Immune Tolerance and Slow-Virus Disease: Skeletons in The Closet of Western Science & Public Health, by A. W. Finnegan

[The Institute of Biological Ethics & Historical Research Initiative]
Immune Tolerance and Slow-Virus Disease: Skeletons in the

Closet of Western Science & Public Health1
By: A. W. Finnegan
Submitted January 1, 2020
Abstract: The mechanisms of chronic disease had been discovered as far back as
1936 by German Virologist Erich Traub, and later termed immunological tolerance,
or immune tolerance, for short. Unfortunately, what could have added voluminous
contributions to the nature and science of chronic disease and its infectious etiology
was occulted in 1960 and attributed to organ transplants. The fields of immunology,
vaccinology, and infectious disease were severely and negatively affected by the
masking of immune tolerance, and public health has suffered irreparable damage.
The spread of chronic disease and para-infectious injury has been steadily
increasing since the early 1950s and beyond. Western prophylactics and over-
vaccination have added significant burden to already overwhelmed immune systems
and vulnerabilities in manufacturing have greatly contributed to exacerbating the
problem. The secondary neurotropic effect on society has been disastrous and far-
reaching. The cumulative effect of incomplete science and immunology on society
may be unfathomable in scope.
In 1960, Western science and public health systems began their long, treasonous
descent into a one-sided, misleading paradigm of immunology and infectious
disease. This step not only ignored the science of this discovery, but went as far as
to mask the infectious origins of what is known as "immune tolerance" by giving the
Nobel Prize to two scientists, Burnet and Medawar, not even nominated together,
and attributing the condition to be restricted to the same condition seen in organ
transplants.i This scientific discovery which underlies chronic disease was actually
discovered by German researcher Erich Traub starting in 1936 with the virus of
Lymphocytic Choriomeningitis Virus (LCM), but his contribution was not
publicized or credited to Traub, it was instead given to other scientists having to do
with something more obscure.ii This was an act that would bring untold suffering
and unnecessary harm for future generations, because it was at this point that science
and public health began to turn their backs on a large portion of immunology and
1
Special thanks to Ms. Kathleen Dickson for her work and teaching me of the fundamental concepts in immunology
and immune tolerance as a post-sepsis syndrome
one-half of the entire spectrum of disease- chronic disease- and medical progress has
been moving nearly in reverse ever since.iii
This spectrum of disease has been covered in layers of scientific fraud and
unnecessary controversy.iv This act of denying the infectious processes of immune
tolerance many years ago, was a pivotal moment in which profiteering and ego
became more important than the easing of human suffering, because if the science
of immune tolerance had been acknowledged for what it was and the implications
that it implied, those in the established science and public health systems would have
been forced to rethink the entire paradigm of immunology, vaccination,
inflammation, and the very nature of disease itself. This would indicate that the act
was done, not just because of the controversial background of its German pioneer,
Erich Traub, but because of conflicts of interest involving profiteering and the
reputation of establishment science and Big Pharma.
Immune Tolerance, as I will show, is the term that denotes the condition that
underlies most if not all of chronic disease, a relapsing/remitting yet progressive
condition that ties autoimmune conditions, genetic disorders, endless syndromes,
and mental illness to para-infectious mechanisms, through a disrupted immune
system and neurotropism, by the proliferation and persistence of a disseminated viral
syndrome, where latent viruses already present in the body will reactivate and turn
on the body, making the bulk of the condition itself.v, vi, vii, viii It is akin to turning
one’s own microbiome against itself, and no doubt, it is why so many people have
so much trouble getting well.ix This process and proliferation of antigen makes it
hard to absorb and take in nutrients, oxygen, and the like,x because there is a state of
balanced parasitism, and the person will tend to operate at a very low capacity, being
malnourished and highly diminished vitality. It is like being somewhat choked out
or suffocated at the cellular level.xi
Immune tolerance will often present without any outward symptoms, but the effect
is still damaging due to the parasitic nature of the para-infectious invasion which
remains in the body, and active infection can be entirely present without antibodies.xii
Oftentimes this balanced parasitism will turn in favor of the parasitism of virus and
antigen. Erich Traub and his mentor Karl Beller noted this in his studies of an AIDS
virus of horses, Equine Infectious Anemia Virus (EIA), and the resultant condition
was not a true immunity:
It is therefore very questionable whether this is a true immunity. The term
"prämunity", which originates from French and becomes more and more natural in
German terminology, would probably be more appropriate here. In human and
animal protozoal diseases, after the survival of the clinical phenomena existing
equilibrium state between parasite and organism, in which the latter is usually
resistant to a new infection. Due to damaging influences on the body, this state of
equilibrium can change in favor of the parasite. Similar observations were made in
[infectious anemia].xiii
As a result of this truly immoral act of burying the science of immune tolerance, a
harmful butterfly effect would take root and branch into every facet of life, every
angle of health would be significantly affected, to such an extent that it would change
the course of history in untold, unprecedented ways.xiv This fraud would have a
cumulative effect that would only grow in intensity over the years, each decade
would become more unstable, and to cover these tracks, more unsound explanations
would have to be layered over it, and the more out of line with Truth reality became,
the more chaotic and unstable its consequences would follow in public health. If
there is any hope of fixing this diabolical treason, we must revisit the past, and recall
the events in which it took root...
Traub’s Discovery of Immune Tolerance

In 1935, a German student studying on a scholarship at the Rockefeller Institute,
Erich Traub, was studying the reactivation of latent viruses in equine
encephalomyelitis and swine fever virus (hog cholera),xv after transmission studies
with Richard Shope in Iowa on the pseudorabies virus, a swine herpesvirus unrelated
to rabies,xvi after which Traub began a series of experiments in mice, where he was
attempting to reactivate a latent virus to cause an epidemic in the mouse colony at
the Institute. He injected the mice intracerebrally with foreign proteins or antigen
suspended in sterile bouillon, and within short order, an outbreak ensued and
infected most of the other mice in the colony, though they all had been otherwise
healthy mice before the experiment:
During recent work with the viruses of equine encephalomyelitis and hog cholera
an infective agent was obtained from white mice which was pathologically and
serologically distinct from both viruses. Its origin was not definitely known, but it
seemed likely that the natural host of the agent was the mouse, in spite of the fact
that in our mouse colony no disease had been previously recognized. In an
experiment designed to trace the origin of the infectious agent, 60 five-week-old,
healthy-looking mice from our colony were each given an intracerebral injection of
a small amount of sterile bouillon. Fifty-one of these mice showed no evidence of
illness during the three weeks that they were under observation. Four died in from
3 to 13 days following the inoculation, and three were killed on from the sixth to the
eighth day when they showed symptoms similar to those observed in the mice
inoculated with the unknown agent. On the sixth day two additional mice that
showed photophobia but no other symptoms were killed. From one of these mice no
material was obtained for inoculation, but bacteriologically sterile suspensions of
the brain of each of the other eight when injected into guinea-pigs caused symptoms
which could not be differentiated from those produced by the original material. This
experiment, together with others, suggests that the infectious agent is carried by
apparently healthy mice in our colony and that symptoms may be brought out by the
intracerebral injection of foreign protein.xvii
The outbreak, however, was not just restricted to the mice, because soon some of the
lab technicians and animal keepers contracted the virus and had to be hospitalized,
infected with this new virus, which they called Lymphocytic Choriomeningitis Virus
(LCM):
In 1935 an epidemic of meningitis of a special type (acute lymphocytic
choriomeningitis) broke out in the mouse colony in Princeton. Erich Traub
recovered a virus to which he gave several years of intensive study before he left the
Institute in 1938. [Interestingly], one of the Princeton staff contracted this disease
during the mouse epidemic and was studied at The Rockefeller Institute Hospital by
T.M. Rivers and T.F.M. Scott, who isolated from him a filtrable agent identical with
Traub’s. Traub’s observations show that the mouse is the natural host of this
potentially serious infection, which, fortunately, only occasionally causes illness in
human beings. The virus, kept alive in mice by healthy carriers, is acquired by infant
mice from their mother while still in utero or shortly after birth. Usually remaining
latent, like many other viruses it is occasionally awakened to virulence by some
accidental circumstance, causing a disease which spreads with epidemic rapidity.xviii
This was transmissible and able to spread from animal to human. Traub studied this
virus in the colony for several years and noted its persistence in the colony,
establishing acute and chronic infections, which set the discovery of immune
tolerance in 1936.xix He noted congenital transmission from mother to newborn,
where the virus would persist and become increasingly more silent in the colonies,xx
but later in life the infected stock would present with blood cancers and leukemia,xxi
as well as chronic, progressive neurodegenerative diseases in what was later termed
early and late onset disease,xxii resembling debilitating chronic conditions that
progressively destroyed the central nervous systems of these mice as they got
older.xxiii The study would be some of the early models of progressive
neurodegenerative chronic diseases which would later become so prevalent in
Western countries.xxiv
Traub’s seminal work on LCM virus became the model of an immunosuppressive,
relapsing chronic disease, which would become so prevalent in Western societies in
later generations. It was termed immune tolerance, in that the body’s immune system
was not immune to the virus, as in actively neutralizing it, but rather tolerized to the
virus, meaning it would not respond to it, little to no antibodies would be produced
after the initial infection, and the virus would be able to continue its replication and
spread, establishing in the infected subject a slow-virus disease.xxv The slow-virus
disease meant that the disease took a much slower, chronic course. The mother
would transmit the virus to the young, and the young would be born tolerized to the
virus, not showing outward symptoms but having a slow, chronic infection which
later in life would turn to chronic disease and result in neurodegeneration,
lymphomatosis and cancers.xxvi, xxvii
Traub noticed that antibodies were minimal in the infected colonies, even though
they were carriers and shedders of active virus. However, when the rest of the stock
was more or less immune tolerant, it would be hard to tell this was the case, but the
effect could be spotted if these mice were put into an uninfected colony.xxviii Another
phenomenon observed in the infected stock, was that the virus only remained in the
blood for several weeks and then disappeared from the blood, taking to organ,
tissues, brain, and some of the secretions and excretions.xxix
What Traub’s studies in LCM taught us was that, first, there were such thing as
infections that failed to produce noticeable antibodies, making today’s diagnostic
methods and approaches to infectious disease somewhat meaningless.xxx If viral
infections that fail to produce antibodies were diagnosed through the detection of
antibodies, it would miss these cases and falsely declare them negative for infection.
Also, with viral infections known to clear from the blood on top of the little to no
antibodies produced, serological testing was not sufficient to confirm infection. This
would indicate that the state of surveillance for many infectious diseases could be
vastly underestimated and incompetent.
Secondly, the process in which Traub was describing immune tolerance, was
actually the way that vaccines were imparting their so-called immunity, they were
not necessarily neutralizing virus from the body, the body was rather being tolerized
to certain antigen.xxxi It would trade the acute expression for a slow, chronic course,
but came with neurologic side effects.xxxii The antibodies produced by vaccination
were only initially generated, and eventually would just fail to produce a response
when tolerance was achieved. The vaccine then does not induce immunity, as in
neutralizing the virus from the body when future infection is contracted or present,
it just tolerizes the immune system not to respond to antigen, and thus, the shedding
of virus still occurs.xxxiii Live virus vaccines are claimed as setting up harmless, latent
infections, but rather, these are slow-virus infections, and the harm can still be
destructive in the end, but the course is much slower, while reactivation can also
occur.xxxiv Tolerization is not a harmless process and the condition brings a multitude
of other complex and chronic health problems,xxxv such as progressive
neurodegenerative diseases,xxxvi autistic spectrum disorders,xxxvii gastrointestinal
problems,xxxviii secondary opportunistic infections not commonly seen or understood
by the average physician or even infectious disease specialist.xxxix This was the trade
off to acute disease for most people with the introduction of vaccinations.xl
There were still some cases whose immune systems mounted a more robust immune
response to these class of antigens, remaining with the acute presentations, also
higher allergic response to vaccines with these antigens. It was a small percentage
of roughly 15%, while 85% presented with immunosuppression. Lyme disease was
one example of a pathogen that induced this immunosuppressive condition and the
problems associated with it. This disease also showed that this 85% tested negative
to the disease while actively infected, as the antibody response was minimal. The
other 15% had a more noticeable disease but these cases were also more responsive
to antibiotic therapy. The same agent produced a variable response in different ratios
of people with variable HLA makeup. There was even a certain small percentage
appearing to present with both outcomes simultaneously.xli
Vaccines like the polio vaccine, contaminated with adventitious virus like SV40
virus, is an example of a virus that mirrored the LCM virus.xlii Many of the human
herpesviruses like Epstein-Barr Virus, HHV-6, Herpes-simplex, were also viruses
that could act much like LCM virus when reactivated to active form.xliii In many
ways LCM served as an animal model of the herpesviruses in humans.xliv Vaccines
were oftentimes contaminated with adventitious animal viruses that mirrored LCM
virus,xlv and the lipoprotein antigens used in vaccines could also cause the effect of
reactivating latent viruses in its human recipients,xlvi much like Traub’s experiment
in mice with foreign antigen injected into the brain.xlvii
I. Immune Tolerance & Vaccinations: A Forecast of Unforeseen
Complication and Crisis
There are serious issues of concern in attempting to mass immunize or compulsively-

immunize a population with varying degrees of immunosuppression from stealth
infections,xlviii, xlix, l along with failure to detect adventitious virus, that is, hidden
viruses and pathogenic microorganisms within the material as contaminants in
commercial vaccines and gene therapies, which has not been given a significant level
of attention, but presents a significant danger to those receiving them with the
aforementioned dilemmas left unaddressed, and common people are paying the price
with their livelihood in such unfortunate situations.li Unfortunately, this topic has
become so laden with controversy and heated debates, that very few will speak freely
about it. However, it must be addressed one way or another, because safety comes
before reputation, if one is truly a health practitioner.lii There are two main factors
involved that make the overall picture a serious problem in regards to this picture,
where stealth infections have proliferated the population causing a crisis of both
chronic disease and mental illness. The two main points in the risk of immunizations
in the Western countries are:
a. Stealth Infections and Immune deficiencies.
Those already dealing with one or several stealth infections causing

immunodeficiencies, immunosuppressed at the time of vaccination, and the
immune response is poor or suboptimal, giving latent viruses within the
host or vaccine material itself a chance to revert to active virulence.liii
When a child or individual is overburdened by stealth, subclinical viral infections at
the time of birth, born immune tolerant through congenital transmission, along with
the older age groups who have picked up several infections in life such as the
arthropod-borne diseases, hepatitis, and so on, the stacking of additional
immunizations present a dilemma and danger to the public.liv, lv There is very little
done to check a child for immunodeficiencies at the time of immunization. Blood
work is likely to appear normal, without antibodies to the infections, and thus,
declared healthy. Traub’s work on LCM virus shows that the virus would pass
through generations of mice, with the virus gaining momentum with each subsequent
generation.lvi This same picture could be playing out in the population today. The
rates of chronic disease, autism, cognitive and behavioral health problems,lvii, lviii
have reached an all-time high in Western civilizations, yet have the highest rates of
immunization.lix
Even so-called non-infectious antigen disseminates to the brain and CNS,lx, lxi
causing cognitive difficulties,lxii arthritis and fibromyalgia.lxiii Essentially, it appears
we are trading acute diseases for chronic diseases; especially disseminated slow-
virus infections through the reactivation of latent herpesvirus.lxiv Compounding this
problem, the number of immunizations a child needs to have just to attend school
are significantly higher, and growing.lxv The compulsory vaccine practices of the
West are essentially fanning the flames of a complex picture of immune tolerance
and systemic proliferation of many stealth infections circulating the environment.lxvi,
lxvii
Stealth infections are evading standard medical diagnostics for infectious disease,
little to no antibodies despite circulating virus or pathogen, the doctors see no clinical
symptoms and designate them healthy.lxviii Next, they suggest several
immunizations, such as influenza, meningococcus, pneumonia, and so on.lxix These
antigenic and live virus vaccines add significantly more burden on the individual’s
immune system, with injurious, and in some instances deadly consequences.lxx If we
continue this path, the problem will get exponentially more complex and problematic
for the public health system,lxxi already overwhelmed by the massive rise in chronic
diseases,lxxii mental health problems,lxxiii and so on.
Risks associated with immunizing an already overburdened immune system poses a
very real danger. Researchers out of John Hopkins University Paul Auwaerter et al.,
published Altered Virulence of Vaccine Strains of Measles Virus after Prolonged
Replication in Human Tissue, whereby an infant was killed by a vaccine strain of
measles after its immunization, reflecting the mechanism laid out within this work:
However, fatal infections have been documented in immunodeficient children
vaccinated with these strains (1, 12, 14, 15). The symptoms of infection occur
many months after immunization, and the viruses isolated are similar to the
original LA vaccine (1, 15), suggesting that in the absence of an effective host
immune response, persistent infection with the vaccine strain can lead to fatal
disease. Viruses isolated from these children could potentially represent
virulent revertants of the original LA vaccine.lxxiv
A second, more indirect manner in which vaccine viruses may revert to active form,
is by transmission of arthropod diseases after bites from ticks, mosquitoes, and other
insects, which Traub had revealed in his 1939 thesis on immunity against viral
disease, with the classical horse-sickness as an example:
In contrast to the neurotrophic yellow fever virus in monkeys, the neurotropic
horse-sickness virus in an intracerebrally-vaccinated horse caused only a
febrile temperature increase, but no encephalitis, while producing in mice
always fatal encephalitis. The virus circulates in the blood of the horses during
the fever period, which may be a drawback in that the horse sickness is most
likely transmitted by blood-sucking insects; which is not guaranteed that the
vaccine virus in the body of the presumed recipient cannot be transformed back
into the original agent. However, this risk should be lessened by the fact that
the vaccinations are carried out in a season in which blood-sucking insects do
not occur or only in small numbers.lxxv
The herpesvirus group, which includes 8 predominant forms, including Herpes-
simplex (HHV 1, HHV-2), Varicella Zoster Virus (VZV, or chicken pox, HHV-3),
Epstein-Barr Virus (EBV, as HHV-4), Cytomegalovirus (CMV, as HHV-5), Human
Herpesvirus 6 (HHV-6), Human Herpesvirus 7 (HHV-7), Human Herpesvirus 8
(Kaposi’s Sarcoma-associated Virus, as HHV-8) is latent in most if not all human
beings in one of the aforementioned forms. EBV and Herpes-simplex tends to be the
most common of the herpesviruses seen, followed by VZV and CMV. The
herpesvirus family can be neurotropic and greatly contribute to the neurological
disorders commonly seen today, as laid out in a publication, Herpesvirus Infections
of the Nervous System, by two researchers of neurological diseases in human
beings:
RECENT FINDINGS: As more patients are becoming therapeutically
immunosuppressed, human herpesvirus infections are increasingly common.
Historically, infections with human herpesviruses were described as temporal
lobe encephalitis caused by herpes simplex virus type 1 or type 2. More
recently, however, additional pathogens, such as varicella-zoster virus,
Epstein-Barr virus, cytomegalovirus, and human herpesvirus 6 have been
identified to cause serious neurologic infections. As literature emerges, clinical
presentations of herpesvirus infections have taken on many new forms,
becoming heterogeneous and involving nearly every location along the
neuraxis. Advanced diagnostic methods are now available for each specific
pathogen in the herpesvirus family. As data emerge on viral resistance to
conventional therapies, newer antiviral medications must be considered.
SUMMARY: Infections from the herpesvirus family can have devastating
neurologic outcomes without prompt and appropriate treatment. Clinical
recognition of symptoms and appropriate advanced testing are necessary to
correctly identify the infectious etiology. Knowledge of secondary neurologic
complications of disease is equally important to prevent additional morbidity
and mortality. This article discusses infections of the central and peripheral
nervous systems caused by herpes simplex virus type 1 and type 2, varicella-
zoster virus, Epstein-Barr virus, cytomegalovirus, and human herpesvirus 6.
The pathophysiology, epidemiology, clinical presentations of disease,
diagnostic investigations, imaging characteristics, and treatment for each
infectious etiology are discussed in detail.lxxvi
The herpesvirus group of viruses are not limited to human beings, but are found in
nearly all animal species. Many diverse animal tissues are used to produce vaccine
material, which brings us to the second factor of major concern in the problem of
para-infectious and post-vaccinal encephalitis:
b. Contaminants (adventitious or latent viruses, Mycoplasma,

Toxoplasma, Acholeplasma, fungal and bacterial pathogens, etc.).
Commercial vaccines with pathogenic contaminants present in the vaccine

material can produce a suboptimal immune response following the
immunization, with latent viral contaminants concurrently inherited by the
recipient,lxxvii resulting in immune deficiency or encephalitis,lxxviii giving the
vaccine material an opportunity to reactivate and regain virulence,lxxix
especially live vaccines, which then circulate and shed from the body by
their secretions and excretions, transmitting the pathogen to circulate the
surrounding environment and those within close proximity to the
individual.lxxx
Contaminants in commercial vaccines is not a new problem. lxxxi It has been a thorn
in the side of vaccine manufacturers all along, who have continuously been plagued
by contamination in batches of vaccines already on the market circulating widely.
For instance, Salk’s inactivated polio vaccine (IPV) was circulating and given out
widely to millions of Americans and Western countries, but took five years before
they discovered the vaccine was contaminated by several viruses, such as SV40, and
additional polyoma viruses.lxxxii The estimation of those potentially infected was
more than half the population, and close to 100% in some age groups, stated by NIH
and John Hopkins researchers in Reviews and Commentary Human Exposure to
SV40: Review and Comment:
Highly reliable information is available on the numbers and age distribution
of the population immunized with inactivated poliovirus vaccines. These data
were collected in yearly national household sample surveys conducted by the
Bureau of the Census, in collaboration with the Communicable Disease Center
(64). A summary of their report for the September, 1961 survey (51) is given in
table 2. Over 98 million people, or 62 per cent of the population, had received
one or more doses of the vaccine during the period when a proportion of the
vaccine was contaminated with SV40. In 1961, potentially contaminated
vaccine had been administered to almost 90 per cent of individuals under 20
years, 60 per cent of those 20-39 years old and 19 per cent of those 40-59 years
old.lxxxiii
Later in the report shows higher cancer rates from those born to mothers who had
the polio vaccine, mirroring Traub’s studies of LCM during his Rockefeller years,
as the virus is passed continually through generations:
Heinonen et al. (74) have recently reported a higher incidence of malignancies
in children born to mothers who received inactivated poliovirus vaccine during
pregnancy. In a prospective study of over 50,000 pregnant women between
1959 and 1965 (Collaborative Perinatal Project of the National Institute of
Neurological Diseases and Blindness), 24 malignancies in their newborns were
detected in the first 4 years of life. The rate of malignancy was about two-fold
greater in children born to mothers immunized during pregnancy.lxxxiv
These adventitious or hidden viral contaminants are potentially carcinogenic,
especially when reactivated from latent to active infection. Even after the problem
was discovered, they were not able to rid the vaccines of these contaminants, as
Konstantin Chumakov et al., stated in a paper, Some oral poliovirus vaccines were
contaminated with infectious SV40 after 1961:
Inactivated poliovirus vaccine (IPV) and live oral poliovirus vaccines (OPV)
were prepared in primary cell cultures derived from rhesus monkey kidneys.
Studies of these vaccines led to the discovery of a new virus called SV40 in
1959. This DNA virus caused vacuolization of green monkey cell cultures and
was found to be highly oncogenic in hamsters (reviewed in refs. 1, 2). It was
found that SV40 was endemic in rhesus monkeys. For this reason, the rhesus
kidney cell cultures used to manufacture poliovirus vaccines as well as some
seed stocks of poliovirus contained infectious SV40. (2–5). Therefore, the early
batches of OPV contained infectious SV40 (3–5). Because formaldehyde
treatment used to prepare IPV failed to completely inactivate SV40, some
batches of IPV contained infectious SV40 (2, 4). As a result, it has been
estimated that > 100 million people in the United States and many more
worldwide received potentially contaminated vaccines prepared during the
years 1954 to 1961 (2).lxxxv
This problem is still plaguing the manufacture of commercial vaccines, with
inadequate testing, or contamination that occurs later in the vaccine material.lxxxvi
Many papers have been written in recent years discussing this dilemma.
Furthermore, many of these animal virus contaminants are oncogenic viruses or
immunosuppressive retroviruses like avian leukosis virus, discussed in Evidence of
avian leukosis virus subgroup E and endogenous avian virus in measles and
mumps vaccines derived from chicken cells: investigation of transmission to
vaccine recipients:
Reverse transcriptase (RT) activity has been detected recently in all chicken
cell-derived measles and mumps vaccines. A study of a vaccine manufactured
in Europe indicated that the RT is associated with particles containing
endogenous avian retrovirus (EAV-0) RNA and originates from the chicken
embryonic fibroblasts (CEF) used as a substrate for propagation of the
vaccine. We investigated the origin of RT in measles and mumps vaccines from
a U.S. manufacturer and confirm the presence of RT and EAV RNA.
Additionally, we provide new evidence for the presence of avian leukosis virus
(ALV) in both CEF supernatants and vaccines. lxxxvii
In Viral Nucleic Acids in Live-Attenuated Vaccines: Detection of Minority
Variants and an Adventitious Virus, the process is admitted, yet rather downplayed
in its extent, where viral material within a vaccine can recombine and turn
pathogenic:
In very rare instances, one attenuated viral vaccine, the oral poliovirus vaccine
(OPV), can accumulate mutations as well as recombine with other coinfecting
enteroviruses and revert to a pathogenic state. (18, 24). Attenuated live
vaccines also carry a potential risk of contamination with adventitious viruses
introduced during the attenuation process, from the cell lines used, and/or from
the animal sera or other biologics often used in cell cultures. Very early
Theiler’s yellow fever attenuated virus was once “stabilized” with human
plasma thought to contain hepatitis B virus, resulting in many cases of hepatitis
(5, 28). Some early Sabin poliovirus vaccines were contaminated with the
simian virus 40 (SV40) polyomavirus from the monkey cells used to amplify
polioviruses.lxxxviii
The Division of Viral Products, Office of Vaccines Research and Review, Center for
Biologics Evaluation and Research, Food and Drugs Administration (FDA),
admitted in a publication, Issues associated with residual cell-substrate DNA in
viral vaccines, that the contamination with viral substrates of commercial lots of
vaccines was inevitable, as well as being potential carcinogens:
The presence of some residual cellular DNA derived from the production-cell
substrate in viral vaccines is inevitable. Whether this DNA represents a safety
concern, particularly if the cell substrate is derived from a tumor or is
tumorigenic, is unknown. DNA has two biological activities that need to be
considered. First, DNA can be oncogenic; second, DNA can be infectious. As
part of our studies to assess the risk of residual cell-substrate DNA in viral
vaccines, we have established assays that can quantify the biological activities
of DNA. From data obtained using these assays, we have estimated the risk of
an oncogenic or an infectious event from DNA.lxxxix
II. Immune Tolerance, HLA Variation, Disease Transmission and Outcome
Fast-forwarding to the present day, there has been a sudden change in some of the
public opinion of those who question the narrative and recklessly jump to the
conclusion that there are no such things as transmissible germs and viruses, at the
suggestion of Thomas S. Cowan and his book, the Contagion Myth, who claims that
germs and viruses cannot be transmitted, that all problems are toxic chemicals in
nature, the underlying tone is that we live in harmony with germs and viruses.xc Are
we currently living in harmony with nature? Not at all, so why wouldn’t these viruses
and microbes turn against us, with such widespread engineering and introduction of
foreign biological constructs that the planet has never seen before? xci
The germ denial discussion would polarize the two sides in typical fashion, making
an already divided discussion polarized and more complicated. It pops up almost
overnight in the middle of the COVID-19 situation after one of his videos went viral
and received millions of views. It appears a convincing argument, since both Koch’s
postulates and Rivers’ postulates do not satisfy the situation, and this remains with
many other forms of infectious disease. However, Traub’s discovery in LCM and
his later creation of widespread weapons and some natural diseases with a slow,
debilitating nature, no outward symptoms and hard-to-isolate viruses and microbes
that took to tissue and brain matter, obliterated both Koch’s and Rivers’ postulates,
because the slow-virus disease, also seen in bacterial and fungal infections, basically,
anything containing the immunosuppressive lipoproteins would cause a very
different picture of disease, but nonetheless equally painful and misery-inducing,
producing chronic disease and mental illness simultaneously, that was extremely
difficult to diagnose and treat.
It would be hard to prove something caused the same disease in every subject, when
the HLA-makeup of each person is variable, producing a variable response, most
(roughly 85%) getting a disease that takes years to incubate and really gain
momentum, while others may have an acute response (the much smaller minority of
roughly 15%),xcii just as some people have aggressive hyperallergic reactions to
vaccines, while some get a pronounced immunosuppression. Some may not get
either forms, and that is the nature of the variable immune systems different people
have. Not all people have the same immune system, and germs will produce a
different picture in different people, so there is no unanimous result which these
postulates can be satisfied.
There were some viruses, such as Newcastle Disease Virus and various retroviruses,
that undressed their virions into small exosome-like particles, some have termed
these viromicrosomes,xciii and the undressing occurs right before entering the cell,
then hijacks parts of the cell lipids, before exiting as a complete virion, now
containing pieces of the cell’s genetic material.xciv This would produce endless
variants as the virus passed through any population with a variation of genes.xcv This
result is typical with any virus or agent that contains these certain antigens and
lipoproteins called fusion proteins.xcvi The influenza groups, foot-and-mouth disease
virus, HIV, and many additional pathogens contained such components.
The problem, however, is that the medical and science establishments of the time
decided to bury Traub’s findings, first, because there was a deeper biological
weapons context to all of this. Secondly, and perhaps more so, the discovery was
also showing that the mechanisms of immune tolerance were quite similar to how
western vaccination was playing out, being that there was indeed harm being done,
a rebound effect in which the immune systems were being slowly burned out not to
respond, but the harm was in some ways more extensive, proliferative, and
permanent.xcvii, xcviii, xcix, c The immune system not only fights off foreign invaders,
but keeps us vital and vigorous to live life. Without this, one will have the full
spectrum of chronic fatigue, fibromyalgia, meningo-encephalitis, and of course,
mental illness. This is still, very much a disease, but can take some time to develop.
This was discussed in two prior articles I’ve written. Tainted Immunity: Post-
Vaccinal Encephalitis and the Chronic Plague of Societal Degeneration and
Medical Misanthropology: A Tale of Vaccination, Clinical Psychiatry, and Mental
Illness.
Cowan put forward the argument that the Spanish Flu was not transmissible, because
Pfiffer’s bacillus isolated from cases of Spanish Flu used in experiments did not
cause Spanish Flu in other subjects.ci Though bacterial agents of many various kinds
were isolated in brain and tissue samples of fatalities resulting from the epidemic, cii
the cause was viral in origin,ciii but of course, the effect of this virus on the immune
system in the initial viral infection would cause a viral-bacterial synergy resulting in
bronchopneumonia, which was a combined secondary infection, back in those days
this was also called infectious respiratory catarrh, or ”mixed infections” another
important line of work that Erich Traub studied in more depth.civ, cv This was a
process in which the virus came through and obliterated the immune system and the
bacterial meningitis would often deliver the deathblow.cvi This is the nature of
targeting and modulating the immune system,cvii throwing the immune system out of
whack would open the flood gates for reactivation of latent germs and the invitation
of secondary opportunistics, such as those seen in AIDS, but in a much more
accelerated manner. It was not the result of Pfiffer’s Bacillus, and attempts to
reproduce the disease with such a germ would typically be fruitless. This was just
one example of some of the mistakes I noted reading Cowan’s book, he did not take
into account many of these factors I am laying out now. In light of the slow-virus
disease and variable HLA-makeup of different people, Cowan’s theories should be
reconsidered and seen through the light of immune tolerance and HLA variation.
At any rate, it can be agreed that there are serious problems in the realm of
vaccination having serious detrimental effects in public health amidst these immune
complexes.cviii There are so many issues and complications involved within the inner
mechanics of vaccination, especially in populations riddled with varying degrees of
immunodeficiency. Vaccines may quiet the acute expression of disease by inducing
tolerance to specific pathogens and viruses, which would be a good thing if not for
the horrific rebound effect of causing severe long-term permanent chronic
diseases.cix, cx One can think of disease not just as an acute expression, but the other
end of this spectrum is disease of a chronic nature that accompanies
immunosuppression.cxi
The process is one in which the person contracts an infectious disease, say a virus or
spirochetal infection from a tick bite, the onset of septicemia occurs, and this would
present with a flu-like illness. This septicemic reaction then disables the immune
system,cxii and while the immune system reaches a compromised state, latent
herpesviruses like Epstein-Barr Virus (EBV), Human Herpesvirus-6 (HHV-6),
Herpes-simplex (HSV-1), and many others, reactivate from within the B-cells,
causing B-cell Immortalization, where unhealthy cells are in a sense zombified yet
transformed into a veritable virus factory following the reactivation.cxiii In normal
subjects, these infected cells would normally undergo a process called apoptosis,
where the unhealthy cell destroys itself so the immune system can then produce new
healthy B-cells.cxiv In this case, however, the B-cell Immortalization causes
inhibition of apoptosis,cxv and the transformed, these zombified B-cell lives on
indefinitely in a state of incompetence.cxvi
The viruses that reactivate in this state of immunodeficiency may be numerous. The
Epstein-Barr Virus (EBV) may be just one among many concurrent and persistent
viral infections of the herpesvirus family among other latent vaccine viruses that
come in with the multiple vaccines given during the lifetime of the individual. cxvii
Following this state of immunodeficiency, opportunistic infections will often present
concurrent infections like Mycoplasma pneumoniae, Chlamydia pneumoniae, and
many other pathogens that normally are not a problem for healthy individuals.cxviii
Occasionally, animal diseases not known to infect humans can be present which
most doctors and even infectious disease specialists will have no experience in
dealing with, such as granulencephalitis (Encephalitizoon caniculi), a blood parasite
of rabbits.cxix
The secondary effect of the immune tolerant state is one of neurotropic effects on
the brain and central nervous system. The neuroimmune system has to work
overtime to keep up with the infectious invasion of para-infectious material like
antigen, virus, and pathogenic microbes. Neurotropism is the degradation of the
central nervous system and brain, and the result will impart neurological and
psychological manifestations due to the continuous low-level neuroinflammation
that results.cxx I covered the para-infectious nature of neurotropism and its effects in
Medical Misanthropology: A Tale of Vaccination, Mental Illness, and Clinical
Psychiatry, an article which cites many papers illustrating the nature of these
processes on the brain and central nervous system. antigen alone will have the same
neurotropic effect, invading the brain leading to chronic neuroinflammation and
neurologic disease.cxxi, cxxii
III. Viral Proliferation, Immune Tolerance, and Resulting Mental Illness in

Targeted Nations of Bioterrorism & Over-Vaccination
If one studied the life work and publications of Erich Traub, this picture mirrors
much of what Traub discussed in his published work, and the picture would present
a very risky, dangerous road we’re going down. The massive rise in chronic disease,
mental illness, and neurological disorders reflects the effect of both a collectively,
overburdened immune system of the population, and, the unrestricted contamination
of commercial vaccines, with many of the contaminant viruses being
neurotropic,cxxiii the antigens and viral envelopes have an affinity for brain tissue and
the nerves,cxxiv and the reactivation of latent herpesviruses, along with new viruses
passed in through immunizations, harbors the conditions for such to occur.cxxv This
results in deterioration of the brain and central nervous system, resulting in
neurological problems, and progressive degenerative diseases.cxxvi The autistic
spectrum would also fall into this category. Extensive mental health problems would
be expected.
Traub’s work with Borna Disease Virus, Eastern Equine Encephalitis, Pseudorabies,
swine encephalomyelitis, neurotropic-modified Foot-and-Mouth Disease Virus
(FMD), among other disease agents, gave him a superior understanding of the
complex interrelationships between immune tolerance and neurotropism. In human
beings, some of these same neurotropic viral infections have been documented and
studied, and present with neurological, cognitive, and mental health problems.cxxvii
The autistic spectrum of disease becomes relevant here, as a form of neurotropism
which is many times accompanied by immune deficiencies, allergies, auto-immune
problems, and so on.cxxviii This would lend more weight to the relevance of Traub’s
work in these areas,cxxix which brings in crucial elements relevant to biodefense and
National Security, because a very serious health risk is present as several stealth
pathogens are filtering through the population, without signs of slowing down, and
the stacking of additional vaccines on top of those infected makes the problem
infinitely more complex and problematic. cxxx, cxxxi
Vaccine revertants such as those described by Auwaerter et al., along with the
reactivated herpesviruses are generally of the neurotropic class, and highly
variable.cxxxii The action of systemic viral infections with a predilection for brain and
nerve tissue has a profound effect on psychological process. Mental illness would
be the inevitable expression of these viral infections and antigen, as German
researcher Karl Bechter pointed out in his 2013 paper, Virus Infection as a Cause
of Inflammation in Psychiatric Disorders:
Many neurotropic viruses exist and may cause classical inflammation but
also low-level neuroinflammation. However, viruses may be dormant within
the CNS and become active later. The role of neurotropic virus infections in
the causation of psychiatric disorders may be underestimated, because the
diagnostic approach to the CNS is difficult and to dormant infections in
general, but especially within the CNS. Evidence is increasing that infections
increase the risk of psychiatric disorders, not only prenatal infections but
also infections during the lifetime. The question how low level
neuroinflammation may be involved in severe psychiatric disorders like
affective and schizophrenic spectrum disorders is intriguing but remains to be
studied. Experimental data clearly show that low-level neuroinflammation
can be induced by viruses, but the definitions of inflammation and low level
neuroinflammation appear to be blurred and apparently the previous
classical definition of inflammation has to be widened. Virus infection itself
or virus-related products or virus-induced autoimmunity may play a role in
disease pathogenesis. More sensitive diagnostic approaches from
neuroimaging and CSF investigations may hold the key to a better
understanding and definition of CNS viral infections as an etiopathogenetic
sub-group of severe psychiatric disorders.cxxxiii
Bechter also studied this further with one of Traub’s later colleagues, Rudolf Rott,
published MRI in psychiatric patients with serum antibodies against Borna disease
virus regarding the findings of Borna Disease Virus antibodies in psychiatric
patients, as evidence of infection among the mentally ill, but not in the healthy
controls:
Investigations in the FRG and the USA revealed the presence of serum
antibodies against the [Borna Disease (BD)] virus in psychiatric patients with
affective psychoses, but not in healthy controls. We demonstrated the
occurrence of BD virus-specific antibodies in sera of about 7% of the
psychiatric inpatients at our hospital, not only in patients with affective
psychoses but also in those with other psychiatric disorders. By comparison, in
surgical patients from an endemic region, only 3% were found to be
seropositive, some of whom had psychiatric or neurological disturbances. The
question was whether the BD virus-positive seroreaction was a coincidental
and harmless finding or whether it could represent the cause of psychiatric
disorders.cxxxiv
Theoretically, a strike with several biological agents on a population for strategic
purposes as a long-term strategy could realistically be carried out whereby a nation
is systematically destroyed very slowly, starting from a few small incidents or
maneuvers, (i.e. attacks that release infected arthropods into the ecosystem,
contamination of commercial vaccines, gene therapies, and prophylactics),cxxxv the
target nation would be largely unaware that it was even under attack, and if they
began to catch on, it would seem smaller in scope, yet infinitely worse in
actuality.cxxxvi Its effects would be cumulative and gain momentum over many
decades, with a culminating snowball effect restricting the response to the point of
being overwhelmed and too heavily burdened by it to recuperate,cxxxvii and for that
reason it becomes a subject most relevant to Western & American National Security.
Part of such an attack depends on the target country’s response, namely the
deployment of vaccines, which make the agents released more vicious, as mentioned
in The Darker Bioweapons Future, an intelligence assessment on biological
warfare and bioterrorism:
According to the scientists convened, other classes of unconventional
pathogens that may arise over the next decade and beyond include binary BW
agents that only become effective when two components are combined (a
particularly insidious example would be a mild pathogen that when combined
with its antidote becomes virulent); “designer” BW agents created to be
antibiotic resistant or to evade an immune response; weaponized gene therapy
vectors that effect permanent change in the victim’s genetic makeup; or a
“stealth” virus, which could lie dormant inside the victim for an extended
period before being triggered. For example, one panelist cited the possibility
of a stealth virus attack that could cripple a large portion of people in their
forties with severe arthritis, concealing its hostile origin and leaving a country
with massive health and economic problems.cxxxviii
IV. A Theoretical Picture of Systemic Immune Tolerance and Neurotropic

Injury in Societal Structures
Because the immune tolerant condition tends to happen in varying degrees, usually
simultaneously to auto-immune problems,cxxxix oftentimes subclinical or inapparent
in outward appearance,cxl and since the condition of Traub’s immune tolerance was
not given significant enough attention in public health’s attack on human infectious
disease even after his publishing on it all the way up to his death in 1985,cxli a
continual failure to detect chronic infections would inevitably result. Traub noted
the lack of antibody response in tolerant carriers, which were also chronically
infected and later displayed signs of chronic health problems, the picture of such an
immunological burden in a society would, in theory, present very similarly to the
conditions we are seeing today, which Traub first modeled in white mice:
Mice infected in utero with LCM virus carry large amounts of active virus in their
blood and organs for many months and discharge considerable quantities of the
agent with their secretions and excretions (11). This observation indicates
continuous multiplication of the virus in the organs of such animals, which look like
normal individuals in spite of their chronic infection. Intrauterine infection leads to
a stable equilibrium between virus and body. It was also observed that mice infected
in utero would develop practically no specific antibodies, while the sera of mature
mice infected experimentally or by contact at least gave positive complement-
fixation (CF) tests (9, 14).
Attempts to demonstrate neutralizing antibodies in the sera of such animals using
customary techniques gave essentially negative results (for references see 13). More
sensitive methods, however, have made the demonstration of such antibodies
possible (7), Recent experiments have shown that their titer is relatively low and
their antiviral action exceptionally slow (13). The failure to recognize this fact no
doubt accounts for the negative results obtained earlier by different investigators.
For several reasons it is still doubtful whether these antibodies represent the
immunity factor of prime importance in mice recovered from experimental infection
(10, 7, 13). The solid immunity of mice infected in utero is obviously not due to
antibodies, since none could be demonstrated with sensitive methods even after
repeated inoculations of virus (12, 13) and since life-long persistence of large
amounts of active virus in the body would hardly be compatible with the antibody
theory.cxlii
Later in the same paper, Traub talked about the mechanisms of immune tolerance
found in other antigens like pneumococcal polysachharides:
The organs of congenitally infected mice, young or old, generally contained more
antigen than those of mature mice infected artificially. The very high antigen content
of the lymphatic system in tolerant animals has been correlated with the tolerance
phenomenon and the possibility considered that their immunological apparatus may
be blocked by specific antigen as in mice showing "immunological paralysis"
following inoculation with large doses of pneumococcal polysaccharide.cxliii
The academic and biodefense specialists have known about the mechanisms of
immune tolerance in other areas. For instance, immune tolerance has gone by other
names, such as endotoxin tolerance,cxliv and, as Traub pointed out, immune paralysis
which is perhaps a more fitting term.cxlv With early studies supported by the National
Institute of Allergy and Infectious Disease, this is not a case where the science of
immune tolerance was unknown and seen as a harmless or even healthy, it was
deliberately settled on as a form of immunity, to allow vaccine practices and
monopolies on public health operating under an outdated and incomplete picture of
immunology to take precedence over well-being. Profiteering and selfish corporate
agendas certainly come into play, giving them a reason to bury what Traub had
contributed to immunology, in the public health system and medicine, by awarding
the discovery to Burnet and Medawar regarding organ transplants.cxlvi The result
would be chronic disease paralyzing the immune system like those who survive
sepsis.cxlvii
One of Traub’s protégés in Germany, Zvonimir Dinter, writing in a later article
submitted to the German medical journal Berliner und Münchener tierärztliche
Wochenschrift in 1984, told his experience working with Traub, and suggested that
he should have been part of the Nobel Prize for his discovery of immune tolerance
in LCM virus:
Traub, "who started the whole thing." The TRAUB's lived on Riems in a mansion
when I entered the villa, it was already twilight, you could hear the monotonous
splashing of the Baltic Sea, it was a mood where memories are easily awakened.
After coffee with cake, TRAUB told me how he came to the US when RICHARD E.
SHOPE, the famous virologist from the Rockefeller Institute, spent his time in
Giessen, among other places, in the early 1930s. Zwick's work on the Borna Disease
attracted researchers who poured in... like TRAUB, who was already considered a
student proficient in English, to SHOPE he was assigned as a language facilitator.
SHOPE soon saw TRAUB's interest and talent in viral research and invited him to
the Rockefeller Institute. Shope was right in his assumption. Much later (1959), he
told me himself.
TRAUB's talent soon starts after he began research at Princeton, the Rockefeller
Institute branch. The topic to which he dedicated his work in the first place was the
behavior of the mouse lymphocytic choriomeningitis virus. A number of
communications that began in Science and continued in the Journal of Experimental
Medicine, and which are generally regarded as classical today, since "... many
pioneering series of investigations in many of the key biological properties of the
virus were established. "(1) When BURNET and MEDAWAR received the Nobel
Prize in 1960 for their seminal immunological work, I, as a veterinarian and
TRAUB’s employee, would have liked to see that he, too, had participated in the
prize because what TRAUB already described in 1936 and 1939, was the mode of
origin of the condition, which was later baptized with "immune tolerance" and
which represented a pillar of Burnet's clone-selective theory (2). The American
researcher HOTCHIN has appreciated TRAUB's dedication by giving him his
monograph with the words: "To ERIC TRAUB who started the whole thing"
(3).cxlviii
The condition and its process would also be increasingly more complex as
vaccinations were stacked on top of those already dealing with one or several chronic
viral infections congenitally transmitted from mother to newborn, with no outward
signs of disease upon birth, tolerized to virus, yet the effect of infection in the
lifetime of that individual would be cumulative,cxlix with early and late onset
disease,cl while the increasing number of vaccines given in the schedule adds
infinitely more burden to an already confused and exhausted immune system,cli it is
akin to trying to put a fire out with gasoline. Many people today can think of several
families with autistic or mentally ill children, and the effect on a nation, if such
angles were used in special weapons of bioterrorism geared for strategic, long-term
attacks, a target country would be plagued by chronic health problems and mental
illness, with failure to detect infection, and simply labelled ‘somatoform’ or
‘mentally ill’ with no infectious disease,clii yet was very likely dealing with one or
several infections. The course of disease by the immune tolerance complex would
present only general similarities between subjects, with different courses and
outcomes for each.cliii The result could be so various that the condition is grouped
into many separate conditions between physical and mental health, leaving health
practitioners and public health systems little to no understanding of the causal factors
of immune tolerance and neurotropism.
The effect on mental health would present with varying degrees and combinations
of neurotropism and damage to the brain,cliv expressing mood swings, lack of
organization and cognitive abilities, irritability, rage, senseless acts of violence,
irrational behaviors, lowered inhibitions resulting in an increase in criminal activity
or deranged behavior, such that is very common in the headlines of Western nations
today.clv Simply put, to target the brain of a population would affect the societal
structure as a whole, because the chaos of one effects the many, and when large
numbers of unstable citizens with unpredictable and irrational or violent behaviors
from neurotropic infections of the brain and CNS are scattered throughout its society
presenting in this manner,clvi that is, infections passing congenitally through
generations gaining more momentum in the subsequent generations infected,clvii the
implications are systemic and far-reaching, tearing at the fabric of the society itself,
as we are seeing the effects today, with Western countries leading in chronic disease
and mental illnesses.clviii, clix
If this idea on the current picture of public health is taken as a whole, perhaps just
as devastating effects would be felt by even the upper echelons of society,
particularly in the science and medical communities, as the incompetence to deal
with this problem inevitably reached its conclusion,clx and the truth made its way
out.clxi Perhaps the fact of the matter may be that the scientists and public health
systems have no definitive game plan since treating virus infections is not curative
but supportive. The integrity of the medical and scientific establishments in countries
where the problem was consistently denied, covered-up, ignored, missed, or left
unaddressed, would have its reputation greatly challenged in the public eye of trust.
If specialists, along with pharmaceutical firms sought to influence the public health
system’s officials and academic scientists in attempts to avoid dealing with this
problem for profit-driven agendas, such as testing methods that miss the problem in
very clever ways as to conceal the presence of infections and the immune tolerant
state, there would have to be prosecutions, penalties, and reparations paid to those
affected if trust were to be re-established.clxii Laws have been pushed in place which
allow the drug firms, unprecedented control through experimental conditions on a
public without its knowledge of experimental phases.clxiii
Lyme disease and SARS-CoV-2 serve as modern examples of the corrupt practices
in effect today, where science and public health officials are perpetuating the same
fraud overlapping other diseases.clxiv For example, the diagnostic approach to Lyme
disease at the 1994 Dearborn conference attempted to phase out and scrub the
seronegative infections in immune tolerant outcomes from the books, changing the
definition of the disease to be only an acute presentation with a more robust immune
response.clxv, clxvi This outcome would only present in a small minority of cases, while
the rest would be immunosuppressed and overrun by chronic health problems,
brushed under the rug and slandered as psychiatric cases.clxvii, clxviii
The LYMErix vaccine would also induce the immune tolerant state and injuries
would follow.clxix Indeed, the vaccine had to be pulled from the market within two
years of its implementation when reports of severe adverse events began to
surface,clxx but the public health officials attempted to coverup the problem by
having the doctors label the reactions unrelated to the vaccine.clxxi Finally, an FDA
hearing led to its ultimate removal from the market, with disturbing testimonies from
those who had now permanent health problems and unable to work or live the active
life they once had.clxxii
SARS-CoV-2 is another example of the same class of antigen on a virus disease.clxxiii
This problem spells out very clearly the problematic nature of public health’s
reliance on antibody response and acute expression.clxxiv The immune tolerance
would be achieved in the first two weeks of infection, with the end result of reaching
an indefinite state of immune tolerance.clxxv They began to employ semantical terms
like “COVID long-haulers,” and “long-COVID,” to mask the idea of chronic
infection.clxxvi This post-viral syndrome described was the same as that described in
other immune tolerant states following infection, such as post-polio syndrome,clxxvii
post-Ebola syndrome,clxxviii post-treatment Lyme disease,clxxix and post- anything was
a term for the immune tolerance imparted from the initial infection, as Kathleen
Dickson of the TruthCures organization accurately described this as a post-sepsis
syndrome.clxxx
The SARS-CoV-2 vaccines would also be inducing immune tolerance as its
mechanism for immunity, with lipids polyethylene glycol (PEG) and other synthetic
chemicals to mimic the lipid envelope expressed on the virus.clxxxi PEG is known to
cause immune tolerance,clxxxii and this would indicate that the mechanism for
immunity they were aiming for was immune tolerance, or burning out the immune
system.clxxxiii This same mechanism of immune tolerance was also decided as an
acceptable form of immunity in the poultry industry after widespread epidemics in
the 1950s.clxxxiv, clxxxv They considered burning out the immune system a viable
solution to infectious disease,clxxxvi and this same approach carried over into the
approach to public health in humans.clxxxvii In effect, they were adding insult to injury
with more severe and permanent degrees of immunosuppression and tolerance to
mask the acute diseases, which they considered the only form of disease, while
labelling the chronic cases a social phenomenon or construct of somatoform.clxxxviii
Had Traub’s discovery of the para-infectious role of immunological tolerance been
accepted and included into the science back in the early 1960s,clxxxix public health
and medicine would look very different today.cxc The entire spectrum of disease and
immunology would have been reconfigured to be aligned with truth and legitimate
science, which could have put forward legitimate solutions and remove the unsound
and destructive health effects of vaccination.cxci, cxcii Instead, the entire spectrum of
chronic disease was purposefully buried and denied,cxciii, cxciv in opposition to
legitimate science.cxcv Profiteering took precedence over genuine well-being,cxcvi
monopoly on health practices and experimental testing programs would be
safeguarded by laws passed to protect Big Pharma under the pretext of
biodefense,cxcvii allowing the people of the country vulnerable and lied to.cxcviii The
science was not working in the aims of Greater Good, but a greater greed with an
insatiable appetite for power and control,cxcix as the chronic health problems and
secondary neurotropism set the pretext in which the implosion of Western society
would inevitably result.cc, cci
In the words of Dr. Erich Traub’s protégé, Werner Schafer, a telling revelation
spoken at a conference for the Office of Naval Research in Kansas in 1962 may once
again hold the key to understanding the situation, albeit through the overtones of
unbelievable irony:
I hope I have been able to demonstrate that the concentrated action of our group on
a very small sector of animal virology was not quite unsuccessful but has created at
least some ideas of possibly a more general importance for the fight against virus
diseases. At the present time, when concentrated scientific activity is often directed
toward the destruction and not toward the preservation of life, one might fear that
the phrase "concentration is the prudence of life" will someday be inverted into
"concentration is the stupidity of life." To avoid such an occurrence, scientific
workers all over the world would do well to return to the ethics of the stoicist Seneca,
who noted in his Naturales Quaestiones that "by exploration of the facts natural
sciences base the moral life of man on a solid foundation; they liberate him from
fear and let him recognize the glory and magnitude of divine creation."
- Dr. Werner Schäfer, ONR lecture, presented at the Annual Meeting of the American
Society for Microbiology, Kansas City, Mo., 6 May 1962.ccii
i
Silverstein, Arthur M. “The curious case of the 1960 Nobel Prize to Burnet and Medawar.” Immunology
vol. 147,3 (2016): 269-74. doi:10.1111/imm.12558
ii
Dinter, Z. Persönaliches, Begegnungen mit Erich Traub.[Personal Encounters with Erich Traub] Berl.
Munch. Tier. Woch. 96. 70-72. (1984)
iii
with the para-infectious origins of immune tolerance obscured and attributed to organ transplants only,
the entire spectrum of chronic disease would be buried, and one-half of the spectrum of immunology
would be ignored. The science and public health would keep antibody response and acute presentation of
disease the only form of disease, and this was erroneous, incomplete, and unscientific.
iv
Lyme disease being one example. See: Dickson, Kathleen. TRUTHCURES.ORG CRIMINAL
CHARGE SHEETS JUNE 2017, 2017, pp. 39.
https://docs.wixstatic.com/ugd/47b066_01d68b1309ae457b81df1e06e6beae1e.pdf
v
See the mechanisms described in Traub’s first paper: Traub, E. A Filterable Virus Recovered from
White Mice. Science, 81. (2099), 298-299. doi:10.1126/science.81.2099.298. (1935).
vi
Schmidt RE, Grimbacher B, Witte T. Autoimmunity and primary immunodeficiency: two sides of the
same coin? Nat Rev Rheumatol. 2017 Dec 19;14(1):7-18. doi: 10.1038/nrrheum.2017.198. PMID:
29255211.
vii
Pierangeli A, Antonelli G, Gentile G. Immunodeficiency-associated viral oncogenesis. Clin Microbiol
Infect. 2015 Nov;21(11):975-83. doi: 10.1016/j.cmi.2015.07.009. Epub 2015 Jul 18. PMID: 26197213.
viii
Lomonte P. Herpesvirus Latency: On the Importance of Positioning Oneself. Adv Anat Embryol Cell
Biol. 2017;223:95-117. doi: 10.1007/978-3-319-53168-7_5. PMID: 28528441.
ix
Kerr JR. Epstein-Barr virus (EBV) reactivation and therapeutic inhibitors. J Clin Pathol. 2019
Oct;72(10):651-658. doi: 10.1136/jclinpath-2019-205822. Epub 2019 Jul 17. PMID: 31315893.
x
Di Lorenzo, Antonino et al. “Toll-Like Receptor 2 at the Crossroad between Cancer Cells, the Immune
System, and the Microbiota.” International journal of molecular sciences vol. 21,24 9418. 10 Dec. 2020,
doi:10.3390/ijms21249418
xi
Mottahedin A, Svedin P, Nair S, Mohn CJ, Wang X, Hagberg H, Ek J, Mallard C. Systemic activation
of Toll-like receptor 2 suppresses mitochondrial respiration and exacerbates hypoxic-ischemic injury in
the developing brain. J Cereb Blood Flow Metab. 2017 Apr;37(4):1192-1198. doi:
10.1177/0271678X17691292. Epub 2017 Jan 1. PMID: 28139935; PMCID: PMC5453473.
xii
Traub, E. Observations on immunological tolerance and "Immunity" in mice infected congenitally with
the virus of lymphocytic choriomeningitis (LCM). Archiv Fur Die Gesamte Virusforschung, 10(3), 303-
314. doi:10.1007/bf01250677. (1960).
xiii
Traub, E. & K. Beller. Untersuchungen uber die ansteckende Blutarmut der Pferde.
Immunitatsversuche. [Immunization experiments in equine infectious anaemia]. Monatshefte fur
Praktische Tierheilkunde; 3:193-206. (1942, 1951). [translated to English by A. Finnegan 2019]
xiv
One excellent example of this is the research put together by Miss Kathleen Dickson, through her
organization, Truthcures, and especially the YouTube video, Lyme Cryme, seen here:
https://www.youtube.com/watch?v=f8DU1Z6R-ms&t=692s
xv
Traub, E. A Filterable Virus Recovered from White Mice. Science, 81. (2099), 298-299.
doi:10.1126/science.81.2099.298. (1935).
xvi
Shope, R. E. (1935). Experiments on The Epidemiology Of Pseudorabies: I. Mode Of Transmission Of
The Disease In Swine And Their Possible Role In Its Spread To Cattle. Journal of Experimental
Medicine, 62(1), 85-99. doi:10.1084/jem.62.1.85
xvii
doi:10.1126/science.81.2099.298. (1935).
xviii
Corner, G. W. (1965). A history of the Rockefeller Institute: 1901-1953: Origins and growth. New
York: The Rockefeller Institute Press., pp. 408-409
xix
Traub, E. Persistence of Lymphocytic Choriomeningitis Virus in Immune Animals and Its Relation To
Immunity. Journal of Experimental Medicine, 63(6), 847-861. doi:10.1084/jem.63.6.847. (1936).
xx
Traub, E. Epidemiology of Lymphocytic Choriomeningitis In A Mouse Stock Observed for Four Years.
Journal of Experimental Medicine, 69(6), 801-817. doi:10.1084/jem.69.6.801. (1939).
xxi
Traub, E. Ueber den Einfluß der latenten Choriomeningitis-Infektion auf die Entstehung der
Lymphomatose bei weißen Mause [On the Influence of Latent Choriomeningitis Infection on the
Development of Lymphomatosis in White Mice]. Zentrl. Bakt. I. Orig. 147 (16). 1-25. (1941). [Translated
to English by A. Finnegan, 2019]
xxii
Traub, E. Observations on “Late onset disease” and Tumor Incidence in Different Strains of
Laboratory Mice infected Congenitally with LCM Virus I. Experiments with Random-bred NMRI Mice.
Zentralblatt Für Veterinärmedizin Reihe B, 22(9), 764-782. (1975, 2010). doi:10.1111/j.1439-
0450.1975.tb00643.x
xxiii
Traub, E. LCM Virus Research, Retrospect and Prospects. Lymphocytic Choriomeningitis Virus and
Other Arenaviruses, 3-10. doi:10.1007/978-3-642-65681-1_1. (1973)
xxiv
Hotchin, John. 1971. Monographs in Virology: Persistent and Slow Virus Infections. Karger.
xxv
Traub, E. Observations on immunological tolerance and "Immunity" in mice infected congenitally
with the virus of lymphocytic choriomeningitis (LCM). Archiv Fur Die Gesamte Virusforschung, 10(3),
303-314. doi:10.1007/bf01250677. (1960).
xxvi
Development of Lymphomatosis in White Mice]. Zentrl. Bakt. I. Orig. 147 (16). 1-25. (1941). [Translated
to English by A. Finnegan, 2019]
xxvii
Traub, E. Can LCM virus cause lymphomatosis in mice? Archiv Fur Die Gesamte Virusforschung,
11(5), 667-682. doi:10.1007/bf01243307. (1962).
xxviii
Traub, E. Choriomenigitis der Mäuse. Handbuch der Viruskrankheiten, (E. Gildenmeister, E. Haagen
& O. Waldmann, eds). Fischer-Verlag, Jena, Vol. 2, Ch. 7. 355-364. (1939). [Translated to English by A.
Finnegan (2019)]
xxix
303-314. doi:10.1007/bf01250677. (1960).
xxx
Lyme disease being a good example, see: Dickson, K. “CRIMINAL CHARGE SHEETS JUNE 2017
Lobbying for a Hearing for Referral to the USDOJ for a Prosecution of the Lyme Disease Crimes.” 2017.
Published at: https://docs.wixstatic.com/ugd/47b066_01d68b1309ae457b81df1e06e6beae1e.pdf
xxxi
This is observable in the relation to chronic neurologic outcomes of vaccine adverse events: Marks,
Donald H. ‘Neurological Complications of Vaccination with Outer Surface Protein A (OspA)’. 1 Jan.
2011 : 89 – 96.
xxxii
Sejvar, James. "Vaccines and Neurologic Disease." Seminars in Neurology 31, no. 03 (2011): 338-55.
doi:10.1055/s-0031-1287655.
xxxiii
Traub, E. Persistence of Lymphocytic Choriomeningitis Virus in Immune Animals and Its Relation
To Immunity. Journal of Experimental Medicine, 63(6), 847-861. doi:10.1084/jem.63.6.847. (1936).
xxxiv
Cherkasova, Elena A., Ekaterina A. Korotkova, Maria L. Yakovenko, Olga E. Ivanova, Tatyana P.
Eremeeva, Konstantin M. Chumakov, and Vadim I. Agol. 2002. “Long-Term Circulation of Vaccine-
Derived Poliovirus That Causes Paralytic Disease.” Journal of Virology 76 (13): 6791–99.
https://doi.org/10.1128/jvi.76.13.6791-6799.2002.
xxxv
Cassisi, G, P Sarzi-Puttini, and M Cazzola. 2011. “Chronic Widespread Pain and Fibromyalgia: Could
There Be Some Relationships with Infections and Vaccinations?” Clinical and Experimental
Rheumatology. U.S. National Library of Medicine. 2011.
https://www.ncbi.nlm.nih.gov/pubmed/22243559.
xxxvi
Cherkasova, E. A., et al. “Spread of Vaccine-Derived Poliovirus from a Paralytic Case in an
Immunodeficient Child: an Insight into the Natural Evolution of Oral Polio Vaccine.” Journal of
Virology, vol. 79, no. 2, 2004, pp. 1062–1070., doi:10.1128/jvi.79.2.1062-1070.2005.
xxxvii
Croft, P. B. (1969). Para-infectious and post-vaccinal encephalomyelitis. Postgraduate Medical
Journal, 45(524), 392–400. doi:10.1136/pgmj.45.524.392
xxxviii
Schmidt, Reinhold E, et al. “Autoimmunity and Primary Immunodeficiency: Two Sides of the Same
Coin?” Nature Reviews. Rheumatology, U.S. National Library of Medicine, 19 Dec. 2017,
https://www.ncbi.nlm.nih.gov/pubmed/29255211
xxxix
Abu-Akkada SS, El Kerdany ED, Mady RF, Diab RG, Khedr GA, Ashmawy KI, Lotfy WM.
Encephalitozoon cuniculi infection among immunocompromised and immunocompetent humans in
Egypt. Iran J Parasitol. 2015 Oct-Dec;10(4):561-70. PMID: 26811722; PMCID: PMC4724832.
xl
Duintjer Tebbens RJ, Thompson KM. Comprehensive screening for immunodeficiency-associated
vaccine-derived poliovirus: an essential oral poliovirus vaccine cessation risk management strategy.
Epidemiol Infect. 2017 Jan;145(2):217-226. doi: 10.1017/S0950268816002302. Epub 2016 Oct 20.
PMID: 27760579; PMCID: PMC5197684
xli
see: Dickson, K. “CRIMINAL CHARGE SHEETS JUNE 2017 Lobbying for a Hearing for Referral to
the USDOJ for a Prosecution of the Lyme Disease Crimes.” 2017. Published at:
xlii
Shah, Keerti, and Neal Nathanson. “Human Exposure To Sv40: Review And Comment.” American
Journal of Epidemiology, vol. 103, no. 1, 1976, pp. 1–12., doi:10.1093/oxfordjournals.aje.a112197
Retrieved from https://academic.oup.com/aje/article-abstract/103/1/1/151919?redirectedFrom=fulltext,
or: https://www.scribd.com/document/409584571/Human-Exposure-to-SV40-Review-and-Comment-
Carcinogens-in-Vaccines
xliii
Baldwin, K. J., & Cummings, C. L. (2018). Herpesvirus Infections of the Nervous System.
CONTINUUM: Lifelong Learning in Neurology, 24, 1349–1369. doi:10.1212/con.0000000000000661
xliv
This is essentially the same mechanism in the human with the herpesvirus class, as the LCM virus in
mice.
xlv
xlvi
Drăgănescu N, Gheorghiu V, Mihalache G. Encefalita postvaccinală la un sugar cu infecţie herpetică
latentă [Post-vaccinal encephalitis in an infant with latent herpes infection]. Stud Cercet Inframicrobiol.
1970;21(5):385-9. Romanian. PMID: 4395307.
xlvii
doi:10.1126/science.81.2099.298. (1935).
xlviii
Cassisi, G, P Sarzi-Puttini, and M Cazzola. 2011. “Chronic Widespread Pain and Fibromyalgia:
Could There Be Some Relationships with Infections and Vaccinations?” Clinical and Experimental
xlix
A case in point: Valsamakis, A, P G Auwaerter, B K Rima, H Kaneshima, and D E Griffin. 1999.
“Altered Virulence of Vaccine Strains of Measles Virus after Prolonged Replication in Human Tissue.”
Journal of Virology. American Society for Microbiology. October 1999.
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC112900/
l
Croft, P. B. (1969). Para-infectious and post-vaccinal encephalomyelitis. Postgraduate Medical Journal,
45(524), 392–400. doi:10.1136/pgmj.45.524.392
li
“The Growing Crisis of Chronic Disease in the United States.” The Relief Work, 13 Jan. 2016,
https://thereliefwork.com/2016/01/the-growing-crisis-of-chronic-disease-in-the-united-states/
lii
Hunter, Philip. “Is Political Correctness Damaging Science? Peer Pressure and Mainstream Thinking
May Discourage Novelty and Innovation.” EMBO reports. U.S. National Library of Medicine, May 2005.
liii
Case in point: Valsamakis, A, P G Auwaerter, B K Rima, H Kaneshima, and D E Griffin. 1999.
liv
Torisu, Hiroyuki, and Kenji Okada. “Vaccination-Associated Acute Disseminated Encephalomyelitis.”
Vaccine, U.S. National Library of Medicine, 14 Feb. 2019, www.ncbi.nlm.nih.gov/pubmed/30683508
lv
Drăgănescu, N, et al. “Post-Vaccinal Encephalitis in an Infant with Latent Herpes Infection.” Studii Si
Cercetari De Inframicrobiologie, U.S. National Library of Medicine, 1970,
www.ncbi.nlm.nih.gov/pubmed/4395307
lvi
Traub, E. Epidemiology of Lymphocytic Choriomeningitis In A Mouse Stock Observed for Four Years.
Journal of Experimental Medicine, 69(6), 801-817. doi:10.1084/jem.69.6.801. (1939).
lvii
lviii
Walton, Alice G. “Why More Americans Suffer From Mental Disorders Than Anyone Else.” The
Atlantic, Atlantic Media Company, 3 Oct. 2011, www.theatlantic.com/health/archive/2011/10/why-more-
americans-suffer-from-mental-disorders-than-anyone-else/246035/
lix
“Vaccination and Immunization Statistics.” UNICEF DATA, data.unicef.org/topic/child-
health/immunization/.
lx
Londoño, Diana, and Diego Cadavid. “Bacterial lipoproteins can disseminate from the periphery to
inflame the brain.” The American journal of pathology vol. 176,6 (2010): 2848-57.
doi:10.2353/ajpath.2010.091235
lxi
Marks, D. H., “Neurological Complications of Vaccination with Outer Surface Protein A (OspA).”
International Journal of Risk & Safety in Medicine. IOS Press. January 1, 2011.
https://content.iospress.com/articles/international-journal-of-risk-and-safety-in-medicine/jrs527
lxii
doi:10.1055/s-0031-1287655.
lxiii
lxiv
Krueger, G. R. F., & Ramon, A. (1988). Overview of immunopathology of chronic active herpesvirus
infection. Journal of Virological Methods, 21(1-4), 11–18. doi:10.1016/0166-0934(88)90048-1
lxv
“Birth-18 Years Immunization Schedule | CDC.” Centers for Disease Control and Prevention, Centers
for Disease Control and Prevention, https://www.cdc.gov/vaccines/schedules/hcp/imz/child-
adolescent.html
lxvi
lxvii
lxviii
A case in point: Helaly, G. F., Elsheredy, A. G., El Basset Mousa, A. A., Ahmed, H. K. F., &
Oluyemi, A. E.-G. S. (2016). Seronegative and occult hepatitis C virus infections in patients with
hematological disorders. Archives of Virology, 162(1), 63–69. doi:10.1007/s00705-016-3049-7
lxix
currently, children are expected to have around 15 vaccines, with up to 4 followup shots by the time
they are 15 months old, which is extremely burdensome, when children that young don’t have fully
developed immune systems yet, and many of them are already carrying a burden from RNA viruses
transmitted to them congenitally by the mother. These same recipients are expected to have another 15
vaccines with several folowups, spanning to the time they reach adulthood. They are also recommended
to have seasonal flu shots. If even one of these vaccines contain either Mycoplasma or viral contaminants,
the damage could be extensive, permanent, and even deadly. For list and schedule, refer to: “Birth-18
Years Immunization Schedule | CDC.” n.d. Centers for Disease Control and Prevention. Centers for
Disease Control and Prevention. Accessed July 28, 2019.
https://www.cdc.gov/vaccines/schedules/hcp/imz/child-adolescent.html
lxx
Croft, P. B. “Para-Infectious and Post-Vaccinal Encephalomyelitis.” Postgraduate Medical Journal,
vol. 45, no. 524, Jan. 1969, pp. 392–400., doi:10.1136/pgmj.45.524.392.,
lxxi
As discussed in: Office of Transnational Issues (OTI),”The Darker Bioweapons Future,” (Unclassified
Intelligence Report). Directorate of Intelligence, Central Intelligence Agency (CIA). 3 December 2003.
Retrieved from: https://www.cia.gov/library/readingroom/docs/DOC_0001298811.pdf
lxxii
lxxiii
Siddique, Haroon. 2018. “Mental Health Disorders on Rise among Children.” The Guardian.
Guardian News and Media. November 22, 2018.
https://www.theguardian.com/society/2018/nov/22/mental-health-disorders-on-rise-among-children-nhs-
figures
lxxiv
Valsamakis, A, P G Auwaerter, B K Rima, H Kaneshima, and D E Griffin. 1999. “Altered Virulence
of Vaccine Strains of Measles Virus after Prolonged Replication in Human Tissue.” Journal of Virology.
American Society for Microbiology. October 1999.
lxxv
Traub, E. Uber Immunität und aktive Immunisierung gegen Viruskrankheiten [About Immunity and
Active Immunization Against Viral Diseases.] Zeit. Infek. Krank. Hyg. Haus. Vol. 45 (1). 1-36. (1939).
[Translated to English by A. Finnegan 2019]
lxxvi
lxxvii
Sheng-Fowler, L., Lewis, A. M., & Peden, K. (2009). Issues associated with residual cell-substrate
DNA in viral vaccines. Biologicals, 37(3), 190–195. doi:10.1016/j.biologicals.2009.02.015
lxxviii
Croft, P. B. (1969). Para-infectious and post-vaccinal encephalomyelitis. Postgraduate Medical
Journal, 45(524), 392–400. doi:10.1136/pgmj.45.524.392
lxxix
A case in point: Valsamakis, A, P G Auwaerter, B K Rima, H Kaneshima, and D E Griffin. 1999.
lxxx
Altamirano, Jonathan et al. “OPV Vaccination and Shedding Patterns in Mexican and US Children.”
Clinical infectious diseases : an official publication of the Infectious Diseases Society of America vol.
67,suppl_1 (2018): S85-S89. doi:10.1093/cid/ciy636
lxxxi
lxxxii
“PREVENTING ANOTHER SV40 TRAGEDY: ARE TODAY'S VACCINE SAFETY
PROTOCOLS EFFECTIVE? : Committee on Government Reform.” 2003. Internet Archive. Government
Publishing Office. November 13, 2003. https://archive.org/details/gov.gpo.fdsys.CHRG-
108hhrg92772?q=SV40
lxxxiii
lxxxiv
Ibid.
lxxxv
Cutrone, Rochelle, et al. “Some Oral Poliovirus Vaccines Were Contaminated with Infectious SV40
after 1961.” Cancer Research, American Association for Cancer Research, 15 Nov. 2005,
cancerres.aacrjournals.org/content/65/22/10273.article-info
lxxxvi
Victoria, Joseph G et al. “Viral nucleic acids in live-attenuated vaccines: detection of minority
variants and an adventitious virus.” Journal of virology vol. 84,12 (2010): 6033-40.
doi:10.1128/JVI.02690-09
lxxxvii
Tsang, S X et al. “Evidence of avian leukosis virus subgroup E and endogenous avian virus in
measles and mumps vaccines derived from chicken cells: investigation of transmission to vaccine
recipients.” Journal of virology vol. 73,7 (1999): 5843-51.
lxxxviii
Victoria, Joseph G et al. “Viral nucleic acids in live-attenuated vaccines: detection of minority
variants and an adventitious virus.” Journal of virology vol. 84,12 (2010): 6033-40.
doi:10.1128/JVI.02690-09
lxxxix
xc
Cowan, T. S., & Fallon, S. (2020). The contagion myth: Why viruses (including "coronavirus") are not
the cause of disease. New York, NY: Skyhorse Publishing.
xci
“The Potential Environmental Consequences of Genetic Engineering : Hearings before the
Subcommittee on Toxic Substances and Environmental Oversight of the Committee on Environment and
Public Works, United States Senate, Ninety-Eighth Congress, Second Session, September 25 and 27,
1984 : United States. Congress. Senate. Committee on Environment and Public Works. Subcommittee on
Toxic Substances and Environmental Oversight” Internet Archive, Washington : U.S. G.P.O., 1 Jan.
1984, www.archive.org/details/potentialenviron00unit/page/n1
xcii
See: Dickson, K. & Beaux Reliosis. Truthcures Video: Lyme Cryme YouTube video:
https://www.youtube.com/watch?v=f8DU1Z6R-ms&t=692s
xciii
Waterson, A.P., Rott, R. The viromicrosomes of fowl plague virus. Archiv f Virusforschung 13, 577–
581 (1963). https://doi.org/10.1007/BF01267799
xciv
Schäfer, W. (1963). Structure of Some Animal Viruses and Significance of their Components.
Bacteriological Review, 27,
xcv
Schäfer, Werner. ”Some Aspects of Animal Virus Multiplication.” Perspectives in Virology ; a
Symposium. (Ed. Morris Pollard). John Wiley & Sons, Inc., New York. Chapman & Hlll, Ltd., London.
xcvi
Kohn, A, and P Fuchs. “Cell fusion by various strains of Newcastle disease virus and their virulence.”
Journal of virology vol. 3,5 (1969): 539-40. doi:10.1128/JVI.3.5.539-540.1969
xcvii
xcviii
Cassisi, G, P Sarzi-Puttini, and M Cazzola. 2011. “Chronic Widespread Pain and Fibromyalgia:
Could There Be Some Relationships with Infections and Vaccinations?” Clinical and Experimental
xcix
doi:10.1055/s-0031-1287655.
c
Croft, P. B. (1969). Para-infectious and post-vaccinal encephalomyelitis. Postgraduate Medical Journal,
45(524), 392–400. doi:10.1136/pgmj.45.524.392
ci
Cowan, T. S., & Fallon, S. (2020). The contagion myth: Why viruses (including "coronavirus") are not
the cause of disease. New York, NY: Skyhorse Publishing.
cii
Morens, David M et al. “Predominant role of bacterial pneumonia as a cause of death in pandemic
influenza: implications for pandemic influenza preparedness.” The Journal of infectious diseases vol.
198,7 (2008): 962-70. doi:10.1086/591708
ciii
He, Cheng-Qiang et al. “The matrix segment of the "Spanish flu" virus originated from intragenic
recombination between avian and human influenza A viruses.” Transboundary and emerging diseases
vol. 66,5 (2019): 2188-2195. doi:10.1111/tbed.13282
civ
Nelson, J B. “INFECTIOUS CATARRH OF MICE : I. A NATURAL OUTBREAK OF THE
DISEASE.” The Journal of experimental medicine vol. 65,6 (1937): 833-41. doi:10.1084/jem.65.6.833
cv
Traub, E. & K. Beller. Stand und Aussichten der Erforschung des ansteckenden Katarrhs der Luftwege
beim Pferd. [The Position and Outlook in Research on Infectious Respiratory Catarrh of the Horse]. Z.
Veterinark, 53: 88-97. (1941). [Translated to English by A. Finnegan, 2019]
cvi
McCullers, Jonathan A. “Insights into the interaction between influenza virus and pneumococcus.”
Clinical microbiology reviews vol. 19,3 (2006): 571-82. doi:10.1128/CMR.00058-05
cvii
Aho, K, and R Pyhälä. “Agglutinins against human erythrocytes modified by Newcastle disease virus
in Mycoplasma pneumoniae infections.” Clinical and experimental immunology vol. 17,4 (1974): 607-16.
cviii
cix
see LYMErix Vaccine Victim Testimony to the FDA 2001:
https://www.scribd.com/document/485041723/LYMErix-Vaccine-Victim-Testimony-to-the-FDA-2001
cx
doi:10.1055/s-0031-1287655.
cxi
cxii
Golightly, M et al. “Modulation of natural killer cell activity by Borrelia burgdorferi.” Annals of the
New York Academy of Sciences vol. 539 (1988): 103-11. doi:10.1111/j.1749-6632.1988.tb31843.x
cxiii
Stevenson, M, et al. “Immortalization of Human T Lymphocytes after Transfection of Epstein-Barr
Virus DNA.” Science, vol. 233, no. 4767, 1986, pp. 980–984., doi:10.1126/science.3016899
cxiv
Elmore, Susan. “Apoptosis: a Review of Programmed Cell Death.” Toxicologic Pathology, U.S.
National Library of Medicine, June 2007, https://www.ncbi.nlm.nih.gov/pubmed/17562483.
cxv
Gerlic, Motti et al. “The inhibitory effect of Mycoplasma fermentans on tumour necrosis factor (TNF)-
alpha-induced apoptosis resides in the membrane lipoproteins.” Cellular microbiology vol. 9,1 (2007):
142-53. doi:10.1111/j.1462-5822.2006.00774.x
cxvi
Busca A, Saxena M, Kryworuchko M, Kumar A. Anti-apoptotic genes in the survival of monocytic
cells during infection. Curr Genomics. 2009 Aug;10(5):306-17. doi: 10.2174/138920209788920967.
PMID: 20119528; PMCID: PMC2729995.
cxvii
cxviii
Nicolson GL, Gan R, Haier J. Multiple co-infections (Mycoplasma, Chlamydia, human herpesvirus-
6) in blood of chronic fatigue syndrome patients: association with signs and symptoms. APMIS 2003;
111:557-66
cxix
Abu-Akkada SS, El Kerdany ED, Mady RF, Diab RG, Khedr GA, Ashmawy KI, Lotfy WM.
Encephalitozoon cuniculi infection among immunocompromised and immunocompetent humans in
Egypt. Iran J Parasitol. 2015 Oct-Dec;10(4):561-70. PMID: 26811722; PMCID: PMC4724832.
cxx
Bechter, K. (2013). Virus Infection as a Cause of Inflammation in Psychiatric Disorders. Modern
Trends in Pharmacopsychiatry, 49–60. doi:10.1159/000343967
cxxi
doi:10.2353/ajpath.2010.091235
cxxii
Li, Jing Jing et al. “In vivo evidence for the contribution of peripheral circulating inflammatory
exosomes to neuroinflammation.” Journal of neuroinflammation vol. 15,1 8. 8 Jan. 2018,
doi:10.1186/s12974-017-1038-8
cxxiii
Rubin, Steven A et al. “Changes in mumps virus gene sequence associated with variability in
neurovirulent phenotype.” Journal of virology vol. 77,21 (2003): 11616-24. doi:10.1128/jvi.77.21.11616-
11624.2003
cxxiv
doi:10.2353/ajpath.2010.091235
cxxv
Petricciani, J., Sheets, R., Griffiths, E., & Knezevic, I. (2014). Adventitious agents in viral vaccines:
Lessons learned from 4 case studies. Biologicals, 42(5), 223–236. doi:10.1016/j.biologicals.2014.07.003
cxxvi
Sejvar, J. (2011). Vaccines and Neurologic Disease. Seminars in Neurology, 31(03), 338–355.
doi:10.1055/s-0031-1287655
cxxvii
RIVERS, T. M. (1946). VIRUS DISEASES OF THE NERVOUS SYSTEM. Journal of the American
Medical Association, 132(8), 427. doi:10.1001/jama.1946.02870430007003
cxxviii
Croft, P. B. “Para-Infectious and Post-Vaccinal Encephalomyelitis.” Postgraduate Medical Journal,
vol. 45, no. 524, Jan. 1969, pp. 392–400., doi:10.1136/pgmj.45.524.392.,
cxxix
Traub, E. Serological Evidence for Antigenic Variation in Brains of Mice Infected Persistently with
the Virus of Lymphocytic Choriomeningitis. Zentralblatt Für Veterinärmedizin Reihe B, 27(9-10), 806-
822. (1980, 2010). doi:10.1111/j.1439-0450.1980.tb02035.x
cxxx
cxxxi
figures
cxxxii
Rubin, Steven A et al. “Changes in mumps virus gene sequence associated with variability in
neurovirulent phenotype.” Journal of virology vol. 77,21 (2003): 11616-24. doi:10.1128/jvi.77.21.11616-
11624.2003
cxxxiii
cxxxiv
Bechter, K., Herzog, S., Schüttler, R., & Rott, R. (1989). MRI in psychiatric patients with serum
antibodies against Borna disease virus. Psychiatry Research, 29(3), 281–282. doi:10.1016/0165-
1781(89)90062-0
cxxxv
PROCEEDINGS of COLLABORATORS MEETING on FOREIGN POULTRY DISEASES. USDA,
1953., Retrieved from: https://archive.org/details/CAT10678555/page/n5
cxxxvi
Office of Transnational Issues (OTI),”The Darker Bioweapons Future,” (Unclassified Intelligence
Report). Directorate of Intelligence, Central Intelligence Agency (CIA). 3 December 2003. Retrieved
from: https://www.cia.gov/library/readingroom/docs/DOC_0001298811.pdf
cxxxvii
reflecting that of LCM in mice, see: Traub, E. Epidemiology of Lymphocytic Choriomeningitis In A
Mouse Stock Observed for Four Years. Journal of Experimental Medicine, 69(6), 801-817.
doi:10.1084/jem.69.6.801. (1939).
cxxxviii
Office of Transnational Issues (OTI),”The Darker Bioweapons Future,” (Unclassified Intelligence
Report). Directorate of Intelligence, Central Intelligence Agency (CIA). 3 December 2003. Retrieved
from: https://www.cia.gov/library/readingroom/docs/DOC_0001298811.pdf
cxxxix
cxl
Development of Lymphomatosis in White Mice]. Zentrl. Bakt. I. Orig. 147 (16). 1-25. (1941).
cxli
Traub, E. Serological Evidence for Antigenic Variation in Brains of Mice Infected Persistently with
the Virus of Lymphocytic Choriomeningitis. Zentralblatt Für Veterinärmedizin Reihe B, 27(9-10), 806-
822. (1980, 2010). doi:10.1111/j.1439-0450.1980.tb02035.x
cxlii
303-314. doi:10.1007/bf01250677. (1960).
cxliii
cxliv
MULHOLLAND, J H et al. “QUANTITATIVE STUDIES OF FEBRILE TOLERANCE AND
LEVELS OF SPECIFIC ANTIBODY EVOKED BY BACTERIAL ENDOTOXIN.” The Journal of
clinical investigation vol. 44,6 (1965): 920-8. doi:10.1172/JCI105209
cxlv
Berger, F. M., & Fukui, G. M. (1963). Endotoxin Induced Resistance to Infections and Tolerance.
Experimental Biology and Medicine, 114(3), 780–783. doi:10.3181/00379727-114-28796
cxlvi
Silverstein, Arthur M. “The Curious Case of the 1960 Nobel Prize to Burnet and Medawar.”
Immunology. John Wiley and Sons Inc., March 2016. https://www.ncbi.nlm.nih.gov/pubmed/26790994
cxlvii
Arens, C et al. “Sepsis-induced long-term immune paralysis--results of a descriptive, explorative
study.” Critical care (London, England) vol. 20 93. 29 Feb. 2016, doi:10.1186/s13054-016-1233-5
cxlviii
Dinter, Z. Persönaliches, Begegnungen mit Erich Traub.[Personal Encounters with Erich Traub]
Berl. Munch. Tier. Woch. 96. 70-72. (1984)
cxlix
Traub, E. . Observations on “Late onset disease” and Tumor Incidence in Different Strains of
Laboratory Mice infected Congenitally with LCM Virus: II. Experiments with Inbred CBA/J mice.
Zentralblatt Für Veterinärmedizin Reihe B, 22(9), 783-792. (1975, 2010) doi:10.1111/j.1439-
0450.1975.tb00644.x.
cl
Traub, E. Observations on “Late onset disease” and Tumor Incidence in Different Strains of Laboratory
Mice infected Congenitally with LCM Virus I. Experiments with Random-bred NMRI Mice. Zentralblatt
Für Veterinärmedizin Reihe B, 22(9), 764-782. (1975, 2010). doi:10.1111/j.1439-0450.1975.tb00643.x
cli
“Birth-18 Years Immunization Schedule | CDC.” n.d. Centers for Disease Control and Prevention.
Centers for Disease Control and Prevention. Accessed July 28, 2019.
https://www.cdc.gov/vaccines/schedules/hcp/imz/child-adolescent.html
clii
Tavel, Morton E. “Somatic Symptom Disorders without Known Physical Causes: One Disease with
Many Names?” The American journal of medicine. U.S. National Library of Medicine, October 2015.
cliii
This would be based on the assessment of genetic variability and predispositions or susceptibilities of
each, the variability of pathogens accumulated, as well as secondary opportunistics, and the combinations
therein are virtually endless.
cliv
Brody, J. A., W. J. Hadlow, J. Hotchin, R. T. Johnson, H. Koprowski, and L. T. Kurland. “Soviet
Search for Viruses That Cause Chronic Neurologic Diseases in the U.S.S.R.” Science 147, no. 3662 (May
1965): 1114–16. https://doi.org/10.1126/science.147.3662.1114
clv
clvi
Siddique, Haroon. 2018. “Mental Health Disorders on Rise among Children.” The Guardian. Guardian
News and Media. November 22, 2018. https://www.theguardian.com/society/2018/nov/22/mental-health-
disorders-on-rise-among-children-nhs-figures
clvii
as seen in Traub’s studies of mouse colonies: Traub, E. Epidemiology of Lymphocytic
Choriomeningitis In A Mouse Stock Observed for Four Years. Journal of Experimental Medicine, 69(6),
801-817. doi:10.1084/jem.69.6.801. (1939).
clviii
figures
clix
clx
Tim, Kae. “Goldman Sachs Asks in Biotech Research Report: 'Is Curing Patients a Sustainable
Business Model?'.” CNBC, CNBC, 11 Apr. 2018, www.cnbc.com/2018/04/11/goldman-asks-is-curing-
patients-a-sustainable-business-model.html
clxi
Persing, David H. INFECTION, CANCER, AND THE IMMUNE RESPONSE. The Infectious
Etiology of Chronic Diseases Defining the Relationship, Enhancing the Research, and Mitigating the
Effects; Workshop Summary. National Acad. Press, 2004., pp. 154-173
clxii
Dickson, K. “CRIMINAL CHARGE SHEETS JUNE 2017 Lobbying for a Hearing for Referral to the
USDOJ for a Prosecution of the Lyme Disease Crimes.” 2017. Retrieved from:
https://docs.wixstatic.com/ugd/47b066_f60123f7e0554461a272fc470a98a598.pdf
clxiii
“42 U.S. Code § 247d–7e - Biomedical Advanced Research and Development Authority.” Legal
Information Institute, Legal Information Institute, www.law.cornell.edu/uscode/text/42/247d-7e
clxiv
i.e only focusing on the acute cases while denying the chronic outcomes and after-effects of the
disabled immune system, or masking the problems with semantics. The vaccines were only working to
induce tolerance, which only quieted the acute manifestations, and suppress clinical symptoms. Yet, the
over-exhausted individual would experience a long-term chronic disease that was demoralizing and
incapacitating.
clxv
Proceedings of the Second National Conference on Serologic Diagnosis of Lyme Disease: October 27-
29, 1994, Dearborn, Michigan. Washington, D.C.: Association, 1994.
clxvi
USDOJ for a Prosecution of the Lyme Disease Crimes.” 2017. Published at:
clxvii
Aronowitz, R A. “Lyme Disease: the Social Construction of a New Disease and Its Social
Consequences.” The Milbank quarterly. U.S. National Library of Medicine, 1991.
clxviii
clxix
Dickson, Kathleen. Testimony. VRBPAC (Vaccines and Related Biological Products Advisory
Committee) of the Food and Drug Administration. 2001. Minutes, January 31. Retrieved from:
http://www.actionlyme.org/2001_FDA_LYMErix_Transcripts.pdf
clxx
Noble, Holcomb. “Concerns Grow Over Reactions to Lyme Shots.” The New York Times, The New
York Times, 21 Nov. 2000, www.nytimes.com/2000/11/21/science/concerns-grow-over-reactions-to-
lyme-shots.html
clxxi
This can be realized from the testimony given to the FDA by those with adverse events, including in
their testimony that doctors were telling the patients it wasn’t the vaccine causing the sudden onset of
health problems, and even went as far as to persuade them to continue taking the subsequent shots, which
made them much sicker and injuries more permanent. See FDA testimony on LYMErix vaccine victim
testimony
clxxii
FDA Vaccine Advisory Committee on LYMErix Vaccine. “LYMErix Vaccine Victim’s Stories and
Related Articles.” 11AD, pp. 1–20. [Misc. written testimony] Retrieve from:
https://web.archive.org/web/20030830064730/https://www.fda.gov/ohrms/dockets/ac/01/briefing/3680b2
_17.pdf
clxxiii
The Native Antigen Company. (n.d.). Novel Coronavirus Antigens Now Available. Retrieved
January 09, 2021, from https://thenativeantigencompany.com/novel-coronavirus-antigens-now-available/
clxxiv
Cajamarca-Baron, J., Guavita-Navarro, D., Buitrago-Bohorquez, J., Gallego-Cardona, L., Navas, A.,
Cubides, H., . . . Rojas-Villarraga, A. (n.d.). SARS-CoV-2 (COVID-19) in patients with some degree of
immunosuppression. Retrieved January 09, 2021, from https://www.reumatologiaclinica.org/en-sars-cov-
2-covid-19-in-patients-with-avance-S2173574320301295?newsletter=true&coronavirus
clxxv
Marshall, Michael. The Lasting Misery of Coronavirus Long-Haulers. 14 Sept. 2020,
www.nature.com/articles/d41586-020-02598-6.
Barber, Carolyn. “The Problem of 'Long Haul' COVID.” Scientific American, Scientific American,
clxxvi
29 Dec. 2020, www.scientificamerican.com/article/the-problem-of-long-haul-covid/.
clxxvii
Pastuszak, Żanna et al. “Post-polio syndrome. Cases report and review of literature.” Neurologia i
neurochirurgia polska vol. 51,2 (2017): 140-145. doi:10.1016/j.pjnns.2017.01.009
clxxviii
Burki, Talha Khan. “Post-Ebola syndrome.” The Lancet. Infectious diseases vol. 16,7 (2016): 780-
781. doi:10.1016/S1473-3099(15)00259-5
clxxix
Rebman, Alison W, and John N Aucott. “Post-treatment Lyme Disease as a Model for Persistent
Symptoms in Lyme Disease.” Frontiers in medicine vol. 7 57. 25 Feb. 2020,
doi:10.3389/fmed.2020.00057
clxxx
USDOJ for a Prosecution of the Lyme Disease Crimes.” 2017. Published at:
clxxxi
From https://30g7el1b4b1n28kgpr414nuu-wpengine.netdna-ssl.com/wp-
content/uploads/2020/12/Pfizer-BioNTech-COVID-19-Vaccine-EUA-Fact-Sheet-for-HCP_0.pdf
The Pfizer-BioNTech COVID-19 Vaccine is supplied as a frozen suspension in multiple dose vials; each
vial must be diluted with 1.8 mL of sterile 0.9% Sodium Chloride Injection, USP prior to use to form the
vaccine. Each dose of the Pfizer-BioNTech COVID-19 Vaccine contains 30 mcg of a nucleoside-modified
messenger RNA (modRNA) encoding the viral spike (S) glycoprotein of SARS-CoV-2.
Each dose of the Pfizer-BioNTech COVID-19 Vaccine also includes the following ingredients: lipids
(0.43 mg (4-hydroxybutyl)a zanediyl)bis( hexane-6 ,1-diyl)bis(2-hexyldecanoate), 0.05 mg
2[(polyethylene glycol)-2000]-N,N-ditetradecylacetamide, 0.09 mg 1,2-distearoyl-sn-glycero-3 -
phosphocholine , and 0.2 mg cholesterol), 0.01 mg potassium chloride, 0.01 mg monobasic potassium
phosphate, 0.36 mg sodium chloride, 0.07 mg dibasic sodium phosphate dihydrate, and 6 mg sucrose. The
diluent (0.9% Sodium Chloride Injection, USP) contributes an additional 2.16 mg sodium chloride per
dose.
clxxxii
Saito, T et al. “Immune tolerance induced by polyethylene glycol-conjugate of protein antigen:
clonal deletion of antigen-specific Th-cells in the thymus.” Journal of biomaterials science. Polymer
edition vol. 11,6 (2000): 647-56. doi:10.1163/156856200743922
Hoang Thi, Thai Thanh et al. “The Importance of Poly(ethylene glycol) Alternatives for
clxxxiii
Overcoming PEG Immunogenicity in Drug Delivery and Bioconjugation.” Polymers vol. 12,2 298. 2 Feb.
2020, doi:10.3390/polym12020298
clxxxiv
“Proceedings : Collaborator's Meeting on Foreign Poultry Diseases (1953 : Washington).” Internet
Archive, United States Department of Agriculture (USDA) Washington, 1 Jan. 1970,
www.archive.org/details/CAT10678555/page/n15
clxxxv
USDA, “A Committee Report on Mycoplasma Gallisepticum Infection in Poultry; Commonly
Known as Chronic Respiratory Disease and Air Sac Disease in Chickens and as Infectious Sinusitis and
Airsacculitis in Turkeys : United States. Agricultural Research Service. Animal Disease and Parasite
Research Division : Free Download, Borrow, and Streaming.” Internet Archive. [Washington] :
Agricultural Research Service, U.S. Dept. of Agriculture, January 1, 1962.
https://www.archive.org/details/committeereporto81unit/page/n1
clxxxvi
Welch, Frank J., A New Image for Agriculture. Disease, Environmental, and Management Factors
Related to Poultry Health: a Symposium Held March 20- 22, 1961 at the Jefferson Auditorium, USDA,
Washington, D.C. : United States. Agricultural Research Service. Internet Archive, [Washington, D.C.] :
Agricultural Research Service, U.S. Dept. of Agriculture, 1 Jan. 1961, pp. 1-3, Retrieved from:
https://archive.org/details/CAT31325715/page/1
clxxxvii
Proceedings of the Second National Conference on Serologic Diagnosis of Lyme Disease: October
27-29, 1994, Dearborn, Michigan. Washington, D.C.: Association, 1994.
clxxxviii
clxxxix
303-314. doi:10.1007/bf01250677. (1960).
cxc
For example, chronic disease would have been taken seriously, and profiteering at our expense would
have been harder. As we stand right now, this is where we’re at, with Goldman-Sachs declaring there is
no sustainable money-making in cures: Kae, Tim. “Goldman Sachs Asks in Biotech Research Report: 'Is
Curing Patients a Sustainable Business Model?'.” CNBC, CNBC, 11 Apr. 2018,
www.cnbc.com/2018/04/11/goldman-asks-is-curing-patients-a-sustainable-business-model.html
cxci
cxcii
doi:10.1055/s-0031-1287655.
cxciii
Tavel, Morton E. “Somatic Symptom Disorders without Known Physical Causes: One Disease with
Many Names?” The American journal of medicine. U.S. National Library of Medicine, October 2015.
cxciv
cxcv
Barbour, A., & Fish, D. (1993). The biological and social phenomenon of Lyme disease. Science,
260(5114), 1610–1616. doi:10.1126/science. 8503006
cxcvi
Kae, Tim. “Goldman Sachs Asks in Biotech Research Report: 'Is Curing Patients a Sustainable
Business Model?'.” CNBC, CNBC, 11 Apr. 2018, www.cnbc.com/2018/04/11/goldman-asks-is-curing-
patients-a-sustainable-business-model.html
cxcvii
“42 U.S. Code § 247d–7e - Biomedical Advanced Research and Development Authority.” Legal
Information Institute. Legal Information Institute, n.d. https://www.law.cornell.edu/uscode/text/42/247d-
7e
cxcviii
“50 U.S. Code § 1520a - Restrictions on Use of Human Subjects for Testing of Chemical or
Biological Agents.” Legal Information Institute. Legal Information Institute, n.d.
https://www.law.cornell.edu/uscode/text/50/1520a
This is nullified by:
“50 U.S. Code Chapter 33 - WAR POWERS RESOLUTION.” Legal Information Institute. Legal
Information Institute, n.d. https://www.law.cornell.edu/uscode/text/50/chapter-33
“50 U.S. Code Chapter 34 - NATIONAL EMERGENCIES.” Legal Information Institute. Legal
Information Institute, n.d. https://www.law.cornell.edu/uscode/text/50/chapter-34
cxcix
Gorenstein, Dan. “BARDA: The Venture Capital Firm Buried in the U.S. Government.” Marketplace,
April 26, 2019. https://www.marketplace.org/2014/10/30/barda-venture-capital-firm-buried-us-
government/
cc
cci
Shorter, Edward. “An Alarming Increase in Mental Illness?” Psychology Today. Sussex Publishers,
n.d. https://www.psychologytoday.com/us/blog/how-everyone-became-depressed/201404/alarming-
increase-in-mental-illness, April 27, 2014
ccii
Schäfer, W. (1963). Structure of Some Animal Viruses and Significance of their Components.
Bacteriological Review, 27.

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[The Institute of Biological Ethics & Historical Research Initiative]

Immune Tolerance and Slow-Virus Disease: Skeletons in the

Traub’s Discovery of Immune Tolerance

There are serious issues of concern in attempting to mass immunize or compulsively-

a. Stealth Infections and Immune deficiencies.

Those already dealing with one or several stealth infections causing

b. Contaminants (adventitious or latent viruses, Mycoplasma,

Commercial vaccines with pathogenic contaminants present in the vaccine

II. Immune Tolerance, HLA Variation, Disease Transmission and Outcome

III. Viral Proliferation, Immune Tolerance, and Resulting Mental Illness in

IV. A Theoretical Picture of Systemic Immune Tolerance and Neurotropic

29 Dec. 2020, www.scientificamerican.com/article/the-problem-of-long-haul-covid/.

This is nullified by:

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