IBKV2 UAS Notes 2022
IBKV2 UAS Notes 2022
IBKV2 UAS Notes 2022
EQUINE DENTISTRY
Sings & Symptoms
• Dental disease (broken teeth, irregular dental arcades) = common underlying cause of thriftiness, loss of
condition / poor breeding / nursing performance
• Classic signs = difficulty / slowness in feeding & reluctance to drink cold water
• Stop for few moment & start again during chewing process
• Head held one side if in pain
• Horse may quid e.g.: pick up its food, form it into a bolus but drop the bolus from the mouth after partially
chewed.
• Semi-chewed mass of feed packed between the teeth and the cheek.
• Bolt its food and subsequently suffer indigestion, choke, or coli → avoid using painful tooth / sore mouth
• Lack of desire to eat hard grain & uncrushed grain in the feces
• Excessive salivation & blood-tinged mucus in the mouth + the fetid breath of dental decay
• Extensive dental decay + periostitis & root abscessation → empyema of the paranasal sinuses &
intermittent unilateral nasal discharge
• Facial / mandibular swelling & development of mandibular fistulas from apical infections of the lower cheek
teeth
1 – 3: Incisors
4: Canine (only male)
5: Wolf (premolar)
6 – 8: Premolars
9 – 11: Molars
BY ANNA
IBKV 2 UAS
HEAD & NECK SURGERY
Surgery of Eyes
Nasolacrimal obs
Def: partial / complete obs of nasolacrimal duct occur after inflammatory, neoplastic trauma process
Cause
Internal External
• Dacryocystitis • Trauma
• Foreign bodies • Neoplasia
• Neoplasia (SCC, sarcoid) • Disease of maxillary dental arcade
• Parasitic granuloma (habronemiasis) • Sinusitis
Clinical sign: Epiphora & Mucopurulent discharge Postoperative care: bac culture & AB after catheter
removal
Diagnosis: careful examination
Complication
▪ Dysgenesis tech: fluorescein stain, normagrade
cannulation of duct, dacryocystorhinography ▪ Congenital ostium malformation / scarring of
nasolacrimal duct / puncta
Treatment & Prognosis: Catheterization & Antibiotic ▪ Corneal ulceration
(AB) & Anti-inflammatory (AI)
Ectero / Entropion
Def: inward rolling of eyelid margin Clinical sign
Ectropion: Eyelash
Cause: ▪ Involve lower eyelid outward → dry up of
▪ Excessive tearing tears
Congenital Acquired ▪ Blepharospasm
• Breed • Dehydration ▪ Conjunctivitis & keratitis Entropion: eyelash
predisposition• Septicemia inward, poke eyeball,
• Malnutrition Postoperative care: eye cup irritate eyes, tears
Treatment: Surgery – temporary eyelid tacking / hotz
celsus blepharoplasty | shave excess eyelash Complication: Corneal irritation & Ectropion
Enucleation
Def: removal of eyeball from eye orbit 1. Simple suture or continuous suture on the eyelid
2. Transpalpebral incision 1 cm from the edge of
Cause: the eyelid, around the orbit.
▪ Ruptured globes ▪ Panophthalmitis 3. 360 ° dissection around the orbit
▪ Intraocular neoplasia ▪ Chronic uveitis / 4. All muscles, adipose tissue, lacrimal glands, and
/ eyelid neoplasia: glaucoma the fascia is removed, along with the eyelid and
squamous cell eyeball.
carcinoma (SCC) of 5. If neoplasia, ensure all neoplastic tissue is
3rd eyelid removed.
6. If non-neoplastic condition, some tissue of the
Postop care: retrobulbar can be left out ⇒ reduce the amount
▪ Eye cup for 1 week preventing self-trauma of dead space and bleeding intraoperative.
7. The optic artery can be tied; / control bleeding by
▪ Medications: nonsteroidal AI drugs from 3 – 7
tight skin closure & pressure => impossible to
days ⇒ min associated discomfort & edema
obliterated when filled with blood in dead space.
▪ Suture removal: approx. 14 D
8. The cavity that is filled with blood clots will
Complications: Periorbital swelling & Orbital cysts / disappear during the healing process
mucoceles 9. Closing: interlocking pattern / simple suture
using nonabsorbent
Procedure: 10. Stitches are removed 2–3 weeks postoperatively.
BY ANNA
IBKV 2 UAS
Upper Alimentary System Surgery
Mandibular Fractures
Cause: DDx Other
Teeth Repulsion
Indication Treatment: dental extract → gen anes | affected top
position tooth | oral speculum ⇒ widen mouth
▪ Rlt deciduous teeth
▪ Interventional orthodontics Postop care
▪ Severe periodontal disease
▪ Loose teeth supernumerary teeth ▪ Exercise restriction: stall rest 2 weeks w
▪ Dental impactions | Malocclusions & neoplasia controlled hand walking only
▪ Odontic disease w 2’ osteomyelitis ▪ Medication: broad-spectrum AB (3 – 5 D).
▪ Severe disease / injury to the dental crown / root Infection: AB + Xsteroidal AI drug (1 – 2 D)
▪ Occlusal trauma & biting discomfort ▪ Lavage mouth & clean surgery site daily. Acrylic
▪ Sinus disease 2’ to dental disease plug / dental wax ⇒ self-expelled / removed w/in
30 D | Clean wound until granulation
Clinical sign
Complication
▪ Presence of head tilt while eating
▪ Nasal discharge, sinusitis ▪ Hemorrhage | Wrong tooth removed
▪ Headshaking, facial pain, ptyalism ▪ Damaged to adjacent structure of tooth being
▪ Anorexia / pica removed | Infected tooth root socket
▪ Facial swelling / distortion ▪ Wound dehiscence / persistent drainage
▪ Weight loss, diarrhea, colic, reluctance to eat, resulting formation of fistula
slow / intermittent eating, diff in prehension, ▪ Incomplete removal | Foreign body in wound
choke & epistaxis ▪ Bone sequestrum | Packing breakdown
▪ Mucous membrane healing prior to wound
Diagnosis: PE & dental radiography granulation
BY ANNA
IBKV 2 UAS
Sinusitis
Def: inflammation of sinus Cause: stagnation of mucus on sinus cavities thru
inhabited dynamic clearance
Acute: sinusitis serosa acute 1’ 2’
Chronica: sinusitis chronica purulenta Upper resp infection Periapical abscess
Inflammation due to cold, Dehorning → not heal,
Symptoms strangle, malleus cause encephalitis
Metastase
▪ Lethargy, no appetite Trauma
▪ Head tilt to pain site Clinical sign: nasal discharge & facial swelling & obs
▪ Discharge from nostril (unilat / bilat) dyspnea
▪ Sinus area: sensitive & swollen Common: 5 paired sinuses:
▪ Advance into fistula / alveolitis (rlt to molar) ▪ Sinus maxillaris ▪ Frontal / concho
▪ Acute: high temp & long lasting major et minor frontal
▪ Sinusitis sphenoidalis: around base of brain → otitis ▪ Sinus frontalis ▪ Caudal maxillary
sphenoidalis ▪ Sinus chonco- ▪ Rostral maxillary
▪ Suffer from encephalitis (critical) frontalis ▪ Ethmoidal
▪ Sinus sphenoidalis ▪ Sphenopalatine
Prognosis: dubius
Diagnosis
PE Facial area inspects for deformity of supporting bones thru swelling / trauma
SQ emphysema detected after trauma in cases where sinus walls been disrupted
Paranasal sinuses wall percussion = unreliable
↑ resonance: wall thin / dullness = filled w fluid / soft tis
Airflow at each nostril should be checked to assess obs of nasal meati
Clinical crowns checking for fracture, displacement/impaction of degenerated ingesta
Patency of nasolacrimal duct by catheterization & infusion of saline sol from either end
Radiograph Free fluid interfaces in the sinuses
Loss of normal air contrast thru substitution by fluid or soft tissue
Depression / elevation of supporting bones of the face
Distortion of normal structure e.g.: tooth roots, sinus wall, midline septum & infra-orbital canals
Treatment
▪ Medical: non-surgical treatment i.e.: AB, mucolytics, steam inhalation, volatile inhalation & continued
controlled exercise
▪ Systemic AB
▪ Surgical treatment: sinus trephination & fascial flap surgery
Trephination portal (TP) for frontal sinus: 3 Skin incision & periosteum in same plane b4
– 4 cm caudal to most rostral aspect of periosteum peeled away from underlying bone
frontal sinus & 3 – 4 cm lat to midline Bone flap w 5 cm diameter trephine (oscillating
TP for caudal maxillary sinus: 1 – 2 cm saw), osteotomy at least 1 cm inside the
dorsal to facial crest & 7 – 8 cm caudal to skin/periosteum incision ⇒ repair support by
most rostral aspect of facial crest bone
TP for cranial maxillary sinus: 1 – 2 cm Contents exposed, focus = identified &
dorsal to facial crest & 3 – 4 cm caudal to removed
facial tubercle Closure of incision achieved w single layer
mattress sutures, but accurate alignment for
Sinus trephination = trephination to access cosmetic result in horse w natural facial
for dental repulsion markings
Tracheotomy
Indication: emergency airway → upper airway obs / Medication: broad-spectrum AB & non-steroidal
relieve nasal / laryngeal inflammation AI agents x necessary unless for underlying
problem
Clinical sign: dyspnea – apnea & inspiratory &
expiratory stridor Other:
2 distinct clinical form: viral papillomatosis & aural ▪ Sometimes spontaneous immunity
papilloma (aural plaques) ▪ Become carrier depending on disease duration
Symptoms Treatment
Sharp Teeth
▪ Common in young horse & pigs Symptoms: mastication, digestion disorder, diarrhea
▪ Birth defects, affect 1 – 4% → common cause of
stillborn neonates ▪ Foals loud resp noise & exercise intolerance
▪ Unilat expiration
Molar (horse): ▪ Nasopharynx observe through 1 nostril
▪ Mastication disorder:
▪ Top molar: downwards, outwards Imperfect rumination
▪ Base molar: inwards, upwards Imperfect mastication, part of feed drops
Cause: on ground
Outer & inner edges w sharp edges
▪ Bad chewing / bad food grinding Lower molar inner tip: scratch / cut
▪ Failure of perforation of bucconasal memb @ tongue
choanae / posterior nares Upper molar outer edge: scratch / cut
check
Treatment
Injury of both checks, & tongue
▪ Floating → smooth out teeth ▪ Decrease of body condition
▪ Feed correction ▪ Sometime diarrhea
▪ Bilat neonatal: ops cito, tracheotomy
DDx: malleus → smelly
▪ Unilat: ~ 1 y/o ops, frontonasal bone flap
Prognosis: dubis
Diagnose: endoscope, contract radiograph
BY ANNA
IBKV 2 UAS
Glossitis [Tongue Inflammation]
▪ Acute / chronic | Superficial / profundal
Fractures Os Hyoid
Cause: kicked / horned, rough handling Prognosis
BY ANNA
IBKV 2 UAS
ABDOMEN REGION
Umbilical
1. Infection (SQ abscess / disease w/in umbilical remnants)
2. Herniation (non-strangulating / strangulating) {Strangulation = emergency due to loss of blood supply}
3. Combination of infection & herniation = primary problem associated w umbilicus in calves
*Usually cause enlargement of umbilicus*
Horse Cattle
Omphalitis
Def: umbilical infection Treatment:
Umbilical hernia
Def: abnormal protrusion of part of the organ in a location ➢ Seeded with bac from generalized septicemia /
that is not where it belongs with the formation of a ring bacteremia
around it ➢ Most common bovine congenital defect & can occur
in any breed, most common in HF
Cause: imperfect closure of umbilicus, infection ➢ Often classified as uncomplicated vs complicated,
Normally regress after birth & developed into: depending on existence of 2’ infection
Herniorrhaphy surgery
▪ Small, uncomplicated umbilical hernias & many umbilical abscesses may not rqd surgery
▪ Calf in dorsal recumbency using sedation (Xylazine HCl) & local anesthetic
▪ Large abscess: drained / aspirated & treated medically w antimicrobials for few days b4 surgery to ↓ size
& min # bac
▪ Infected umbilical remnant & abscess shud be resected en bloc if possible, to prevent contamination of
abdomen & incision
▪ Small, umcomplicated hernias in claves repaired w closed herniorrhaphy like those performed in foals
▪ Compared to closed herniorrhaphy, open herniorrhaphy take less time, less traumatic, allows inspection
of abdominal viscera, & permits removal of umbilical remnant
Atresia
Atresia ani ▪ Congenital atresia, prolapse / hernia, fistula
▪ Abnormal congenital @ rectum & anus:
▪ Common in pigs, sheep, horse & (less extend) Atresia ani: anus, hole | common in
cattle cattle, pigs, dog, sheep & goat
▪ Female: rectal fistula into vagina Atresia recti: anus, rectum
▪ SQ bilges when straining: A. ani / et recti Atresia ani et recti: anus, rectum
▪ A. coli: poor prognosis ➢ In Europe, congenital abnormalities animals not
▪ X-ray & USG: distance between rectal pouch & used for farming
perineal skin ➢ Normally include fistula recto-vaginalis on
Atresia female
➢ Rectum run towards anus, but end in VU in
▪ hole form organs w end tube male
▪ Abnormality in anus & rectum ➢ Other abnormalities: fistula recto-urethralis
BY ANNA
IBKV 2 UAS
Atresia ani Atresia recti Atresia ani et recti
Clinical ▪ Known 2 – 3 D post partus → ▪ Anus seems like dead ▪ No pop-up @ anus,
symptoms digestive tract disorder i.e.: end pouch hard when pressed
cholic, pops up anal skin, ▪ Symptoms like atresia ani ▪ Suppose anus, but
fluctuation when pressed on ▪ If fistula recto-vaginalis → only fine line, &
the pop-ups feces out from vagina fluctuations & feel
▪ defecation, enlarged (sometimes, feces accumulation of feces
abdomen accumulate in vestibulum ▪ No appetite, full
▪ Apart from straining, feces vaginae) abdomen, straining,
coming out → intoxication → ▪ if fistula recto-urethralis, long term →
death, appetite decrease accumulation of feces in intoxication → death
▪ Male last long, female live VU
longer → fistula ▪ prognosis: dubius –
rectovaginalis, feces come infausta
out thru vagina
Treatment ▪ Anesthesia / sedation / ▪ Shave skin & disinfect ▪ No treatment →
epidural anesthesia ▪ Local anesthesia euthanized
▪ Local anesthesia, clean area ▪ Incise slightly lower at ▪ Flank fistulation
below the tail, incise skin bottom of tail until SQ ▪ Flank assisted surgery
dorso-ventral around anus ▪ Prepare bluntly to pelvic
▪ Fluid feces come out if incise space
@ correct location ▪ Blunt end of rectum, pull
▪ Oval incision, suture snua out using tang artery
mucosa to the skin w simple ▪ Rectum end suture to
suture skin
▪ Apply laxament soapy water, ▪ The blund end of rectum
boorwater / laxantia i.e.: olium if cut & suture mucosa to
ricini, paraffin the skin
▪ Complication: fecal ▪ Give laxament & change
incontinence diet
▪ Remove suture when
heal
▪ Protruded part of organ thru hole ▪ Straining / loose sphincter withstand pressure
▪ Digestive tract: anus, rectum, colon ▪ Prolapses during pregnant → strong push from
▪ Reproduction: female (vagina, uterus) abnormal birth canal / abnormal fetus (dystocia /
giant fetus)
Rectal Prolapse
Cause: straining from diarrhea, dystocia, intestinal parasitism, cholic, proctitis, rectal tumor & rectal foreign body
Classification
Classification Treatment
Type I Only rectal mucosa & submucosa project ▪ Mucosal edema & irritation reduced by topical
thru anus, sometimes more on one side than application of glycerin, sugar, magnesium
on other side ▪ Purse-string suture
Type II Complete prolapse of the full thickness of all
lesion / part of the rectal ampulla
Type III Variable amount of small colon Submucosal incision
prolapse intussuscepts into the rectum in addition to a ▪ 2 incisions around circumference of prolapse,
type II prolapse peel & remove the mucosal layer
Type IV Peritoneal rectum & a variable length of the Resection & anastomosis
prolapse small colon form an intussusception thru ▪ Full thickness circumferential incisions thru inner
anus & outer walls of the intussuscepted healthy tis
BY ANNA
IBKV 2 UAS
Vaginal & Cervical Prolapse
▪ Common in cattle & sheep Operation
▪ Usually in mature female in last trimester of
pregnancy Buhner suture placement
Treatment
BY ANNA
IBKV 2 UAS
Castration [Orchidectomy]
Aim
Callicrate bander
Post op care
Complications
BY ANNA
IBKV 2 UAS
Scrotal Problem
Hydrocele / Varicocele
▪ Hydrocele: abnormal collection of serous fluid bet the visceral & parietal layers of t. vaginalis
▪ Varicocele: abnormally distended & tortuous pampiniform plexus
Orchitis
Def: testis inflammation Symptoms: pain from scrotum, swelling
Cause: excessive trauma on testis causes inflammation Treatment: alternating cold & warm compression
Tumor of testis
▪ Sertoli cell tumor (SCT), interstitial cell tumor ▪ Teratomas & ICT rare in boars
(ICT) & teratoma in bulls & horse ▪ Most stallion castrated while young, avoid dev
▪ Seminoma, rete testis tumors & leiomyoma of ▪ Majority benign → remove affected testis
testis in rams
Penile Problem
Paraphimosis: glans penis extended from preputium & Balanitis: penis inflammation
x return Balanophatitis: inflammation on preputium
Phimosis: glans penis x come out from preputium
BY ANNA
IBKV 2 UAS
Female Abdomen Region
Mare Caesarian Section [C-sec]
Elective indication Emergency indication
▪ Pelvic fracture / soft tis injury w/in rep tract ▪ Dystocia
▪ Previous dystocia ▪ Near-term undergoing colic surgery
▪ Severe uterine artery hemorrhage ▪ Correction for uterine torsion
▪ Produce gnotobiotic foals for research purposes
BY ANNA
IBKV 2 UAS
C-sec in Cattle
Elective indication Emergency indication
▪ Prolonged gestation ▪ Dystocia
▪ Potentially valuable calf, ehrn a dystocia ▪ Deformities of maternal pelvis
anticipated, e.g.: in Belgian blue ▪ Induration of cervix
▪ Hydrops aminii & allantois, uterine torsion
▪ Emphysematous fetuses
Surgical technique
BY ANNA
IBKV 2 UAS
Teat Laceration
▪ Common in cow → severe deficits in milk production
▪ Laceration do not penetrate mucosa → 2nd intention healing
▪ Laceration penetrate mucosa → suturing:
Maintain normal teat function for milking
Prevent dev of teat fistulae / acute mastitis & loss of quarter
Post op management
BY ANNA
IBKV 2 UAS
LIMB FRACTURE TREATMENT IN LARGE ANIMAL
▪ Axial skeletal fracture – stall rest, external / internal fixation not rqd
▪ Appendicular skeleton fracture:
Is treatment rqd?
Can fracture acceptably reduce closed / internal reduction rqd
Can the fracture adequately immobilize using external coaptation alone / is internal fixation w /
w/o external coaptation rqd?
What is the cost-benefit analysis?
BY ANNA
IBKV 2 UAS
Level 1: Robert Jones bandage w ▪ Prognosis: simple fracture of pastern >> severe comminuted
caudal splint (hind limb) & dorsal ▪ Proximal sesamoid bone fracture & condylar fracture of Mc 3/ Mt 3
splint (front limb) ▪ Traumatic disruptions of fetlock arthrodesis & prognosis for
successful repair = variable → degree of soft tis injury present
Walking block
▪ Cow stand on 1 digit during convalescence of paired digit
▪ Wooden, rubber / plastic bloc (2.5 – 3.5 cm) formed to the size &
shape of hoof
▪ Most suitable for management of P1, P2, P3 fractures of a 1 digit
▪ Confined to a pen for 6 – 10 weeks while the fracture heals
▪ Remove after 6 weeks
Level 2 (mid diaphyseal ▪ 3rd Mc & 3rd Mt: mon soft tis coverage → open due to external trauma
fractures): Robert Jones bandages or penetration of skin by edges of the fractured bone: poor prognosis
w plantar & lat splint (hind limb) & ▪ Unstable fracture of carpus & tarsus: poor prognosis for athletic
caudal & lats splints (fore limb) soundness but salvaged in some breeding case
Metacarpus & metatarsus III / IV
▪ Fractures involving Mc / Mt III / IV: most common in food animal
▪ Due to forced extraction during dystocia
▪ Closed fracture of distal physis of metacarpus (Mc) / Metatarsus
(Mt): half-limb cast
▪ Closed fracture of middle portion of Mc / Mt: full limb cast
▪ Open fractures in mature cattle treated thoroughly debriding,
cleaning, & flushing wound, applying full limb cast, & AB for 10 –
14D
▪ Valuable cattle & young calves, open fractures best treated by use
of external skeletal fixator & daily wound care until healing
Tibia fracture
▪ Result of forces extraction during dystocia & trauma
▪ Fracture of distal physis of tibia: full-limb cast, uncommon
▪ Fracture of middle portion of tibia: Thomas splint-cast, TPC, bone
plate
▪ Thomas splint cast have good prognosis for bone healing, but have
a high rate of injury to the contralateral limb
BY ANNA
IBKV 2 UAS
▪ TPC & bone plate have good – excellent prognosis for healing &
pose min problem w contralateral limb injury
Thomas Splint + Cast
▪ Distal to elbow / stifle fracture
▪ Length of splint shud be measured while standing & using normal
limb for measure
▪ Must assisted to stand for 3 – 5 days until they learn how to rise
under their own power
Transfixation-pin + Cast & ESF
▪ ESF = stabilization of debilitating musculoskeletal injury (typically
fractures but + joint luxation / tendon rupture) using transfixation pin
& any external frame connecting the pins & spanning the region of
instability
▪ TPC aim to provide a sustainable, comfortable means to return the
patient to weight bearing asap postop, to maintain normal joint
mobility, if possible, & to provide an optional environment for
osteosynthesis & wound healing
Level 4: padded bandage with ▪ Disruption of diaphysis of the humerus in adult horse: poor
caudal splint that extends form the prognosis
elbow to the ground is used to fix the ▪ In foals & yearling, some humeral fractures heal w/o surgery
carpus & support forelimbs ▪ Humeral fractures in yearlings & foals undergo surgery >> adults,
unless radial nerve damage
▪ Simple fractures of the neck of the scapula that are repaired w
internal fixation: good prognosis for athletic function
▪ Comminuted fractures of the glenoid / neck of the scapula: poor
prognosis
▪ Fracture of scapula spine: good prognosis
▪ Diaphyseal fractures adult femur: grave prognosis
▪ Diaphyseal femoral fractures in foals younger than 3 Mo: guarded
to fair prognosis
▪ Distal femoral fractures in foals: guarded prognosis
▪ Pelvic fractures: uncommon in horse, overall survival rate 50 – 70%
▪ Fractures of tuber coxae: good prognosis for athletic activity
▪ Pelvic fractures involving the articular surface of the coxofemoral
joint: 20% chance for athletic soundness
BY ANNA
IBKV 2 UAS
Close VS Open fracture Ossues sequestrum
▪ Prognosis: close >> open ▪ Caused by calving chain injuries & extensive
▪ Success recovery rate depends: soft tis damage associated
Severity of soft tis damage ▪ Healing is slow by presence of sequestrum &
Bone affected sequestrectomy usually allow fracture healing
Age to proceed
Duration & degree of contamination ▪ Def not apparent until 4 – 6 weeks after initial
Economic limitations placed on injury
fracture management ▪ Remove sequestrum at earliest time possible
▪ Mature cow w open Mc fracture able to heal & based on radiographs / clinical findings,
return to productivity after thorough cleaning of cleanse the wound until a healthy granulation
the wound, administration of AB, & full limb bed is present, then perform cancellous bone
cast grafting from sternum
▪ By contrast, young calf w similar injury prone to ▪ Sternum provides largest volume of cancellous
septic nonunion / delay union bone for grafting
▪ Alternatively, proximal tibia, proximal humerus,
Infection management for open fractures or ileum may be used for cancellous grafts
▪ Induction of anesth → clean & debride the
wound, & copiously lavage the wound Septic non-union
▪ Ab for therapeutic concentration in bone: ▪ Infected fractures that progress to septic non-
penicillin, cephalosporins, fluoroquinolones & union shud be treated similarly to cortical
trimethoprim-sulfa combine (age dependent) sequestrum cases
▪ Chronic infection (osteomyelitis) of fracture ▪ Aerobic & anerobic cultures & microbial
difficult to resolves susceptibility test aid in AB selection & fracture
▪ Chronic osseous infection: IV infusion of AB site if extensively debrided of fibrous tis
distal to tourniquet achieves high levels of AB ▪ Debridement is continued until healthy bone is
in synovial fluid exposed
▪ A fresh, autologous cancellous bone graft is
harvested & implanted into the fracture site
BY ANNA
IBKV 2 UAS
TUMOR IN HORSE
Sarcoid
▪ Cutaneous, fibroblastic neoplasia w ▪ Usually firmly attached to the skin overlying
proliferative epithelial component them but sometimes freely moveable under the
▪ Classified histopathological as benign tumors surface
→ morphologic characteristics of the
fibroblasts & slow growing causing little of any Ulcerative fibroblastic sarcoid
▪ Fleshy masses that grow quickly, bleed easily
physical problems
& have ulcerated surfaces
▪ Misleading classification & ignores the large #
▪ Like exuberant granulation tissue (‘proud flesh’)
of tumors whose clinical behavior can only be
▪ Develop at the site of a wound
described as malignant
▪ Found anywhere on the horse body
▪ On basis of clinical appearance
Occult Mixed tumors sarcoid
Verrucous ▪ Sub-classification describing a lesion that
Nodular fibroblastic shows qualities of two or more different sarcoid
Ulcerative fibroblastic groups
Mixed tumors ▪ Commonly described as ‘mixed’ as a lot of
Malevolent sarcoid lesions will demonstrate
▪ Most aggressive subtype malevolent sarcoid characteristics of more than one type.
that infiltrate locally along fascial planes &
vessels, grow rapidly & high recurrence rate Malevolent sarcoid
after excision ▪ Most aggressive
▪ Rapidly spread over a wide area of the horse’s
Occult sarcoid body & grows just as quickly
▪ Appear as roughly circular hairless areas of ▪ Most likely appearance of ulcerative nodular-
skin like lesions group in large bundles
▪ Early development: subtle & difficult to ▪ Aggressive in nature that often there are no
recognize treatment options
▪ Mistaken for ‘ring-worm’ or even rub marks
from tack Sites of predilection
▪ Common on nose & side of face, armpit & groin
▪ Vary with geographic location:
▪ Accidentally traumatized, develop rapidly into
Face (muzzle, ears, and periocular
more serious type of sarcoid
region)
Verrucous sarcoid Distal limbs
▪ ‘wart-like’ appearance & often greyish in color Neck / ventral abdomen
▪ Skin crack easily & flakes of scale often be Areas of previous injury & scarring
rubbed off from the surface ▪ Location affect prognosis
▪ Appear singularly / in groups that merge into ▪ Distal limb & periorbital region having a worse
larger lesions prognosis for resolution than in other locations
▪ Manipulation of verrucous sarcoid usually not
Cause
painful
▪ Older horse: common in spontaneous
▪ Interference w verrucous sarcoid lead to rapid
malignancies
transformation into more serious & aggressive
▪ Younger horse: genetic predisposition /
forms
exogenous factor
Nodular fibroblastic sarcoid ▪ Standardbred – ½ x → Thoroughbreds –→
▪ Firm, round nodules appear anywhere on the Quarter horse (2x of Tb)
horse’s body
Surgical excision
▪ Often seen in the armpit, inside edge of the
▪ Transform to a more aggressive phenotype
thigh & groin & under the skin of the eyelids
after incomplete / unsuccessful treatment →
▪ Singular or multiple & variable in size
▪ Usually covered by a layer of normal skin but harder to resolve
▪ Surgical excision w/o adjunctive therapy =
can also be ulcerative
least successful treatment options, recurrence
rate of 15.8% to 82%.
BY ANNA
IBKV 2 UAS
Squamous Cell Carcinoma (SCC)
▪ Malignant, locally invasive neoplasia of Cause:
squamous epithelial cells
▪ Location: ▪ Develop in areas of chronic, poorly healing
Integument, sites of predilection wounds and at sites of previous burn injury
include areas lacking pigmentation, ▪ Breeds with poorly pigmented, pink-skinned
poorly haired regions, & skin near areas including Appaloosas and paint-colored
mucocutaneous junctions horses are more prone to develop SCC→ 69%
Most common neoplasm of the equine of all ocular SCC cases occur in individuals
eye, conjunctiva, ocular adnexal lacking periocular pigmentation
structures, & external genitalia ▪ Draft breeds have an increased incidence of
Nasal cavity, paranasal sinuses, SCC.
pharynx, larynx
Hoof capsule & it should be
considered in horses with chronic,
refractory foot abscesses.
Ulcerative Proliferative
▪ Develop over time, early lesions appearing as ▪ In the ocular structures can invade the orbit,
small nodules underlying normal-haired skin calvarium, tear duct, and sinuses if left untreated.
▪ Mistaken for nonhealing wounds and chronic ▪ Pedunculated lesions on the penis often have a
granulation tissue → delayed treatment cauliflower-like appearance
▪ Ocular lesions begin as small ulcerative lesions on ▪ Typically spreads to surrounding tis & local lymph
lid margins / as keratitic plaques on the cornea nodes, but distant metastasis is rare
▪ Thus, be suspicious whenever raised red lesions ▪ Local / distant metastases or large, invasive
appear on the lid margins, sclera, or conjunctiva, tumors have a poor prognosis for cure.
particularly in unpigmented skin
Treatment
BY ANNA
IBKV 2 UAS
Melanocytic nevi Malignant melanomas
▪ Usually benign ▪ True MM rare & classified on basis of presence
▪ Small, single discrete masses (< 2 – 5 cm) of both histopathologic & clinical
▪ Seen in young horse of any color characteristics of malignancy
▪ Appear anywhere on body ▪ Occurred > 20 y/o, recurred w/in 10 Mo after
surgical excision
Dermal melanomas ▪ Frequently in various size & associated w a
▪ Usually benign
poor prognosis for complete resolution
▪ Large / dev in atypical location can bcome
malignant Location
▪ Vary in size, discrete masses & appears in
clusters ▪ The vast majority:
▪ Mostly in more mature grey horses Around the perineum and base of the
▪ Common under tail, in perineum & external tail
genitalia, but can also invade parotid salivary Lesions around the head (lips, eyes,
glands, lips, eyelids, & neck parotid region) and other sites less
frequent
Dermal melanomastosis ▪ Foot, meninges, thorax, ocular structures, and
▪ More likely malignant & metastatic abdominal cavity
▪ Usually in horse > 15 y/o ▪ These unusual locations appear to be
▪ Multiple, large poorly circumscribed masses of associated with a poor prognosis.
various size
Treatment
▪ Gray horses exhibited normal quality of life regardless of tumor number and type
▪ Treated with benign neglect, as rarely the cause of significant disease in affected horses
▪ However, with age, the risk of progression to dermal melanomatosis and metastases increases
▪ Small nodules are easily removed and rarely recur, but owners need to be informed that new tumors will
quite likely develop over time
▪ Although conservative management is reasonable in the majority of cases, more aggressive treatment,
including early removal of smaller tumors, may decrease the risk of melanomatosis or metastases as the
animal ages
▪ Malignant melanomas have a higher recurrence rate with simple excision
▪ Combination therapy, including surgical debulking and intralesional chemotherapeutic injections, may
offer palliation, but the prognosis for cure is poor
BY ANNA