Case Study: On Alcoholism
Case Study: On Alcoholism
Case Study: On Alcoholism
CASE STUDY
ON ALCOHOLISM
CLIENT-MR.AMRIK SINGH
[THIS CASE STUDY HAS ENCLOSED THE BRIEF HISTORY OF MR.AMRIK SINGH
ADMITTED TO SGRD HOSPITAL PSYCHIATRIC WARD & DIAGNOSED AS ALCOHOL
DEPENDENCE.. INFORMANT IS HIS FATHER AND VARIOUS BOOKS ARE REFERRED FOR
COMPLETION OF CASE STUDY.]
CASE STUDY
OF
Mr. Amrik Singh
WITH
ALCOHOL DEPENDENCE
IDENTIFICATION DATA
PSYCHIATRIC HISTORY
COMPLAINTS AND THEIR DURATION
1. According to patient
Patient is regularly taking alcohol since 2003 daily around half bottle. He started taking alcohol
under peer pressure for first time in 2003. Gradually he developed habits of taking alcohol daily
in Peer Company. The after 3 months he increased the dose of alcohol consumption from half
bottle to full one bottle. At present in 2012 client is chronic alcoholic and used to consume 3-4
bottles a day. After taking alcohol quarrels with father and brother.
CHIEF COMPLAINTS:
Uncontrolled anger
Anorexia
Insomnia
Body aches
2. According to Father
Patient is taking alcohol and cigarette daily with friends, shows anger towards father and brother.
Sleep is reduced and taking less food. Many time tried to hospitalize. This time patient came
voluntarily for treatment.
HISTORY OF PRESENT ILLNESS
a) Onset :- Gradual
b) Precipitating factors :- Peer group pressure His friends use alcohol and
Cigarette. He is working in chicken shop and job
stress.
c) Course of the illness :- 8 years
d) Associate disturbance :- Not taking food, decreased sleep and anger
Outbursts
FAMILY HISTORY
26years 24years
No history of alcohol dependence and psychiatric diseases in family. History of diabetes mellitus
and hypertension present
PERSONAL HISTORY
1. Birth and early development
Full term normal home delivery No pre and post natal complications. Normal growth and
development, Milestones achieved appropriately
4. School
Patient studied till 10th std. Relationship with peers and teachers normal. He was an
average student and left school due to the lack of interest in studies.
5. Occupation
He is working in chicken shop as a helper form 2 years .
6. Sexual history
Not sexually active. No abnormalities reported.
7. Martial History
Unmarried
PREMORBID PERSONALITY
1. Social relations
Normal behavior toward family and friends
2. Intellectual activities
No significant intellectual activities noted.
3. Mood
Subjective – satisfied
Objective – looks cheerful
4. Character
a) Attitude to work and responsibility
Shows a responsibility towards home
b) Interpersonal relationship
Normal interpersonal relationship
6. Fantasy life
Not reported
1. GENERAL APPEARANCE:
- Body built and physical experience: moderately built and healthy, young and wheatish in
complexion
- Grooming : well groomed
- Hygiene : maintained
- Dress : dress well and appropriate to season
- Facial expression : Pleasant
- Eye contact : maintained
- Posture : normal
-Attitude towards examiner : co-operative
- Rapport : rapport maintained
- Motor behaviour : no abnormal motor behaviour like tics,
Mannerism, pacing, motor retardation
B. THOUGHT AND SPEECH:
1. Form of thought:
N. Why do you come here with your father?
P. I am using alcohol a lot and making problems .So to stop alcohol drinking I came
here.
Inference: normal form of thought.
2. Content of thought
N. What is your problem?
P. Alcohol drinking, smoking, anger towards father and brother.
3. Speech:
- Evaluation of speech: appropriate volume, tone, speed
- Disorder of Speech: no aphonia and others disorders
D. PERCEPTION:
N. When you are alone, can you seeing anything that other person can’t see and hear?
P. No
b. Recent memory:
N. What you had in breakfast?
P. Yes
H. ORIENTATION:
N. What must be the time now?
P. It must be 10’o clock
N. Who brought you here?
P. I came along with my father.
N. Who is that lady wearing white saree?
P. She is a ward sister.
N. Where are you now?
P. I am in NIMHANS hospital.
Inference: he is oriented to time, place, and person.
I. ABSTRACTION:
N. What is the different between the potato and stone?
P. Potato is eatable and stone is hard.
N. What is the similarity of banana and orange?
P. Both are fruits
J. COMPREHENSION:
N. What will you do, if u misses the bus?
P. I’ll catch another bus.
N. What you will do if you feel cold?
P. I’ll wear sweater.
K. INTELLIGENCE:
N. What is the capital of India?
P. Delhi
L. JUDGMENT:
- Personal judgment:
N. What are you planning to do after discharging from here?
P. I want to settle my life.
- Social judgment:
N. What will you do when you saw a person drowning?
P. I will call other people because I don’t know swimming
- Test judgment:
N. What you will you do if your ward is fire?
P. I’ll pour water to stop the fire.
Inference: Judgment of the patient is good.
M. INSIGHT:
N. Why you are coming and staying here?
P. I have to stop alcohol drinking.
E. HEAD:
a. Head and cranium : No scar.
b. Hair: black hair, no lice noted, no dandruff noted.
c. Movement of the head: normal ROM of the head and neck.
d. Forehead: No scar or lesion
F. EYES:
a. Expression: pleasant
b. Eye brows: equal, evenly distributed and no dandruff noted.
c. Eye lids: No lesion and scars, eye lashes are equally distributed
d. Lacrimation: clear fluid expressed, no discharges present.
e. Conjunctiva: appears pale and clear
G. EARS
a. Appearance: No masses or lesions present in the external ear.
b. No discharge
c. Hearing: is able to hear in both ears; Weber test-negative; Rinne test-positive
d. No lesions or mass.
H. NOSE:
a. Appearance: septum not deviated; no growth or lumps externally noted
b. Discharge: no discharge present
c. Patency: Both nostrils are patent
d. Sense of smell: good
J. NECK:
a. Trachea is normal position, no palpable mass.
b. Lymph nodes: no palpable
L. CARDIOVUSCULAR SYSTEM:
a. Inspection: size and shape of the chest is with the normal limits; no surgical scar
b. Palpation: carotid pulse and peripheral pulses are regular; normal sinus rhythm; rate-
80/mt
c. Percussion: cardiac borders well within normal limits, no cardiac or supra cardiac
dullness
d. Auscultation: S1 S2 auscultated. No abnormalities noted, pulse-80/minutes
M. ABDOMEN:
a. Inspection: size and shape of the abdomen normal, no distension and tenderness.
b. Palpation: no abnormalities found. Firm musculature noted
c. Percussion: no ascitis and fluid collection.
d. Auscultation: peristalsis heard in the right lower quadrant
N. BACK:
a. Spine and curvature: no abnormalities noted; no lymphs or lesions present
b. Movement: all movements are possible
c. Tenderness: no tenderness noted
O. GENITALIA:
Normal male genitalia; has no discharges
P. UPPER EXTREMITIES:
a. Deformity: no deformities noted
b. Swelling/ oedema: no swelling or oedema
c. Muscles: no emaciate
Q. LOWER EXTREMITIES:
a. Deformities: no abnormality noted
b. Swelling/ oedema: absent
c. Muscles: not emaciated
d. Lymph nodes: not palpable
e. Joints: normal ROM
R. NERVOUS SYSTEM:
a. Higher function: conscious and oriented
b. Memory: immediate is impaired and recent, and remote is intact.
c. Orientation: patient is oriented to time, place and person
d. Insight and judgement: normal
PROCESS RECORDING
CONVERSATION COMMENTS
N: Good morning! Gait normal
P: Good morning, sister!
N: Do you think drinking alcohol is right? Realizes his mistake and analysis
P: No, it’s wrong and feels shame for this habit. his behavior.
N: Do you like your parents and brother? Patient is attached with his family.
P: Yes, I like my parents and brother
N: Why you are quarrelling with brother?
P: When I drink alcohol I will lose my control.
N: What is your plan after discharge?
P: I want to go for a good job or start a business. Willing to work after discharge.
LAB INVESTIGATIONS
Sl Investigations Patient’s value Normal value Interence
No
1. Hemoglobin 11.2 gm % 13-18 gm% Slightly anemic
2. Total WBC count 6.600 cu mm 4,000-11,000 cu mm Normal
3. Polymorph 58% 60-70% Normal
4. Lymphocyte 36% 20-30% Normal
5. Eosinophil 4% 1-4% Normal
6. Serum creatinine 0.6 mg/dL 0.7-1.4 mg/dL Normal
MEDICATIONS
Drug Name Pharmacologic Dose Rout Action Side effects Nurses
al Name e responsibil
ity
T. Chlordiazepoxi Oral 15- Oral Anti- Nausea, vomiting, In
chlordiazepoxide de 100 mg IV and anxiety epigastric pain, injection
50-100 mg IV agent. diarrhea, should be
slowly impotence, given
impairment of deltoid and
driving skills, is gluteal
irritability. muscle.
T. Rantac Ranitidine Oral 150- Oral Histamine Headache,Dizzines Administer
300 mg IV H2receptor s,rarelyhepatitis,th correct
antagonist rombocytopenia,br dose
east symptoms,
hypersensitivity,co
nfusion,
T. BC BC 32.5mg oral Vitamin B Look for
and C Hypervitaminosis, side
complex G.I.Symptoms,yell effects,exp
supplemet. ow coloured urine. lain about
urine
colour
change
T. Liv 52 Sihymanin 140mg oral Liver Occasional See for
protectant laxative effects hypersensit
ivity.
T. Fluoxtine Fluoxtine 20mg oral Antidepres Nervousness,
sant insomnia, anxiety,
tremor, headache,
drowsiness, nausea
and drymouth.
ALCOHOL DEPENDENCE
INTRODUCTION
From time immemorial human beings have looked for substances to make life more pleasurable
and to avoid or decrease pain, discomfort and frustration. Despite definite improvements in
health care in most countries, problems related to drug and alcohol abuse are increasing almost
everywhere.
DEFINITION
Alcohol dependence was earlier called as “alcoholism”. Alcoholism is defined as a chronic
disease by repeated drinking that produces injury to the drunken health or to his social or
economic functioning.
EFFECT
Low to moderate consumption produces a feeling of well-being and reduced inhibitions. At
higher concentrations motor and intellectual functions are impaired, mood becomes very labile
and behavior characteristic of depression, euphoria and aggression are exhibited.
Alcoholic beverages are widely used in many societies because of which their abuse potential is
often under estimated. Commonly used alcohol preparations are beer, wine, brandy, whisky,
rum, gin, arrack and toddy.
EPIDEMIOLOGY
Epidemiological survey carried out in India reveal that 20 to 40 percent of subjects aged
above 15 are current users of alcohol and nearly 10 % of them are regular or excessive users.
Nearly 15 to 30 percent of patients seeking admission in psychiatric facilities are for alcohol
related problems.
Among the acute medical admissions in a general hospital 10to 20 percent are due to alcohol
related problems.
BOOK STUDY PATIENT STUDY
TYPES
A. Alpha alcoholism
Excessive and inappropriate drinking to relieve physical and for
emotional pain.
No loss of control.
Ability to abstain present.
B. Beta alcoholism
Excessive and in appropriate drinking.
Physical complications (e.g. Cirrhosis, gastritis, and neuritis) due to
cultural drinking patterns and poor nutrition.
No dependence.
C. Gamma alcoholism
Also called as malignant alcoholism.
Client belongs to type
Progressive course.
Delta alcoholism.
Physical dependence with tolerance and withdrawal syndrome.
Psychological dependence, with inability to control thinking.
D. Delta alcoholism
Inability to abstain.
Tolerance.
Withdrawal symptoms.
The amount of alcohol consumed can be controlled.
Social disruption is minimal.
E. Epsilon
Dipsomania (compulsive discharge)
BOOK STUDY PATIENT
CLASSIFICATION OF ALCOHOLISM
STUDY
Factors Type I Type II
Synonym Milieu limited Milieu – limited
Sex Both sexes Mostly in males greater
than 25 years .
Age of onset >25 years. <25 years.
Etiological Genetic factors Heritable
Contributory classification
Family history May be positive. Parental alcoholism and client comes
antisocial behavior under
usually present Type-1
Loss of control Present No loss of control
Other features Psychological Drinking followed by
dependence and aggressive behavior,
guilt present spontaneous alcohol
seeking.
Pre-morbid Harm avoidance,
personality traits high reward
dependence
BOOK STUDY PATIENT STUDY
ETIOLOGICAL FACTORS
BIOLOGICAL FACTORS
PSYCHOLOGICAL FACTORS
SOCIAL FACTORS
1. BIOLOGICAL FACTORS
Genetic vulnerability (family history of substance are disorder, eg, is
type II alcoholism).
Co-morbid psychiatric disorder or personality disorder.
Co-morbid medical disorders.
Reinforcing effects of drugs (explains continuation of drugs)
Withdrawal effects and caring. (Explain continuation of drugs).
Client was
Biochemical factors (e.g. role of dopamine and non epinephrine in
influenced both
cocaine, ethanol and opiod dependence.
by psychological
and Social factors.
2. PSYCHOLOGICAL FACTORS
Curiosity, need for novelty seeking.
General rebelliousness and social non-conformity.
Early use of alcohol and tobacco.
Poor impulse control.
Sensation – seeking (high)
Low – self esteem (anomia)
Concerns regarding personal autonomy.
Poor stress management skills.
Child hood trauma or loss.
Relief from fatigue and for boredom.
Escape from reality.
Lack of interest in conventional goals.
Psychological distress.
He was also
3. SOCIAL FACTORS
Peer pressure (often more important than parental factors.)
Modeling (imitating behavior of important other).
Ease of availability of alcohol and drugs.
Strictness of drug law enforcement.
Intra-familiar conflicts.
OTHER CAUSES
Interpersonal factors.
Socioeconomic factors.
influenced by easy
Cultural and ethnic factors.
availability and
Pharmacological factors.
socioeconomic
Ecological factors. factors
AVAILABILITY
Alcohol is easily available and drinking is accepted as a norm in
functioning and social gathering.
GENETIC FACTORS
Some excessive disorders have a family history of excessive
drinking. There is a genetic relation between alcoholism, depression
and antisocial personality disorder.
BIOCHEMICAL FACTORS
LEARNED BEHAIVOR
Children especially boys tend to follow their parents drinking pattern. Some
people drink to get away from pain.
PERSONALITY FACTORS
PSYCHIATRIC DISORDERS
Some patients with depressive disorders take to alcohol is the mistaken hope
that it will activate low mood. Persons suffering from anxiety disorders are
prone to take alcohol as an escape
SOCIAL CAUSES
Isolation, unemployment, loss, injustice and other social causes may lead to
Alcoholism
BOOK STUDY PATIENT STUDY
Client has the same
HIGH RISK GROUPS process of
development of
Persons suffering from chronic physical illness, business executives, traveling alcoholism.
sales persons, industrial workers, urban slum dwellers, students in hostel,
military personnel etc are more prone to develop alcohol abuse.
EXPERIMENTAL
To begin with, persons start drinking alcohol due to pressure and curiosity.
RECREATIONAL
Gradually, whenever they meet in functions like marriages, hostel day or
college day, parties, conferences, they drink occasionally.
RELAXATIONAL
Further, whenever they want relaxation, on holidays and weak ends they start
enjoying their drink and continue to do so. Hence the frequency gradually
increases.
COMPULSIVE
Some people who started drinking occasionally, start drinking almost daily or
drinking heavily for a period of time for pleasure or to avoid the discomfort of
withdrawal symptoms.
STAGES
EARLY STAGE
INCREASED TOLERANCE: Needing more and more alcohol to experience
the same pleasure as experienced earlier.
BLACK OUTS: Inability to recollect incidents which happened under the
influence of alcohol.
PREOCCUPATION: Always thinking about how, when and where to drink.
MIDDLE STAGES
Loss of control over amount, time and occasion of drinking. Keeping away
from alcohol for sometime but going back to obsessive drinking after each
such abstinent period.
CHRONIC STAGE
Getting drunk ever on small amounts of alcohol. Willing to i.e., beg, borrow,
or steal to maintain supply to alcohol. Living to drink – alcohol takes priority
over family or job.
DIAGNOSTIC EVALUATION
Certain laboratory makers of alcohol dependence have been suggested. There
include:
Physical examination. In case of client’s
History collection. physical
Neurological examination. examination, history,
Mental status examination. collection,
GGT (gamma glutyl transfarase) neurological
MCV (mean corpuscular volume) examination, MSE
and blood
GGT is raised to about 40 IU/L in 80% of alcohol dependant individuals. investigation
An increase in GGT of more than 50% in an abstinent individual signifies a done.
resumption of heavy drinking.
MCV is more than 92 fi (normal= 80-90 H) is 60% of alcohol dependent
Individuals.
OTHER LAB MARKETS
It includes,
Alkaline phosphatase.
AST
ALT
Uric acid
Blood triglycerides
CPK
COMPLICATIONS
I PHYSICAL OR MEDICAL COMPLICATIONS
A GASTRO-INTESTINAL SYSTEM
Gastritis.
Client developed the
Dyspepsia
complication of
Vomiting
vomiting, muscle
Peptic ulcer
wastage and vitamin
Cancer
deficiency.
Esophageal varices
Mallory-weiss syndrome
Achlorohydria
Carcinoma stomach and esophagus.
LIVER
Fathy degeneration of the liver.
Alcoholic hepatitis
Cirrhosis
Liver cell carcinoma
Liver failure
PANCREASE
Acute and chronic pancreatitis.
B CENTRAL NERVOUS SYSTEM
Peripheral neuropathy.
Delirium tremors.
Rum fits.
Alcoholic hallucinosis.
Alcoholic Jealousy
Wernicke- Korakoff psychosis
Alcoholic dementia.
Suicide
Cerebellar degeneration
Central posture myelinosis
Head injury and fractures.
C CARDIO VASCULAR
Alcoholic cardiomyopathy
High risk for myocardial infarction.
Cardiac beriberi.
Alcoholic myopathy.
Risk for coronary artery disease.
D BLOOD
Folic acid deficiency anemia.
Decreased WBC production.
Anemia, thrombocytopenia, vilk factor deficiency, hemolytic anemia.
E MUSCLE
Peripheral muscle weakness and wasting of muscles.
F SKIN
Spider angiomas.
Acnerosacea
Palmar erythema
Rhinophyma
Spider revi
Parotid enlargement
Ascitis.
G NUTRITION
Protein malnutrition.
Vitamin deficiency disorders like pellagra and beri-beri.
H JOINTS
Gouts due to increase in uric acid level.
I REPRODUCTIVE SYSTEM
Sexual dysfunction in males.
Failure of ovulation in females. Client does not
Pseudo-cushing’s syndrome, hypogonadium, gynecomastia (in men). develop
Ammenorhea, infertility, decreased testosterone and increased LH levels. the reproductive
complications
J PREGNANCY
Fetal alcohol syndrome- fetal abnormalities like mental retardation and
growth deficiency.
BOOK STUDY PATIENT STUDY
II PSYCHIATRIC COMPLICATIONS
WITHDRAWAL PHENOMENOM
The general withdrawal symptoms are – tremors, nausea and vomiting,
malacia, tachycardia, elevated BP, irritability, anorexia, insomnia, fits.
6. ALCOHOLIC DEMENTIA
A chronic organic mental disorder due to long term alcohol drinking.
Irreversible impairment in memory, orientation, impulse control, ability to
solve problems etc may be there.
OCCULAR SIGNS
Coarse nystagmus and opthalmopligia with bilateral external rectus paralysis
occur early. Pupillary irregularities, retinal haemorhages and papilladema
can occur causing impairment of vision.
2. KORSAKOFF’S PSYCHOSIS
As korsakof’s psychosis often follows wernicke’s encephalopathy, there are
together referred to as wernicke-kossakoff syndrome.Clinically, korsakoff’s
psychosis presents as an annestic syndrome,characterized by gross memory
disturbances with confabulation. In sight
is often impaired.
IV OTHERS
Alcoholic dementia.
Cerebellar degeneration.
Peripheral neuropathy
TREATMENT
Before starting any method of treatment, it is important to follow these steps. In case of client
i) Ruling out or diagnosing any physical disorder. behavior therapy,
ii) Ruling out or diagnosing any psychiatric disorder. psychotherapy,
iii) Assessment of motivation for treatment. group therapy are
iv) Assessment of social support system. carried out.
v) Assessment of personality characteristics of the patient.
vi) Current and past social, interpersonal and occupational functioning.
The treatment can be broadly divided in to two types, which are often
interlinked. There are detoxification and treatment of alcohol dependence.
PHYSICAL METHODS
I) Detoxification
II) Disulfiram therapy.
PSYCHOLOGICAL METHODS
I) Counselling
II) Individual and group psychotherapy.
III) Marital/ family therapy.
IV) Behavioral modification conversion therapy.
V) Relapse prevention therapy.
Rehabilitation
Alcoholic anonymous.
1. DETOXIFICATION
In case of clients’s
Detoxification is the process by which an alcohol dependent person recovers
assessment and
from the intoxicating effects of alcohol in a supervised way. It includes,
psychological
Administration of minor transquilisers (anti-anxiety drugs like
methods of treatment
chlordiazepoxide or diazepam) to control anxiety, insomnia agitation
are carried out.
and tremors.
Assess fluid and electrolyte balance for rehydration-IV fluids are
essential.
Re-establish proper nutrition by giving a diet high is protein (when
there is no liver damage), carbohydrate, vitamins C and B complex.
(especially vitamin B1, B6 and B12) preparation parenterally.
Provide calm, safe environment.
Control nausea and vomiting.
Administer anti-convulsant if there is withdrawal seizure.
2. BEHAIVOR THERAPY
The most commonly used behavior therapy is aversion therapy. Using
either a sub-thrushed electro shock or an emetic, like apomorphine.
3. PSYCHOTHERAPY
Supportive psychotherapy and individual psychotherapy have been used.
The patient should be educated about the risks of continuing alcohol use,
asked to resume personal responsibility for change and given a choice of
options for change.
4. GROUP THERAPY
Of particular importance is a voluntary self help group AA (alcoholics
anonymous) with branches all over the world and a membership in
BOOK STUDY PATIENT STUDY
Hundreds of thousands. Although the approach is partly religious in nature,
many patients desire benefits from group meetings.
5. DETERRENT AGENTS
The deterrent agents are also called sensitizing drugs.
CONTRAINDICATIONS
First trimester of pregnancy.
Coronary artery disease.
Liver failure
Chronic renal failure
Peripheral neuropathy
Muscle disease and history of psychosis in past.
6. PSYYCHOSOCIAL REHABILITAION
Rehabilitation is an integral part of multimodal treatment of alcohol Nursing care of
dependence. alcohol dependents
was given to Client.
NURSING CARE OF ALCOHOL DEPENDENTS
The nurse taking care of an alcoholic in a de-addiction ward should
Understand some basic concepts about the problem.
Alcoholism is a chronic disorder.
It is a relapsing disorder.
It is a disease affecting physical, mental and social well-being.
Not only does the individual suffer but his family, work and
community also suffer.
Accepting drinking as a problem by the patient is an important first
step, because most of the alcoholics deny that they are addicts.
(Denial).
They are prone to pathological lying and manipulative behavior.
The involvement of other significant persons especially the family
members enhances the recovery process.
MEDICATION
Follow medications as advised by doctor.
Anti-anxiety drugs like chlordiazepoxide (Librium) and diazepam, if
necessary, parenterally given.
Plenty of vitamins, especially Inj.B1, B6 land B12 and Tab B complex
and vitamin C.
Antacids to relieve gastritis.
Correct fluid and electrolyte in balanced by IV fluids.
NUTRITION
Take care of the nutrition of the patient.
Document intake, output and calorie content.
Weight daily.
Ensure that the patient receives small frequent feedings rather than
large meals.
Ask family members to bring food that the patient enjoys.
PRIMARY PREVENTION: Aim to avoid the appearance of new cases In nurse’s role in
of alcohol abuse by reducing the consumption of alcohol through heath prevention of alcohol
promotion, especially heath education. abuse tertiary
prevention was not
SECONDARY PREVENTION: Attempts to detect cases early and to carried out.
treat them before serious complications cause disability.
2. Subjective data: Impaired verbal To make the -to assess the -Patient’s speech of -To assess baseline data for The speech pattern of
Patient complains that communication affective patient about the pattern is assessed. further complications. the patient is cleared.
he is unable to speak related to slurred verbal pattern of speech. -rate, volume and -To know the rate of affected
due to shaking voice. speech and side communicat - To assess the rate, clarity of speech is speech.
Objective data: effects of ions by volume & clarity of assessed by conducting -To know that cause of the
Impaired verbal reducing speech. nursing process with the slurred speech.
medication.
communication related (slurred -to compare the patient.
to slurred speech. speech ) the relationship of - Relationship of the
effect of slurred speech with slurred speech is
withdrawal the side effect of compared with the
symptoms prescribed prescribed medication -To make the clear speech of
medication. (sedatives) (it can be the patient & to maintain the
- To provide speech due to the side effect of effective communication
therapy to the the medication). skills
patient. - Speech therapy is
-to report the provided to the patient.
presented sign of -Presented signs of
slurred speech the slurred speech is
psychiatrist. reported to the
psychiatrist.
3. Subjective data: Imbalanced To improve -To assess the -Assessed the condition -To asses the baseline data Nutritional status of the
Patient complaints nutrition: less than the condition of the of the patient. for further complications client is improved.
about the weakness & body nutritional patient - To correct fluid &
weight loss. requirement/fluid status of the - To provide - Parenteral Support is electrolyte imbalance,
Objective data: volume deficit. patient. Parenteral support provided to the patient. hypoglycaemia and vitamins
Nutritional level is may be required deficit.
altered as evidenced by initially. - To facilitate the repair of
loss of weight, poor - To Encourage -Encouraged cessation damage to GI tract.
skin turgor, electrolyte cessation of of substance abuse. - These interventions are
imbalance. substance abuse necessary to maintain an
- To consult with - Consulted with ongoing nutritional
Dietician regarding dietician regarding the assessment.
the no: of calories no: of calories require
required based on based on body size &
body size & level of level of activity. -To assess the effectiveness
activity. of nursing interventions.
-To document
intake & output and - Documented the
calorie count and intake and output & -diseased liver may be
weight client daily. calorie count and incapable of properly
- Ensure that the weight client daily. metabolizing protein
amount of proteins resulting in an accumulation
in the diet is correct - Ensured the amount of of ammonia in the blood.
for the individual proteins in the diet is - To minimize the fluid
client condition. correct for the retention.
- To restrict the individualised client
sodium may be condition. -To encourage the intake &
needed. facilitates client’s
- To provide small - Restricted the sodium achievement of adequate
frequent feeding of in the diet. nutrition.
client’s favourite
foods. - Small frequent feeing
- Supplement of client favourite foods
nutritious meals is provided.
with multiple
vitamin and mineral - Supplemented
tablets. nutritious meals with
multiple vitamin &
mineral tablet.
4. Subjected data: Altered physical To reduce -To assess the -Assessed the condition -To assess baseline data for Physical
Patient complaints of & psychological the condition of the of the client. further complications. &Psychological
body aches. dependence withdrawal patient. -To assess the present dependence is
Objected data: related to symptoms - To identify the -Identified the condition of the patient. improved.
Body aches are related withdrawal & help the withdrawal withdrawal symptoms
to withdrawal symptoms. patient in symptoms present t present in patient. -To provide comfort to the
symptoms of coping of in patient. patient and to treat the
de-addiction. withdrawal -To treat the -Treated the withdrawal symptoms.
symptoms. withdrawal symptoms -To reduce the pain & to
symptoms symptomatically. support the patient in
symptomatically. -Provided analgesics to withdrawal symptoms.
-To provide the patient as prescribed - To support the client in
analgesics to the by the physician withdrawal symptoms.
patient as prescribed (tab.Tramadol 10 mg
to the patient. bd). -To relieve anxiety of the
- To provide -Provided sedatives to patient.
sedatives to the the client (inj.serenace
client as prescribed 10mg od) as prescribed -To enhance the knowledge
by the client. by the physician. of the patient.
- To provide - Provided
psychological Psychological support
support to the client. to the client.
- To educate the -Educated the client
client knowledge knowledge about the
about the withdrawal symptoms
withdrawal of deaddiction.
symptoms of de-
addiction.
5. Subjective data: The patient will Ineffective -To help the patient -Helped the patient to -motivation for change is patient has substituted
Patient complaints that I substitute healthy coping identify the identify the substance related to recognition of a healthy coping
am feeling aggressive, coping responses related to substance abusing abusing behaviour and problem that is upsetting to responses for substance
because I am not for substance withdrawal behaviour and its its consequences. the patient. abuse behaviour.
tolerating body aches. abusing symptoms consequences. -Identification of
Objective data: behaviour. -To help the patient -Helped the patient predisposing factor and
Patient is aggressive identifying the identifying the precipitating stressors must
due to inability to cope substance abuse substance abuse precede planning for more
up withdrawal problems. problems. adaptive behavioural
symptoms. -To involve the -Involved the patient in responses.
patient in describing describing situations
situations leads to leads to the substance
the substance abuse abuse behaviour.
behaviour.
-To offer support -Offered support
consistently & consistently &
expectations that the expectations that the
problem has the problems had the -Denial and rationalization
strength to strength to overcome are dysfunctional coping
overcome the the problem. mechanism that can interfere
problem. with recovery.
- To Encourage the -Encouraged the patient
patient to participate to participate in a
in a treatment treatment programme. -Socially isolated people who
program. use drugs to gain confidence
-To help the patient -helped the patient in social situations.
identify and adopt identify and adopt -substance abusing behaviour
healthier coping healthier coping alienates significant others,
responses. responses. thus increasing the person’s
-To Identify & -identified & assessed isolation.
assess social social support systems
support systems that that are available to the
are available to the patients.
patients.
-To educate the -Educated the patient
patient and and significant others
significant others about the substance
about the substance abuse problems and
abuse problems and available resources.
available resources.
6. Subjective data To improve the Insomnia -To assess the -Assessed the pattern of -To assess the baseline data Sleeping pattern is
Patient complaints that sleeping pattern of related to pattern of sleep. sleep. for further complications. improved of the client.
he is unable to sleep at the patient emotional - To restrict the -Restricted the visitors Or
night from 1 week. disturbances visitors at the time at the time of sleep. -To enhance sleep at night. Insomnia is treated with
Objective Data: of sleep. -To reduce the body activity the following
Disturbed sleeping - To avoid day time -Avoided day time & to enhance the sleep. implemented
pattern is related to naps naps. interventions.
withdrawal symptoms -To avoid the -Avoided the
of deaddiction or consumption of consumption of
emotional disturbances. stimulant stimulants at night time. -To prepare the environment
supplements at to enhance the sleep
night like tea or psychologically.
coffee. -To enhance the sleep of the
-To provide calm & -Provided calm & quiet patient at night.
quiet environment environment to the
to the patient. patient. -To sedate the client & to
-To Encourage the -Encouraged the patient enhance the sleep.
patient to take warm to take warm glass of
glass of milk before milk before going to
going to the bed. bed.
-To administer the -Administered the
sedatives to the sedatives (inj-serenace
patient as prescribed 10mg I/M) as
by the physician. prescribed the
physician.
7. Subjective data: Knowledge deficit Knowledge -To assess the -Assessed the previous -To assess the level of The knowledge is
Patient complaints that related to the deficit previous knowledge knowledge of the understanding of the patient. enhanced of client
what are the hazardous substance abuse related to of the patient. patient. -To enhance the knowledge regarding the substance
effect of substance and its treatment. substance -To provide -Provided knowledge of the Patient. abuse & its treatment.
abuse, advantage of abuse, knowledge regarding the diseased
treatment given to me. treatments regarding condition, its effect on
Objective data: and its (substance abuse ,its health.
Knowledge deficit effects hazardous
related to substance physical
abuse and of its ,environmental
treatment . ,social -To enhance the knowledge
,psychological of the patient regarding the
effect) -Provided knowledge treatment.
-To provide the regarding advantages of
knowledge the treatment given to
regarding the the patient. -To enhance the knowledge
treatment given to about the benefits of
the patient. compliance of regimen &
-To provide -Provided knowledge side effects of non
knowledge about about the compliance of compliance.
the compliance of
treatment regimen. Treatment regimen.
-To provide proper -Proper time is provided
time to the patient to the patient to clear
to clear his enquires his doubts regarding the
& doubts regarding provided knowledge.
provided
knowledge.
CLIENT EDUCATION
Help the patient to quit the plan
Set a quit date-ideally
Tell family and friend support
Anticipate changes like alcohol withdrawal syndromes
FAMILY EDUCATION
FAMILY EDUCATION
Help family members to recognize the danger situations and explain the chance of
relapse.
Avoid others drinking in front of patient
Remove all products of alcohol from the surro8unding prior to alcohol cessation
Accomplish life styles that reduce stress.
Improve quality of life or produce pressure learning cognitive and behavioral
activities to cope.
CONCLUSION
As a part of my clinical requirement I selected Amrik singh with diagnosis of alcohol
dependence for my case presentation and by treatment his condition is improving. By taking this
case presentation I attained adequate knowledge about the disease condition and its management
in detail, which will help me in caring such patients in future.
BIBLOGRAPHY
2. Sreevani.R.A Guide to Mental and Psychiatric nursing.2 nd edition. New Delhi. Jaypee
publishers.2007; p.129-134