Visual Fields
Visual Fields
Slide
2
Slide
3 Visual Fields
Learning outcomes
Know-how and skill
• Show proficiency in performing a
structured examination of visual fields by
confrontation using both static and
kinetic techniques to detect either ocular
or visual pathway-related defects.
• Recognize any abnormal signs and apply
their knowledge when determining their
possible causes.
Slide
4 Visual Fields
Learning outcomes
Competence
• Adopt an empathic and holistic
approach to patient assessment.
• Extrapolate from any abnormal signs
the key relevant features to form a
differential diagnosis with appropriate
management strategies.
Examination of Visual
Fields by
Slide
6
Confrontation:
Introduction/explanation to
subject.
http://emedicine.medscape.com/ar
ticle/2094663-
technique#aw2aab6b4b1aa
https://www.youtube.com/watch?
v=2-9FVywV2j4
Slide
Case Report:
7 77 yr old diabetic complaining of vague visual disturbance, with
hallucinations along the right hand side of her vision
Slide
11 Humphrey Visual Field Analyzer
Slide Slide curtesy of Alcon’s
Glaucoma Slide Resource
12 Visual field examination – perimetry
• Testing of visual field is
mandatory in glaucoma
assessment and management in
order to define the state of optic
nerve function and visual
impairment.
• All patients with glaucoma or
suspected glaucoma should
undergo frequent visual field
examinations.
•
Standard automated perimetry
Mild loss of visual field Severe loss of visual field (SAP) is the preferred technique
for evaluating the visual field in
glaucoma subjects.
• Characteristics of glaucomatous
visual field defects include:
• Asymmetrical across
horizontal midline in
early/moderate cases
• Located in mid-
periphery, 5–25 degrees
from fixation, in
early/moderate cases
• Reproducible
• Not attributable to other
pathology
• Clustered in
neighbouring test points
(localised)
• Defect should correlate with the
appearance of the optic disc and
neighbourhood.
1. Photoreceptors.
2. Bipolar cells.
3. Ganglion cells – axon passes thro’
optic nerve – chiasm – optic tract –
lateral geniculate body – synapses
with:
4. Neurone – optic radiation – visual
cortex.
Slide
14 Clinical features relating to
site of visual pathway lesion
Eye & Optic Nerve:
Field loss:
• Visual loss in one eye.
• Central loss suggests macular or optic nerve
disease.
• Peripheral loss suggests retinal disease or
disease of optic nerve head.
• Altitudinal field loss (see slides 16 & 17) implies
anterior ischaemic optic neuropathy, but can also
be due to retinal detachment, hemispherical retinal
vein or artery occlusion, or advanced glaucoma.
• NB Glaucomatous field loss – arcuate scotoma
(see upcoming slides), central vision spared til
late.
Slide Visual field of a patient showing superior Some arcuate scotomas can
22 arcuate scotoma, who had a cup-to-disc affect one half of the visual
ratio of 0.5, and very thin inf. NRR field more than the other. The
defect in this slide is similar to
the altitudinal defect seen in
cases of AION. However, the
degree of optic disc cupping
with thinning of the inferior
neuroretinal rim (NRR) helps
differentiate the two
conditions.
Slide Visual field of a patient showing Small central spared island of
23 extensive scotoma, who had a cup-to- vision means that visual
disc ratio of 0.9, and very thin NRR acuity can remain excellent
even in advanced cases of
glaucoma, demonstrating how
the condition can present very
late unless picked up early
through routine screening.
Slide
24 Clinical features relating to
site of visual pathway lesion
Optic Chiasm:
Field loss:
• Bitemporal hemianopia (frequently asymmetrical).
• Colour desaturation across vertical midline is earliest sign.
• May present late, until central vision affected from pressure on
fibres serving macula.
• Eventual complete blindness.
Causes:
• Pituitary adenoma (usually chromophobe – superotemporal
field initially).
• Craniopharyngioma (from vestigial remnants of Rathke pouch
along pituitary stalk - inferotemporal field initially).
• Meningioma (typically compresses junction of optic chiasm
with optic nerve – ipsilateral central scotoma, contralateral
upper temporal defect).
• Aneurysms (ICA)/vascular accidents/trauma less common
cause.
Causes:
• Uncommon.
• Posterior extensions of pituitary tumours.
Causes:
• Tumours.
• Vascular insults.
• Basal meningitis.
• Aneurysms.
• Abscesses.
• Trauma.
• Migraine.
Slide Binocular visual field showing
29
L
R left inferior homonymous
quadrantanopia. Where is the
likely site of the
corresponding visual pathway
lesion? Is this patient allowed
to drive with this defect? (see
http://www.rsa.ie/Documents/
Both
eyes
Licensed%20Drivers/Eyesight
_Report_D_502_Form.pdf).
Causes:
• Vascular lesions.
• Migraine.
• Trauma.
• Tumours.
Slide
33 Case Report Answers
To be discussed in face-to-face tutorial