The Acute Painful Red Eye: History of Presenting Complaint - The Time and Speed of Onset

Download as docx, pdf, or txt
Download as docx, pdf, or txt
You are on page 1of 4

Red eye

History
 History of presenting complaint –
o The time and speed of onset
o Ocular associations (e.g.photophobia, blurry vision, discharge etc)
o Systemic associations (e.g. headaches, nausea, rash on the forehead)
o Don’t forget the other eye
 Past ocular history - similar episodes, other episodes, surgery, 'lazy eye' (which would
guide you as to whether the recorded visual acuity is worrying or not). Do they or have they
worn contact lenses and ask about their level of hygiene (do they forget to clean them?
Forget to take daily disposables out at night?).
 Social history – in contact with other patient with same symptoms

Examinations
 Visual acuity (VA)
 Pupils and their reactions should also be checked (look for the distorted or small pupil of
acute anterior uveitis or the mid dilated fixed pupil of acute angle closure glaucoma).
 Is the cornea intact? – fluorescein eyedrops

The acute painful red eye 2

Suspected Common symptoms Common signs Referral urgency


condition

Acute angle Severely painful, haloes Decreased VA, hazy Refer immediately.


closure glaucoma around lights, may be cornea, fixed, semi-
systemically unwell dilated or oval pupil.
(nausea, vomiting,
headache). Usually > 50yo.

Keratitis/corneal Photophobia, foreign body VA depends on exact Fluorescein drop


ulceration/abrasion sensation ± history nature of problem -
of contact lens wear ± peripheral lesions may
previous episodes (e.g. cause little change but
herpes simplex infection). some decrease is
expected. Corneal defect
on staining ± hypopyon
(pus seen in anterior
chamber).

Acute anterior Photophobia, blurred VA may be reduced, A slit lamp reveals white


uveitis/iritis vision, headache, pain on redness more localised precipitate on the back of
accommodating. May have around corneal edge the cornea & cells in the
been unresponsive to (ciliary injection), pupils anterior chamber
previous treatment for may be constricted or
conjunctivitis. irregular. When severe,
white cells precipitate on
Associated w:- corneal endothelial
- AS surface (seen as white
- IBD clumps - keratic
- RA precipitates).

Trauma e.g. foreign Pain depends on type of Depends on trauma. Patient needs to have a full
body (FB) trauma, severity and slit-lamp examination -
location. refer immediately if risk of
serious trauma.
The acute non painful red eye 2

Suspected condition Common symtpoms Common signs Tx

Conjunctivitis • Discomfort or itch. Normal VA unless Chloramphenicol


- Bilateral w discharge • Discharge (watery or corneal involvement,
purulent). uni or bilateral, Chlamydia (use
Causes:- • Crusting of lid margins discharge expert help) –
- Viral (adenovirus) – • General flu-like symptoms in infective corneal scarring
small lymphoid in viral cases. conjunctivitis,
aggregates appear as  History of contact with follicles or papillae, Staph – if
follicles on conjunctiva people with red eyes. may be eyelid resistant to cipro
- Bacterial (purulent swelling then use
discharge prominent) – ±conjunctival gentamicin
oedema.

Episcleritis Mild discomfort, few Normal VA, localised Topical/systemic


- Inflammation below th symptoms. patch of NSAIDs
econjuctiva in the - Associated w SLE/PAN redness/injection
episclera is often seen rheumatic fever which blanches on
with an inflammatory application of a drop
nodule. of phenlyepherine
2.5%. No discharge.

Subconjunctival May be spontaneous or Blood under Refer if traumatic.


haemorrhage traumatic, can occur after conjunctiva covering If not, check BP in
prolonged coughing. part or all of eye elderly patients
Asymptomatic. which is otherwise (can occur with
quiet with normal hypertension) and
VA. reassure: should
resolve over a
fortnight
Trauma
 High Velocity Foreign Bodies
 Penetrating injuries
 Blunt Trauma
 Burns
 Lacerations and Abrasions

History
 Time of injury
 Mode of injury:
o Physical vs chemical
o Superficial vs blunt
o Speed of impact
o Nature and size of object
 Were glasses or goggles worn?
 Possible foreign body (on the surface or penetrating)?
 Other injuries sustained and treatment received so far
 Previous acuity (even if just a rough estimate) and any eye problems
 Past medical history
 Medication (e.g. anticoagulants), allergies, tetanus immunisation

Examination
Your examination will be dictated by the patient's ability to co-operate (level of consciousness,
pain, intoxication, age - although children as young as 3 or 4 can manage a slit lamp in the right
conditions) and to a certain extent, your confidence.

1. Your examination must be complete - assume the worst until you have ruled it out.
2. Start with visual acuities of both eyes - the patient can often give an indication of
whether the current acuity seems about right for them or not. Document what you find:
this is invaluable when assessing how things are evolving from the earliest assessment
following injury.
3. Examine the eye from front to back, doing as much as your equipment allows (you may
need a drop of local anaesthetic if the patient cannot open their eyes due to pain):
o Orbits and lids: lacerations, subcutaneous emphysema, bruising, oedema. If you
think there may be a fracture, measure medial intercanthal distance (this should
be 35-40mm in adults). Could the bilateral bruising actually be due to a base
of skull fracture rather than an eye injury? (And conversely, rule out eye injury in
the patient with 'panda eyes' from a base of skull fracture). Evert lids.
o Conjunctiva: look for haemorrhage and lacerations (these show up on staining with
fluorescein).
o Cornea: lacerations may be small and missed. Perform a Seidel test first (to assess
for leakage from cornea) and then assess for corneal abrasion with dilute
fluorescein.
o Anterior chamber: blood there collects inferiorly in the erect patient to produce a
fluid level (hyphaema) and damage to the iris will result in an irregular or
abnormally reacting pupil.
o Fundus: a loss of red reflex could be due to opacification from blood in the
vitreous or a large retinal detachment.
o Ideally, intraocular pressure should also be assessed unless you suspect a
perforating injury.
4.Do a functional examination: movement of the eyes (ask about diplopia before and during
examination), pupil reactions and a confrontational visual field test.

Note that the degree of pain in ocular trauma does not necessarily correlate with the severity
of injury.

Time may be of the essence where a periocular haematoma develops: if this is severe, the window
of opportunity to examine the eye may close quickly and not reopen for several days.

You may find our record on Examination of the Eye useful. Techniques are outlined at the end of
this record.
Orbital injuries
Blow-out floor fracture
This is typically caused by a sudden increase of orbital pressure caused by a striking object (e.g.
fist or tennis ball). Clinical features vary with the severity of the trauma and the time between
trauma and presentation. Common findings include:

 Bruising and oedema ± subcutaneous emphysema.


 Anaesthesia over the region supplied by the infraorbital nerve (lower lid, cheek, side of
nose, upper lip, upper teeth and gums).
 If the inferior rectus gets trapped within the fractured bone, there will be restriction of
upper gaze with associated diplopia (which also occurs on downward gaze). These
movements are associated with pain.
 Diplopia may also occur due to haemorrhage and oedema within the orbit.
 There may also be a degree of enophthalmos and globe damage should be ruled out.
Other findings may include epistaxis, ptosis and trismus.

Other orbital fractures


 Medial wall fracture - this tends to be associated with orbital floor fractures. They are
characterised by periorbital subcutaneous emphysema (crepitus) which develops when
the patient blows their nose. This should be discouraged (infected sinus contents can be
forced into the orbit). If the medial rectus gets entrapped, there will also be restriction
of ocular adduction and abduction.
 Lateral wall fracture - this is the most solid of the orbital walls and so lateral wall
fractures most often occur in association with extensive facial damage.
 Roof fracture - this is a less common fracture. In children, it is more commonly caused by
minor trauma (e.g. falling on a sharp object or a blow to the forehead). In adults, it
tends to be caused by major trauma associated with other craniofacial bone disturbances
(e.g. displacements) or fractures. Patients typically have a haematoma of the upper
eyelid that rapidly spreads around the eye (over a few hours) and sometimes to the
fellow eye.There may be globe displacement and in severe cases, there may be pulsation
of the globe.
Fractures should be imaged (X-ray or CT; avoid MRI if there is the possibility of a metallic foreign
body) and the patient referred (there will often be a joint ophthalmology and maxillofacial input -
the neurosurgeons may also be involved depending on the nature of the injury). Meanwhile, provide
antibiotic cover and instruct the patient not to blow their nose.

You might also like