrease
incidence of lipodystrophy.
Reference:
● HUMULIN R rxlist.com
DRUG DETAILS MECHANISM OF ACTION INDICATION AND SIDE EFFECTS AND NURSING
CONTRAINDICATION ADVERSE REACTIONS RESPONSIBILITIES
Generic Name: Acts via specific receptor to Indication: Side Effects: 1. Check blood glucose level.
Insulin (Human regulate metabolism of Treatment of insulin-dependent type Localized redness,
Recombinant) carbohydrates, protein, and fats. 1 diabetes mellitus; non–insulin- swelling, itching (due to 2. Discuss lifestyle to
Insulin HUman (rDna) Acts on liver, skeletal muscle, dependent type 2 diabetes mellitus improper insulin injection determine extent of learning,
and adipose tissue. (NIDDM) to improve glycemic technique), lipodystrophy emotional needs.
Brand Name: control. (depression at injection site
Insurgent-N Humulin N: Intermediate due to breakdown of 3. Assess for hypoglycemia
Acting Contraindication: adipose tissue). Somogyi (cool, wet skin, tremors,
Classification: Hypersensitivity, hypoglycemia. effect with chronically dizziness, headache, anxiety,
Insulin and Analogue Onset: 1-2 hours Cautions: Pts at risk for excessive insulin dosage. tachycardia, numbness in
Peak: 6-14 hours hypokalemia mouth, hunger, diplopia.)
Dosage: 16U-0-8U Duration: 16-24 hours Adverse Effects:
Route: SQ Severe hypoglycemia (due 4. Advised diet is an essential
Frequency: ODAM & to hyperinsulinism) may part of treatment; do not
ODPM occur with insulin skip/delay meals.
overdose.
Reference:
● Saunders Nursing Drug Handbook, 2016, page 636
DRUG DETAILS MECHANISM OF ACTION INDICATION AND SIDE EFFECTS AND NURSING
CONTRAINDICATION ADVERSE REACTIONS RESPONSIBILITIES
Generic Name: Happens to inhibit Indication: Side Effects: 1. Assess alcohol consumption
Paracetamol prostaglandin synthesis in the Relief of Mild to moderate pain such Tachycardia, nausea, before administering
CNS and to lesser extent, block as muscle aches, back pain, Hypersensitivity Reaction Paracetamol.
Brand Name: pain impulses through headache; fever reduction (rash, pruritus)
Amcetam peripheral action. It produces 2. Assess For fever, for clinical
antipyresis by inhibiting the Contraindication: Adverse Reaction: improvement of fever.
Classification: hypothalamic heat regulating Severe Hepatic Impairment or Early Signs of
Analgesic and centre. severe active liver disease. Acetaminophen Nausea, 3. Assess Pain onset type,
Antipyretic Fatigue: Location duration of Pain
Late Signs:
Dosage: 300mg Vomiting, Right Upper 4. Assess for symptoms of
Route: IV Quadrant Tenderness hypersensitivity reactions.
Frequency: q4h PRN
for fever and headache
Reference:
● Saunders Nursing Drug Handbook, 2016, page 9-11
A.12 Nursing Care Plan
1. Acute Pain related to increased cerebral blood flow secondary to vasodilation, as evidenced by
facial grimace, guarding behavior on the affected area, and increased heart rate.
3. Unstable blood glucose level related to inadequate knowledge on disease process secondary to
dietary intake, as evidenced by increased random blood sugar, elevated HBa1C, and presence of
glucose in urine.
Assessment Nursing Diagnosis Planning Interventions And Evaluation
(Goals of Care) Rationale
Subjective Hyperthermia related to Short Term Goal Independent Short Term Goal
“Mainit ang increased metabolic rate After 30 to 45 minutes 1. Establish rapport with Goal was met: the
pakiramdam ko,” as secondary to infection, of nursing interventions, the patient and relative. patient’s body
verbalized by the as evidenced by the patient’s body Rationale: To promote temperature and heart
patient. elevated body temperature will return cooperation in the rate returned to normal
temperature, increased to normal range of nursing care. as evidenced by body
Objective heart rate and white 36.5°C to 37.5°C, and temperature of 36.8°C
- Flushed skin blood cells. heart rate of 60- 2. Assess and monitor and heart rate of 80bpm.
- Warm to touch 100bpm. patient’s temperature
- Body Temp- 38.9°C and patterns of Long Term Goal
- Tachycardia (HR Long Term Goal occurrence as well as Goal was met: the
110bpm) After 12 hours of the presence of chill. patient’s vital signs are
- RR- 22cpm nursing interventions, Rationale: This will maintained within the
- WBC- 25,400/mm3 the patient’s vital signs help to identify the normal range as
will be able to maintain development of the evidenced by body
normal range of body patient’s temperature temperature of 36.6°C,
temperature (36.5°C - and guide in HR- 77 bpm, BP- 120/
37.5°C), and heart rate formulation of 80mmhg, and RR-
(60-100bpm). diagnosis and 19cpm. No episodes of
management. fever occurred.
3. Assess patient’s
clothing that is too
warm for the
environment.
Rationale: removing
excess clothing or
blanket will promote
heat loss. Furthermore,
used of many clothes or
blankets inhibits the
body’s natural ability to
reduce body
temperature.
4. Advise patient to
have adequate oral fluid
intake.
Rationale: Water helps
to reduce body
temperature and to
prevent dehydration.
7. Monitor patient’s
intake and output.
Rationale: To monitor
patient’s fluid volume
accurately and to
maintain metabolic
functions during fever.
Collaborative
8. Administer IV fluids
as prescribed.
Rationale: To support
circulating volume and
tissue perfusion.
9. Administer
antipyretic as
prescribed.
Rationale: To reduce
fever by its central
action on
hypothalamus.
10. Administer
antibiotics as
prescribed.
Rationale: To control
the spread of infection
and treats underlying
cause.
Reference:
Williams, L. & Wilkins, L. (2008). Understanding the pathophysiology of fever. Nursing 38(8), 56cc1-56cc2.
https://doi.org/10.1097/01.NURSE.0000327497.08688.47
Baracos, V., Whitmore, W. & Gale, R. (1987). The metabolic cost of fever. Can J Physiol Pharmacol, 65(6),
1248-54. https://doi.org/10.1139/y87-199
5. Encourage patient to
verbalize his feelings.
Rationale: To alleviate
anxiety, tension, and
provide psychological
relief.
6. Provide non-
pharmacological pain
management such as
quiet environment, calm
activities, comfort
measures, use of
relaxation exercise (e.g.,
breathing exercise), and
diversional activity
(e.g., surfing the net,
listening to calm music
or watching tv- if
available).
Rationale: To decrease
environmental factors
which contributes to
pain. To distract
attention from pain and
decrease tension.
Collaborative:
8. Administer
analgesics as
prescribed.
Rationale: Medication
will provide synergistic
effect with non-
pharmacologic
interventions for pain
relief and promote
better circulation by
aiding in vasodilation
for better blood flow to
the brain and altering
prostaglandin synthesis
to decrease pain.
9. Assess the
effectiveness of pain
medications as
prescribed.
Rationale: Evaluation
of effectiveness of pain
relievers must be done
because absorption and
metabolism of
medication differ from
one person to another.
Notify the physician if
the regimen is
inadequate to meet the
pain control goal.
Reference:
7. Monitor blood
glucose levels.
Rationale: To ensure
that the blood glucose
level is within target
range.
8. Encourage patient to
increase physical
activity.
Rationale: Physical
activity helps lower
blood glucose levels.
Regular exercise is a
core part of diabetes
management and
reduces risk for
cardiovascular
complications.
9. Instruct patient to
avoid using heating
pads and always wear
shoes when walking.
Rationale: Patients
have decreased
sensation in the
extremities due to
peripheral neuropathy.
Collaborative:
10. Administer insulin
as prescribed.
Rationale: This helps to
regulate blood glucose
levels by alerting the
liver, muscle and fat
cells to absorb glucose
from the blood.
Herdman, T. H., PhD, RN, FNI. (Ed.) and Kamitsuru, S., PhD, RN, FNI. (Ed). (2018). NANDA Nursing Diagnoses:
Definitions and Classification 2018-2020 (11th ed.). New York, Stuttgart, Delhi, Rio de Janeiro: Thieme.
https://www.studocu.com/ph/document/central-mindanao-university/nursing/vera-sophia-areola-nursing-care-plan/
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Kelley, J. (2021). Oral hypoglycemic drugs. Boulder Medical Center. Retrieved from
https://www.bouldermedicalcenter.com/oral-hypoglycemic-drugs/
Nemar, A., Cavalcante, T., Moreira, R., … Olieveira, L. (2020, May). Nursing diagnosis risk for unstable
blood glucose level in patients with diabetes mellitus. Wiley Online Library, 31(4), 240-245.
https://doi.org/10.1111/2047-3095.12282
A.13 Discharge Planning
M
E
T
H
O
D
Reference:
..
Reference:
Lippincott Williams & Wilkins
● Healthy eating.
● Regular exercise.
● Weight loss.
● Possibly, diabetes medication or insulin therapy.
● Blood sugar monitoring.
Reference:
nih.gov
https://www.ncbi.nlm.nih.gov › articles › PMC4751088